9. ď‚—Arise frfom ventral root
and gray rami
communicants near DRG
ď‚—Innervates PLL,ant
dura,post annulus,blood
vessels
ALL,lat & ant annulus –
sympathetics
ď‚—SP.VIP,CGRP
10. FACET JOINT PAIN
ď‚—Innervated by medial branches of dorsal primary rami
ď‚—Facet capsule-contains encapsulated,non encapsulated & free
nerve endings
ď‚—Mechanoreceptors-inflamation sensitizes these to movements of
facet jt
ď‚—Nociceptors-unmyelinated & plexiform fibres sensitizes to
chemical or mechanical stimulus
11. mechanism
ď‚—Injury to articular cartilage as in OA
ď‚—DEGEN changes of facet jt-static n dynamic compression of
nerve root-lateral recess stenosis
ď‚—Blockage of facet by synovial fold
18. duration of pain
ď‚—Acute- strains, sprains
ď‚—Chronic- degenerative conditions
ď‚—a/c on chronic
ď‚—Radiation of pain
ď‚—Nature of pain
ď‚—Aggravating/relieving factors
19. Nature and intensity of pain
ď‚—Discogenic- focal,aching in nature,increased with activity causing
axial loading,decreased with rest
ď‚—Facetal pain-pain on extension of spine
ď‚— (Can be of muscle strain)
ď‚—Degenerative-Pain and stiffness in morning
ď‚—Inflammatory-prolonged pain with stifness > 1hr
ď‚—Tumour/infection- Night Pain unrelieved by rest
21. Neurogenic claudication
ď‚—Diffuse pain n numbness
ď‚—Progressive loss of walking ability/forward stooping walking
ď‚—Symptoms produced by activities causing extension of spine,
relieved by flexion
ď‚—To r/o vascular claudication
23. ď‚—Occupational history-return to heavy physical work may not
be possible
ď‚—Family n social history- assess pts resources and support for
treatment plan
ď‚—Other systems assessment-CVS,PULMO,GI ,GU,ENDO
25. INSPECTION
ď‚—Gait
ď‚—Antalgic one leg-nerve root irritation,muscle weakness
ď‚—Sciatica :walk with hip more extended & knee more flexed
ď‚—High stepping : foot drop -to clear the ground
ď‚—Spastic:drags the foot
26. ď‚—Trendelenburgs : L5 - abductor lurch
ď‚—S1- extensor lurch
ď‚— toe walking not possible
ď‚—L4-heel walking not possible
56. Piriformis syndrome
ď‚—Entrapment of sciatic nerve by the piriformis as it passes thru the
sciatic notch
ď‚—Causes:hypertrophy
ď‚—Trauma
ď‚—Excessive exercises
ď‚—Spasm n inflammation
ď‚—Anomalies of piriformis
ď‚—Pseudo aneurysm of inf gluteal artery
ď‚—Traumatic myositis ossifcans
57. Clinical features
ď‚—History of trauma to SI or gluteal region
ď‚—Exacerbation of symptoms by lifting leg or
stooping/difficulty in walking
ď‚—Tenderness over sciatic notch
ď‚—Sausage shaped mass over piriformis
ď‚—Felt by rectal exmn-pathognomonic
58. ď‚—Positive SLR,Lasegue sign
ď‚—Freiberg sign-pain with forced int rotation of extended thigh
ď‚—Positive sign of Pace and Nagle-pain with resistance to
abduction n ER the thigh
ď‚—Tibial nerve is less affected than peroneal
66. Plain Xray
ď‚— AP
ď‚—Alignment of vertebral column
ď‚—Lesion of pedicles/ TP
ď‚—Side to side collapse
ď‚—Paravertebral soft tissue shadows
ď‚—scoliosis
67. Lateral view
ď‚—Shape n size of vertebralbody
ď‚—Anterior n posterior walls integrity
ď‚—Superior n inferior surfaces of body
ď‚—Wedging
ď‚—Disc space
ď‚—Spinal canal-between post end of body n lamina-space
occupied by cord
68. ď‚—oblique views-for pars defects
ď‚—Scannograms-to view the entire spinal column
ď‚—Ct-demonstrates bony lesions better
ď‚—Mri- demonstrates soft tissues better
ď‚—Scrrening of whole spine