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Model of Multiple Sclerosis
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Michael Changaris
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A model of MS that is rooted in systemic regulation of tissue.
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Model of Multiple Sclerosis
1.
Adverse Childhood Experiences
(ACEs) Autonomic Dysregula1on Allosta1c Load Telomerase Telemeres ROS, CASP9, CREB, Bcl2 etc. Apoptosis Regulators Cell Markers of Cell Health and Self Removal of Healthy Cells Neuroplas1c Immunofunc1ons Neuroplas1c Changes Remove Brake On Plas1city Increase Rate of Brain Tissue Change Glial Func1oning • Glutamate Reuptake • Immune Barrior • Synap1c Plas1city • Apop1c Signal Rapid Tissue Loss Cell Repair Cycles • Neurogenesis • LTP • Cell Repair Lesion Regrowth Metabolic Changes (e.g. Sugar/Insulin, Mitochondria fxn) Model of Mul1ple Sclerosis Balance Decrease Cell Replacement And Reduced Cell Health Gene1c & Other Factors
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