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Head Injuries
         Dr. Ketan Vagholkar
    M.S.(BOM)., D.N.B., M.R.C.S.(GLASGOW)., F.A.C.S.


Consultant General Surgeon & Professor of Surgery
Head Injuries

         Learning Objectives
v Understanding the basic sciences related
 to pathophysiology of head injuries
v Clinical evaluation
v Management
v Medicolegal documentation
Pathophysiology
• Monro Kellie
  Hypothesis.
• Cerebral Edema
 vasogenic-bbb affected
 cytotoxic-bbb unaffected
 osmotic-bbb intact
 interstitial- csf brain barrier affected

• Conning
 subfalceal
 trans tentorial- Kernohans notch
 trans foramen magnum
Primary management of trauma
               patient
•   Airway
•   Breathing
•   Circulation
•   Clinical assessment
Clinical Features
• History                     •   PhysicalExamination
Mechanism of injury/impact,   Pulse
Unconsciousness,              Blood pressure
Vomiting,                     Respiratory rate
Convulsions,                  Glasgow coma scale
ENT bleed,
Alcoholism,                   •   Local examination of the head
Diabetes,                         for scalp injuries & depressed
Hypertension,                     fractures
TIA’s
Glasgow Coma Scale
• Detailed
  documentation of vital
  and neurological
  parameters
Neurological Examination
• Position of patient
• Depth of unconsciousness
• Eyes ; Black eye (Periorbital ecchymosis)
       : Pupils – size, equality and reaction to light
• Examination of ears and nose for blood or csf leak
• Power in the limbs and reflexes
• Cranial nerves
• Examination of neck
Coup and Contre- coup injuries
•   Scalp laceration          •   -
•   Skull fracture            •   -
•   Extradural hematoma       •   -
•   Subdural hematoma         •   Subdural hematoma
•   Cerebral laceration and   •   Cerebral laceration and
    contusion                     contusion
•   Brain edema               •   Brain edema
•   Intracerebral hematoma    •   Intracerebral hematoma
Examination of Neck
•   Every head injury patient is assumed         •   Cervical (neck) injuries usually result in full or partial
                                                     tetraplegia (Quadriplegia). However, depending on
    to have sustained a neck injury unless           the specific location and severity of trauma, limited
    proved otherwise hence detatiled                 function may be retained.
    evaluation of neck for injuries is pivitol   •   Injuries at the C-1/C-2 levels will often result in loss
                                                     of breathing, necessitating mechanical ventilators or
                                                     phrenic nerve pacing.
                                                 •   C3 vertebrae and above : Typically results in loss of
                                                     diaphragm function, necessitating the use of a
                                                     ventilator for breathing.
                                                 •   C4 : Results in significant loss of function at the
                                                     biceps and shoulders.
                                                 •   C5 : Results in potential loss of function at the
                                                     shoulders and biceps, and complete loss of function
                                                     at the wrists and hands.
                                                 •   C6 : Results in limited wrist control, and complete
                                                     loss of hand function.
                                                 •   C7 and T1 : Results in lack of dexterity in the hands
                                                     and fingers, but allows for limited use of arms.
                                                 •   Patients with complete injuries above C7 typically
                                                     cannot handle activities of daily living and cannot
                                                     function independently.[citation needed]
                                                 •   Additional signs and symptoms of cervical injuries
                                                     include:
                                                 •   Inability or reduced ability to regulate heart rate,
                                                     blood pressure, sweating and hence body
                                                     temperature.
                                                 •   Autonomic dysreflexia or abnormal increases in
                                                     blood pressure, sweating, and other autonomic
                                                     responses to pain or sensory disturbances.
Other Examinations
• Thorax
External injuries
Movements of the chest
Auscultation of the chest

• Abdomen
External injuries
Tenderness, rebound tenderness
Guarding , rigidity
4 quadrant tap if required

Skeletal examinations
Limb fractures
Admission to hospital for all head
   injury patients is a safe practice
• Always give a equivocal or a accurate picture regarding
  the severity of injury and a uncertainty regarding the
  variable outcome to avoid illusion in the minds of the
  relatives
• Close observation of neurological status
• Early detection of neurological deterioration and avoiding
  delay in neurosurgical intervention
• Allays the anxiety of relatives
• Quick transfer to a neurosurgical facility if in a peripheral
  hospital
• Medico legally safe and defensive for the attending
  clinician
Investigations
• Hematological                  • Radiolgy
CBC                              •   Plain CT scan of brain
Blood sugar levels               •   Cervical spine x-ray preferably
BUN                                  in all head injury patients
Creatinine                       •   3D CT scan of the skull in
Electrolytes                         suspected facial bone fractures
Blood grouping in severe cases   •   Plain x-rays depending upon
                                     the injuries e.g. open mouth
Toxicology scan in suspected         view, nasal spine, paranasal
   drug ingestions                   sinuses,
Treatment Objectives
• Treatment of raised intracranial tension
  due to cerebral edema
• Treatment /prevention of convulsions
• Treatment of scalp injuries
• Treatment of skull fractures
• Treatment of intracranial hematomas
Treatment of raised intracranial
   tension due to cerebral edema
• Elevation of the patient’s head to promote venous
  drainage
• Hyperventilation if GCS greater than 5
• Increasing the serum osmolality to approx 300-310
  mOsm/lit by administering mannitol ( in adults bolus
  dose of 1 gm followed by maintainance dose of 0.25-
  0.50 gms evey 6 hourly) however the effect remains for
  not more than 48 hours
• Loop diuretics such as lasix in patients with comorbid
  problems
• Barbiturates as the last resort, best given in a
  neurosurgical facility
Treatment /prevention of
                 convulsions
• Eptoin sodium is the drug of choice
• In patients who have had a convulsion
Loading dose of 300mgms followed by maintenance dose of 100mgms every 8
   hourly converted to oral dosage at discharge and continued for 2 years

• In patients who have not convulsed but
  have intracranial pathology
Prophylactic dosage of 100mgm every 8 hourly converted to oral dosage at the
   time of discharge and continued for a period of 6 months
Treatment of scalp injuries
•   Shave the surrounding hair to have minimum clearance of 1 inch all
    around the wound
•   Rigourous cleansing of the wound ensuring complete removal of all
    foreign materials
•   Adequate hemostasis of bleeding scalp vessels by eversion of scalp
    layers or by pressure tamponade
•   Exact assessment of the depth of the wound with regards the layers
    of the scalp and if breached whether aponeurosis breached or intact
•   Limited debridement only in badly contused and irregularly lacerated
    wounds.
•   Chromic catgut 1-0 for aponeurosis suturing
•   Prolene or ethilon 1-0 for skin and superficial layers of scalp
•   Antibiotics, analgesics and tetanus prophylaxis
Treatment of skull fractures
• Linear fracture- conservative treatment
• Depressed fracture- requires surgery if significantly
  depressed
• Diastatic fractures which involve sutures usually
  lambdoid - conservative treatment
• Basilar fractures- conservative tratment- if CSF leak
  persist than osteoplastic flap surgery at a latter date
• Compound skull fractures- conservative treatment to
  start with followed by surgical intervention at a latter date
  if required.
Treatment of intracranial
                 hematomas
• Extradural Hematoma

  LocationBetween the skull and the dura

  Involved vessel
  Temperoparietal locus (most likely) - Middle meningeal artery
  Frontal locus - anterior ethmoidal artery
  Occipital locus - transverse or sigmoid sinuses
  Vertex locus - superior sagittal sinus

  SymptomsLucid interval followed by unconsciousnessCT

  AppearanceBiconvex lens

  Treatment Exploratory craniotomy with evacuation of the hematoma
Treatment of intracranial
              hematoma
• Subdural Hematoma
 Lcation ;Between the dura and the arachnoid

 Involved vessel Bridging veins

 Symptoms Gradually increasing headache and confusion
 ( acute,subacute and chronic presentation)

 Appearance Crescent-shaped

 Treatment craniotomy with evacuation of hematoma
Treatment of intracranial
            hematoma
• Intracerebral hematomas have very poor
  prognosis
• Surgical debridement may be attempted in
  a few cases but with poor results
• Post traumatic Subarachnoid hemorrhage
  is treated conservatively and has doubtful
  prognosis.
Late Complications of head injuries

•   Post traumatic headache
•   Post-traumatic epilepsy
•   Hydrocephalus
•   Memory Changes
Medicolegal documentation
•   Note should be made about the informant
•   Detail list of injuries along with diagrams is strongly advised
•   Dimensions in cms should be mentioned
•   Injury certificate should contain clinical examination injury findings as well
    as lesions picked on Ct or any other radiological examination.
•   While in hospital the level of consciousness (GCS) and vital parameters
    should be documented on a periodic basis till the patient achieves
    neurological and hemodynamic stability
•   All short term and long term sequelae of the lesion should be explained to
    the patient and his close relatives at the time of transfer or discharge.
Pupillary Changes in head injury
            patients
Clinical signs in head injury
           patients
Extradural Hematoma
Subdural Hematoma
Intracerebral Hematoma
Skull fractures
Thank You

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Head injuries: Prompt diagnosis and immediate treatment.

  • 1. Head Injuries Dr. Ketan Vagholkar M.S.(BOM)., D.N.B., M.R.C.S.(GLASGOW)., F.A.C.S. Consultant General Surgeon & Professor of Surgery
  • 2. Head Injuries Learning Objectives v Understanding the basic sciences related to pathophysiology of head injuries v Clinical evaluation v Management v Medicolegal documentation
  • 3. Pathophysiology • Monro Kellie Hypothesis. • Cerebral Edema vasogenic-bbb affected cytotoxic-bbb unaffected osmotic-bbb intact interstitial- csf brain barrier affected • Conning subfalceal trans tentorial- Kernohans notch trans foramen magnum
  • 4. Primary management of trauma patient • Airway • Breathing • Circulation • Clinical assessment
  • 5. Clinical Features • History • PhysicalExamination Mechanism of injury/impact, Pulse Unconsciousness, Blood pressure Vomiting, Respiratory rate Convulsions, Glasgow coma scale ENT bleed, Alcoholism, • Local examination of the head Diabetes, for scalp injuries & depressed Hypertension, fractures TIA’s
  • 7. • Detailed documentation of vital and neurological parameters
  • 8. Neurological Examination • Position of patient • Depth of unconsciousness • Eyes ; Black eye (Periorbital ecchymosis) : Pupils – size, equality and reaction to light • Examination of ears and nose for blood or csf leak • Power in the limbs and reflexes • Cranial nerves • Examination of neck
  • 9. Coup and Contre- coup injuries • Scalp laceration • - • Skull fracture • - • Extradural hematoma • - • Subdural hematoma • Subdural hematoma • Cerebral laceration and • Cerebral laceration and contusion contusion • Brain edema • Brain edema • Intracerebral hematoma • Intracerebral hematoma
  • 10. Examination of Neck • Every head injury patient is assumed • Cervical (neck) injuries usually result in full or partial tetraplegia (Quadriplegia). However, depending on to have sustained a neck injury unless the specific location and severity of trauma, limited proved otherwise hence detatiled function may be retained. evaluation of neck for injuries is pivitol • Injuries at the C-1/C-2 levels will often result in loss of breathing, necessitating mechanical ventilators or phrenic nerve pacing. • C3 vertebrae and above : Typically results in loss of diaphragm function, necessitating the use of a ventilator for breathing. • C4 : Results in significant loss of function at the biceps and shoulders. • C5 : Results in potential loss of function at the shoulders and biceps, and complete loss of function at the wrists and hands. • C6 : Results in limited wrist control, and complete loss of hand function. • C7 and T1 : Results in lack of dexterity in the hands and fingers, but allows for limited use of arms. • Patients with complete injuries above C7 typically cannot handle activities of daily living and cannot function independently.[citation needed] • Additional signs and symptoms of cervical injuries include: • Inability or reduced ability to regulate heart rate, blood pressure, sweating and hence body temperature. • Autonomic dysreflexia or abnormal increases in blood pressure, sweating, and other autonomic responses to pain or sensory disturbances.
  • 11. Other Examinations • Thorax External injuries Movements of the chest Auscultation of the chest • Abdomen External injuries Tenderness, rebound tenderness Guarding , rigidity 4 quadrant tap if required Skeletal examinations Limb fractures
  • 12. Admission to hospital for all head injury patients is a safe practice • Always give a equivocal or a accurate picture regarding the severity of injury and a uncertainty regarding the variable outcome to avoid illusion in the minds of the relatives • Close observation of neurological status • Early detection of neurological deterioration and avoiding delay in neurosurgical intervention • Allays the anxiety of relatives • Quick transfer to a neurosurgical facility if in a peripheral hospital • Medico legally safe and defensive for the attending clinician
  • 13. Investigations • Hematological • Radiolgy CBC • Plain CT scan of brain Blood sugar levels • Cervical spine x-ray preferably BUN in all head injury patients Creatinine • 3D CT scan of the skull in Electrolytes suspected facial bone fractures Blood grouping in severe cases • Plain x-rays depending upon the injuries e.g. open mouth Toxicology scan in suspected view, nasal spine, paranasal drug ingestions sinuses,
  • 14. Treatment Objectives • Treatment of raised intracranial tension due to cerebral edema • Treatment /prevention of convulsions • Treatment of scalp injuries • Treatment of skull fractures • Treatment of intracranial hematomas
  • 15. Treatment of raised intracranial tension due to cerebral edema • Elevation of the patient’s head to promote venous drainage • Hyperventilation if GCS greater than 5 • Increasing the serum osmolality to approx 300-310 mOsm/lit by administering mannitol ( in adults bolus dose of 1 gm followed by maintainance dose of 0.25- 0.50 gms evey 6 hourly) however the effect remains for not more than 48 hours • Loop diuretics such as lasix in patients with comorbid problems • Barbiturates as the last resort, best given in a neurosurgical facility
  • 16. Treatment /prevention of convulsions • Eptoin sodium is the drug of choice • In patients who have had a convulsion Loading dose of 300mgms followed by maintenance dose of 100mgms every 8 hourly converted to oral dosage at discharge and continued for 2 years • In patients who have not convulsed but have intracranial pathology Prophylactic dosage of 100mgm every 8 hourly converted to oral dosage at the time of discharge and continued for a period of 6 months
  • 17. Treatment of scalp injuries • Shave the surrounding hair to have minimum clearance of 1 inch all around the wound • Rigourous cleansing of the wound ensuring complete removal of all foreign materials • Adequate hemostasis of bleeding scalp vessels by eversion of scalp layers or by pressure tamponade • Exact assessment of the depth of the wound with regards the layers of the scalp and if breached whether aponeurosis breached or intact • Limited debridement only in badly contused and irregularly lacerated wounds. • Chromic catgut 1-0 for aponeurosis suturing • Prolene or ethilon 1-0 for skin and superficial layers of scalp • Antibiotics, analgesics and tetanus prophylaxis
  • 18. Treatment of skull fractures • Linear fracture- conservative treatment • Depressed fracture- requires surgery if significantly depressed • Diastatic fractures which involve sutures usually lambdoid - conservative treatment • Basilar fractures- conservative tratment- if CSF leak persist than osteoplastic flap surgery at a latter date • Compound skull fractures- conservative treatment to start with followed by surgical intervention at a latter date if required.
  • 19. Treatment of intracranial hematomas • Extradural Hematoma LocationBetween the skull and the dura Involved vessel Temperoparietal locus (most likely) - Middle meningeal artery Frontal locus - anterior ethmoidal artery Occipital locus - transverse or sigmoid sinuses Vertex locus - superior sagittal sinus SymptomsLucid interval followed by unconsciousnessCT AppearanceBiconvex lens Treatment Exploratory craniotomy with evacuation of the hematoma
  • 20. Treatment of intracranial hematoma • Subdural Hematoma Lcation ;Between the dura and the arachnoid Involved vessel Bridging veins Symptoms Gradually increasing headache and confusion ( acute,subacute and chronic presentation) Appearance Crescent-shaped Treatment craniotomy with evacuation of hematoma
  • 21. Treatment of intracranial hematoma • Intracerebral hematomas have very poor prognosis • Surgical debridement may be attempted in a few cases but with poor results • Post traumatic Subarachnoid hemorrhage is treated conservatively and has doubtful prognosis.
  • 22. Late Complications of head injuries • Post traumatic headache • Post-traumatic epilepsy • Hydrocephalus • Memory Changes
  • 23. Medicolegal documentation • Note should be made about the informant • Detail list of injuries along with diagrams is strongly advised • Dimensions in cms should be mentioned • Injury certificate should contain clinical examination injury findings as well as lesions picked on Ct or any other radiological examination. • While in hospital the level of consciousness (GCS) and vital parameters should be documented on a periodic basis till the patient achieves neurological and hemodynamic stability • All short term and long term sequelae of the lesion should be explained to the patient and his close relatives at the time of transfer or discharge.
  • 24. Pupillary Changes in head injury patients
  • 25. Clinical signs in head injury patients