Death to Diabetes: Type 2 Diabetes Pathology

What is Diabetes: The Pathology cell

Normal Biological Cell Function Normal Cellular Function Cell Cell When we eat food: 1. Food converted to glucose 2. Insulin released ,[object Object],Glycoforms (Glycoproteins) Mitochondria Insulin receptors Cell 6 6. Communication (Network op system) 3b Glucose level goes down 3a 3. Glucose absorbed (Insulin) (Endocytosis) 5 5. Waste/toxins removed (Exotysosis) 3c Glucose stored in blood; liver, muscle cells Cytoplasm 4 4. Energy produced, Fat is burned DNA repaired CoQ10, ALC, ALA -- > ATP:

Type 2 Diabetes: Cellular Level Insulin Resistance & Inflammation Cell Cell ,[object Object],Glycoforms (Glycoproteins) Cell plasma membrane (with cell receptors) Mitochondria Damaged insulin receptors Cell 4 4. Can’t Communicate (Network op system) 1b 1b. Glucose backs up in bloodstream 1c. Blood glucose level rises 1d. More insulin secreted, Inflammation 1a 1. Can’t absorb glucose (Insulin) (Endocytosis) 3 3. Can’t remove waste/toxins (Exocytosis) Cytoplasm 2 2. Can’t produce energy, Can’t burn fat (Fat cells “locked”) Can’t repair DNA CoQ10, ALC, ALA -- > ATP:

Why Diabetics Remain in Diabetic State ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],BG Rises Insulin Secreted BG Continues to Rise More Insulin Secreted BG  Insulin Stays High Cravings Return, Low Energy “ Dead” Foods Hunger/ Cravings Go Away Temporarily BG Goes Down Too Far (Hypoglycemia) or ,[object Object],[object Object],Liver Converts Extra Glucose to Fat Glucose Stored In Liver, Muscle Cells Fat Storage Insulin resistance ,[object Object],[object Object],[object Object],Lack of Knowledge, Motivation, Negative Attitude, Weak Spirit Internal Inflammation Spreads Damage to arteries, kidneys, nerves, etc. Insulin inhibits fat metabolism (Fat cells locked) & homocysteine breakdown BG Continues to Rise ,[object Object],[object Object],[object Object],[object Object],[object Object],(Insulinemia) Leptin: No appetite control Nutrient depletion

Blood Glucose & Insulin Levels Blood Glucose & Insulin Levels ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Blood Glucose Insulin Blood Glucose ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Non-diabetic Diabetic BG “Crash” Insulin

Risk Factors for Pre-Diabetes Poor nutrition · Age (>45) · Sedentary · Family Background · Emotional Health: Stress, Anger, Depression · Gestational Diabetes Tobacco, Alcohol, Drugs · Ethnic Background · High BG, Blood Pressure, High Triglycerides, LDL, Homocysteine, CRP, TNF- α , IL-6 Abdominal fat ·

Death to Diabetes: Type 2 Diabetes Pathology

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Death to Diabetes: Type 2 Diabetes Pathology