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1) Definition of PUD 2) Pathogenesis of PUD 3) Risk Factors for PUD 4) Morphology of PUD 5) Clinical features 6) Complications 7) Summary This presentation was presented in MULTI SYSTEM SEMINAR by a 2nd year MBBS-2015 student of BPKIHS, Dharan, NEPAL.
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Gastrointestinal Diseases Group 5: Leticia Bernal Leon Daydig Rodriguez Maria Rodriguez Karina Silveira Instructor: Dr. Alain Llanes Rojas, DNP, APRN, FNP-BC Miami Regional University Diagnosis, Symptoms & Illness Management MSN5600 Gastroesophageal Reflux Gastroesophageal reflux that does not cause symptoms is known as physiologic reflux. In nonerosive reflux disease (NERD), individuals have symptoms of reflux disease but no visible or minimal esophageal mucosal injury Gastroesophageal reflux disease (GERD) is the reflux of acid and pepsin or bile salts from the stomach to the esophagus that causes esophagitis. The severity of the esophagitis depends on the composition of the gastric contents and esophageal mucosa exposure time. Definition & Classification Gastroesophageal Reflux Causes GERD can be caused by abnormalities or alterations in 1. Lower esophageal sphincter function 2. Esophageal motility 3. Gastric motility or emptying Esophageal function studies include the following: Determination of the lower esophageal sphincter (LES) pressure (manometry) Graphic recording of esophageal swallowing waves, or swallowing pattern (manometry) Detection of reflux of gastric acid back into the esophagus (acid reflux) Detection of the ability of the esophagus to clear acid (acid clearing) An attempt to reproduce symptoms of heartburn (Bernstein test) Gastroesophageal Reflux Risk Factors Obesity Hiatal hernia Use of drugs or chemicals that relax the LES (anticholinergics, nitrates, calcium channel blockers, nicotine) Cigarette smoke. Trigger Factors Coughing Vomiting Straining at stool Asthma Chronic cough Sinusitis. Gastroesophageal Reflux Common Symptoms Heartburn that occurs 30 to 60 minutes after meals and when the patient bends over or lies down. Regurgitation of sour or bitter gastric contents Belching, and fullness of the stomach Upper abdominal pain within 1 hour of eating. Atypical Symptoms chronic cough asthma attacks chronic laryngitis sinusitis discomfort during swallowing. Noncardiac chest pain. Dysphagia Gastroesophageal Reflux Clinical manifestations are related to mucosal injury from acid regurgitation and the frequency and duration of reflux events. The symptoms worsen if the individual lies down or if intraabdominal pressure increases because of coughing, vomiting, or straining at stool. Uncomplicated GERD that is responsive to first-line therapy does not require an endoscopy. Patients who do not respond to therapy and those with suspected complications should undergo an endoscopic examination Management & Evaluation Differential diagnosis Gastritis Peptic ulcer Gastric cancer Cholelithiasis Angina pectoris. Gastroesophageal Reflux Diagnosis of GERD is based on the history and clinical manifestations. An upper endoscopy with biopsy is the standard diagnostic procedure for GERD. It confirms the diagnosis and documents the type and extent of tissue damage. Esophageal endoscopy: shows hyperemia ...
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Dr. Faiza delivers an insightful lecture on the distinguishing characteristics of skeletal, smooth, and cardiac muscles, offering a comprehensive understanding of their histology, physiological anatomy, electrophysiological properties, and contractile mechanisms. Through meticulous tabulation and comparison, she aims to equip learners with a detailed comprehension of the unique features and functionalities of each muscle type. Beginning with a detailed exploration of histological differences, Dr. Faiza outlines key distinctions such as multinucleation in skeletal muscle, mononucleation in smooth and cardiac muscles, and the presence of striations in skeletal and cardiac muscles. She meticulously elucidates the structural arrangement of each muscle type, highlighting their cellular morphology and organization within tissues. Transitioning to physiological anatomy, Dr. Faiza navigates through various aspects including innervation, level of control, initiation of contraction, and modification by hormones. By delineating the role of the somatic nervous system in skeletal muscle control, and contrasting it with the autonomic nervous system's influence on smooth and cardiac muscles, she offers a nuanced understanding of neural regulation in muscle physiology. Furthermore, Dr. Faiza delves into electrophysiological properties, elucidating the mechanisms underlying action potential generation, duration, and ionic basis in each muscle type. Through insightful analysis, she reveals how differences in action potential characteristics contribute to variations in muscle contraction speed, duration, and resistance to fatigue. Finally, Dr. Faiza meticulously examines the contractile mechanisms of skeletal, smooth, and cardiac muscles, shedding light on the sliding filament theory, mechanisms of calcium action, and speed of contraction. By comprehensively comparing these mechanisms, she provides learners with a deep understanding of the intricate processes governing muscle contraction and function. Through engaging presentation and meticulous analysis, Dr. Faiza's lecture serves as a valuable resource for students seeking a thorough understanding of the fundamental differences between skeletal, smooth, and cardiac muscles.
Difference Between Skeletal Smooth and Cardiac Muscles
Difference Between Skeletal Smooth and Cardiac Muscles
MedicoseAcademics
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These are simplified slides discussing the regulation of cardiac output and venous return. Learning objectives: 1. Comprehend the determinants of cardiac output and factors affecting cardiac output 2. Comprehend the factors affecting stroke volume and heart rate and total peripheral resistance 3. Identify the factors regulating venous return 4. Discuss the causes of high and low output cardiac failure 5. Enlist the functions of veins and recognise the significance of venous reservoirs Study Resources: 1. Chapter 20, Guyton and Hall Textbook of Medical Physiology, 14th edition 2. Chapter 30 and 32, Ganong’s Review of Medical Physiology, 26th edition 3. Chapter 10, Human Physiology by Lauralee Sherwood, 9th edition 4. Physiology, Cardiac Output - StatPearls https://www.ncbi.nlm.nih.gov/books/NBK470455/
Cardiac Output, Venous Return, and Their Regulation
Cardiac Output, Venous Return, and Their Regulation
MedicoseAcademics
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7 steps How to prevent Thalassemia
7 steps How to prevent Thalassemia : Dr Sharda Jain & Vandana Gupta
7 steps How to prevent Thalassemia : Dr Sharda Jain & Vandana Gupta
Lifecare Centre
Explore the fundamentals of the human reproductive system in this concise presentation, suitable for medical students and professionals alike. Covering anatomy, physiology, and Pregnancy, it offers essential knowledge for understanding reproductive health.
ANATOMY AND PHYSIOLOGY OF REPRODUCTIVE SYSTEM.pptx
ANATOMY AND PHYSIOLOGY OF REPRODUCTIVE SYSTEM.pptx
Swetaba Besh
In this lecture on circulatory shock, Dr. Faiza, an Assistant Professor of Physiology, delves into the profound implications of inadequate blood flow throughout the body, leading to tissue damage. The session begins by defining circulatory shock and elucidating its physiological causes, including decreased cardiac output, diminished blood volume, decreased vascular tone, obstruction to blood flow, excess metabolic rate, and abnormal perfusion patterns. Dr. Faiza categorizes shock into various types, such as cardiogenic shock, hypovolemic shock, neurogenic shock, obstructive shock, anaphylactic shock, and septic shock, and explores the pathophysiological basis of each. Furthermore, the lecture examines the stages of shock, from the non-progressive phase where compensatory mechanisms aim for full recovery to the progressive phase where shock worsens steadily without intervention, potentially leading to irreversible damage. Dr. Faiza discusses therapeutic interventions for shock, including replacement therapy, blood and plasma transfusion, sympathomimetic drugs, and other adjunctive treatments like head-down positioning, oxygen therapy, and glucocorticoids.
Circulatory Shock, types and stages, compensatory mechanisms
Circulatory Shock, types and stages, compensatory mechanisms
MedicoseAcademics
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Difference Between Skeletal Smooth and Cardiac Muscles
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Cardiac Output, Venous Return, and Their Regulation
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ANATOMY AND PHYSIOLOGY OF REPRODUCTIVE SYSTEM.pptx
ANATOMY AND PHYSIOLOGY OF REPRODUCTIVE SYSTEM.pptx
Circulatory Shock, types and stages, compensatory mechanisms
Circulatory Shock, types and stages, compensatory mechanisms
Pud Gastritis Lecture[1]
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55 yo male
c/ epigastric pain on/off x 1 mo
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Reflux Esophagitis Peptic
Ulcer Disease, Gastritis, & GI Bleeding Pascale Gèhy-Andrè, PA-C
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Screw the environment
carpooling is bad
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GI Bleeding
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Guiaic/ Hemoccult
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CXR Pneumoperitoneum
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Flex Sig
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Anoscopy/ Rigid Sigmoidoscopy
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Super Duper Pooper
Scooper
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Colonoscopy
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