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High risk pregnancy
By: ShenellD
Bleeding Disorders of
pregnancy
• First Trimester bleeding- Abortion
and ectopic pregnancy
• Second trimester bleeding-
Hydatidiform mole and incompetent
cervix
• Third trimester bleeding- Placenta
previa and abruption placenta
abortion
• Abortion- is the most common bleeding disorder of
early pregnancy. Abortion is the termination of
pregnancy before viability,that is, before 20 weeks.
• Abortus- a fetus that is aborted before it is 500
gms in weight.
• Blighted ovum- a small macerated fetus,
sometimes there is no fetus, surrounded by a fluid
inside the sac.
• Maceration- a dead fetus undergoing necrosis.
• Early abortion- termination of pregnancy before
16 weeks.
• Late abortion- abortion that occurs between 16 to
20 weeks.
Causes of abortion:
• FETAL CAUSES-
• The most common cause of early
spontaneous abortion is abnormal
development of the zygote, embryo,
and fetus.
• This abnormalities are incompatible
with life and would have resulted to
severe congenital anomalies if
pregnancy has not been aborted.
Causes of abortion:
• MATERNAL CAUSES-
• These are congenital or acquired
conditions of the mother and
environmental factors that had
adversely affected the pregnancy
outcome and led to abortion.
• Such conditions include DM,
incompetent cervix, exposure to
radiation and infection.
Types of abortion:
• Threatened abortion
• Inevitable abortion
• Incomplete Abortion
• Complete Abortion
• Missed Abortion
• Habitual Abortion
• Septic abortion
Threatened abortion- possible
loss of product of conception
• Light vaginal bleeding
• None to mild uterine cramping
• Vaginal examination at this
stage usually reveals a closed
cervix. 25% to 50% of
threatened abortion eventually
result in loss of the pregnancy.
The development of abortion is as follows:
continuing
pregnancy
• complete
inevitable abortion
abortion
incomplete
abortion
threatened
abortion
• Inevitable abortion- the loss of
the products of conception
cannot be prevented
• Moderate to profuse bleeding,
moderate to severe uterine
cramping
• Open cervix
• Rupture of membrane
• Complete abortion-
spontaneous expulsion of the
products of conception after the
fetus has died in utero
• Light bleeding
• Mild uterine cramping
• Passage of tissue
• Closed cervix
• Incomplete abortion- expulsion of
some parts and retention of other
parts of conceptus in uterus
• Heavy vaginal bleeding
• Severe uterine cramping
• Open cervix
• Passage of tissue
• Missed abortion- retention of all
products of conception after the
death of the fetus in the uterus
• No FHT
• Signs of pregnancy disappear
• Habitual abortion- abortion
occurring in 3 or more successive
pregnancies
• The most common cause is a
significant genetic abnormality of
the conceptus.
• Septic abortion- abortion
complicated by infection
• Foul smelling vaginal discharge
• Uterine cramping
• Fever
Nursing
responsibilities
• Save all tissue passed
(histopathology examination)
• Strict bed rest and monitor bleeding
• Increased fluid PO or IV as ordered
• Prepare client for surgical
intervention (D & C or suction
evacuation) if needed
ECTOPIC PREGNANCY
• Ectopic pregnancy is any gestation
located outside the uterine cavity.
• extra uterine pregnancy is the
second leading cause of bleeding in
early pregnancy.
Causes of Ectopic pregnancy
• Mechanical Factors- factors that delay
the passage of ovum in the oviducts
and prevent it from reaching the
uterus in time for implantation.
• Salphingitis
• Peritubal adhesions- kinking and
narrowing
• Previous ectopic pregnancy
• Tumors that distort the tube
Causes of Ectopic pregnancy
• Functional and failed
contraception factors
– External migration of the ovum
– IUD
– Oral contraception
– Tubal ligation- 15-50 %
– Hysterectomy
Causes of Ectopic pregnancy
• Assisted reproduction
– Ovulation induction- clomid
– Gamete intrafallopian transfer
– In vitro fertilization
– Ovum transfer
siteS OF ECTOPIC
PREGNANCY
Most frequent site is in the fallopian tube,
so rupture of the site usually occurs
before 12 weeks
• Ectopic pregnancy usually occurs
99% of cases in the uterine tube. It
can be found in
• 1. The ampulla (64%)
• 2. The Isthmus (25%)
• 3. The infundibulum (9%)
• 4. The intramural junction (2%)
• 5. Ovarian (0.5%)
• 6. Cervical (0.4%)
• 7. Abdominal (0.1%)
• 8. Intraligamental (0.05%)
•The classic
symptom triad:
amenorrhea,
vaginal bleeding,
abdominal pain.
Assessment findings:
• History of missed periods & symptoms of
early pregnancy.
• Abdominal pain, may be localized on one
side
• Rigid. Tender abdomen; sometimes
abnormal pelvic mass
• Bleeding: if severe may lead to shock
• Low Hgb & Hct, rising white cell count
• Pelvic pain- sudden knife like pain is the
most common symptom when the tube
ruptures
• Signs of hemorrhage:
– Cullen’s sign- bluish discoloration of the
umbilicus due to the presence of blood in
the peritoneal cavity
– Hard rigid board like abdomen due to
presence of blood in the peritoneal cavity.
– Signs of shock- cyanosis, pallor, cold
clammy skin, rapid pulse, dec BP
Blood loss
dec. intravascular volume
dec. venous return, cardiac output & BP
Vasoconstriction of peripheral blood vessels & inc. respiratory rate.
Cold, clammy skin, dec. uterine perfusion
Reduced renal, uterine & brain perfusion
Lethargy, coma, dec. renal output
Renal failure
Matenal and fetal death
The
process of
shock due
to blood
loss
Management:
ectopic pregnancy.flv
• If not yet ruptured, therapeutic
abortion is performed.
• If ruptured, removal or repair of
ruptured tube. Many physician
choose to remove the ruptured
tube because the presence of scar
if the tube repaired and left can
lead to another tubal pregnancy.
• Prevent and treat hemorrhage
which is the main danger of
ectopic pregnancy.
• Prevent infection as the woman
who lost so much blood is
susceptible to infection
• Prepare client for surgery
• Institute measures to control?
Treat shock if hemorrhage is
severe; continue to monitor
postoperatively.
• Allow client to express feelings
about loss of pregnancy &
concern about future pregnancies.
HYDATIDIFORM MOLE
h-mole
• A benign disorder characterized by
degeneration of the chorion and
death of the embryo. The chorionic
villi rapidly proliferate and become
grape like vesicles that produce
large amount of HCG.
• Gestational trophoblastic disease
• Cause essentially unknown
Risk factors:
• A molar pregnancy creates a 20-
40 times higher risk of having it
again.
• Increased incidence with advanced
maternal age.
• Unusual chromosomal patterns
seen. ( either no genetic material
in ovum or 69 chromosomes)
Diagnostics:
• Ultrasonography reveals no
fetal skeleton
• Elevated HCG level
Signs and symptoms
• excessive vomiting due to elevated HCG
levels
• passage of grape like vesicles around the
4th month (dark red to brownish vaginal
bleeding)
• rapid increase of uterine size which is out of
proportion to the actual age of gestation.
• absence of FHT and fetal skeleton
• ultrasound reveal a mass of fluid filled
vesicles instead of a developing fetus.
Management:
– D and C to remove the mole. If the
woman is more than 40 years old,
hysterectomy since she has a higher
chance of developing choriocarcinoma
– Anticancer drug prescribed to the
woman for one year to prevent
development of malignant or cancer
cells in the uterus.
Nursing responsibilities:
• Provide pre-postoperative care for
evacuation of uterus (usually suction
curettage).
• Teach contraceptive use so that
pregnancy is delayed for at least a year.
• Teach client’s need for follow-up lab
work to detect rising HCG levels
indicative of choriocarcinoma.
Hyper emesis gravidarum
Hyper emesis gravidarum
• -is intractable vomiting during
pregnancy that results in
dehydration and electrolyte
imbalance.
• It occurs in one of every 1000
pregnancies; the cause is
uncertain
• Risk factors: unknown
• Diagnostics: by symptoms
• Sign and symptoms:
1. Severe, persistent vomiting that
leads to dehydration or nutritional
deficiency
2. Progresses to fluid electrolyte
imbalance and alkalosis from loss
of hydrochloric acid.
Management:
• Medical: replacement of fluids,
electrolytes, and vitamins, along with
tranquilizer or antiemetic
• NPO for 48 hours, after condition
improves, six small feedings are
alternated with liquid nourishment in
small amount every 1-2 hours.
• If vomiting recurs, NPO status is
resumed and administration of IV is
restarted.
PLACENTA PREVIA
• Placenta previa is the abnormal
implantation of placental near or
over the internal os.
• It is the most common bleeding
disorder of the third trimester.
Causes of Placenta previa:
• Multiparity
• Multiple pregnancy
• Advance maternal age- over 35
years old
• Smoking
• Previous cesarean section and
abortion
• Sign and symptoms:
• Painless bright red vaginal bleeding
is the most significant sign near the
end of early of the 3rd trimester.
• Ultrasound revealed placenta
implanted over or near the cervix.
Nursing intervention:
• Ensure complete bed rest.
• Maintain sterile conditions for any
invasive procedure.
• Make provisions for emergency
cesarean birth
• Continue to monitor maternal/fetal
vital signs
Management:
• Cesarian is the delivery of
choice for all kinds of placenta
previa.
• Manage bleeding episodes
• Watchful waiting- delay delivery
until fetus is mature enough
• No IE is performed in diagnosed
placenta previa
ABRUPTIO PLACENTA
• Abruptio placenta is the
premature separation of
placenta from part or all normal
implantation site, usually
accompanied by pain.
• Usually occurs after 20 weeks of
gestation and before delivery of
the fetus
Causes of abruptio placenta:
• Maternal hypertension
• Advance maternal age
• Multiparity
• Trauma to the uterus
• Short umbilical cord
• Cigarette smoking and cocaine
abuse
Signs and symptoms:
• Painful Vaginal bleeding
• Board-like abdomen caused by
accumulation of blood behind the
placenta with fetal parts hard to
palpate
• Sharp pain over the fundus as the
placenta separates
• Signs of shock and fetal distress if
bleeding is severe.
Nursing interventions:
• Ensure bed rest
• Check maternal/fetal vital signs
frequently
• Vaginal delivery if there is no sign
of fetal distress, CS if bleeding is
severe and fetus cannot be
delivered with vaginal method.
Incompetent cervix
• Premature dilation of the
cervix
• Is a defect related trauma of
the cervix or a congenitally
short cervix, which leads to
habitual abortion and
premature labor.
Risk factors: cervical trauma related
to D&C, cervical lacerations from
previous deliveries
Sign & symptoms:
• Dilated cervix without painful
uterine contractions.
• Rupture membranes, labor begins
and premature fetus is delivered.
Surgical treatment:
• Reinforcement of the weakened cervix by
a purse string suture, which encircles the
internal os.
• Shidorkar-barter cerclage; permanent
suture that allows the cervix to remain
closed for all pregnancies; cesarian
delivery is required.
• McDonald cerclage; left in place until
term, then remove before labor.
hydramnios
Polyhydramnios: (More than
2L of fluid). Excess of amniotic
fluid.
Causes:
• Fetal abnormalities- excessive
urination of fetus
• Esophageal atresia- fetus cannot
swallow amniotic fluid.
• Multiple pregnancy
• Diabetes mellitus
Complications:
• Premature labor & delivery
• Abruptio placenta
• Postpartum hemorrhage due to
over distension of uterus
• Cord prolapsed
• malpresentation
oligohydramnios
Oligohydramnios: (Less than
500 ml of fluid) ↓ of the
amniotic fluid.
Causes:
• Fetal renal anomalies that
results in anuria
• Premature rupture of
membranes
Complications:
• Club foot
• Amputation- due to adhesion of
fetal parts to the amnion
• Abortion
• Stillbirth
• Fetal growth retardation
• Abruptio placenta
Complication during labor and
delivery
• Cord compression
• Fetal hypoxia as a result of
cord compression
• Prolonged labor
Pregnancy induced
hypertension- PIH
• Gestational hypertension replaces the
term PIH and is used for hypertensive
disorders that are specifically
associated with pregnancy,
preeclampsia, and eclampsia.
Incidence:
• Occur in 5-7% of all pregnancies
• Seen more often to primigravidas,
teenagers of low socioeconomic class.
• May be related to decreased production
of some vasodilating prostaglandins,
vasospasm occurs.
• Onset after 20th week of pregnancy,
may appear in labor or up to 48 hours
postpartum.
• Cause essentially unknown
vasospasm
Vascular effect Kidney effect Interstitial effects
vasoconstriction
Poor organ
perfusion
Inc. BP
Dec. glomeruli
filtration rate &
inc. permeability of
glomeruli
membranes
Inc. serum blood urea
nitrogen, uric acid, &
creatinine
Dec. urine output &
protenuria
Diffusion of
fluid from
blood stream
into interstitial
tissue
edema
–Danger Signs of Pregnancy-
Induced Hypertension
• Swelling of the face or fingers
• Flashes of light or dots
• Blurring of vision
• Severe continuous headache
Mild preeclampsia
• Bp of 140/90 or +30/+15 mmhg
on two consecutive occasions at
least 6 hours apart.
• Sudden weight gain
• Proteinuria of 300 mg/l in 24 hour
urine collection
Nursing intervention:
• Promote bed rest as long as signs of
edema or proteinuria are minimal,
preferably side lying.
• Provide well-balanced diet with
adequate protein.
• Explain need for close follow-up, weekly
or twice-weekly visits to physician.
Severe preeclampsia
• Headaches, epigastric pain, nausea
and vomiting, visual disturbances,
irritability
• Bp of 150-160/100-110 mmhg
• Increased edema and weight gain
• Proteinuria (5g/24hrs) 4+
Management:
• Magnesium sulfate- acts upon the
myoneural junction, diminishing
neuromuscular transmission
• It promotes maternal vasodilatation,
better tissue perfusion and has
anticonvulsant effect.
• Antidote: calcium gluconate
• Nursing responsibilities:
mgs04
• Monitor client’s respirations,
blood pressure and reflexes,
as well as urinary output
• Adm.med. Either IV or IM
Nursing interventions:
• Bed rest, side lying
• Carefully monitor maternal/fetal vital
signs
• Monitor I&O, results of laboratory
test
• Take daily weights
• Institute seizure precautions
• Continue to monitor 24-48 hours
post delivery
eclampsia
• Increased HPN precede convulsion
followed by hypotension and
collapse
• Coma may ensue
• Labor may begin, putting fetus in
great jeopardy
• Convulsion may occur
Medical mgt. same with severe
preeclampsia
Nursing intervention:
• Minimize all stimuli
• Have airway, oxygen and suction
equipment available
• Administer medication as ordered
• Prepare for C-section with seizures
stabilized
• Continue observations 24-48 hours
postpartum.
Complication of PIH:
• Maternal complications:
• Inc. intraocular pressure leading to
retinal detachment.
• HELLP (Hemolysis, Elevated Liver
function test, Low platelet count)
syndrome has been associated with
severe preeclampsia.
Fetal complications:
• Usually small for gestational age
• May be born prematurely
• Newborn maybe born over sedated
because of medications given to mother
• May have hypermagnesemia because
of maternal treatment with mgs04.
Danger signs of pregnancy
SIGN POSSIBLE CAUSE
Swelling of face, fingers & legs HPN of pregnancy
Headache, continuous & severe HPN of pregnancy
Abdominal/ chest pain Ectopic pregnancy, uterine rupture,
pulmonary embolism
Vaginal bleeding Placental problems , abortion
Vomiting, persistent Infection, hyperemesis gravidarum
Visual changes HPN of pregnancy
Escape of vaginal fluids PROM
Gestational diabetes
• disorder of late gestation
• disorder induced by pregnancy:
from exaggerated physiological
changes in glucose metabolism
• Reversal after termination of
pregnancy with 20-50% chances of
developing type 2 diabetes later in
life.
RISK FACTORS
• Age over 30
• Family Hx of DM
• Prior macrosomic,
malformed or stillborn infant
• Obesity
• Hypertension
TWO TYPES OF DIABETES
Assessment for gestational diabetes
• 3 P’s (polyphagia, polyuria, polydipsia)
• Dizziness, if hypoglycemic
• Confusion, if hyperglycemic
• Congenital anomalies
• Inc.risk of PIH
• Macrosomia
• Poor tissue perfusion of fetus
• Glycosoria
• Hyperglycemia
• Hydramios
• Possibility of inc. monilial infection
Diagnostic Tests for DM
Glycosylated hemoglobin
 Provides information about blood
glucose level during the previous 3
months
 because glucose in the
bloodstream attaches to some of
the hemoglobin and stay attached
during the 120-day lifespan of the
RBC
Diagnostic Tests for DM
Oral glucose challenge test values for
pregnancy:
Test type pregnancy glucose level
Fasting 95
1 hour 180
2 hours 155
3 hours 140
Following a 100g glucose load. Rate is
abnormal if two values are exceeded.
GDM - ADVERSE EFFECTS
MACROSOMIA
• Excessive fat deposition on
shoulders/trunk
• Predisposes to shoulder dystocia
• Maternal hyperglycemia  transfer of
excess glucose to fetus  stimulate fetal
insulin secretion which is a potent
growth factor
HYPOGLYCEMIA at birth
99
MACROSOMIA
Pathogenesis
D-I-A-B-E-T-E-S
• D- DIET: 50-60% CHO, 20-30% FATS,
10-20% CHON
• I- INSULIN– TYPE 1
• A- ANTIDIABETIC AGENTS– TYPE 2
• B- BLOOD SUGAR MONITORING
• E- EXERCISE
• T- TRANSPLANT OF PANCREAS
• E- ENSURE ADEQUATE FOOD INTAKE
• S- SCRUPULOUS FOOT CARE
Heart disease
HEART DISEASE
Normal hemodynamic of pregnancy that
adversely affect the client with heart
disease
1. Oxygen consumption increased 10% to
20% ; related to needs of growing fetus
2. Plasma level and blood volume
increase; RBCs remain the same
(physiologic anemia)
Functional or therapeutic classification of heart
disease during pregnancy
1. Class I: no limitation of physical activity; no
symptoms of cardiac insufficiency or angina
2. Class II: slight limitation of physical activity;
may experience excessive fatigue,
palpitation, angina, or dyspnea; slight
limitation as indicated
3. Class III: moderate to marked limitation of
physical activity; dyspnea, angina, and fatigue
occur with slight activity, and bed rest is
indicated during most pregnancy
4. Class IV; marked limitation of physical
activity; angina, dyspnea, and discomfort
occur at rest; pregnancy should be avoided;
indication for termination of pregnancy
Prenatal period assessment:
• Evidenced of cardiac decompensation
especially when blood volume peaks (
weeks 28-32)
• Cough & dyspnea
• Edema
• Heart murmur
• Palpitations
• rales
Nursing intervention
prenatal period:
• Teach client to recognize & report signs
of infection, importance of prophylactic
antibiotics
• Compare vital signs to baseline
• Instruct in diet to limit weight gain to 15
lbs, low na+
Highriskpregnancydelfin202 101102174717-phpapp02

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Highriskpregnancydelfin202 101102174717-phpapp02

  • 2. Bleeding Disorders of pregnancy • First Trimester bleeding- Abortion and ectopic pregnancy • Second trimester bleeding- Hydatidiform mole and incompetent cervix • Third trimester bleeding- Placenta previa and abruption placenta
  • 4. • Abortion- is the most common bleeding disorder of early pregnancy. Abortion is the termination of pregnancy before viability,that is, before 20 weeks. • Abortus- a fetus that is aborted before it is 500 gms in weight. • Blighted ovum- a small macerated fetus, sometimes there is no fetus, surrounded by a fluid inside the sac. • Maceration- a dead fetus undergoing necrosis. • Early abortion- termination of pregnancy before 16 weeks. • Late abortion- abortion that occurs between 16 to 20 weeks.
  • 5. Causes of abortion: • FETAL CAUSES- • The most common cause of early spontaneous abortion is abnormal development of the zygote, embryo, and fetus. • This abnormalities are incompatible with life and would have resulted to severe congenital anomalies if pregnancy has not been aborted.
  • 6. Causes of abortion: • MATERNAL CAUSES- • These are congenital or acquired conditions of the mother and environmental factors that had adversely affected the pregnancy outcome and led to abortion. • Such conditions include DM, incompetent cervix, exposure to radiation and infection.
  • 7. Types of abortion: • Threatened abortion • Inevitable abortion • Incomplete Abortion • Complete Abortion • Missed Abortion • Habitual Abortion • Septic abortion
  • 8. Threatened abortion- possible loss of product of conception • Light vaginal bleeding • None to mild uterine cramping • Vaginal examination at this stage usually reveals a closed cervix. 25% to 50% of threatened abortion eventually result in loss of the pregnancy.
  • 9. The development of abortion is as follows: continuing pregnancy • complete inevitable abortion abortion incomplete abortion threatened abortion
  • 10. • Inevitable abortion- the loss of the products of conception cannot be prevented • Moderate to profuse bleeding, moderate to severe uterine cramping • Open cervix • Rupture of membrane
  • 11. • Complete abortion- spontaneous expulsion of the products of conception after the fetus has died in utero • Light bleeding • Mild uterine cramping • Passage of tissue • Closed cervix
  • 12. • Incomplete abortion- expulsion of some parts and retention of other parts of conceptus in uterus • Heavy vaginal bleeding • Severe uterine cramping • Open cervix • Passage of tissue
  • 13. • Missed abortion- retention of all products of conception after the death of the fetus in the uterus • No FHT • Signs of pregnancy disappear
  • 14. • Habitual abortion- abortion occurring in 3 or more successive pregnancies • The most common cause is a significant genetic abnormality of the conceptus.
  • 15. • Septic abortion- abortion complicated by infection • Foul smelling vaginal discharge • Uterine cramping • Fever
  • 17. • Save all tissue passed (histopathology examination) • Strict bed rest and monitor bleeding • Increased fluid PO or IV as ordered • Prepare client for surgical intervention (D & C or suction evacuation) if needed
  • 19. • Ectopic pregnancy is any gestation located outside the uterine cavity. • extra uterine pregnancy is the second leading cause of bleeding in early pregnancy.
  • 20. Causes of Ectopic pregnancy • Mechanical Factors- factors that delay the passage of ovum in the oviducts and prevent it from reaching the uterus in time for implantation. • Salphingitis • Peritubal adhesions- kinking and narrowing • Previous ectopic pregnancy • Tumors that distort the tube
  • 21. Causes of Ectopic pregnancy • Functional and failed contraception factors – External migration of the ovum – IUD – Oral contraception – Tubal ligation- 15-50 % – Hysterectomy
  • 22. Causes of Ectopic pregnancy • Assisted reproduction – Ovulation induction- clomid – Gamete intrafallopian transfer – In vitro fertilization – Ovum transfer
  • 24. Most frequent site is in the fallopian tube, so rupture of the site usually occurs before 12 weeks
  • 25. • Ectopic pregnancy usually occurs 99% of cases in the uterine tube. It can be found in • 1. The ampulla (64%) • 2. The Isthmus (25%) • 3. The infundibulum (9%) • 4. The intramural junction (2%) • 5. Ovarian (0.5%) • 6. Cervical (0.4%) • 7. Abdominal (0.1%) • 8. Intraligamental (0.05%)
  • 27. Assessment findings: • History of missed periods & symptoms of early pregnancy. • Abdominal pain, may be localized on one side • Rigid. Tender abdomen; sometimes abnormal pelvic mass • Bleeding: if severe may lead to shock • Low Hgb & Hct, rising white cell count
  • 28. • Pelvic pain- sudden knife like pain is the most common symptom when the tube ruptures • Signs of hemorrhage: – Cullen’s sign- bluish discoloration of the umbilicus due to the presence of blood in the peritoneal cavity – Hard rigid board like abdomen due to presence of blood in the peritoneal cavity. – Signs of shock- cyanosis, pallor, cold clammy skin, rapid pulse, dec BP
  • 29. Blood loss dec. intravascular volume dec. venous return, cardiac output & BP Vasoconstriction of peripheral blood vessels & inc. respiratory rate. Cold, clammy skin, dec. uterine perfusion Reduced renal, uterine & brain perfusion Lethargy, coma, dec. renal output Renal failure Matenal and fetal death The process of shock due to blood loss
  • 31. • If not yet ruptured, therapeutic abortion is performed. • If ruptured, removal or repair of ruptured tube. Many physician choose to remove the ruptured tube because the presence of scar if the tube repaired and left can lead to another tubal pregnancy.
  • 32. • Prevent and treat hemorrhage which is the main danger of ectopic pregnancy. • Prevent infection as the woman who lost so much blood is susceptible to infection
  • 33. • Prepare client for surgery • Institute measures to control? Treat shock if hemorrhage is severe; continue to monitor postoperatively. • Allow client to express feelings about loss of pregnancy & concern about future pregnancies.
  • 35. • A benign disorder characterized by degeneration of the chorion and death of the embryo. The chorionic villi rapidly proliferate and become grape like vesicles that produce large amount of HCG. • Gestational trophoblastic disease • Cause essentially unknown
  • 36.
  • 37. Risk factors: • A molar pregnancy creates a 20- 40 times higher risk of having it again. • Increased incidence with advanced maternal age. • Unusual chromosomal patterns seen. ( either no genetic material in ovum or 69 chromosomes)
  • 38. Diagnostics: • Ultrasonography reveals no fetal skeleton • Elevated HCG level
  • 39. Signs and symptoms • excessive vomiting due to elevated HCG levels • passage of grape like vesicles around the 4th month (dark red to brownish vaginal bleeding) • rapid increase of uterine size which is out of proportion to the actual age of gestation. • absence of FHT and fetal skeleton • ultrasound reveal a mass of fluid filled vesicles instead of a developing fetus.
  • 40. Management: – D and C to remove the mole. If the woman is more than 40 years old, hysterectomy since she has a higher chance of developing choriocarcinoma – Anticancer drug prescribed to the woman for one year to prevent development of malignant or cancer cells in the uterus.
  • 41. Nursing responsibilities: • Provide pre-postoperative care for evacuation of uterus (usually suction curettage). • Teach contraceptive use so that pregnancy is delayed for at least a year. • Teach client’s need for follow-up lab work to detect rising HCG levels indicative of choriocarcinoma.
  • 43. Hyper emesis gravidarum • -is intractable vomiting during pregnancy that results in dehydration and electrolyte imbalance. • It occurs in one of every 1000 pregnancies; the cause is uncertain
  • 44. • Risk factors: unknown • Diagnostics: by symptoms • Sign and symptoms: 1. Severe, persistent vomiting that leads to dehydration or nutritional deficiency 2. Progresses to fluid electrolyte imbalance and alkalosis from loss of hydrochloric acid.
  • 45. Management: • Medical: replacement of fluids, electrolytes, and vitamins, along with tranquilizer or antiemetic • NPO for 48 hours, after condition improves, six small feedings are alternated with liquid nourishment in small amount every 1-2 hours. • If vomiting recurs, NPO status is resumed and administration of IV is restarted.
  • 47. • Placenta previa is the abnormal implantation of placental near or over the internal os. • It is the most common bleeding disorder of the third trimester.
  • 48.
  • 49. Causes of Placenta previa: • Multiparity • Multiple pregnancy • Advance maternal age- over 35 years old • Smoking • Previous cesarean section and abortion
  • 50. • Sign and symptoms: • Painless bright red vaginal bleeding is the most significant sign near the end of early of the 3rd trimester. • Ultrasound revealed placenta implanted over or near the cervix.
  • 51. Nursing intervention: • Ensure complete bed rest. • Maintain sterile conditions for any invasive procedure. • Make provisions for emergency cesarean birth • Continue to monitor maternal/fetal vital signs
  • 52. Management: • Cesarian is the delivery of choice for all kinds of placenta previa. • Manage bleeding episodes • Watchful waiting- delay delivery until fetus is mature enough • No IE is performed in diagnosed placenta previa
  • 54. • Abruptio placenta is the premature separation of placenta from part or all normal implantation site, usually accompanied by pain. • Usually occurs after 20 weeks of gestation and before delivery of the fetus
  • 55.
  • 56.
  • 57. Causes of abruptio placenta: • Maternal hypertension • Advance maternal age • Multiparity • Trauma to the uterus • Short umbilical cord • Cigarette smoking and cocaine abuse
  • 58. Signs and symptoms: • Painful Vaginal bleeding • Board-like abdomen caused by accumulation of blood behind the placenta with fetal parts hard to palpate • Sharp pain over the fundus as the placenta separates • Signs of shock and fetal distress if bleeding is severe.
  • 59. Nursing interventions: • Ensure bed rest • Check maternal/fetal vital signs frequently • Vaginal delivery if there is no sign of fetal distress, CS if bleeding is severe and fetus cannot be delivered with vaginal method.
  • 60. Incompetent cervix • Premature dilation of the cervix • Is a defect related trauma of the cervix or a congenitally short cervix, which leads to habitual abortion and premature labor.
  • 61. Risk factors: cervical trauma related to D&C, cervical lacerations from previous deliveries Sign & symptoms: • Dilated cervix without painful uterine contractions. • Rupture membranes, labor begins and premature fetus is delivered.
  • 62. Surgical treatment: • Reinforcement of the weakened cervix by a purse string suture, which encircles the internal os. • Shidorkar-barter cerclage; permanent suture that allows the cervix to remain closed for all pregnancies; cesarian delivery is required. • McDonald cerclage; left in place until term, then remove before labor.
  • 63.
  • 64. hydramnios Polyhydramnios: (More than 2L of fluid). Excess of amniotic fluid.
  • 65.
  • 66. Causes: • Fetal abnormalities- excessive urination of fetus • Esophageal atresia- fetus cannot swallow amniotic fluid. • Multiple pregnancy • Diabetes mellitus
  • 67. Complications: • Premature labor & delivery • Abruptio placenta • Postpartum hemorrhage due to over distension of uterus • Cord prolapsed • malpresentation
  • 68. oligohydramnios Oligohydramnios: (Less than 500 ml of fluid) ↓ of the amniotic fluid.
  • 69.
  • 70. Causes: • Fetal renal anomalies that results in anuria • Premature rupture of membranes
  • 71. Complications: • Club foot • Amputation- due to adhesion of fetal parts to the amnion • Abortion • Stillbirth • Fetal growth retardation • Abruptio placenta
  • 72. Complication during labor and delivery • Cord compression • Fetal hypoxia as a result of cord compression • Prolonged labor
  • 74. • Gestational hypertension replaces the term PIH and is used for hypertensive disorders that are specifically associated with pregnancy, preeclampsia, and eclampsia. Incidence: • Occur in 5-7% of all pregnancies • Seen more often to primigravidas, teenagers of low socioeconomic class.
  • 75. • May be related to decreased production of some vasodilating prostaglandins, vasospasm occurs. • Onset after 20th week of pregnancy, may appear in labor or up to 48 hours postpartum. • Cause essentially unknown
  • 76. vasospasm Vascular effect Kidney effect Interstitial effects vasoconstriction Poor organ perfusion Inc. BP Dec. glomeruli filtration rate & inc. permeability of glomeruli membranes Inc. serum blood urea nitrogen, uric acid, & creatinine Dec. urine output & protenuria Diffusion of fluid from blood stream into interstitial tissue edema
  • 77. –Danger Signs of Pregnancy- Induced Hypertension • Swelling of the face or fingers • Flashes of light or dots • Blurring of vision • Severe continuous headache
  • 78. Mild preeclampsia • Bp of 140/90 or +30/+15 mmhg on two consecutive occasions at least 6 hours apart. • Sudden weight gain • Proteinuria of 300 mg/l in 24 hour urine collection
  • 79. Nursing intervention: • Promote bed rest as long as signs of edema or proteinuria are minimal, preferably side lying. • Provide well-balanced diet with adequate protein. • Explain need for close follow-up, weekly or twice-weekly visits to physician.
  • 80. Severe preeclampsia • Headaches, epigastric pain, nausea and vomiting, visual disturbances, irritability • Bp of 150-160/100-110 mmhg • Increased edema and weight gain • Proteinuria (5g/24hrs) 4+
  • 81. Management: • Magnesium sulfate- acts upon the myoneural junction, diminishing neuromuscular transmission • It promotes maternal vasodilatation, better tissue perfusion and has anticonvulsant effect. • Antidote: calcium gluconate
  • 82. • Nursing responsibilities: mgs04 • Monitor client’s respirations, blood pressure and reflexes, as well as urinary output • Adm.med. Either IV or IM
  • 83. Nursing interventions: • Bed rest, side lying • Carefully monitor maternal/fetal vital signs • Monitor I&O, results of laboratory test • Take daily weights • Institute seizure precautions • Continue to monitor 24-48 hours post delivery
  • 84. eclampsia • Increased HPN precede convulsion followed by hypotension and collapse • Coma may ensue • Labor may begin, putting fetus in great jeopardy • Convulsion may occur Medical mgt. same with severe preeclampsia
  • 85. Nursing intervention: • Minimize all stimuli • Have airway, oxygen and suction equipment available • Administer medication as ordered • Prepare for C-section with seizures stabilized • Continue observations 24-48 hours postpartum.
  • 86. Complication of PIH: • Maternal complications: • Inc. intraocular pressure leading to retinal detachment. • HELLP (Hemolysis, Elevated Liver function test, Low platelet count) syndrome has been associated with severe preeclampsia.
  • 87. Fetal complications: • Usually small for gestational age • May be born prematurely • Newborn maybe born over sedated because of medications given to mother • May have hypermagnesemia because of maternal treatment with mgs04.
  • 88. Danger signs of pregnancy SIGN POSSIBLE CAUSE Swelling of face, fingers & legs HPN of pregnancy Headache, continuous & severe HPN of pregnancy Abdominal/ chest pain Ectopic pregnancy, uterine rupture, pulmonary embolism Vaginal bleeding Placental problems , abortion Vomiting, persistent Infection, hyperemesis gravidarum Visual changes HPN of pregnancy Escape of vaginal fluids PROM
  • 90. • disorder of late gestation • disorder induced by pregnancy: from exaggerated physiological changes in glucose metabolism • Reversal after termination of pregnancy with 20-50% chances of developing type 2 diabetes later in life.
  • 91. RISK FACTORS • Age over 30 • Family Hx of DM • Prior macrosomic, malformed or stillborn infant • Obesity • Hypertension
  • 92.
  • 93. TWO TYPES OF DIABETES
  • 94.
  • 95. Assessment for gestational diabetes • 3 P’s (polyphagia, polyuria, polydipsia) • Dizziness, if hypoglycemic • Confusion, if hyperglycemic • Congenital anomalies • Inc.risk of PIH • Macrosomia • Poor tissue perfusion of fetus • Glycosoria • Hyperglycemia • Hydramios • Possibility of inc. monilial infection
  • 96. Diagnostic Tests for DM Glycosylated hemoglobin  Provides information about blood glucose level during the previous 3 months  because glucose in the bloodstream attaches to some of the hemoglobin and stay attached during the 120-day lifespan of the RBC
  • 97. Diagnostic Tests for DM Oral glucose challenge test values for pregnancy: Test type pregnancy glucose level Fasting 95 1 hour 180 2 hours 155 3 hours 140 Following a 100g glucose load. Rate is abnormal if two values are exceeded.
  • 98. GDM - ADVERSE EFFECTS MACROSOMIA • Excessive fat deposition on shoulders/trunk • Predisposes to shoulder dystocia • Maternal hyperglycemia  transfer of excess glucose to fetus  stimulate fetal insulin secretion which is a potent growth factor HYPOGLYCEMIA at birth
  • 100.
  • 101. D-I-A-B-E-T-E-S • D- DIET: 50-60% CHO, 20-30% FATS, 10-20% CHON • I- INSULIN– TYPE 1 • A- ANTIDIABETIC AGENTS– TYPE 2 • B- BLOOD SUGAR MONITORING • E- EXERCISE • T- TRANSPLANT OF PANCREAS • E- ENSURE ADEQUATE FOOD INTAKE • S- SCRUPULOUS FOOT CARE
  • 102.
  • 104. HEART DISEASE Normal hemodynamic of pregnancy that adversely affect the client with heart disease 1. Oxygen consumption increased 10% to 20% ; related to needs of growing fetus 2. Plasma level and blood volume increase; RBCs remain the same (physiologic anemia)
  • 105. Functional or therapeutic classification of heart disease during pregnancy 1. Class I: no limitation of physical activity; no symptoms of cardiac insufficiency or angina 2. Class II: slight limitation of physical activity; may experience excessive fatigue, palpitation, angina, or dyspnea; slight limitation as indicated 3. Class III: moderate to marked limitation of physical activity; dyspnea, angina, and fatigue occur with slight activity, and bed rest is indicated during most pregnancy 4. Class IV; marked limitation of physical activity; angina, dyspnea, and discomfort occur at rest; pregnancy should be avoided; indication for termination of pregnancy
  • 106. Prenatal period assessment: • Evidenced of cardiac decompensation especially when blood volume peaks ( weeks 28-32) • Cough & dyspnea • Edema • Heart murmur • Palpitations • rales
  • 107. Nursing intervention prenatal period: • Teach client to recognize & report signs of infection, importance of prophylactic antibiotics • Compare vital signs to baseline • Instruct in diet to limit weight gain to 15 lbs, low na+