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Head Injury

            Zafar Iqbal
           Sr. Lecturer
Jinnah College Of Nursing Karachi
Head Injury

• Any trauma to the scalp, skull, or brain

• Head trauma includes an alteration in
  consciousness no matter how brief
Head Injury

• Causes
  – Motor vehicle accidents
  – Firearm-related injuries
  – Falls
  – Assaults
  – Sports-related injuries
  – Recreational accidents
Road Traffic Crashes
Sports injuries




                  A&E(VMH)
Assaults
(Sickle injuries)




                    A&E(VMH)
MECHANISM

• BLUNT INJURY
     High Velocity
     Low Velocity


• PENETRATING INJURY
     Gunshot
     Sharp instruments
Head Injury

• High potential for poor outcome
• Deaths occur at three points in time after
  injury:
   – Immediately after the injury
   – Within 2 hours after injury
   – 3 weeks after injury
Classification

• By Nature of insult; penetrating or blunt.
• Concomitant injuries; isolated head injury or
  multiple trauma.
• Timing of the injury; Primary or Secondary.
Classification
• Primary injury is that occurring at the scene and is
  usually outside the control of the intensivist.
• Secondary injury is anything that occurs to augment
  the primary injury; the prevention of this is
  predominantly where intensive therapy is aimed.
Types of Head Injuries


• Scalp lacerations
  – The most minor type of
    head trauma
  – Scalp is highly
    vascular → profuse
    bleeding
  – Major complication is
    infection                     Cephal Hematoma
Minor Head Trauma
        Manifestation
– Concussion
   • A sudden transient mechanical head injury
     with disruption of neural activity and a
     change in LOC
   • Brief disruption in LOC
   • Amnesia
   • Headache
   • Short duration
Minor Head Trauma
       Manifestation

– Postconcussion syndrome
   • 2 weeks to 2 months
   • Persistent headache
   • Lethargy
   • Personality and behavior changes
Types of Head Injuries


• Skull fractures
   – Linear or depressed
   – Simple,
     comminuted, or
     compound
   – Closed or open
   – Direct & Indirect
Types of Head Injuries


• Skull fractures
   – Location of fracture alters the
     presentation of the manifestations
   – Facial paralysis
   – Deviation of gaze
   – Battle’s sign
Types of Head Injuries

• Basal Skull fractures
  – CSF leak (extravasation) into ear (Otorrhea) or
    nose (Rhinorrhea)
  – High risk infection or meningitis
  – “HALO Sign (Battle Sign)”
  – Possible injury to Internal carotid artery
  – Permanent CSF leaks possible
Basilar :
         with/with out CSF leak
         with/with out seventh-nerve palsy




                        Raccoon eyes
   Battle sign                           CSF rhinorrhea
INTRACRANIAL LESIONS

• Focal : epidural hematoma
          subdural hematoma
          intracerebral hematoma
INTRACRANIAL LESIONS


Intracerebral -in the brain



Epidural Hematoma -between
the skull and the dura




                              Subdural Hematoma -between the brain and the dura)
Manifestation of Major Head
          Trauma

– Includes cerebral contusions and
  lacerations
– Both injuries represent severe trauma to
  the brain
Manifestation of Major Head
          Trauma
– Contusion (“brain bruises” )
   • bruising’ within the brain with relatively
     localised cellular damage, haemorrhage and
     oedema or The bruising of brain tissue
     within a focal area that maintains the
     integrity of the pia mater and arachnoid
     layers
– Lacerations
   • Involve actual tearing of the brain tissue
   • Intracerebral hemorrhage is generally
     associated with cerebral laceration
Pathophysiology

• Diffuse axonal injury (DAI)
  – Widespread axonal damage occurring
    after a mild, moderate, or severe TBI
  – Process takes approximately 12-24 hours
Pathophysiology

• Diffuse axonal injury (DAI)
  – Clinical signs:
     ∀↓ LOC
     ∀↑ ICP
     • Decerebration or decortication
     • Global cerebral edema
Approach to a Patient With Head
                Injury
•   History

• Initial Assessment
        Primary Survey
        Secondary Survey
Diagnostic Studies and
           Collaborative Care
• CT scan considered the best diagnostic test to
  determine craniocerebral trauma
• MRI
• Cervical spine x-ray
• Glasgow Coma Scale (GCS)
Management of Traumatic Head
             Injury
• Maximize oxygenation and ventilation

• Support circulation / maximize cerebral perfusion
  pressure
• Decrease intracranial pressure

• Decrease cerebral metabolic rate
Nursing Management
     Nursing Assessment


– GCS score
– Neurologic status (GCS)
– Presence of CSF leak
Nursing Management
      Nursing Diagnoses


– Ineffective tissue perfusion
– Hyperthermia
– Acute pain
– Anxiety
– Impaired physical mobility
Nursing Management
             Planning

– Overall goals:
   • Maintain adequate cerebral perfusion
   • Remain normothermic
   • Be free from pain, discomfort, and
     infection
   • Attain maximal cognitive, motor, and
     sensory function
Nursing Management
              PRIMARY SURVEY
Airway maintenance with cervical spine protection
Nursing Management
Intubation with Cervical inline stabilization
• Breathing and ventilation : Intubation precautions
    Pre-medicate with Lidocaine, 1mg/kg IV 2
  minutes prior to attemptICP Spike
• Laryngoscopy produces an
Nursing Management
                Circulation
• Maintain MAP >90mmhg- adequate
• Hematocrit >30%
• Cushing reflex
Conti…..
• Isolated intracranial injuries do not cause
  hypotension
• LOOK FOR THE CAUSE OF HYPOTENSION
Decreasing Intracranial Pressure
Diuretic Therapy
Osmotic Diuretic                 Loop Diuretic
• Mannitol (0.25-1 gm / kg)      • Furosemide
• Increases serum osmolarity     • Decreased CSF formation
• Vasoconstriction               • Decreased systemic and
  (adenosine) / less effect if     cerebral blood volume
  autoregulation is impaired       (impairs sodium and water
  and if CPP is < 70               movement across blood
• Initial increase in blood        brain barrier)
  volume, BP and ICP             • May have best affect in
  followed by decrease             conjunction with mannitol
• Questionable mechanism of
  lowering ICP
Nursing Management of Skull
              Fractures
• Minimize CSF leak
  – Bed flat
  – Never suction orally; never insert NG tube; never use Q-Tips
    in nose/ears; caution patient not to blow nose

• Place sterile gauze/cotton ball around area

• Verify CSK leak:
   – DEXTROSTIX: positive for glucose

• Monitor closely: Respiratory status+++
Nursing Management
     Nursing implementation


Health Promotion
  • Prevent car and motorcycle accidents
  • Wear safety helmets
Nursing Management
     Nursing implementation


Acute Intervention
  • Maintain cerebral perfusion and prevent
    secondary cerebral ischemia
  • Monitor for changes in neurologic status
Nursing Management
        Nursing implementation

Ambulatory and Home Care
   • Nutrition
   • Bowel and bladder management
   • Spasticity
   • Dysphagia
   • Seizure disorders
   • Family participation and education
Nursing Management
           Evaluation

Expected Outcomes
  • Maintain normal cerebral perfusion
    pressure
  • Achieve maximal cognitive, motor, and
    sensory function
  • Experience no infection, hyperthermia, or
    pain
Summary of Recommended Practices
• Decrease intracranial pressure
  – Evacuate mass occupying hemorrhages
  – Consider draining CSF with ventriculostomy when possible
  – Hyperosmolar therapy, +/- diuresis (cautious use to avoid
    hypovolemia and decreased BP)
  – Mid-line neck, elevated head of bead (some research supports
    elevation not > 30 degrees)
  – Treat pain and agitation - consider pre-medication for nursing
    activities, +/- neuromuscular blockade (only when needed)
  – Careful monitoring of ICP during nursing care, cluster nursing
    activities and limit handling when possible
  – Suction only as needed, limit passes, pre-oxygenate / +/- pre-
    hyperventilate (PaCo2 not < 30) / use lidocaine IV or IT when
    possible
                                                        A&E(VMH)
  – After careful preparation of visitors, allow calm contact
Complications

• Epidural hematoma
  – Results from bleeding between the dura
    and the inner surface of the skull
  – A neurologic emergency
  – Venous or arterial origin
Complications

• Subdural hematoma
   – Occurs from bleeding between the dura
     mater and arachnoid layer of the
     meningeal covering of the brain
Complications

• Subdural hematoma
   – Usually venous in origin
   – Much slower to develop into a mass large
     enough to produce symptoms
   – May be caused by an arterial hemorrhage
Complications

• Subdural hematoma
   – Acute subdural hematoma
      • High mortality
      • Signs within 48 hours of the injury
      • Associated with major trauma (Shearing
        Forces)
      • Patient appears drowsy and confused
      • Pupils dilate and become fixed
Complications


• Subdural hematoma
   – Subacute subdural hematoma
      • Occurs within 2-14 days of the injury
      • Failure to regain consciousness may be
        an indicator
Complications


• Subdural hematoma
   – Chronic subdural hematoma
      • Develops over weeks or months after a
        seemingly minor head injury
Surgical Management


•   Craniotomy
•   Craniectomy
•   Cranioplasty
•   Burr-hole

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Head injury

  • 1. Head Injury Zafar Iqbal Sr. Lecturer Jinnah College Of Nursing Karachi
  • 2. Head Injury • Any trauma to the scalp, skull, or brain • Head trauma includes an alteration in consciousness no matter how brief
  • 3. Head Injury • Causes – Motor vehicle accidents – Firearm-related injuries – Falls – Assaults – Sports-related injuries – Recreational accidents
  • 5. Sports injuries A&E(VMH)
  • 7. MECHANISM • BLUNT INJURY  High Velocity  Low Velocity • PENETRATING INJURY  Gunshot  Sharp instruments
  • 8. Head Injury • High potential for poor outcome • Deaths occur at three points in time after injury: – Immediately after the injury – Within 2 hours after injury – 3 weeks after injury
  • 9. Classification • By Nature of insult; penetrating or blunt. • Concomitant injuries; isolated head injury or multiple trauma. • Timing of the injury; Primary or Secondary.
  • 10. Classification • Primary injury is that occurring at the scene and is usually outside the control of the intensivist. • Secondary injury is anything that occurs to augment the primary injury; the prevention of this is predominantly where intensive therapy is aimed.
  • 11. Types of Head Injuries • Scalp lacerations – The most minor type of head trauma – Scalp is highly vascular → profuse bleeding – Major complication is infection Cephal Hematoma
  • 12. Minor Head Trauma Manifestation – Concussion • A sudden transient mechanical head injury with disruption of neural activity and a change in LOC • Brief disruption in LOC • Amnesia • Headache • Short duration
  • 13. Minor Head Trauma Manifestation – Postconcussion syndrome • 2 weeks to 2 months • Persistent headache • Lethargy • Personality and behavior changes
  • 14. Types of Head Injuries • Skull fractures – Linear or depressed – Simple, comminuted, or compound – Closed or open – Direct & Indirect
  • 15. Types of Head Injuries • Skull fractures – Location of fracture alters the presentation of the manifestations – Facial paralysis – Deviation of gaze – Battle’s sign
  • 16. Types of Head Injuries • Basal Skull fractures – CSF leak (extravasation) into ear (Otorrhea) or nose (Rhinorrhea) – High risk infection or meningitis – “HALO Sign (Battle Sign)” – Possible injury to Internal carotid artery – Permanent CSF leaks possible
  • 17. Basilar : with/with out CSF leak with/with out seventh-nerve palsy Raccoon eyes Battle sign CSF rhinorrhea
  • 18. INTRACRANIAL LESIONS • Focal : epidural hematoma subdural hematoma intracerebral hematoma
  • 19. INTRACRANIAL LESIONS Intracerebral -in the brain Epidural Hematoma -between the skull and the dura Subdural Hematoma -between the brain and the dura)
  • 20. Manifestation of Major Head Trauma – Includes cerebral contusions and lacerations – Both injuries represent severe trauma to the brain
  • 21. Manifestation of Major Head Trauma – Contusion (“brain bruises” ) • bruising’ within the brain with relatively localised cellular damage, haemorrhage and oedema or The bruising of brain tissue within a focal area that maintains the integrity of the pia mater and arachnoid layers – Lacerations • Involve actual tearing of the brain tissue • Intracerebral hemorrhage is generally associated with cerebral laceration
  • 22. Pathophysiology • Diffuse axonal injury (DAI) – Widespread axonal damage occurring after a mild, moderate, or severe TBI – Process takes approximately 12-24 hours
  • 23. Pathophysiology • Diffuse axonal injury (DAI) – Clinical signs: ∀↓ LOC ∀↑ ICP • Decerebration or decortication • Global cerebral edema
  • 24. Approach to a Patient With Head Injury • History • Initial Assessment  Primary Survey  Secondary Survey
  • 25. Diagnostic Studies and Collaborative Care • CT scan considered the best diagnostic test to determine craniocerebral trauma • MRI • Cervical spine x-ray • Glasgow Coma Scale (GCS)
  • 26. Management of Traumatic Head Injury • Maximize oxygenation and ventilation • Support circulation / maximize cerebral perfusion pressure • Decrease intracranial pressure • Decrease cerebral metabolic rate
  • 27. Nursing Management Nursing Assessment – GCS score – Neurologic status (GCS) – Presence of CSF leak
  • 28. Nursing Management Nursing Diagnoses – Ineffective tissue perfusion – Hyperthermia – Acute pain – Anxiety – Impaired physical mobility
  • 29. Nursing Management Planning – Overall goals: • Maintain adequate cerebral perfusion • Remain normothermic • Be free from pain, discomfort, and infection • Attain maximal cognitive, motor, and sensory function
  • 30. Nursing Management PRIMARY SURVEY Airway maintenance with cervical spine protection
  • 31. Nursing Management Intubation with Cervical inline stabilization • Breathing and ventilation : Intubation precautions Pre-medicate with Lidocaine, 1mg/kg IV 2 minutes prior to attemptICP Spike • Laryngoscopy produces an
  • 32. Nursing Management Circulation • Maintain MAP >90mmhg- adequate • Hematocrit >30% • Cushing reflex
  • 33. Conti….. • Isolated intracranial injuries do not cause hypotension • LOOK FOR THE CAUSE OF HYPOTENSION
  • 34. Decreasing Intracranial Pressure Diuretic Therapy Osmotic Diuretic Loop Diuretic • Mannitol (0.25-1 gm / kg) • Furosemide • Increases serum osmolarity • Decreased CSF formation • Vasoconstriction • Decreased systemic and (adenosine) / less effect if cerebral blood volume autoregulation is impaired (impairs sodium and water and if CPP is < 70 movement across blood • Initial increase in blood brain barrier) volume, BP and ICP • May have best affect in followed by decrease conjunction with mannitol • Questionable mechanism of lowering ICP
  • 35. Nursing Management of Skull Fractures • Minimize CSF leak – Bed flat – Never suction orally; never insert NG tube; never use Q-Tips in nose/ears; caution patient not to blow nose • Place sterile gauze/cotton ball around area • Verify CSK leak: – DEXTROSTIX: positive for glucose • Monitor closely: Respiratory status+++
  • 36. Nursing Management Nursing implementation Health Promotion • Prevent car and motorcycle accidents • Wear safety helmets
  • 37. Nursing Management Nursing implementation Acute Intervention • Maintain cerebral perfusion and prevent secondary cerebral ischemia • Monitor for changes in neurologic status
  • 38. Nursing Management Nursing implementation Ambulatory and Home Care • Nutrition • Bowel and bladder management • Spasticity • Dysphagia • Seizure disorders • Family participation and education
  • 39. Nursing Management Evaluation Expected Outcomes • Maintain normal cerebral perfusion pressure • Achieve maximal cognitive, motor, and sensory function • Experience no infection, hyperthermia, or pain
  • 40. Summary of Recommended Practices • Decrease intracranial pressure – Evacuate mass occupying hemorrhages – Consider draining CSF with ventriculostomy when possible – Hyperosmolar therapy, +/- diuresis (cautious use to avoid hypovolemia and decreased BP) – Mid-line neck, elevated head of bead (some research supports elevation not > 30 degrees) – Treat pain and agitation - consider pre-medication for nursing activities, +/- neuromuscular blockade (only when needed) – Careful monitoring of ICP during nursing care, cluster nursing activities and limit handling when possible – Suction only as needed, limit passes, pre-oxygenate / +/- pre- hyperventilate (PaCo2 not < 30) / use lidocaine IV or IT when possible A&E(VMH) – After careful preparation of visitors, allow calm contact
  • 41. Complications • Epidural hematoma – Results from bleeding between the dura and the inner surface of the skull – A neurologic emergency – Venous or arterial origin
  • 42. Complications • Subdural hematoma – Occurs from bleeding between the dura mater and arachnoid layer of the meningeal covering of the brain
  • 43. Complications • Subdural hematoma – Usually venous in origin – Much slower to develop into a mass large enough to produce symptoms – May be caused by an arterial hemorrhage
  • 44. Complications • Subdural hematoma – Acute subdural hematoma • High mortality • Signs within 48 hours of the injury • Associated with major trauma (Shearing Forces) • Patient appears drowsy and confused • Pupils dilate and become fixed
  • 45. Complications • Subdural hematoma – Subacute subdural hematoma • Occurs within 2-14 days of the injury • Failure to regain consciousness may be an indicator
  • 46. Complications • Subdural hematoma – Chronic subdural hematoma • Develops over weeks or months after a seemingly minor head injury
  • 47. Surgical Management • Craniotomy • Craniectomy • Cranioplasty • Burr-hole

Notes de l'éditeur

  1. Subgaleal_hemorrhage Subgaleal hemorrhage or hematoma is bleeding in the potential space between the skull periosteum and the scalp galea aponeurosis. Causes Majority (90%) result from vac Cephalhematoma is a hemorrhage of blood between the skull and the periosteum of a newborn baby secondary to ru ...
  2. Battles&apos;s Sign- Periauricular ecchymosis Periauricular - around the external ear Ecchymoisis - bleeding under the skin
  3. Raccoon Eyes - Ecchymosis in the periorbital area, resulting from bleeding from a fracture site in the anterior portion of the skull base
  4. Subdural hemorrhage is bleeding due to trauma that occurs between the outer and middle membranes (meninges) covering the brain. The outer membrane is called the dura, the middle is called the arachnoid, and the inner membrane is known as the pia mater. Subdural hemorrhage, therefore, is bleeding beneath (below) the dura and above the arachnoid. This type of hemorrhage can result from blunt head trauma as minimal as a mild bump.
  5. Cerebral contusions are “brain bruises” which occur from acceleration and de-acceleration of the head. Head trauma can also produce microscopic changes that are scattered throughout the brain. This category of injury is called diffuse axonal injury (DAI) and refers to the microscopic severing of axons (fibers which allow brain neurons to communicate with each other). If enough axons are injured in this way, then the ability of nerve cells to integrate and function may be lost or greatly impaired
  6. Craniotomy Craniectomy Cranioplasty Burr-hole