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Core Hip and Slings -
Intelligent prescription
                            PRESENTED BY:
              Max MARTIN BAppSc (Hons) AEP
Movement is a behaviour
  Developmental and learned
Quality over quantity
Posture is a good baseline for movement
Posture is not the cause of dysfunction but a SYMPTOM
Such dysfunction corresponds to compromised activity of muscles
Stabilisers typically become hypotonic/inhibited – ‘allowing’ faulty
posture
Gross movers typically become hypertonic/facilitated – ‘driving’
faulty posture


             Prescription Paradigms
synergist

tightness   weakness

                       antagonist
Why weakness?

Muscle inhibition due to pain/injury
Muscle susceptibility – eg. VMO vs VL atrophy post surgery
Muscle inactivity in chronic postures – eg. Sedentary behaviours
CNS driven protection
Why tightness?
Joint ROM can be limited by the following factors

1. Joint constraints

2. connective tissue (40%) –
   protective, inactivity, hypertonicity

3. Neurogenic constraints (voluntary and reflexive) -
   protective

4. Myogenic constraints – overload protective
tightness?

        Or

gaining stability??
Clinical/Practical findings



                                      synergist
                                      Glute max

tightness          weakness
Hamstrings
 Hip Flexors
 • Psoas
                                      antagonist
 • Iliacus                             Glute max
 • TFL
 • Rec fem                             TrA (+core)
 Lumbar Erectors
Joint by joint approach

Foot            Stable       unstable
Ankle           Mobile       Stiff
Knee            Stable       unstable
Hip             Mobile       Stiff
Lx Spine        Stable       unstable
Tx Spine        Mobile       Stiff
Scapula         Stable       unstable
GH Joint        Mobile       Stiff



           Prescription Paradigms
CORE Anatomy
The research journey
1992: TrA found to exhibit anticipatory function (activation prior to activation of prime
movers in arm movements) in healthy subjects (Cresswell)
1996-97: TrA disrupted in multi-directional arm movements in LBP subjects
1998: TrA also disrupted in lower limb movements among LBP patients
2001: TrA latency in LBP patients shown to increase with increasing task demand
2001: Experimentally induced pain causes disruption (hypoactivity) in the TrA
2002: TrA contraction shown to increase stiffness of the sacro-illiac joint to a greater
extent than a more global abdominal contraction
2007: Pelvic floor shown to share the same pre-emptive quality as TrA and MU
2009: LBP patients shown to have greater lumbo-pelvic instability in simple open-chain
stability exercises (eg Leg Loads) compared to controls.
Lumbar Vertebrae
Largest and strongest due to compressive
load.
Cortical bone shell with cancellous bone
core (trabeculae). Vertical Column
alignment.



Aids shock absorption quality of L1-5.
Age and repetitious loading degenerate
horizontal trabeculae ‘struts’
Lumbar facet joints

Bony articulations between vertebrae.
Synovial Joints- articular surfaces covered in
hyaline cartilage.
Allow flexion and extension
Movement pumps fluid in and out of joint
space. Fixed postures lead to joint dehydration
and degeneration.
Constant compression caused by hypertonicity
of paraspinals can accelerate degeneration.
Sacroiliac Joints
     Junction point between spine and pelvis.
     Synovial Joint- innervated by pain
     receptors.
     Corrugated design to assist stability.
     Allows forward and backward tilting of the
     sacrum.
     Sublaxation possible, resulting in dull ache
     or sharp pain that may refer inferiorly.
Intervertebral Discs
Colloidal gel nucleus
Concentric rings of fibrocartilage (lamellae)
form the annulus.
Outer third ONLY innervated by pain and
mechanoreceptors.
Slight movement of the vertebrae helps
rehydrate discs.
Repetitious torsion forces can derange
annulus, allowing nucleus to seep out.
Late warning of this process due to lack of
pain receptors amongst inner 2/3 of annulus.
Intervertebral Discs Cont’d


Discs are poor shock absorbers
   – Very little compressive potential
   – Nucleus facilitates movement rather
     than compression
Thoracolumbar Fascia

Dense multilayered sheet of
connective tissue.
Insertion point for many muscles
Overactive lats and/or glutes can
cause excess collagen
deposition, making TLF more stiff.
This can restrict the ability of TrA to
slide freely as it pulls on deep layer.
Transversus Abdominis
Transversus Abdominis

Intra-abdominal pressure, thus making this area more stiff (less bendable).
Increases the stiffness of thoraco-lumbar fascia and abdominal aponeurosis.
Line of pull helps to align the ribs and pelvis in anatomically correct
Fibres crossing the sacroiliac joints pull the Ilium and the sacrum closer
together, decreasing laxity in these joints.
Gluteus Maximus




Primary hip extensor and external rotator*
Important for maintaining upright posture
Stabiliser of SIJ via attachment to TLF
Supports hip and knee via ITB attachment
Functional role in stepping, running, climbing etc. and…
DECELERATION
Gluteus Medius
Primary abductor and controller of rotation of the hip*
Functionally supports pelvis during SL stance and gait
Plays rotator cuff-like role
Strongest in neutral or slight adduction
Tensor Fascia Latae


Primary functions are hip
flexion, internal rotation and abduction
(via ITB)
Works in synergy with glute max:
     Tighten ITB to extend
     knee joint
     Control movements of pelvis on
     femur and femur on tibia when
     weight bearing
Iliotibial Band

 Thick, lateral aspect of
 fascia lata
 Attachment point for glute
 max, TFL (and glute med)
 Indirect insertion onto
 patella
 Anatomically impossible to
 stretch effectively
Piriformis & External Hip Rotators

              Primarily lateral rotator of the hip
              In hip flexion, will also abduct the hip
              Secondary phasic stabiliser of the SIJ
              Close relationship to sciatic nerve
                   Piriformis syndrome
Vastus Medialis & Lateralis


               Primary action is knee extension in inner
               range- 15-20deg of knee flexion
               Provide medial and lateral stability
               to patella respectively
               Perform anticipatory role
               Often dysfunctional (knee pain, pronation)
Single Legged Squat




               Functional strength exercise
               Assessment tool
SLSq Research (performance and strength)
 Wilson et al (2006) Frontal Plane Projection Angle measured
 (FPPA)
  Women > FPPA
  Weakness in external rotators correlated most closely to
    FPPA (predisposes to ACL injury & PFP)
 Claiborne et al (2006)
  Hip abductor strength most important for resisting
    valgus alignment
 Crossley, 2006
  Glute med shown to be latent in poor SLQ
  Abduction strength and Trendelenburg test shows
    correlation to SLSq
Slings
Thomas Myers- Anatomy Trains




                     Superficial Front Line
Superficial Back Line
Spiral Line
Correctives!!
Core exercises:
Leg loads (ant oblique, ant superficial and Spiral)
hip extension (post oblique and posterior superficial)
Hip lifts/SL (post oblique and post superficial)
Hip exercises:
Squat (posterior superficial),
SL DL (Lateral), hitches (lateral) and Rots (posterior and
anterior oblique), SL SQ (lateral)
PRESENTED BY:
  Max MARTIN BAppSc (Hons)AEP
      @iNformMaxMartin
max@correctiveexerciseaustralia.com

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Core hip and slings function review oct 2012

  • 1. Core Hip and Slings - Intelligent prescription PRESENTED BY: Max MARTIN BAppSc (Hons) AEP
  • 2. Movement is a behaviour Developmental and learned Quality over quantity Posture is a good baseline for movement Posture is not the cause of dysfunction but a SYMPTOM Such dysfunction corresponds to compromised activity of muscles Stabilisers typically become hypotonic/inhibited – ‘allowing’ faulty posture Gross movers typically become hypertonic/facilitated – ‘driving’ faulty posture Prescription Paradigms
  • 3. synergist tightness weakness antagonist
  • 4. Why weakness? Muscle inhibition due to pain/injury Muscle susceptibility – eg. VMO vs VL atrophy post surgery Muscle inactivity in chronic postures – eg. Sedentary behaviours CNS driven protection
  • 5. Why tightness? Joint ROM can be limited by the following factors 1. Joint constraints 2. connective tissue (40%) – protective, inactivity, hypertonicity 3. Neurogenic constraints (voluntary and reflexive) - protective 4. Myogenic constraints – overload protective
  • 6. tightness? Or gaining stability??
  • 7. Clinical/Practical findings synergist Glute max tightness weakness Hamstrings Hip Flexors • Psoas antagonist • Iliacus Glute max • TFL • Rec fem TrA (+core) Lumbar Erectors
  • 8. Joint by joint approach Foot Stable unstable Ankle Mobile Stiff Knee Stable unstable Hip Mobile Stiff Lx Spine Stable unstable Tx Spine Mobile Stiff Scapula Stable unstable GH Joint Mobile Stiff Prescription Paradigms
  • 10. The research journey 1992: TrA found to exhibit anticipatory function (activation prior to activation of prime movers in arm movements) in healthy subjects (Cresswell) 1996-97: TrA disrupted in multi-directional arm movements in LBP subjects 1998: TrA also disrupted in lower limb movements among LBP patients 2001: TrA latency in LBP patients shown to increase with increasing task demand 2001: Experimentally induced pain causes disruption (hypoactivity) in the TrA 2002: TrA contraction shown to increase stiffness of the sacro-illiac joint to a greater extent than a more global abdominal contraction 2007: Pelvic floor shown to share the same pre-emptive quality as TrA and MU 2009: LBP patients shown to have greater lumbo-pelvic instability in simple open-chain stability exercises (eg Leg Loads) compared to controls.
  • 11. Lumbar Vertebrae Largest and strongest due to compressive load. Cortical bone shell with cancellous bone core (trabeculae). Vertical Column alignment. Aids shock absorption quality of L1-5. Age and repetitious loading degenerate horizontal trabeculae ‘struts’
  • 12. Lumbar facet joints Bony articulations between vertebrae. Synovial Joints- articular surfaces covered in hyaline cartilage. Allow flexion and extension Movement pumps fluid in and out of joint space. Fixed postures lead to joint dehydration and degeneration. Constant compression caused by hypertonicity of paraspinals can accelerate degeneration.
  • 13. Sacroiliac Joints Junction point between spine and pelvis. Synovial Joint- innervated by pain receptors. Corrugated design to assist stability. Allows forward and backward tilting of the sacrum. Sublaxation possible, resulting in dull ache or sharp pain that may refer inferiorly.
  • 14. Intervertebral Discs Colloidal gel nucleus Concentric rings of fibrocartilage (lamellae) form the annulus. Outer third ONLY innervated by pain and mechanoreceptors. Slight movement of the vertebrae helps rehydrate discs. Repetitious torsion forces can derange annulus, allowing nucleus to seep out. Late warning of this process due to lack of pain receptors amongst inner 2/3 of annulus.
  • 15. Intervertebral Discs Cont’d Discs are poor shock absorbers – Very little compressive potential – Nucleus facilitates movement rather than compression
  • 16. Thoracolumbar Fascia Dense multilayered sheet of connective tissue. Insertion point for many muscles Overactive lats and/or glutes can cause excess collagen deposition, making TLF more stiff. This can restrict the ability of TrA to slide freely as it pulls on deep layer.
  • 18. Transversus Abdominis Intra-abdominal pressure, thus making this area more stiff (less bendable). Increases the stiffness of thoraco-lumbar fascia and abdominal aponeurosis. Line of pull helps to align the ribs and pelvis in anatomically correct Fibres crossing the sacroiliac joints pull the Ilium and the sacrum closer together, decreasing laxity in these joints.
  • 19. Gluteus Maximus Primary hip extensor and external rotator* Important for maintaining upright posture Stabiliser of SIJ via attachment to TLF Supports hip and knee via ITB attachment Functional role in stepping, running, climbing etc. and… DECELERATION
  • 20. Gluteus Medius Primary abductor and controller of rotation of the hip* Functionally supports pelvis during SL stance and gait Plays rotator cuff-like role Strongest in neutral or slight adduction
  • 21. Tensor Fascia Latae Primary functions are hip flexion, internal rotation and abduction (via ITB) Works in synergy with glute max: Tighten ITB to extend knee joint Control movements of pelvis on femur and femur on tibia when weight bearing
  • 22. Iliotibial Band Thick, lateral aspect of fascia lata Attachment point for glute max, TFL (and glute med) Indirect insertion onto patella Anatomically impossible to stretch effectively
  • 23. Piriformis & External Hip Rotators Primarily lateral rotator of the hip In hip flexion, will also abduct the hip Secondary phasic stabiliser of the SIJ Close relationship to sciatic nerve Piriformis syndrome
  • 24. Vastus Medialis & Lateralis Primary action is knee extension in inner range- 15-20deg of knee flexion Provide medial and lateral stability to patella respectively Perform anticipatory role Often dysfunctional (knee pain, pronation)
  • 25. Single Legged Squat Functional strength exercise Assessment tool
  • 26. SLSq Research (performance and strength) Wilson et al (2006) Frontal Plane Projection Angle measured (FPPA)  Women > FPPA  Weakness in external rotators correlated most closely to FPPA (predisposes to ACL injury & PFP) Claiborne et al (2006)  Hip abductor strength most important for resisting valgus alignment Crossley, 2006  Glute med shown to be latent in poor SLQ  Abduction strength and Trendelenburg test shows correlation to SLSq
  • 28. Thomas Myers- Anatomy Trains Superficial Front Line
  • 31. Correctives!! Core exercises: Leg loads (ant oblique, ant superficial and Spiral) hip extension (post oblique and posterior superficial) Hip lifts/SL (post oblique and post superficial) Hip exercises: Squat (posterior superficial), SL DL (Lateral), hitches (lateral) and Rots (posterior and anterior oblique), SL SQ (lateral)
  • 32. PRESENTED BY: Max MARTIN BAppSc (Hons)AEP @iNformMaxMartin max@correctiveexerciseaustralia.com

Notes de l'éditeur

  1. Do visual black out after “CNS protection”
  2. ----- Meeting Notes (6/02/11 07:41) -----WHAT DOES IT DO??