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GAIT
movements that produces locomotion
Gait Cycle or Stride
A single gait cycle or stride is defined:
Period when 1 foot contacts the ground to when that
same foot contacts the ground again
Each stride has 2 phases:
Stance Phase
Foot in contact with the ground
Swing Phase
Foot NOT in contact with the ground
2
Stance Phase
Principal events during the stance phase
1. Heel strike
2. Foot-flat (followed by opposite heel-off)
3. Heel-rise (followed by opposite heel strike)
4. Toe-off
3
Principal events during the stance phase:
heel-strike, foot-flat, heel-off, toe-off
4
Activities occur in stance phase
Traditional Method
1. Heel Strike : double support
2. Foot flat : total contact
3. Mid-stance : total weight
bearing
4. Heel-off : heel clears the ground
5. Toe-off : toe clears the ground
RLA Method
1. Initial contact : heel strike
2. Loading response : double
support
3. Mid-stance : begins when
contralateral L/E clears the
ground & when the body come
straight line to supporting limb
4. Terminal stance : end of mid
stance to initial contact of CL
L/E
5. Pre-swing : period of clearance
from the ground
5
Initial Contact
Phase 1
The moment when the
red foot just touches the
floor.
The heel (calcaneous) is
the first bone of the foot
to touch the ground.
Meanwhile, the blue leg
is at the end of terminal
stance.
6
Static Positions at Initial Contact
Shoulder is extended
Pelvis is rotated left
Hip is flexed and
externally rotated
Knee is fully extended
Ankle is dorsiflexed
Foot is supinated
Toes are slightly
extended
7
Loading Response
Phase 2
The double stance period
beginning
Body wt. is transferred
onto the red leg.
Phase 2 is important for
shock absorption,
weight-bearing, and
forward progression.
The blue leg is in the
pre-swing phase.
8
Static Positions at Loading Response
Shoulder is slightly
extended
Pelvis is rotated left
hip is flexed and slightly
externally rotated
knee is slightly flexed
ankle is plantar flexing to
neutral
foot is neutral
Toes are neutral
9
Mid-stance
Phase 3
single limb support
interval.
Begins with the lifting of
the blue foot and
continues until body
weight is aligned over the
red (supporting) foot.
The red leg advances
over the red foot
The blue leg is in its mid-
swing phase.
10
Static Positions at Midstance
Shoulder is in neutral
Pelvis is in neutral
rotation
Hip is in neutral
Knee is fully extended
Ankle is relatively
neutral
Foot is pronated
Toes are neutral
11
Terminal Stance
Phase 4
Begins when the red heel
rises and continues until
the heel of the blue foot
hits the ground.
Body weight progresses
beyond the red foot
12
Static Positions at Terminal Stance
Shoulder is slightly
flexed
Pelvis is rotated left
Hip is extended and
internally rotated
Knee is fully extended
Ankle is dorsi flexed
Foot is slightly
supinated
Toes are neutral 13
Pre-swing
Phase 5
The second double
stance interval in the gait
cycle.
Begins with the initial
contact of the blue foot
and ends with red toe-
off.
Transfer of body weight
from ipsilateral to
opposite limb takes
place. 14
Static Positions at Pre-swing
Shoulder is flexed
Pelvis is rotated right
Hip is fully extended
and internally rotated
Knee is fully extended
Ankle is plantar
flexed
Foot is fully supinated
Toes are fully
extended 15
Swing phase
Principal events during the Swing phase
1. Acceleration: ‘Initial swing’
2. Mid swing : swinging limb overtakes the limb in
stance
3. Deceleration: ‘Terminal swing’
16
Activities occur in swing phase
Traditional Method
1. Acceleration : starts
immediately from toe
off
2. Mid stance : swing
directly beneath body
3. Deceleration : knee
extension and prepare
for heel strike
RLA Method
1. Initial swing : max.
knee flexion
2. Mid swing : from max.
knee flxn. to verticl.
Postn. of tibia
3. Terminal swing : from
verticl. Postn. of tibia
to initial contact
17
Initial Swing
Phase 6
Begins when the red foot
is lifted from the floor
and ends when the red
swinging foot is opposite
the blue stance foot.
It is during this phase
that a foot drop gait is
most apparent.
The blue leg is in mid-
stance.
18
Static Positions at Initial Swing
Shoulder is flexed
Spine is rotated left
Pelvis is rotated right
hip is slightly extended
and internally rotated
Knee is slightly flexed
Ankle is fully plantar
flexed
Foot is supinated
Toes are slightly flexed
19
Mid-swing
Phase 7
Starts at the end of the
initial swing and
continues until the red
swinging limb is in front
of the body
Advancement of the red
leg
The blue leg is in late
mid-stance.
20
Static Positions at Mid-swing
Shoulder is neutral
Spine is neutral
Pelvis is neutral
Hip is neutral
Knee is flexed 60-90°
Ankle is plantar flexed to
neutral
Foot is neutral
Toes are slightly
extended
21
Terminal Swing
Phase 8
Begins at the end of mid-
swing and ends when the
foot touches the floor.
Limb advancement is
completed at the end of
this phase.
22
Static Positions at Terminal Swing
Shoulder is extended
Spine is rotated right
Pelvis is rotated left
Hip is flexed and
externally rotated
Knee is fully extended
Ankle is fully dorsi flexed
Foot is neutral
Toes are slightly
extended
23
DISTANCE VARIABLES
Step length
Stride length
Width of walking base
Degree of toe out
24
Step Length
Distance between corresponding successive points
of heel contact of the opposite feet
Rt step length = Lt step length (in normal gait)
25
Stride Length
Distance between successive points of heel contact
of the same foot
Double the step length (in normal gait)
26
Walking Base
Side-to-side distance between the line of the two feet
Also known as ‘stride width’
Normal is 3.5 inches
27
Degree of toe out
Represents the angle of foot placement
It is the angle formed by each foot’s line of progression
and a line intersecting the centre of the heel and the
second toe
Normal angle is 7°for men at free speed walking
28
TEMPORAL VARIABLES
Stance Time : Is the amount of time that elapses
during the stance phase of one extremity in a gait cycle
Single support time : is the amount of time that
elapses during the period when only one extremity is on
the supporting surface in a gait cycle
Double support time : is the amount of time that a
person spends with both feet on the ground during one
gait cycle
Stride duration : is the amount of time it takes to
accomplish one stride
Step duration : is the amount of time spent during a
single step
29
TEMPORAL VARIABLES
Cadence
Number of steps per unit time
Normal: 100 – 115 steps/min
Cultural/social variations
Velocity
Distance covered by the body in unit time
Usually measured in cm/s
Instantaneous velocity varies during the gait cycle
Average velocity (m/min) = step length (m) x cadence (steps/min)
Comfortable Walking Speed (CWS)
Least energy consumption per unit distance
Average= 80 m/min (~ 5 km/h , ~ 3 mph)
30
DETERMINANTS OF GAIT
1. Lateral pelvic tilt
2. Knee flexion
3. Knee, ankle, foot interactions
4. Pelvic forward and backward rotation
5. Physiologic valgus of knee
DGs represents the adjustments made by above components
that help to keep movements of body’s COG to minimum.
They are credited with decreasing the vertical and lateral
excursions of the body’s COG and therefore decreasing
energy expenditure and making gait more efficient
31
Lateral pelvic tilt
During mid-stance the COG
reaches the peak level & the total
body supported by one lower
extremity
The pelvis slopes downwards
laterally towards the leg which is
in swing phase. i.e. rotation
about an anterior-posterior axis
Only anatomically possible if the
swing leg can be shortened
sufficiently (principally by knee
flexion) to clear the ground.
Where this is not possible (e.g.
through injury), the absence of
pelvic tilt and pronounced
movements of the upper body
are obvious.
32
Knee flexion
Another DG which
helps to reduce the
COG during mid-
stance
As the hip joint passes
over the foot during
the support phase,
there is some flexion
of the knee.
This reduces vertical
movements at the hip,
and therefore of the
trunk and head.
33
Knee, ankle, foot interactions
KAF interaction prevent
abrupt hike in COG from
heel strike to foot flat
After heel strike huge
upward displacement of
COG occurs
This is reduced by Knee
flexion, ankle plantar
flexion & foot pronation.
From mid stance to heel
off there is sudden drop in
COG
Ankle plantar flexion, knee
extension and foot
supination maintain this
34
Pelvic forward and backward rotation
Forward rotn. occurs in swing
phase
It starts during acceleration and
ends in deceleration
During mid-swing pelvis comes to
neutral position
Forward and backward rotation
help to prevent further reduction
in COG which started from mid-
stance
During deceleration both lower
extremities lengthens
This prevents in further reduction
of COG
35
Physiologic valgus of knee
Is minimised by having
a narrow walking base
i.e. feet closer together
than are hips.
Therefore less energy is
used moving hip from
side to side (less lateral
movement needed to
balance body over
stance foot)
Reduced lateral pelvic
displacement
36
Efficiency, and energy considerations
• Walking is very energy-
efficient: little ATP is required.
• This is because of various
mechanisms that ensure the
mechanical energy the body has
is passed on from one step to
the next.
• The two forms of mechanical
energy involved are
•kinetic energy (energy due
to movement
•potential energy (energy
due to position)
Economy (J m-1)
37
A conventional pendulum –
energy interconversion
P.E. – Potential energy
K.E. – Kinetic energy
Three points on a pendulum
swing are illustrated.
As the pendulum swings away
from the midpoint, in either
direction, KE is progressively
converted into PE
At the extreme points in the
swing, there is no KE at all and
all the energy is present as PE
38
Conventional pendulum action
during the swing phase
The legs move as conventional pendulums during the swing
phase (with a little assistance from the hip flexors).
This reduces the amount of muscle energy needed to move
the swinging leg forward
It also accounts for the “natural” frequency of gait that has
optimal energy efficiency
Although the legs swing forwards much like pendulums, they
are prevented from swinging backwards by foot strike.
During the stance phase, the leg can be viewed as an
“inverted pendulum”. This action also involves inter-
conversion of potential and kinetic energy 39
An “inverted” pendulum
The pendulum
“bounces”
backwards and
forwards, using
the springs.
40
“Inverted” pendulum action during the stance phase
During the stance phase, the leg can be viewed as an “inverted
pendulum”.
The forward momentum of the body gives it the necessary
initial angular velocity of rotation (taking the place of the
“spring” on the previous slide).
“Inverted” pendulum action also involves inter-conversion of
potential and kinetic energy, but in this case (unlike a
conventional pendulum) KE reaches a minimum at the
midpoint of the motion, and PE is highest at that point.
When reaching the endpoint of its “inverted swing” the stance
leg does not swing back, as a real inverted pendulum would,
because the foot is taken off the floor, the fulcrum transfers
from the foot to the hip, and the leg swings again as a
conventional pendulum. 41
Positive & negative Work
At a cadence of 105-112 steps/minute
between heel strike and foot flat
1. a brief burst of positive work (energy generation) occurs as the hip extensors contract
concentrically
2. while the knee extensors perform negative work (energy absorption) by acting
eccentrically to control knee flexion
from foot flat through mid-stance
1. Negative work is done by plantar flexors as the leg rotates over the foot during the
period of stance
2. Positive work of the knee extensors occurs during this period to extend the knee
late stance and in early swing
1. Positive work of plantar flexors and hip flexors increase the energy level of the body
In late swing
1. negative work is performed by the hip extensors as they work eccentrically to
decelerate the leg in preparation for initial contact
42
Forces
The principal forces are:
body weight (BW)
ground reaction force (GRF)
muscle force (MF)
BW and GRF are external forces; so the movement of
the centre of mass (CoM) can be predicted from them alone.
MF must be examined however if we wish to
consider either of the following:
movements of individual limbs or body segments,
why GRF changes in magnitude and direction
during the gait cycle.
43
Vitally important point:
Muscle forces can only influence
the movement of the body as a
whole indirectly, by their effects
on the GRF
44
Walking as a “controlled fall”
One way of beginning to understand the mechanics of
walking is to view the movements as a “controlled fall”
When starting a walk, we lean forward, overbalancing
from the equilibrium position.
This gives the upper part of the body forwards (and
downwards) motion
As the body falls forwards, a leg is extended forwards
and halts the fall
At the same time, the other leg “kicks off” in order to
keep the body moving forwards.
This forward momentum carries the body forward into
the next forward fall, i.e. the start of the next step
45
Walking as a controlled fall: forces
involved
When starting to move, we lean forward (MF)
As the body starts to fall (BW), a leg is extended
forwards and halts the fall (MF; GRF)
At the same time, the other leg “kicks off, upwards and
forwards” (MF; GRF) in order to keep the body
moving forwards.
This forward momentum carries the body forward into
the next forward fall, i.e. the start of the next step
46
Body weight
Always acts vertically downwards from the CoM
If its line of action does not pass through a joint, it
will produce a torque about that joint
The torque will cause rotation at the joint unless it is
opposed by another force (e.g. muscle, or ligament)
BW contributes to GRF
47
Ground reaction force
The force that the foot exerts on
the floor due to gravity & inertia
is opposed by the ground reaction
force
Ground reaction force (RF) may
be resolved into horizontal (HF)
& vertical (VF) components.
Understanding joint position &
RF leads to understanding of
muscle activity during gait
Forces are typically resolved into:
1. Vertical Compression (z)
2. Anterior-Posterior Shear (y)
3. Medial-Lateral Shear (x)
48
Muscle force
In gait, as in all human movement, muscle
activation generates internal joint moments
(torques) that:
Contribute to ground reaction force
Ensure balance
Increase energy economy
Allow flexible gait patterns
Slow down and/or prevent limb movements
Much muscle activity during gait is
eccentric or isometric, rather than
concentric 49
Center Of Pressure (COP)
Represents the centroid of foot
forces on the floor
This is an idealization, because
pressures are distributed all over
It is important, because we want
to know where the GRF is applied
to the body
When measured by a force plate, it
is more correctly called the point
of application of the GRF
Plotting the COP as it moves
under the foot:
1. Normal Path: Center of the
calcaneus or slightly lateral, curving
laterally and then medial
(pronation) and ending between
the 1st and second toes
2. Variable: Normal individuals can
have many COP trajectories, just
by changing the footgear
50
Muscles, Joints, and Forces
Eccentric
Concentric
Ground Reaction Force
51
Sagittal Plane Analysis
Initial Contact
Hip 30° of flexion,
knee is extended
ankle is neutral
GRF
Ant. to hip, drives the hip into
flexion
Ant. to knee drives the knee
into extension
Ankle into plantar flexion
Hip: hamstrings, gluteus maximus,
and adductor magnus (i to e)
Knee: quadriceps (c to e)
Tibiotalar joint: tibialis anterior (e)
Subtalar joint: anterior and lateral
compartment muscles (e)
52
Sagittal Plane Analysis
Loading Response
Hip extension 25°
Knee flexion to 20°
Ankle plantar flexion to 10°
Contralateral pelvis rotates
anterior
GRF
Ant. to hip
posterior to knee
posterior to ankle
Hip : extensors (e),
Abductors (e) limit contralateral
drop to 5°
Knee : Quadriceps fire (c)
Ankle :Tibialis anterior (e)
53
Sagittal Plane Analysis
Mid Stance
GRF through hip, knee, and
ankle
Muscular activity terminates
Hip and knee stability provided
by ligamentous restraints
GRF
Posterior to hip
Anterior to knee and ankle
Gastrosoleus complex fires to
initiate knee flexion
Pelvis continues to rotate,
abductors continue to resist
pelvic drop
54
Sagittal Plane Analysis
Terminal Stance
No change in GRF
Free forward fall
Strong activation of
gastrosoleus complex
55
Sagittal Plane Analysis
Pre-Swing
Hip 20° of hyperextansion
Knee 30° of flexion
Ankle 20° of plantarflexion
Toes 50° of hyper extension
GRF
posterior to hip, knee
anterior to ankle
Rapid flexion of knee from rapid
heel rise and unweighting of limb
Rectus femoris initiates hip flexion
Adductor longus fires
Hip : iliopsoas, adductor magnus,
adductor longus
Knee : Quadriceps
Ankle :Gastrosoleus complex
Toes : Ab.hal., FDB, FHB,
Introssei, lumb. 56
Sagittal Plane Analysis
Initial Swing
Hip 0-30° of flexion
Knee from 30-60° of flexion and
extension from 60-30°
Ankle 20° of plantarflexion to neutral
Foot clearance is passive due to rapid
hip flexion, unless gait is very slow
In slow gait, tibialis anterior and
hamstrings fire to help
Gait cadence (speed) governed by
accelerations of hip flexion during this
phase
Hip flexion
Rectus femoris
Iliacus
Adductor longus
Gracilis
Sartorius
Rest of limb is passive pendulum
57
Sagittal Plane Analysis
Mid Swing
Tibialis anterior fires to
maintain foot position
Knee extension and hip
flexion continue by inertia
58
Sagittal Plane Analysis
Terminal Swing
Decelerate knee extension
and hip flexion
Hamstrings
Gluteus max
Quads co-contract
Tibialis anterior maintains
ankle position
59
Frontal Plane Analysis
IC to LR
Pelvis : forward rotn.
Hip : med. rotn. of femur
Knee : increased valgus
Ankle : increased pronation
Thorax : Post. postion at
initial contact and begins
moving forward
Shoulder : extended and
moving forward
Gracilis, vastus medialis,
semitendinosus, LH of
biceps femoris
Tibialis post. to control
valgus thrust
60
Frontal Plane Analysis
LR to Mid-stance
Pelvis : Rt. side rotating
backward to reach neutral.
Lat. tilt towards the swinging
extremity
Hip : Med. rotn. of femur
continues to neutral.
Knee : Reduction in valgus
and tibia begins to rotate
laterally
Ankle-foot : neutral at mid-
stance
Thorax : Rt. side move
forward
Shoulder : Move forward
Hip abductors are active
Tibialis post. produce
supination
61
Frontal Plane Analysis
Mid-stance to TS
Pelvis : Rt. side move
Posteriorly
Hip : Lat. rotn. of femur and
adduction
Knee : lat. rotn. of tibia
Ankle & foot : supination of
subtalar jt. increases
Thorax : Rt. side move
forward
Shoulder : Rt. shoulder move
forward
Hip : inconsistent adductor
activity
Ankle plantar flexor activity
62
Frontal Plane Analysis
TS to PS
Pelvis : Lt. side move
forward, lat. tilting to swing
side ends as double support
begins
Hip : Abduction as wt.
shifted to opp. extremity, Lat.
rotn. of femur
Knee : Lat rotn. of tibia
Foot/Ankle : Wt. shifted to
toes. Supination of sub talar
joint
Thorax : Translation to the
left
Shoulder : Moving forward
Hip adductors control pelvis
Plantar flexion
63
Frontal Plane Analysis
IS to MS
Pelvis : Lat. pelvic tilt to the
rt. Right side move forward
Hip : Lat. rotn to med. rotn.
Knee : From lat. to med. rotn
Foot/Ankle : NWBing
subtalar joint returns to
supination
Thorax : Rt. side move
Posteriorly
Shoulder : Rt. side move
Posteriorly
Left gluteus medius on pelvis
64
Frontal Plane Analysis
MS to TS
Pelvis : Rt. side move
anteriorly
Hip : Lat. tilting to the left
med. rotn.
Knee : Med. rotn.
Ankle/Foot :
Thorax : Rt. side move
posteriorly
Shoulder : Rt. shoulder move
posteriorly
Right gluteus medius
65
A word on running
Walking is biomechanically like a
pendulum, KE to PE to KE
Running is biomechanically like a
spring
No double leg stance phase
Aerial phase or float period
Ground Reaction Force during
stance phase loads spring
(quads, achilles)
Unloading in preparation for
aerial phase is passive recoil
from tendons and connective
tissue and dynamic concentric
muscular contraction
66
Running: swing phase
Muscular rather than pendular
motion at hip.
Knee flexion, and ankle
dorsiflexion, bring CoM of the leg
closer to the hip. This reduces
moment of inertia and increases
angular velocity.
Knee movements largely passive
(i.e not due to muscle activity), and
result from transfer of momentum
from thigh.
Depending on the speed of
running, initial ground contact may
be with heel, whole foot, or ball of
foot.
67
Running: support phase
Hip: slight flexion followed by
extension. Gluteus maximus
activity initially eccentric
Knee: degree of flexion increases
with speed; that of extension
decreases. Quadriceps active at
knee, initially eccentrically
Ankle : dorsiflexion followed by
plantarflexion. Gastrocnemius and
soleus active during whole phase,
particularly so at the end.
Stretch shortening/energy
storage activity occurs at all
three joints
68
STAIR GAIT
Stair Ascent
Stance Phase
1. Weight acceptance
2. Pull up
3. Forward continuance
Swing Phase
1. Foot clearance
2. Foot Placement
Ascending stairs involves a large
amount of positive work that is
accomplished by concentric
action of the rectus femoris,
vastus lateralis, soleus and
medial gastrocnemeus
69
STAIR GAIT
Stair Descending
Swing Phase
1. Foot clearance
2. Foot Placement
Stance Phase
1. Weight acceptance
2. Pull up
3. Forward continuance
Descending stairs is achieved
mostly through eccentric activity of
same muscles and involves energy
absorption
The support moments exhibit
similar pattern in stair and level gait
But magnitude is greater in stair
gait
70
Sagittal Plane analysis of Stair gait
WA to PU
Hip : Extension from 60-
30° of flexion
Knee : Extension from
80-35° of flexion
Ankle : DF 20-25° of
DF, PF 25-15° of DF
Hip : Gluteus maximus,
Semitendinosus, Gluteus
medius
Knee : Vastus Lateralis,
Rectus femoris
Ankle : Tibialis anterior,
soleus, gastronemius
71
Sagittal Plane analysis of Stair gait
PU to FC
Hip : extension 30 - 5° of
flexion, flexion 5-20° of
flexion
Knee : Extension 35-10°
of flexion, flexion 5- 10°
of flexion
Ankle : PF 15°of DF to
15-10° of PF
Hip : Gluteus maximus,
gluteus medius,
semitendinosus
Knee : Vastus lateralis,
rectus femoris
Ankle : Soleus,
gastronemius, tibialis
anterior
72
Sagittal Plane analysis of Stair gait
Foot clearance through foot
placement
Hip : flexion 10-20° to 40-
60° of flexion, extension 40-
60° of flexion to 50° of
flexion
Knee : Flexion 10° of flexion
to 90-100° of flexion,
extension 90-100° of flexion
to 85° of flexion
Ankle : DF 10° of PF to 20°
of DF
Hip : Gluteus medius
Knee : semitendinosus,
vastus lateralis, rectus femoris
Ankle : Tibialis anterior
73
FACTORS INFLUENCING GAIT
Age
Gender
Assistive devices
abnormalities
74
FACTORS INFLUENCING GAIT
Age
A toddler has a higher COG, wider BOS, decreased
single leg support time, a shorter step length , a slower
velocity and a higher cadence in comparison to adult
3-5 year old showed increase in stride length adjusted to
leg length, step length and a faster speed in gait
From 6-13 years ROM of L/E were almost identical to
adults. However linear displacement, velocities and
accelerations are larger.
Elderly demonstrate a decrease in natural walking
speed, shorter stride and step length, longer duration of
double support periods and smaller swing to support
phase ratios
75
FACTORS INFLUENCING GAIT
Gender
Men Women
Joint angle increases as speed
increases
Gait speed faster i.e.. 118-134
cm/s
Step length larger
Not much joint angle increase
as compared to men
Gait speed slower i.e.. 110-129
cm/s
Step length smaller
Increased hip flexion and
decreased knee extension during
gait initiation
Increased knee flexion in pre-
swing
Increased stride length
Greater cadence 76
FACTORS INFLUENCING GAIT
Assistive devices
Canes are typically been used on the
contralateral side to an affected limb to reduce
forces acting at the affected limb
Use of cane on the contralateral side increase the
BOS and decrease muscle, GRF forces acting at
the affected hip and hip abductor & gluteus
maximus activity was reduced to 45%
Walker gait
77
COMMON GAIT ABNORMALITIES
A. Deformity(Contracture)
B. Muscle Weakness
C. Sensory Loss
D. Pain
E. Impaired Motor Control(Spasticity)
78
Ankle and Foot Gait Deviation
79
Knee Abnormal Gait
80
Knee Abnormal Gait
81
Knee Abnormal Gait
82
Knee Abnormal Gait
83
Hip Abnormal Gait
84
Hip Abnormal Gait
85
Pelvis and Trunk Pathological Gait
86
Pelvis and Trunk Pathological Gait
87
Pelvis and Trunk Pathological Gait
88
Pelvis and Trunk Pathological Gait
89
COMMON TYPES OF ABNORMAL GAITS
Scissor gait
Antalgic gait
Cerebellar ataxia
Festinating gait
Pigeon gait
Propulsive gait
Steppage gait
Stomping gait
Spastic gait
Myopathic gait
Magnetic gait
Trendelenburg gait
90
Scissor gait
Hypertonia in the legs, hips and pelvis means these areas
become flexed, to various degrees, giving the appearance of
crouching, while tight adductors produce extreme adduction,
presented by knees and thighs hitting or crossing in a scissors-
like movement, while the opposing muscles, the abductors,
become comparatively weak from lack of use. Most common in
patients with spastic cerebral palsy,
usually diplegic and paraplegic varieties. The individual is forced
to walk on tiptoe unless the dorsiflexor muscles are released by
an orthaepedic surgical procedure.
91
Antalgic gait
Person tries to avoid pain associated with the
ambulation. Often quick, short and soft foot
steps.
92
Ataxic gait
Spinal - proprioceptive pathways of the spine or
brainstem are interrupted. There is loss of
position and motion sense. The person will walk
with a wide base of gait with foot slap at heel
contact. Often watch feet as they walk.
Cerebellar - coordinating functions of the
cerebella are interfered with, so the person tends
to walk with a wide base of gait with an unsteady
irregular gait, even if watching feet.
93
Festinating gait
The patient has difficulty starting, but also has
difficulty stopping after starting. This is due to
muscle hypertonicity. The patient moves with
short, jerky steps.
94
Pigeon gait
In-toe gait is a very common problem among
children and even adults. Fortunately, most in-
toeing that is seen in children is a growth and
developmental condition and will correct itself
without medical or surgical intervention.
95
Propulsive gait
Disturbance of gait typical of Parkinsonism in
which, during walking, steps become faster and
faster with progressively shorter steps that pass
from a walking to a running pace and may
precipitate falling forward.
96
Steppage gait
A manner of walking in which the advancing
foot is lifted high so that the toes clear the
ground. Steppage gait is a sign of foot-drop.
97
Stomping gait
Sensory ataxia presents with an unsteady
"stomping" gait with heavy heel strikes, as well
as postural instability that is characteristically
worsened when the lack of proprioceptive input
cannot be compensated by visual input, such as
in poorly lit environments.
98
Spastic gait
An unbalanced muscle action of certain muscle
groups leads to deformity. Prime example is
"Scissor gait" - adduction and internal rotation
of the hips with an equinus of the feet and
flexion of the knee.
the legs are held together and move in a stiff
manner, the toes seeming to drag and catch.
99
Myopathic gait
The "waddling" is due to the weakness of the
proximal muscles of the pelvic girdle.
The patient uses circumduction to compensate
for gluteal weakness.
exaggerated alternation of lateral trunk
movements with an exaggerated elevation of the
hip.
100
Magnetic gait
Normal pressure hydrocephalus (NPH) gait
disturbance is often characterized as a "magnetic
gait," in which feet appear to be stuck to the
walking surface until wrested upward and
forward at each step. The gait may mimic a
Parkinsonian gait, with short shuffling steps and
stooped, forward-leaning posture, but there is
no rigidity or tremor. A broad-based gait may be
employed by the patient in order to compensate
for the ataxia.
101
Trendelenburg gait
The Trendelenburg gait is an abnormal gait caused by
weakness of the abductor muscles of the lower
limb, gluteus medius and gluteus minimus.
During the stance phase, the weakened abductor
muscles allow the pelvis to tilt down on the opposite
side. To compensate, the trunk lurches to the weakened
side to attempt to maintain a level pelvis throughout the
gait cycle. The pelvis sags on the opposite side of the
lesioned superior gluteal nerve.
102
Hemiplegic Gait Demonstration
The patient has unilateral
weakness and spasticity with
the upper extremity held in
flexion and the lower
extremity in extension. The
foot is in extension so the leg
is "too long" therefore, the
patient will have to
circumduct or swing the leg
around to step forward. This
type of gait is seen with a
UMN lesion.
This girl has a right
hemiparesis. Note how she
holds her right upper
extremity flexed at the elbow
and the hand with the thumb
tucked under the closed
fingers (this is "cortical
fisting"). There is
circumduction of the right
lower extremity.
103
Diplegic Gait Demonstration
The patient has spasticity in the
lower extremities greater than the
upper extremities. The hips and
knees are flexed and adducted with
the ankles extended and internally
rotated. When the patient walks
both lower extremities are
circumducted and the upper
extremities are held in a mid or low
guard position. This type of gait is
usually seen with bilateral
periventricular lesions. The legs are
more affected than the arms
because the corticospinal tract
axons that are going to the legs are
closest to the ventricles.
This man has an UMN lesion
affecting both lower extremities.
He has spasticity and weakness of
the legs and uses a walker to steady
himself. There is bilateral
circumduction of the lower
extremities.
104
Neuropathic Gait Demonstration
This type of gait is most
often seen in peripheral
nerve disease where the
distal lower extremity is
most affected. Because
the foot dorsiflexors are
weak, the patient has a
high stepping gait in an
attempt to avoid
dragging the toe on the
ground.
This girl has weakness of
the distal right lower
extremity so she can't
dorsiflex her foot. In
order to walk she has to
lift her right leg higher
then the left to clear the
foot and avoid dragging
her toes on the ground.
105
Myopathic Gait Demonstration
With muscular diseases, the
proximal pelvic girdle
muscles are usually the most
weak. Because of this the
patient will not be able to
stabilize the pelvis as they lift
their leg to step forward, so
the pelvis will tilt toward the
non-weight bearing leg which
results in a waddle type of
gait.
This young boy has pelvic
girdle weakness, which
produces a waddling type of
gait. Note the lumbar
hyperlordosis with the
shoulders thrust backwards
and the abdomen being
protuberant. This posture
places the center of gravity
behind the hips so the patient
doesn't fall forward because
of weak back and hip
extensors.
106
Parkinsonian Gait Demonstration
This type of gait is seen
with rigidity and
hypokinesia from basal
ganglia disease. The
patient's posture is
stooped forward. Gait
initiation is slow and
steps are small and
shuffling; turning is en
bloc like a statue.
This man's gait is
bradykinetic and his
steps are smaller then
usual. There is also the
pill-rolling tremor in his
hands. He turns en bloc
and there is decreased
facial expression
107
Choreiform Gait Demonstration
This is a hyperkinetic gait
seen with certain types of
basal ganglia disorders. There
is intrusion of irregular, jerky,
involuntary movements in
both the upper and lower
extremities.
Note the involuntary,
irregular, jerky movements of
this woman's body and
extremities, especially on the
right side. There are also
choreiform movements of
the face. A lot of her
movements have a writhing,
snake-like quality to them,
which could be called
choreoathetoisis.
108
Ataxic Gait Demonstration
The patient's gait is wide-
based with truncal instability
and irregular lurching steps
which results in lateral
veering and if severe, falling.
This type of gait is seen in
midline cerebellar disease. It
can also be seen with severe
lose of proprioception
(sensory ataxia)
This woman's gait is wide-
based and unsteady. She has
to use a walker or hold on to
someone in order to maintain
her balance (note how hard
she has to work with the
hand that she's holding on
with in order to maintain her
balance). Her ataxia is even
more apparent when she tries
to turn.
109

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Gait, movements that produce locomotion

  • 2. Gait Cycle or Stride A single gait cycle or stride is defined: Period when 1 foot contacts the ground to when that same foot contacts the ground again Each stride has 2 phases: Stance Phase Foot in contact with the ground Swing Phase Foot NOT in contact with the ground 2
  • 3. Stance Phase Principal events during the stance phase 1. Heel strike 2. Foot-flat (followed by opposite heel-off) 3. Heel-rise (followed by opposite heel strike) 4. Toe-off 3
  • 4. Principal events during the stance phase: heel-strike, foot-flat, heel-off, toe-off 4
  • 5. Activities occur in stance phase Traditional Method 1. Heel Strike : double support 2. Foot flat : total contact 3. Mid-stance : total weight bearing 4. Heel-off : heel clears the ground 5. Toe-off : toe clears the ground RLA Method 1. Initial contact : heel strike 2. Loading response : double support 3. Mid-stance : begins when contralateral L/E clears the ground & when the body come straight line to supporting limb 4. Terminal stance : end of mid stance to initial contact of CL L/E 5. Pre-swing : period of clearance from the ground 5
  • 6. Initial Contact Phase 1 The moment when the red foot just touches the floor. The heel (calcaneous) is the first bone of the foot to touch the ground. Meanwhile, the blue leg is at the end of terminal stance. 6
  • 7. Static Positions at Initial Contact Shoulder is extended Pelvis is rotated left Hip is flexed and externally rotated Knee is fully extended Ankle is dorsiflexed Foot is supinated Toes are slightly extended 7
  • 8. Loading Response Phase 2 The double stance period beginning Body wt. is transferred onto the red leg. Phase 2 is important for shock absorption, weight-bearing, and forward progression. The blue leg is in the pre-swing phase. 8
  • 9. Static Positions at Loading Response Shoulder is slightly extended Pelvis is rotated left hip is flexed and slightly externally rotated knee is slightly flexed ankle is plantar flexing to neutral foot is neutral Toes are neutral 9
  • 10. Mid-stance Phase 3 single limb support interval. Begins with the lifting of the blue foot and continues until body weight is aligned over the red (supporting) foot. The red leg advances over the red foot The blue leg is in its mid- swing phase. 10
  • 11. Static Positions at Midstance Shoulder is in neutral Pelvis is in neutral rotation Hip is in neutral Knee is fully extended Ankle is relatively neutral Foot is pronated Toes are neutral 11
  • 12. Terminal Stance Phase 4 Begins when the red heel rises and continues until the heel of the blue foot hits the ground. Body weight progresses beyond the red foot 12
  • 13. Static Positions at Terminal Stance Shoulder is slightly flexed Pelvis is rotated left Hip is extended and internally rotated Knee is fully extended Ankle is dorsi flexed Foot is slightly supinated Toes are neutral 13
  • 14. Pre-swing Phase 5 The second double stance interval in the gait cycle. Begins with the initial contact of the blue foot and ends with red toe- off. Transfer of body weight from ipsilateral to opposite limb takes place. 14
  • 15. Static Positions at Pre-swing Shoulder is flexed Pelvis is rotated right Hip is fully extended and internally rotated Knee is fully extended Ankle is plantar flexed Foot is fully supinated Toes are fully extended 15
  • 16. Swing phase Principal events during the Swing phase 1. Acceleration: ‘Initial swing’ 2. Mid swing : swinging limb overtakes the limb in stance 3. Deceleration: ‘Terminal swing’ 16
  • 17. Activities occur in swing phase Traditional Method 1. Acceleration : starts immediately from toe off 2. Mid stance : swing directly beneath body 3. Deceleration : knee extension and prepare for heel strike RLA Method 1. Initial swing : max. knee flexion 2. Mid swing : from max. knee flxn. to verticl. Postn. of tibia 3. Terminal swing : from verticl. Postn. of tibia to initial contact 17
  • 18. Initial Swing Phase 6 Begins when the red foot is lifted from the floor and ends when the red swinging foot is opposite the blue stance foot. It is during this phase that a foot drop gait is most apparent. The blue leg is in mid- stance. 18
  • 19. Static Positions at Initial Swing Shoulder is flexed Spine is rotated left Pelvis is rotated right hip is slightly extended and internally rotated Knee is slightly flexed Ankle is fully plantar flexed Foot is supinated Toes are slightly flexed 19
  • 20. Mid-swing Phase 7 Starts at the end of the initial swing and continues until the red swinging limb is in front of the body Advancement of the red leg The blue leg is in late mid-stance. 20
  • 21. Static Positions at Mid-swing Shoulder is neutral Spine is neutral Pelvis is neutral Hip is neutral Knee is flexed 60-90° Ankle is plantar flexed to neutral Foot is neutral Toes are slightly extended 21
  • 22. Terminal Swing Phase 8 Begins at the end of mid- swing and ends when the foot touches the floor. Limb advancement is completed at the end of this phase. 22
  • 23. Static Positions at Terminal Swing Shoulder is extended Spine is rotated right Pelvis is rotated left Hip is flexed and externally rotated Knee is fully extended Ankle is fully dorsi flexed Foot is neutral Toes are slightly extended 23
  • 24. DISTANCE VARIABLES Step length Stride length Width of walking base Degree of toe out 24
  • 25. Step Length Distance between corresponding successive points of heel contact of the opposite feet Rt step length = Lt step length (in normal gait) 25
  • 26. Stride Length Distance between successive points of heel contact of the same foot Double the step length (in normal gait) 26
  • 27. Walking Base Side-to-side distance between the line of the two feet Also known as ‘stride width’ Normal is 3.5 inches 27
  • 28. Degree of toe out Represents the angle of foot placement It is the angle formed by each foot’s line of progression and a line intersecting the centre of the heel and the second toe Normal angle is 7°for men at free speed walking 28
  • 29. TEMPORAL VARIABLES Stance Time : Is the amount of time that elapses during the stance phase of one extremity in a gait cycle Single support time : is the amount of time that elapses during the period when only one extremity is on the supporting surface in a gait cycle Double support time : is the amount of time that a person spends with both feet on the ground during one gait cycle Stride duration : is the amount of time it takes to accomplish one stride Step duration : is the amount of time spent during a single step 29
  • 30. TEMPORAL VARIABLES Cadence Number of steps per unit time Normal: 100 – 115 steps/min Cultural/social variations Velocity Distance covered by the body in unit time Usually measured in cm/s Instantaneous velocity varies during the gait cycle Average velocity (m/min) = step length (m) x cadence (steps/min) Comfortable Walking Speed (CWS) Least energy consumption per unit distance Average= 80 m/min (~ 5 km/h , ~ 3 mph) 30
  • 31. DETERMINANTS OF GAIT 1. Lateral pelvic tilt 2. Knee flexion 3. Knee, ankle, foot interactions 4. Pelvic forward and backward rotation 5. Physiologic valgus of knee DGs represents the adjustments made by above components that help to keep movements of body’s COG to minimum. They are credited with decreasing the vertical and lateral excursions of the body’s COG and therefore decreasing energy expenditure and making gait more efficient 31
  • 32. Lateral pelvic tilt During mid-stance the COG reaches the peak level & the total body supported by one lower extremity The pelvis slopes downwards laterally towards the leg which is in swing phase. i.e. rotation about an anterior-posterior axis Only anatomically possible if the swing leg can be shortened sufficiently (principally by knee flexion) to clear the ground. Where this is not possible (e.g. through injury), the absence of pelvic tilt and pronounced movements of the upper body are obvious. 32
  • 33. Knee flexion Another DG which helps to reduce the COG during mid- stance As the hip joint passes over the foot during the support phase, there is some flexion of the knee. This reduces vertical movements at the hip, and therefore of the trunk and head. 33
  • 34. Knee, ankle, foot interactions KAF interaction prevent abrupt hike in COG from heel strike to foot flat After heel strike huge upward displacement of COG occurs This is reduced by Knee flexion, ankle plantar flexion & foot pronation. From mid stance to heel off there is sudden drop in COG Ankle plantar flexion, knee extension and foot supination maintain this 34
  • 35. Pelvic forward and backward rotation Forward rotn. occurs in swing phase It starts during acceleration and ends in deceleration During mid-swing pelvis comes to neutral position Forward and backward rotation help to prevent further reduction in COG which started from mid- stance During deceleration both lower extremities lengthens This prevents in further reduction of COG 35
  • 36. Physiologic valgus of knee Is minimised by having a narrow walking base i.e. feet closer together than are hips. Therefore less energy is used moving hip from side to side (less lateral movement needed to balance body over stance foot) Reduced lateral pelvic displacement 36
  • 37. Efficiency, and energy considerations • Walking is very energy- efficient: little ATP is required. • This is because of various mechanisms that ensure the mechanical energy the body has is passed on from one step to the next. • The two forms of mechanical energy involved are •kinetic energy (energy due to movement •potential energy (energy due to position) Economy (J m-1) 37
  • 38. A conventional pendulum – energy interconversion P.E. – Potential energy K.E. – Kinetic energy Three points on a pendulum swing are illustrated. As the pendulum swings away from the midpoint, in either direction, KE is progressively converted into PE At the extreme points in the swing, there is no KE at all and all the energy is present as PE 38
  • 39. Conventional pendulum action during the swing phase The legs move as conventional pendulums during the swing phase (with a little assistance from the hip flexors). This reduces the amount of muscle energy needed to move the swinging leg forward It also accounts for the “natural” frequency of gait that has optimal energy efficiency Although the legs swing forwards much like pendulums, they are prevented from swinging backwards by foot strike. During the stance phase, the leg can be viewed as an “inverted pendulum”. This action also involves inter- conversion of potential and kinetic energy 39
  • 40. An “inverted” pendulum The pendulum “bounces” backwards and forwards, using the springs. 40
  • 41. “Inverted” pendulum action during the stance phase During the stance phase, the leg can be viewed as an “inverted pendulum”. The forward momentum of the body gives it the necessary initial angular velocity of rotation (taking the place of the “spring” on the previous slide). “Inverted” pendulum action also involves inter-conversion of potential and kinetic energy, but in this case (unlike a conventional pendulum) KE reaches a minimum at the midpoint of the motion, and PE is highest at that point. When reaching the endpoint of its “inverted swing” the stance leg does not swing back, as a real inverted pendulum would, because the foot is taken off the floor, the fulcrum transfers from the foot to the hip, and the leg swings again as a conventional pendulum. 41
  • 42. Positive & negative Work At a cadence of 105-112 steps/minute between heel strike and foot flat 1. a brief burst of positive work (energy generation) occurs as the hip extensors contract concentrically 2. while the knee extensors perform negative work (energy absorption) by acting eccentrically to control knee flexion from foot flat through mid-stance 1. Negative work is done by plantar flexors as the leg rotates over the foot during the period of stance 2. Positive work of the knee extensors occurs during this period to extend the knee late stance and in early swing 1. Positive work of plantar flexors and hip flexors increase the energy level of the body In late swing 1. negative work is performed by the hip extensors as they work eccentrically to decelerate the leg in preparation for initial contact 42
  • 43. Forces The principal forces are: body weight (BW) ground reaction force (GRF) muscle force (MF) BW and GRF are external forces; so the movement of the centre of mass (CoM) can be predicted from them alone. MF must be examined however if we wish to consider either of the following: movements of individual limbs or body segments, why GRF changes in magnitude and direction during the gait cycle. 43
  • 44. Vitally important point: Muscle forces can only influence the movement of the body as a whole indirectly, by their effects on the GRF 44
  • 45. Walking as a “controlled fall” One way of beginning to understand the mechanics of walking is to view the movements as a “controlled fall” When starting a walk, we lean forward, overbalancing from the equilibrium position. This gives the upper part of the body forwards (and downwards) motion As the body falls forwards, a leg is extended forwards and halts the fall At the same time, the other leg “kicks off” in order to keep the body moving forwards. This forward momentum carries the body forward into the next forward fall, i.e. the start of the next step 45
  • 46. Walking as a controlled fall: forces involved When starting to move, we lean forward (MF) As the body starts to fall (BW), a leg is extended forwards and halts the fall (MF; GRF) At the same time, the other leg “kicks off, upwards and forwards” (MF; GRF) in order to keep the body moving forwards. This forward momentum carries the body forward into the next forward fall, i.e. the start of the next step 46
  • 47. Body weight Always acts vertically downwards from the CoM If its line of action does not pass through a joint, it will produce a torque about that joint The torque will cause rotation at the joint unless it is opposed by another force (e.g. muscle, or ligament) BW contributes to GRF 47
  • 48. Ground reaction force The force that the foot exerts on the floor due to gravity & inertia is opposed by the ground reaction force Ground reaction force (RF) may be resolved into horizontal (HF) & vertical (VF) components. Understanding joint position & RF leads to understanding of muscle activity during gait Forces are typically resolved into: 1. Vertical Compression (z) 2. Anterior-Posterior Shear (y) 3. Medial-Lateral Shear (x) 48
  • 49. Muscle force In gait, as in all human movement, muscle activation generates internal joint moments (torques) that: Contribute to ground reaction force Ensure balance Increase energy economy Allow flexible gait patterns Slow down and/or prevent limb movements Much muscle activity during gait is eccentric or isometric, rather than concentric 49
  • 50. Center Of Pressure (COP) Represents the centroid of foot forces on the floor This is an idealization, because pressures are distributed all over It is important, because we want to know where the GRF is applied to the body When measured by a force plate, it is more correctly called the point of application of the GRF Plotting the COP as it moves under the foot: 1. Normal Path: Center of the calcaneus or slightly lateral, curving laterally and then medial (pronation) and ending between the 1st and second toes 2. Variable: Normal individuals can have many COP trajectories, just by changing the footgear 50
  • 51. Muscles, Joints, and Forces Eccentric Concentric Ground Reaction Force 51
  • 52. Sagittal Plane Analysis Initial Contact Hip 30° of flexion, knee is extended ankle is neutral GRF Ant. to hip, drives the hip into flexion Ant. to knee drives the knee into extension Ankle into plantar flexion Hip: hamstrings, gluteus maximus, and adductor magnus (i to e) Knee: quadriceps (c to e) Tibiotalar joint: tibialis anterior (e) Subtalar joint: anterior and lateral compartment muscles (e) 52
  • 53. Sagittal Plane Analysis Loading Response Hip extension 25° Knee flexion to 20° Ankle plantar flexion to 10° Contralateral pelvis rotates anterior GRF Ant. to hip posterior to knee posterior to ankle Hip : extensors (e), Abductors (e) limit contralateral drop to 5° Knee : Quadriceps fire (c) Ankle :Tibialis anterior (e) 53
  • 54. Sagittal Plane Analysis Mid Stance GRF through hip, knee, and ankle Muscular activity terminates Hip and knee stability provided by ligamentous restraints GRF Posterior to hip Anterior to knee and ankle Gastrosoleus complex fires to initiate knee flexion Pelvis continues to rotate, abductors continue to resist pelvic drop 54
  • 55. Sagittal Plane Analysis Terminal Stance No change in GRF Free forward fall Strong activation of gastrosoleus complex 55
  • 56. Sagittal Plane Analysis Pre-Swing Hip 20° of hyperextansion Knee 30° of flexion Ankle 20° of plantarflexion Toes 50° of hyper extension GRF posterior to hip, knee anterior to ankle Rapid flexion of knee from rapid heel rise and unweighting of limb Rectus femoris initiates hip flexion Adductor longus fires Hip : iliopsoas, adductor magnus, adductor longus Knee : Quadriceps Ankle :Gastrosoleus complex Toes : Ab.hal., FDB, FHB, Introssei, lumb. 56
  • 57. Sagittal Plane Analysis Initial Swing Hip 0-30° of flexion Knee from 30-60° of flexion and extension from 60-30° Ankle 20° of plantarflexion to neutral Foot clearance is passive due to rapid hip flexion, unless gait is very slow In slow gait, tibialis anterior and hamstrings fire to help Gait cadence (speed) governed by accelerations of hip flexion during this phase Hip flexion Rectus femoris Iliacus Adductor longus Gracilis Sartorius Rest of limb is passive pendulum 57
  • 58. Sagittal Plane Analysis Mid Swing Tibialis anterior fires to maintain foot position Knee extension and hip flexion continue by inertia 58
  • 59. Sagittal Plane Analysis Terminal Swing Decelerate knee extension and hip flexion Hamstrings Gluteus max Quads co-contract Tibialis anterior maintains ankle position 59
  • 60. Frontal Plane Analysis IC to LR Pelvis : forward rotn. Hip : med. rotn. of femur Knee : increased valgus Ankle : increased pronation Thorax : Post. postion at initial contact and begins moving forward Shoulder : extended and moving forward Gracilis, vastus medialis, semitendinosus, LH of biceps femoris Tibialis post. to control valgus thrust 60
  • 61. Frontal Plane Analysis LR to Mid-stance Pelvis : Rt. side rotating backward to reach neutral. Lat. tilt towards the swinging extremity Hip : Med. rotn. of femur continues to neutral. Knee : Reduction in valgus and tibia begins to rotate laterally Ankle-foot : neutral at mid- stance Thorax : Rt. side move forward Shoulder : Move forward Hip abductors are active Tibialis post. produce supination 61
  • 62. Frontal Plane Analysis Mid-stance to TS Pelvis : Rt. side move Posteriorly Hip : Lat. rotn. of femur and adduction Knee : lat. rotn. of tibia Ankle & foot : supination of subtalar jt. increases Thorax : Rt. side move forward Shoulder : Rt. shoulder move forward Hip : inconsistent adductor activity Ankle plantar flexor activity 62
  • 63. Frontal Plane Analysis TS to PS Pelvis : Lt. side move forward, lat. tilting to swing side ends as double support begins Hip : Abduction as wt. shifted to opp. extremity, Lat. rotn. of femur Knee : Lat rotn. of tibia Foot/Ankle : Wt. shifted to toes. Supination of sub talar joint Thorax : Translation to the left Shoulder : Moving forward Hip adductors control pelvis Plantar flexion 63
  • 64. Frontal Plane Analysis IS to MS Pelvis : Lat. pelvic tilt to the rt. Right side move forward Hip : Lat. rotn to med. rotn. Knee : From lat. to med. rotn Foot/Ankle : NWBing subtalar joint returns to supination Thorax : Rt. side move Posteriorly Shoulder : Rt. side move Posteriorly Left gluteus medius on pelvis 64
  • 65. Frontal Plane Analysis MS to TS Pelvis : Rt. side move anteriorly Hip : Lat. tilting to the left med. rotn. Knee : Med. rotn. Ankle/Foot : Thorax : Rt. side move posteriorly Shoulder : Rt. shoulder move posteriorly Right gluteus medius 65
  • 66. A word on running Walking is biomechanically like a pendulum, KE to PE to KE Running is biomechanically like a spring No double leg stance phase Aerial phase or float period Ground Reaction Force during stance phase loads spring (quads, achilles) Unloading in preparation for aerial phase is passive recoil from tendons and connective tissue and dynamic concentric muscular contraction 66
  • 67. Running: swing phase Muscular rather than pendular motion at hip. Knee flexion, and ankle dorsiflexion, bring CoM of the leg closer to the hip. This reduces moment of inertia and increases angular velocity. Knee movements largely passive (i.e not due to muscle activity), and result from transfer of momentum from thigh. Depending on the speed of running, initial ground contact may be with heel, whole foot, or ball of foot. 67
  • 68. Running: support phase Hip: slight flexion followed by extension. Gluteus maximus activity initially eccentric Knee: degree of flexion increases with speed; that of extension decreases. Quadriceps active at knee, initially eccentrically Ankle : dorsiflexion followed by plantarflexion. Gastrocnemius and soleus active during whole phase, particularly so at the end. Stretch shortening/energy storage activity occurs at all three joints 68
  • 69. STAIR GAIT Stair Ascent Stance Phase 1. Weight acceptance 2. Pull up 3. Forward continuance Swing Phase 1. Foot clearance 2. Foot Placement Ascending stairs involves a large amount of positive work that is accomplished by concentric action of the rectus femoris, vastus lateralis, soleus and medial gastrocnemeus 69
  • 70. STAIR GAIT Stair Descending Swing Phase 1. Foot clearance 2. Foot Placement Stance Phase 1. Weight acceptance 2. Pull up 3. Forward continuance Descending stairs is achieved mostly through eccentric activity of same muscles and involves energy absorption The support moments exhibit similar pattern in stair and level gait But magnitude is greater in stair gait 70
  • 71. Sagittal Plane analysis of Stair gait WA to PU Hip : Extension from 60- 30° of flexion Knee : Extension from 80-35° of flexion Ankle : DF 20-25° of DF, PF 25-15° of DF Hip : Gluteus maximus, Semitendinosus, Gluteus medius Knee : Vastus Lateralis, Rectus femoris Ankle : Tibialis anterior, soleus, gastronemius 71
  • 72. Sagittal Plane analysis of Stair gait PU to FC Hip : extension 30 - 5° of flexion, flexion 5-20° of flexion Knee : Extension 35-10° of flexion, flexion 5- 10° of flexion Ankle : PF 15°of DF to 15-10° of PF Hip : Gluteus maximus, gluteus medius, semitendinosus Knee : Vastus lateralis, rectus femoris Ankle : Soleus, gastronemius, tibialis anterior 72
  • 73. Sagittal Plane analysis of Stair gait Foot clearance through foot placement Hip : flexion 10-20° to 40- 60° of flexion, extension 40- 60° of flexion to 50° of flexion Knee : Flexion 10° of flexion to 90-100° of flexion, extension 90-100° of flexion to 85° of flexion Ankle : DF 10° of PF to 20° of DF Hip : Gluteus medius Knee : semitendinosus, vastus lateralis, rectus femoris Ankle : Tibialis anterior 73
  • 75. FACTORS INFLUENCING GAIT Age A toddler has a higher COG, wider BOS, decreased single leg support time, a shorter step length , a slower velocity and a higher cadence in comparison to adult 3-5 year old showed increase in stride length adjusted to leg length, step length and a faster speed in gait From 6-13 years ROM of L/E were almost identical to adults. However linear displacement, velocities and accelerations are larger. Elderly demonstrate a decrease in natural walking speed, shorter stride and step length, longer duration of double support periods and smaller swing to support phase ratios 75
  • 76. FACTORS INFLUENCING GAIT Gender Men Women Joint angle increases as speed increases Gait speed faster i.e.. 118-134 cm/s Step length larger Not much joint angle increase as compared to men Gait speed slower i.e.. 110-129 cm/s Step length smaller Increased hip flexion and decreased knee extension during gait initiation Increased knee flexion in pre- swing Increased stride length Greater cadence 76
  • 77. FACTORS INFLUENCING GAIT Assistive devices Canes are typically been used on the contralateral side to an affected limb to reduce forces acting at the affected limb Use of cane on the contralateral side increase the BOS and decrease muscle, GRF forces acting at the affected hip and hip abductor & gluteus maximus activity was reduced to 45% Walker gait 77
  • 78. COMMON GAIT ABNORMALITIES A. Deformity(Contracture) B. Muscle Weakness C. Sensory Loss D. Pain E. Impaired Motor Control(Spasticity) 78
  • 79. Ankle and Foot Gait Deviation 79
  • 86. Pelvis and Trunk Pathological Gait 86
  • 87. Pelvis and Trunk Pathological Gait 87
  • 88. Pelvis and Trunk Pathological Gait 88
  • 89. Pelvis and Trunk Pathological Gait 89
  • 90. COMMON TYPES OF ABNORMAL GAITS Scissor gait Antalgic gait Cerebellar ataxia Festinating gait Pigeon gait Propulsive gait Steppage gait Stomping gait Spastic gait Myopathic gait Magnetic gait Trendelenburg gait 90
  • 91. Scissor gait Hypertonia in the legs, hips and pelvis means these areas become flexed, to various degrees, giving the appearance of crouching, while tight adductors produce extreme adduction, presented by knees and thighs hitting or crossing in a scissors- like movement, while the opposing muscles, the abductors, become comparatively weak from lack of use. Most common in patients with spastic cerebral palsy, usually diplegic and paraplegic varieties. The individual is forced to walk on tiptoe unless the dorsiflexor muscles are released by an orthaepedic surgical procedure. 91
  • 92. Antalgic gait Person tries to avoid pain associated with the ambulation. Often quick, short and soft foot steps. 92
  • 93. Ataxic gait Spinal - proprioceptive pathways of the spine or brainstem are interrupted. There is loss of position and motion sense. The person will walk with a wide base of gait with foot slap at heel contact. Often watch feet as they walk. Cerebellar - coordinating functions of the cerebella are interfered with, so the person tends to walk with a wide base of gait with an unsteady irregular gait, even if watching feet. 93
  • 94. Festinating gait The patient has difficulty starting, but also has difficulty stopping after starting. This is due to muscle hypertonicity. The patient moves with short, jerky steps. 94
  • 95. Pigeon gait In-toe gait is a very common problem among children and even adults. Fortunately, most in- toeing that is seen in children is a growth and developmental condition and will correct itself without medical or surgical intervention. 95
  • 96. Propulsive gait Disturbance of gait typical of Parkinsonism in which, during walking, steps become faster and faster with progressively shorter steps that pass from a walking to a running pace and may precipitate falling forward. 96
  • 97. Steppage gait A manner of walking in which the advancing foot is lifted high so that the toes clear the ground. Steppage gait is a sign of foot-drop. 97
  • 98. Stomping gait Sensory ataxia presents with an unsteady "stomping" gait with heavy heel strikes, as well as postural instability that is characteristically worsened when the lack of proprioceptive input cannot be compensated by visual input, such as in poorly lit environments. 98
  • 99. Spastic gait An unbalanced muscle action of certain muscle groups leads to deformity. Prime example is "Scissor gait" - adduction and internal rotation of the hips with an equinus of the feet and flexion of the knee. the legs are held together and move in a stiff manner, the toes seeming to drag and catch. 99
  • 100. Myopathic gait The "waddling" is due to the weakness of the proximal muscles of the pelvic girdle. The patient uses circumduction to compensate for gluteal weakness. exaggerated alternation of lateral trunk movements with an exaggerated elevation of the hip. 100
  • 101. Magnetic gait Normal pressure hydrocephalus (NPH) gait disturbance is often characterized as a "magnetic gait," in which feet appear to be stuck to the walking surface until wrested upward and forward at each step. The gait may mimic a Parkinsonian gait, with short shuffling steps and stooped, forward-leaning posture, but there is no rigidity or tremor. A broad-based gait may be employed by the patient in order to compensate for the ataxia. 101
  • 102. Trendelenburg gait The Trendelenburg gait is an abnormal gait caused by weakness of the abductor muscles of the lower limb, gluteus medius and gluteus minimus. During the stance phase, the weakened abductor muscles allow the pelvis to tilt down on the opposite side. To compensate, the trunk lurches to the weakened side to attempt to maintain a level pelvis throughout the gait cycle. The pelvis sags on the opposite side of the lesioned superior gluteal nerve. 102
  • 103. Hemiplegic Gait Demonstration The patient has unilateral weakness and spasticity with the upper extremity held in flexion and the lower extremity in extension. The foot is in extension so the leg is "too long" therefore, the patient will have to circumduct or swing the leg around to step forward. This type of gait is seen with a UMN lesion. This girl has a right hemiparesis. Note how she holds her right upper extremity flexed at the elbow and the hand with the thumb tucked under the closed fingers (this is "cortical fisting"). There is circumduction of the right lower extremity. 103
  • 104. Diplegic Gait Demonstration The patient has spasticity in the lower extremities greater than the upper extremities. The hips and knees are flexed and adducted with the ankles extended and internally rotated. When the patient walks both lower extremities are circumducted and the upper extremities are held in a mid or low guard position. This type of gait is usually seen with bilateral periventricular lesions. The legs are more affected than the arms because the corticospinal tract axons that are going to the legs are closest to the ventricles. This man has an UMN lesion affecting both lower extremities. He has spasticity and weakness of the legs and uses a walker to steady himself. There is bilateral circumduction of the lower extremities. 104
  • 105. Neuropathic Gait Demonstration This type of gait is most often seen in peripheral nerve disease where the distal lower extremity is most affected. Because the foot dorsiflexors are weak, the patient has a high stepping gait in an attempt to avoid dragging the toe on the ground. This girl has weakness of the distal right lower extremity so she can't dorsiflex her foot. In order to walk she has to lift her right leg higher then the left to clear the foot and avoid dragging her toes on the ground. 105
  • 106. Myopathic Gait Demonstration With muscular diseases, the proximal pelvic girdle muscles are usually the most weak. Because of this the patient will not be able to stabilize the pelvis as they lift their leg to step forward, so the pelvis will tilt toward the non-weight bearing leg which results in a waddle type of gait. This young boy has pelvic girdle weakness, which produces a waddling type of gait. Note the lumbar hyperlordosis with the shoulders thrust backwards and the abdomen being protuberant. This posture places the center of gravity behind the hips so the patient doesn't fall forward because of weak back and hip extensors. 106
  • 107. Parkinsonian Gait Demonstration This type of gait is seen with rigidity and hypokinesia from basal ganglia disease. The patient's posture is stooped forward. Gait initiation is slow and steps are small and shuffling; turning is en bloc like a statue. This man's gait is bradykinetic and his steps are smaller then usual. There is also the pill-rolling tremor in his hands. He turns en bloc and there is decreased facial expression 107
  • 108. Choreiform Gait Demonstration This is a hyperkinetic gait seen with certain types of basal ganglia disorders. There is intrusion of irregular, jerky, involuntary movements in both the upper and lower extremities. Note the involuntary, irregular, jerky movements of this woman's body and extremities, especially on the right side. There are also choreiform movements of the face. A lot of her movements have a writhing, snake-like quality to them, which could be called choreoathetoisis. 108
  • 109. Ataxic Gait Demonstration The patient's gait is wide- based with truncal instability and irregular lurching steps which results in lateral veering and if severe, falling. This type of gait is seen in midline cerebellar disease. It can also be seen with severe lose of proprioception (sensory ataxia) This woman's gait is wide- based and unsteady. She has to use a walker or hold on to someone in order to maintain her balance (note how hard she has to work with the hand that she's holding on with in order to maintain her balance). Her ataxia is even more apparent when she tries to turn. 109