Head injury is one of the commonest form of trauma in urban settings. Prompt diagnosis of the underlying damage followed by immediate treatment is the mainstay of treatment.
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Head injuries: Prompt diagnosis and immediate treatment.
1. Head Injuries
Dr. Ketan Vagholkar
M.S.(BOM)., D.N.B., M.R.C.S.(GLASGOW)., F.A.C.S.
Consultant General Surgeon & Professor of Surgery
2. Head Injuries
Learning Objectives
v Understanding the basic sciences related
to pathophysiology of head injuries
v Clinical evaluation
v Management
v Medicolegal documentation
3. Pathophysiology
• Monro Kellie
Hypothesis.
• Cerebral Edema
vasogenic-bbb affected
cytotoxic-bbb unaffected
osmotic-bbb intact
interstitial- csf brain barrier affected
• Conning
subfalceal
trans tentorial- Kernohans notch
trans foramen magnum
5. Clinical Features
• History • PhysicalExamination
Mechanism of injury/impact, Pulse
Unconsciousness, Blood pressure
Vomiting, Respiratory rate
Convulsions, Glasgow coma scale
ENT bleed,
Alcoholism, • Local examination of the head
Diabetes, for scalp injuries & depressed
Hypertension, fractures
TIA’s
7. • Detailed
documentation of vital
and neurological
parameters
8. Neurological Examination
• Position of patient
• Depth of unconsciousness
• Eyes ; Black eye (Periorbital ecchymosis)
: Pupils – size, equality and reaction to light
• Examination of ears and nose for blood or csf leak
• Power in the limbs and reflexes
• Cranial nerves
• Examination of neck
10. Examination of Neck
• Every head injury patient is assumed • Cervical (neck) injuries usually result in full or partial
tetraplegia (Quadriplegia). However, depending on
to have sustained a neck injury unless the specific location and severity of trauma, limited
proved otherwise hence detatiled function may be retained.
evaluation of neck for injuries is pivitol • Injuries at the C-1/C-2 levels will often result in loss
of breathing, necessitating mechanical ventilators or
phrenic nerve pacing.
• C3 vertebrae and above : Typically results in loss of
diaphragm function, necessitating the use of a
ventilator for breathing.
• C4 : Results in significant loss of function at the
biceps and shoulders.
• C5 : Results in potential loss of function at the
shoulders and biceps, and complete loss of function
at the wrists and hands.
• C6 : Results in limited wrist control, and complete
loss of hand function.
• C7 and T1 : Results in lack of dexterity in the hands
and fingers, but allows for limited use of arms.
• Patients with complete injuries above C7 typically
cannot handle activities of daily living and cannot
function independently.[citation needed]
• Additional signs and symptoms of cervical injuries
include:
• Inability or reduced ability to regulate heart rate,
blood pressure, sweating and hence body
temperature.
• Autonomic dysreflexia or abnormal increases in
blood pressure, sweating, and other autonomic
responses to pain or sensory disturbances.
11. Other Examinations
• Thorax
External injuries
Movements of the chest
Auscultation of the chest
• Abdomen
External injuries
Tenderness, rebound tenderness
Guarding , rigidity
4 quadrant tap if required
Skeletal examinations
Limb fractures
12. Admission to hospital for all head
injury patients is a safe practice
• Always give a equivocal or a accurate picture regarding
the severity of injury and a uncertainty regarding the
variable outcome to avoid illusion in the minds of the
relatives
• Close observation of neurological status
• Early detection of neurological deterioration and avoiding
delay in neurosurgical intervention
• Allays the anxiety of relatives
• Quick transfer to a neurosurgical facility if in a peripheral
hospital
• Medico legally safe and defensive for the attending
clinician
13. Investigations
• Hematological • Radiolgy
CBC • Plain CT scan of brain
Blood sugar levels • Cervical spine x-ray preferably
BUN in all head injury patients
Creatinine • 3D CT scan of the skull in
Electrolytes suspected facial bone fractures
Blood grouping in severe cases • Plain x-rays depending upon
the injuries e.g. open mouth
Toxicology scan in suspected view, nasal spine, paranasal
drug ingestions sinuses,
14. Treatment Objectives
• Treatment of raised intracranial tension
due to cerebral edema
• Treatment /prevention of convulsions
• Treatment of scalp injuries
• Treatment of skull fractures
• Treatment of intracranial hematomas
15. Treatment of raised intracranial
tension due to cerebral edema
• Elevation of the patient’s head to promote venous
drainage
• Hyperventilation if GCS greater than 5
• Increasing the serum osmolality to approx 300-310
mOsm/lit by administering mannitol ( in adults bolus
dose of 1 gm followed by maintainance dose of 0.25-
0.50 gms evey 6 hourly) however the effect remains for
not more than 48 hours
• Loop diuretics such as lasix in patients with comorbid
problems
• Barbiturates as the last resort, best given in a
neurosurgical facility
16. Treatment /prevention of
convulsions
• Eptoin sodium is the drug of choice
• In patients who have had a convulsion
Loading dose of 300mgms followed by maintenance dose of 100mgms every 8
hourly converted to oral dosage at discharge and continued for 2 years
• In patients who have not convulsed but
have intracranial pathology
Prophylactic dosage of 100mgm every 8 hourly converted to oral dosage at the
time of discharge and continued for a period of 6 months
17. Treatment of scalp injuries
• Shave the surrounding hair to have minimum clearance of 1 inch all
around the wound
• Rigourous cleansing of the wound ensuring complete removal of all
foreign materials
• Adequate hemostasis of bleeding scalp vessels by eversion of scalp
layers or by pressure tamponade
• Exact assessment of the depth of the wound with regards the layers
of the scalp and if breached whether aponeurosis breached or intact
• Limited debridement only in badly contused and irregularly lacerated
wounds.
• Chromic catgut 1-0 for aponeurosis suturing
• Prolene or ethilon 1-0 for skin and superficial layers of scalp
• Antibiotics, analgesics and tetanus prophylaxis
18. Treatment of skull fractures
• Linear fracture- conservative treatment
• Depressed fracture- requires surgery if significantly
depressed
• Diastatic fractures which involve sutures usually
lambdoid - conservative treatment
• Basilar fractures- conservative tratment- if CSF leak
persist than osteoplastic flap surgery at a latter date
• Compound skull fractures- conservative treatment to
start with followed by surgical intervention at a latter date
if required.
19. Treatment of intracranial
hematomas
• Extradural Hematoma
LocationBetween the skull and the dura
Involved vessel
Temperoparietal locus (most likely) - Middle meningeal artery
Frontal locus - anterior ethmoidal artery
Occipital locus - transverse or sigmoid sinuses
Vertex locus - superior sagittal sinus
SymptomsLucid interval followed by unconsciousnessCT
AppearanceBiconvex lens
Treatment Exploratory craniotomy with evacuation of the hematoma
20. Treatment of intracranial
hematoma
• Subdural Hematoma
Lcation ;Between the dura and the arachnoid
Involved vessel Bridging veins
Symptoms Gradually increasing headache and confusion
( acute,subacute and chronic presentation)
Appearance Crescent-shaped
Treatment craniotomy with evacuation of hematoma
21. Treatment of intracranial
hematoma
• Intracerebral hematomas have very poor
prognosis
• Surgical debridement may be attempted in
a few cases but with poor results
• Post traumatic Subarachnoid hemorrhage
is treated conservatively and has doubtful
prognosis.
22. Late Complications of head injuries
• Post traumatic headache
• Post-traumatic epilepsy
• Hydrocephalus
• Memory Changes
23. Medicolegal documentation
• Note should be made about the informant
• Detail list of injuries along with diagrams is strongly advised
• Dimensions in cms should be mentioned
• Injury certificate should contain clinical examination injury findings as well
as lesions picked on Ct or any other radiological examination.
• While in hospital the level of consciousness (GCS) and vital parameters
should be documented on a periodic basis till the patient achieves
neurological and hemodynamic stability
• All short term and long term sequelae of the lesion should be explained to
the patient and his close relatives at the time of transfer or discharge.