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THE PATIENT WITH HEAD,
OCULAR, OR FACIAL PAIN
EVALUATION OF
HEADACHE
• HISTORY
  – Nature of the headache
  – Daily pattern of hedache
  – Location
  – Associated symptoms
  – Precipitating or alleviating factors
  – Overall pattern
  – Family history
• VAST MAJORITY OF PATIENTS WITH
  HEADACHE HAVE NORMAL VISUAL ACUITY
• PE:
  • BP, Pulse
  • Meningeal signs
  • Symmetry of cranial nerve and motor functions
TYPES OF HEADACHE
• PRIMARY
  – Migraine
  – Tension- type
  – Cluster
• SECONDARY
NEUROIMAGING?
•   Sudden onset of severe headache
•   Unexplained change in headache pattern
•   Unresponsive to medical therapies
•   HA related to physical exertion or change in body position
•   New onset of headache after the age of 50 years
•   New headaches in immunosuppressed patients
•   (+) focal neurologic signs
•   (+) fever, neck stiffness, change in mental status, behavioral
    changes
MIGRAINE
• Condition consisting of repetitive bouts of
  headache
• F>M
• (+) familial tendency
• (+) history of motion sickness in childhood
• Onset- puberty or young adulthood
• Decrease after menopause
Characteristic:
• unilaterality, pulsating, N/V, photophobia,
  aggravated by physical activities
Exacerbated by:
• menstruation, pregnancy, hunger, stress,
  certain foods and sleep deprivation
I. Migraine with aura
• Classic migraine
• 30%
• Visual aura: begins w/ a small scotoma
  near fixation that gradually expands,
  then breaks up
• < 45 minutes
• Followed by HA on the contralateral side
  of the head
• Untreated: 4 to 72 hrs
II. Migraine without aura
•   Common migraine
•   65%
•   No preceding neurologic symtoms
•   Global
•   Can last hours to days
III. Migraine aura w/o
Headache
•   Acephalgic migraine
•   5%
•   Visual aura:
•   Scintillating scotoma, transient
    homonymous hemianopia, peripheral VF
    constriction, transient monocular visual
    loss, episodic diplopia ( vertical)
EVALUATION


• Headache or aura always occur on the same
  side
• Headache precedes the aura
• Neurologic deficit, persists after aura resolves
• Features of aura are atypical
Tension -type headache
•   Chronic
•   Aching
•   Worse at the end of day
•   Precipitated by stress
•   Associated with depression
TREATMENT
• Reassurance
• Avoid precipitating factors:
• Chocolates, nitrates, MSG, cheese,
  caffeine, red wine, alcohol, nuts,
  shellfish
• OCPs
• Stress, change in sleep patterns,
  strong scents such as perfume,
  cigarette smoke and exercise
• Acute relief: dihydroergotamine, sertonergic
  agents, NSAIDS
• * analgesic rebound headache
• Prophylactic treatment:
• Disrupted functions of daily life
• Beta blockers, Ca channel blockers, TCA, SSRIs,
  sodium valproate, NSAIDS
CLUSTER HEADCHE
• Men
• 30’s to 40’s
• Cigarette smokers
• Pain localized behind 1 eye
• Tearing, conjunctival injection,
  rhinorrhea
• < 2 hrs
• Difficult to treat
• Inhaled oxygen, methysergide, subcutaneous
  sumatriptan or dihydroergotamine
• Prednisone tapered for 10- 14 days
• Verapamil- prophylaxis
OCULAR AND ORBITAL
CAUES OF PAIN
• Refractive errors and starbismus
• KS, Keratitis, AACG, intraocular
  inflammation
• Recurrent erosion syndrome
• Scleritis
• Optic neuritis
PHOTOPHOBIA
•   Keratitis
•   Uveitis
•   Chorioretinitis
•   Menigeal irritation
•   migraine
FACIAL PAIN
• Most often, pain in the eye area is a
  manifestation of headache
• Dental disorders, sinus disease
1. Trigeminal neuralgia
• Tic douloureux
• Caused by vascular compression of CN V
• Demyelinating dse, posterior fossa mass lesion
• Unilateral
• chewing , tooth brushing, cold wind
• Normal sensory function
• MRI
• Treatment:
•  gabapentin, carbamezepine, phenytoin, baclfen,
  clonazepam, valproic acid
• Rhizotomy, decompression of CN V
1. Trigeminal neuralgia
• Tic douloureux
• Caused by vascular compression of CN V
• Demyelinating dse, posterior fossa mass lesion
• Unilateral
• chewing , tooth brushing, cold wind
• Normal sensory function
• MRI
• Treatment:
•  gabapentin, carbamezepine, phenytoin, baclfen,
  clonazepam, valproic acid
• Rhizotomy, decompression of CN V
• 2. Glossopharyngeal neuralgia
• 3. Carotidynia- neck pain that radiates to
  ipsilateral face and ear
• 4. Carotid dissection- (+) sympathetic
  dysfunction
• 5. Temporomandibular disease
6. HZO-
• pain before vesicle eruption
• Gabapentin, TCA, lidocaine patch
7. Neoplastic process
8. Mental nerve neuropathy- numb chin
• Saroidosis, lymphoma, mets breast Ca
Thank you

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evaluation of headache,eye pain

  • 1. THE PATIENT WITH HEAD, OCULAR, OR FACIAL PAIN
  • 2. EVALUATION OF HEADACHE • HISTORY – Nature of the headache – Daily pattern of hedache – Location – Associated symptoms – Precipitating or alleviating factors – Overall pattern – Family history
  • 3. • VAST MAJORITY OF PATIENTS WITH HEADACHE HAVE NORMAL VISUAL ACUITY • PE: • BP, Pulse • Meningeal signs • Symmetry of cranial nerve and motor functions
  • 4. TYPES OF HEADACHE • PRIMARY – Migraine – Tension- type – Cluster • SECONDARY
  • 5. NEUROIMAGING? • Sudden onset of severe headache • Unexplained change in headache pattern • Unresponsive to medical therapies • HA related to physical exertion or change in body position • New onset of headache after the age of 50 years • New headaches in immunosuppressed patients • (+) focal neurologic signs • (+) fever, neck stiffness, change in mental status, behavioral changes
  • 6. MIGRAINE • Condition consisting of repetitive bouts of headache • F>M • (+) familial tendency • (+) history of motion sickness in childhood • Onset- puberty or young adulthood • Decrease after menopause
  • 7. Characteristic: • unilaterality, pulsating, N/V, photophobia, aggravated by physical activities Exacerbated by: • menstruation, pregnancy, hunger, stress, certain foods and sleep deprivation
  • 8. I. Migraine with aura • Classic migraine • 30% • Visual aura: begins w/ a small scotoma near fixation that gradually expands, then breaks up • < 45 minutes • Followed by HA on the contralateral side of the head • Untreated: 4 to 72 hrs
  • 9.
  • 10. II. Migraine without aura • Common migraine • 65% • No preceding neurologic symtoms • Global • Can last hours to days
  • 11. III. Migraine aura w/o Headache • Acephalgic migraine • 5% • Visual aura: • Scintillating scotoma, transient homonymous hemianopia, peripheral VF constriction, transient monocular visual loss, episodic diplopia ( vertical)
  • 12.
  • 13. EVALUATION • Headache or aura always occur on the same side • Headache precedes the aura • Neurologic deficit, persists after aura resolves • Features of aura are atypical
  • 14. Tension -type headache • Chronic • Aching • Worse at the end of day • Precipitated by stress • Associated with depression
  • 15. TREATMENT • Reassurance • Avoid precipitating factors: • Chocolates, nitrates, MSG, cheese, caffeine, red wine, alcohol, nuts, shellfish • OCPs • Stress, change in sleep patterns, strong scents such as perfume, cigarette smoke and exercise
  • 16. • Acute relief: dihydroergotamine, sertonergic agents, NSAIDS • * analgesic rebound headache • Prophylactic treatment: • Disrupted functions of daily life • Beta blockers, Ca channel blockers, TCA, SSRIs, sodium valproate, NSAIDS
  • 17. CLUSTER HEADCHE • Men • 30’s to 40’s • Cigarette smokers • Pain localized behind 1 eye • Tearing, conjunctival injection, rhinorrhea • < 2 hrs
  • 18. • Difficult to treat • Inhaled oxygen, methysergide, subcutaneous sumatriptan or dihydroergotamine • Prednisone tapered for 10- 14 days • Verapamil- prophylaxis
  • 19. OCULAR AND ORBITAL CAUES OF PAIN • Refractive errors and starbismus • KS, Keratitis, AACG, intraocular inflammation • Recurrent erosion syndrome • Scleritis • Optic neuritis
  • 20. PHOTOPHOBIA • Keratitis • Uveitis • Chorioretinitis • Menigeal irritation • migraine
  • 21. FACIAL PAIN • Most often, pain in the eye area is a manifestation of headache • Dental disorders, sinus disease
  • 22. 1. Trigeminal neuralgia • Tic douloureux • Caused by vascular compression of CN V • Demyelinating dse, posterior fossa mass lesion • Unilateral • chewing , tooth brushing, cold wind • Normal sensory function • MRI • Treatment: • gabapentin, carbamezepine, phenytoin, baclfen, clonazepam, valproic acid • Rhizotomy, decompression of CN V
  • 23. 1. Trigeminal neuralgia • Tic douloureux • Caused by vascular compression of CN V • Demyelinating dse, posterior fossa mass lesion • Unilateral • chewing , tooth brushing, cold wind • Normal sensory function • MRI • Treatment: • gabapentin, carbamezepine, phenytoin, baclfen, clonazepam, valproic acid • Rhizotomy, decompression of CN V
  • 24. • 2. Glossopharyngeal neuralgia • 3. Carotidynia- neck pain that radiates to ipsilateral face and ear • 4. Carotid dissection- (+) sympathetic dysfunction • 5. Temporomandibular disease
  • 25. 6. HZO- • pain before vesicle eruption • Gabapentin, TCA, lidocaine patch 7. Neoplastic process 8. Mental nerve neuropathy- numb chin • Saroidosis, lymphoma, mets breast Ca
  • 26.