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Surgery

      Haemorrhoids




      Presenter: Dr. Sachin
Haemorrhoids ?




Engorgement of the haemorrhoidal venous plexuses
        with redundancy of their coverings.
Haemorrhoids

         haimorrhoides          bleeding



haima=blood              rhoos=flowing
Piles

           pila (a ball)


swelling
          in anal canal
     which may or may not bleed
Anal sphincters
        Internal
            Involuntary
            Circular muscle layer
            Upper ¾ of anal canal
            Upto to white line of hilton
        External
            Voluntary
            Striated muscle layer
            Inferior rectal nerve &
             sacral nerve
            Three parts:
                Subcut., superficial, deep
above dentate line
Arterial
           
                superior rectal A.
supply        below dentate line
                inferior rectal A.
Venous drainage
     Internal haemorrhoidal plexus
        in submucosa
        drain in superior rectal vein
        Communicate with external
         plexus
        Site of communication between
         portal and systemic veins
        Veins at 3,7 and 11 o’clock
         position are large
        Potential site for primary
         haemorrhoid
Venous drainage
     External haemorrhoidal plexus
        Lies outside muscular coat of
         anal canal
        Communicate freely with internal
         plexus
Potential sites for
Primary Haemorrhoids
Anal veins
 Arrange  radially around anal margin
 Communicates with internal plexus & IRV

 Straining rupture these vein

 Ruptured vein present
    as subcutaneous perianal hematoma
Venous           SRV
drainage


           MRV




                  IRV
Aetiology
                    Straining
                  Constipation
            Prolonged lavatory sitting
                     Trauma
                     Ageing
                    Diarrhoea
              Lack of fibre rich diet
                   hereditary
Secondary causes
       Local              anorectal deformity,
                          hypotonic sphincter

      Abdominal                    ascites

                              gravid uterus,
      Pelvic                  uterine neoplasm,
                              ovarian neoplasm,
    Pregnancy
    Portal hypertension

                                 paraplegia,
      Neurological             multiple sclerosis
Pathogenesis

 Various  theories are :
1. Portal hypertension and varicose veins
2. Upright posture of human beings
3. Hyperplasia of corpus cavernosum recti
4. Erosion and weakening of wall of veins due
    to infection secondary to trauma
5. Hard faecal matter obstructing venous return
6. Raised anal canal resting pressure
CURRENT VIEW
 Shearing  forces acting on anus
 Caudal displacement of anal cushions and
  mucosal trauma
 Fragmentation of supporting structures

 Loss of elasticity of anal cushions

 Loss of retraction of cushions
Anal Cushions

 Haemorrhoidal venous plexuses together with

some arteriovenous anastomoses surrounded by

    smooth muscle, elastic and fibrous tissue

in the subepithelial space both above & below the

                  pecinate line.
   Shield anal canal and sphincter during evacuation.

   Complete the closure of the anal canal.

   Contribute 15% of the anal canal’s pressure.

   Congest during Valsalva manoeuvre or increased
    intra-abdominal pressure.

   Increase in the size is the starting point of
    haemorrhoids.
11 o’clock

             3 o’clock




7 o’clock
Incidence

    Difficult to evaluate.

    Prevalence ~ 5%.

    Peak of prevalence is between 45 and 65.

    unusual before the age of 20.

    Caucasians > Afro-Caribbeans.
Earliest symptom
                       Bleeding        { A splash in the pan }




                                           Discharge
             Prolaps   H’oids
                       Haemorrhoids
                                           & Pruritus




                          Pain
                                      ( If complication )




                   Symptoms
Physical Examination
 Left   lateral decubitus position

 Any    rashes, condylomata, or eczema

 Any    abscesses, fissures or fistulae
Digital Rectal Examination
 The    resting tone of the anal canal

 voluntary contraction of the puborectalis and
  external anal sphincter.

 mass     / any area of tenderness.

 Int.   hemorrhoids are generally not palpable

 Appear     as bulging mucosa on Anoscopy
Diagnostic Tests

 Physical  examination.
 Proctoscopy.

 Flexible sigmoidoscopy

 Evaluation under anaesthesia in acute pain

 Anal manometry
    if h/o soiling & incontinence
• Classified according to origin of haemorrhoid.
• Above or below the Pecinate line?




             External or Internal
External hemorrhoid Internal hemorrhoid
    Below dentate line        Above dentate line
   Varicosities of veins     Varicosities of veins
         draining                  draining
   inferior rectal artery    superior rectal artery
        Lined by                 Lined by
   squamous epithelium      columnar epithelium
        Painful               Pain insensitive

  Prone to thrombosis if     May prolapse outside
      vein ruptures               anal canal
  (Thrombosed pile)         (prolapsed hemorrhoid)
Gr I               Gr II                  Gr III                  Gr IV
not prolapse   returns spontaneously   manually returned   remains prolapsed

  Grading of hemorrhoids (on history)
Complications of hemorrhoids
                      prolapse
                               Gripped by Ext. sphincter
                Impeded venous return




  Gangrene            Strangulation         Fibrosis



         Ulceration               Thrombosis


                        Suppuration


                                      Portal pyaemia
Thrombosed
External haemorrhoids
Prolapsed
Internal haemorhoids
TREATMENT

 Conservative Dietary and lifestyle modification.
 Non operative/office procedures.

 Operative hemorrhoidectomy

 Minimal invasive procedures
Dietary & Lifestyle
modifications




       Minimize straining at stool.
                   and
       Prevention of constipation
Dietary & Lifestyle
modifications
 Drinking Fluids
 High-fiber diet

 Use of Fiber
  supplements
 Stool softeners

 Exercise

 Local hygiene
Dietary & Lifestyle
modifications




    “you don't defecate in the library
                    so
   you shouldn't read in the bathroom”
Dietary & Lifestyle
modifications

  Ifprolapses,
   gently push back
   into anal canal



  Use  moist towelettes or wet toilet paper
   instead of dry toilet paper.
Topical Treatment
 Include:
     Pads,
     Ointments,
     Creams,
     Gels,
     Lotions
     Suppositories.
 Used   now a days includes

    Calcium dobesilate .25%

    Anhydrous lignocaine 3%

    Hydrocortisone acetate .25%

    Zinc 5%
Sitz bath
 Sitz mean to sit
 Used in treatment of
  Gr. IV hemorrhoids
 Duration:15-20 minutes

 Cold water is used
     Draw heat out of sore piles
     Reduce blood flow in them
     Reduce pressure inside
      swollen piles
Sitz bath
 Postoperative
 Warm water is used
    Dialatation of blood vessels
    Allow blood to pass through
     swollen piles more quickly
    Relaxes muscles so ease anal
     sphincter tone
Oral Medications

 Oral vasotopic drugs.
 Most common - purified flavonoid fraction.
 Actions:
     Increases vascular tone
     Increases lymphatic drainage
     Anti-inflammatory effects.
     Several recent studies have shown it to be
      effective.
Topical medications
         Commonaly used is

            Combination of

 Calcium dobesilate & docusate sodium
 Calcium   dobesilate:
    Decrease   capillary permeablity,

    Decrease   platelet aggregation

    Stops   bleeding

    Reduce   thrombus formation

    Improves   mucosal inflammation
 Docusate     sodium:

    Stimulant laxative,

    makes bowel movement softer and easier
     to pass

    Reduces pain or rectal damage caused by
     hard stools or straining
Office procedures
 Sclerotherapy

 Infra-redCoag
 Band Ligation

 Cryosurgery

 Manual Dilation of anus.

 Sphincterotomy (lateral)

 Bicap electrocoagulation

 haemorrhoidolysis
Sclerotherapy(Mitchell)
 For Gr I to II haemorrhoids.
 phenol, vegetable oil,
  quinine, and urea
  hydrochloride.
 Albright solution:
     5% phenol
       in almond or arachis oil

     with 140 mg of menthol
     to make 30 ml

                                   Injected in submucosa
                                        around pedicle
Sclerotherapy
 Causes
    oedema,
    inflammatory reaction
    & intravascular thrombosis.
 Submucosal      fibrosis &
 scarring
    minimises the extent of
     mucosal prolapse
    and potentially shrinks the
     haemorrhoid as well.
                                   Injected in submucosa
                                        around pedicle
Sclerotherapy
 Quick

 painless

 Follow  up after 6 weeks
 2-3 further injections may
  be required
 Free from major
  complications



                               Injected in submucosa
                                    around pedicle
Sclerotherapy
 Contraindications
    Prolapsed pile
    Infection
 Complications
    retroperitoneal sepsis,
    portal pyemia
    necrotising fascitis
    Prostatitis
    Impotence
    Rectovaginal fistula
                               Injected in submucosa
                                    around pedicle
Barron’Band Ligation
 Large Gr I & Gr II witout
  external component
 2 bands

 Not >2 hemorrhoids at a
  time
 Follow up after 1 month

 Success rate:50-100%



                              occlude base of hemorrhoid
                                   above dentate line
Barron’Band Ligation
 Band causes ischemic
  necrosis ulceration and
  scarring
 Fix connective tissue to
  rectal wall
 necrosis in 24-48 hrs &
  slough off in 7 days
 May cause pain for 24-48
  hrs and secondary
  hemorrhage
                             occlude base of hemorrhoid
                                  above dentate line
Barron’Band Ligation
 Complications:
  anal stenosis
  Inclusion of dentate line
   cause pain
  vasovagal shock

  sepsis




                               occlude base of hemorrhoid
                                    above dentate line
cryosurgery

 Freezing    of hemorrhoidal tissue
    liquid Nitrogen probe at -160oC for 3 minutes


 Applied   for 10-15 minutes
    Over upper part of hemorrhoidal area


 Profusewatery discharge is most common
 complication (in first 3 hrs)
Cryosurgery

 Painless



 Causes     necrosis of hemorrhoidal tissue

 Healing    completes in 4-6 wks

 Little   efficacy in prolapsed hemorrhoids
Infrared photocoagulation




 High  intensity infra red light
 3-6 pulses of 1.5 sec each

 appllied to mucosa
Infrared photocoagulation




 Coagulate   vessels & fix underlying mucosa
 Useful for actively bleeding piles

 Painless and uncomplicated
BICAP Electrocoagulation

 Theoretically   similar to photocoagulation

 Probe   must be left in place for ten minutes.

 Poor  patient tolerance minimizes the effect of
  this procedure.
Lord’s Maximal anal dilatation

 Spasmof int sphincter responsible for many
 symptoms of hemorrhoids

 Reserved    for large Gr II & Gr III hemorrhoids

 NOT    eliminate redundant tissue

 Risk   of incontinence
Haemorrhoidolysis

 Therapeutic   galvanic waves

 Produce   chemical reaction

 Shrink   and dissolve hemorrhoidal tissue

 Most   effective on internal hemorrhoids
Indications of surgery
 Mainly  driven by impact of symptoms on
  quality of life
 3rd and 4th degree piles

 2nd degree not cured by conservative means

 Fibrosed hemorrhoid

 Interno-external hemorrhoid

 Bleeding sufficient to cause anemia

 Soiling

 Ulceration,thrombosis,gangrene
Milligan-Morgan (open)
Haemorrhoidectomy

           First described
         over 2 centuries ago.
Milligan-Morgan (open)
Haemorrhoidectomy
Milligan-Morgan (open)
Haemorrhoidectomy
Final Operative Aspect in a Haemorrhoidectomy.

               Wound left open
Ferguson’s (Closed)
Haemorrhoidectomy
 Developed   in 1952




 Haemorrhoidal  tissue excised.
 Mucosal wound and skin sutured completely
  with a continuous absorbable suture.
Harmonic Scalpel
 sutureless   technique

 shorter   operative time

 less   post-op pain.

 hospital   stay not required.

 Comparative    Increased
  cost to other techniques.
MIPH




Longo introduced the technique in 1995.
MIPH




Stappler haemorrhoidopexy
Advantages
   Lesser pain
   Quick return to normal activity
   Lesser mean hospital stay


Risks
   Higher chances of recurrence and prolapse
   May be unsuccessful in large hemorrhoids
   Pelvis sepsis and sphincter dysfunction
Laser surgery of hemorrhoid

 Pile mass excised or vaporised using laser
  beam
 Allow precision and accuracy

 Rapid and unimpaired healing

 Lesser bleeding and pain as laser seal off
  tiny blood vessels and nerves
 Can be combined with other modalities
HAL-RAR Hemorrhoidectomy

   HAL - Doppler guided haemorrhoidal artery ligation
   RAR - Recto anal repair proctoplasty (mucopexy)
   Combine two methods
   Artery ligated 3-4 cm proximal to dentate line
   Reducing blood flow to inner hemorrhoidal plexus
   Mucopexy combined for grade 3-4 hemorrhoid
A.M.I. (DG) HAL/RAR®
System
Complications of surgery

 Early   complications
    Post operative pain lasting 2-3 weeks
    Wound infection rarely
    Post op bleeding
    Swelling of skin bridges
    Short term incontinence
    Difficult urination
Complications of surgery

 Late   complications
    Anal stenosis
    Anal fissure
    Fecal impaction
    Mild incontinence
    Submucous abscess
    Delayed bleeding
    Skin tags
    Recurrence
Prevention
 Eat high fiber diet
 Drink Plenty of Liquids

 Fiber Supplements

 Exercise

 Avoid long periods of standing or sitting

 Don’t Strain

 Go as soon as you feel the urge
Thank you for your patience

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Hemorrhoids-

  • 1. Surgery Haemorrhoids Presenter: Dr. Sachin
  • 2. Haemorrhoids ? Engorgement of the haemorrhoidal venous plexuses with redundancy of their coverings.
  • 3. Haemorrhoids haimorrhoides bleeding haima=blood rhoos=flowing
  • 4. Piles pila (a ball) swelling in anal canal which may or may not bleed
  • 5.
  • 6. Anal sphincters  Internal  Involuntary  Circular muscle layer  Upper ¾ of anal canal  Upto to white line of hilton  External  Voluntary  Striated muscle layer  Inferior rectal nerve & sacral nerve  Three parts:  Subcut., superficial, deep
  • 7. above dentate line Arterial   superior rectal A. supply  below dentate line  inferior rectal A.
  • 8. Venous drainage Internal haemorrhoidal plexus  in submucosa  drain in superior rectal vein  Communicate with external plexus  Site of communication between portal and systemic veins  Veins at 3,7 and 11 o’clock position are large  Potential site for primary haemorrhoid
  • 9. Venous drainage External haemorrhoidal plexus  Lies outside muscular coat of anal canal  Communicate freely with internal plexus
  • 11. Anal veins  Arrange radially around anal margin  Communicates with internal plexus & IRV  Straining rupture these vein  Ruptured vein present  as subcutaneous perianal hematoma
  • 12. Venous SRV drainage MRV IRV
  • 13. Aetiology Straining Constipation Prolonged lavatory sitting Trauma Ageing Diarrhoea Lack of fibre rich diet hereditary
  • 14. Secondary causes Local anorectal deformity, hypotonic sphincter Abdominal ascites gravid uterus, Pelvic uterine neoplasm, ovarian neoplasm, Pregnancy Portal hypertension paraplegia, Neurological multiple sclerosis
  • 15. Pathogenesis  Various theories are : 1. Portal hypertension and varicose veins 2. Upright posture of human beings 3. Hyperplasia of corpus cavernosum recti 4. Erosion and weakening of wall of veins due to infection secondary to trauma 5. Hard faecal matter obstructing venous return 6. Raised anal canal resting pressure
  • 16. CURRENT VIEW  Shearing forces acting on anus  Caudal displacement of anal cushions and mucosal trauma  Fragmentation of supporting structures  Loss of elasticity of anal cushions  Loss of retraction of cushions
  • 17. Anal Cushions Haemorrhoidal venous plexuses together with some arteriovenous anastomoses surrounded by smooth muscle, elastic and fibrous tissue in the subepithelial space both above & below the pecinate line.
  • 18. Shield anal canal and sphincter during evacuation.  Complete the closure of the anal canal.  Contribute 15% of the anal canal’s pressure.  Congest during Valsalva manoeuvre or increased intra-abdominal pressure.  Increase in the size is the starting point of haemorrhoids.
  • 19. 11 o’clock 3 o’clock 7 o’clock
  • 20. Incidence  Difficult to evaluate.  Prevalence ~ 5%.  Peak of prevalence is between 45 and 65.  unusual before the age of 20.  Caucasians > Afro-Caribbeans.
  • 21. Earliest symptom Bleeding { A splash in the pan } Discharge Prolaps H’oids Haemorrhoids & Pruritus Pain ( If complication ) Symptoms
  • 22. Physical Examination  Left lateral decubitus position  Any rashes, condylomata, or eczema  Any abscesses, fissures or fistulae
  • 23. Digital Rectal Examination  The resting tone of the anal canal  voluntary contraction of the puborectalis and external anal sphincter.  mass / any area of tenderness.  Int. hemorrhoids are generally not palpable  Appear as bulging mucosa on Anoscopy
  • 24. Diagnostic Tests  Physical examination.  Proctoscopy.  Flexible sigmoidoscopy  Evaluation under anaesthesia in acute pain  Anal manometry  if h/o soiling & incontinence
  • 25. • Classified according to origin of haemorrhoid. • Above or below the Pecinate line? External or Internal
  • 26. External hemorrhoid Internal hemorrhoid Below dentate line Above dentate line Varicosities of veins Varicosities of veins draining draining inferior rectal artery superior rectal artery Lined by Lined by squamous epithelium columnar epithelium Painful Pain insensitive Prone to thrombosis if May prolapse outside vein ruptures anal canal (Thrombosed pile) (prolapsed hemorrhoid)
  • 27. Gr I Gr II Gr III Gr IV not prolapse returns spontaneously manually returned remains prolapsed Grading of hemorrhoids (on history)
  • 28. Complications of hemorrhoids prolapse Gripped by Ext. sphincter Impeded venous return Gangrene Strangulation Fibrosis Ulceration Thrombosis Suppuration Portal pyaemia
  • 31. TREATMENT  Conservative Dietary and lifestyle modification.  Non operative/office procedures.  Operative hemorrhoidectomy  Minimal invasive procedures
  • 32. Dietary & Lifestyle modifications Minimize straining at stool. and Prevention of constipation
  • 33. Dietary & Lifestyle modifications  Drinking Fluids  High-fiber diet  Use of Fiber supplements  Stool softeners  Exercise  Local hygiene
  • 34. Dietary & Lifestyle modifications “you don't defecate in the library so you shouldn't read in the bathroom”
  • 35. Dietary & Lifestyle modifications  Ifprolapses, gently push back into anal canal  Use moist towelettes or wet toilet paper instead of dry toilet paper.
  • 36. Topical Treatment  Include:  Pads,  Ointments,  Creams,  Gels,  Lotions  Suppositories.
  • 37.  Used now a days includes  Calcium dobesilate .25%  Anhydrous lignocaine 3%  Hydrocortisone acetate .25%  Zinc 5%
  • 38. Sitz bath  Sitz mean to sit  Used in treatment of Gr. IV hemorrhoids  Duration:15-20 minutes  Cold water is used  Draw heat out of sore piles  Reduce blood flow in them  Reduce pressure inside swollen piles
  • 39. Sitz bath  Postoperative  Warm water is used  Dialatation of blood vessels  Allow blood to pass through swollen piles more quickly  Relaxes muscles so ease anal sphincter tone
  • 40. Oral Medications  Oral vasotopic drugs.  Most common - purified flavonoid fraction.  Actions:  Increases vascular tone  Increases lymphatic drainage  Anti-inflammatory effects.  Several recent studies have shown it to be effective.
  • 41. Topical medications Commonaly used is Combination of Calcium dobesilate & docusate sodium
  • 42.  Calcium dobesilate:  Decrease capillary permeablity,  Decrease platelet aggregation  Stops bleeding  Reduce thrombus formation  Improves mucosal inflammation
  • 43.  Docusate sodium:  Stimulant laxative,  makes bowel movement softer and easier to pass  Reduces pain or rectal damage caused by hard stools or straining
  • 44. Office procedures  Sclerotherapy  Infra-redCoag  Band Ligation  Cryosurgery  Manual Dilation of anus.  Sphincterotomy (lateral)  Bicap electrocoagulation  haemorrhoidolysis
  • 45. Sclerotherapy(Mitchell)  For Gr I to II haemorrhoids.  phenol, vegetable oil, quinine, and urea hydrochloride.  Albright solution:  5% phenol  in almond or arachis oil  with 140 mg of menthol  to make 30 ml Injected in submucosa around pedicle
  • 46. Sclerotherapy  Causes  oedema,  inflammatory reaction  & intravascular thrombosis.  Submucosal fibrosis & scarring  minimises the extent of mucosal prolapse  and potentially shrinks the haemorrhoid as well. Injected in submucosa around pedicle
  • 47. Sclerotherapy  Quick  painless  Follow up after 6 weeks  2-3 further injections may be required  Free from major complications Injected in submucosa around pedicle
  • 48. Sclerotherapy  Contraindications  Prolapsed pile  Infection  Complications  retroperitoneal sepsis,  portal pyemia  necrotising fascitis  Prostatitis  Impotence  Rectovaginal fistula Injected in submucosa around pedicle
  • 49. Barron’Band Ligation  Large Gr I & Gr II witout external component  2 bands  Not >2 hemorrhoids at a time  Follow up after 1 month  Success rate:50-100% occlude base of hemorrhoid above dentate line
  • 50. Barron’Band Ligation  Band causes ischemic necrosis ulceration and scarring  Fix connective tissue to rectal wall  necrosis in 24-48 hrs & slough off in 7 days  May cause pain for 24-48 hrs and secondary hemorrhage occlude base of hemorrhoid above dentate line
  • 51. Barron’Band Ligation  Complications:  anal stenosis  Inclusion of dentate line cause pain  vasovagal shock  sepsis occlude base of hemorrhoid above dentate line
  • 52. cryosurgery  Freezing of hemorrhoidal tissue  liquid Nitrogen probe at -160oC for 3 minutes  Applied for 10-15 minutes  Over upper part of hemorrhoidal area  Profusewatery discharge is most common complication (in first 3 hrs)
  • 53. Cryosurgery  Painless  Causes necrosis of hemorrhoidal tissue  Healing completes in 4-6 wks  Little efficacy in prolapsed hemorrhoids
  • 54. Infrared photocoagulation  High intensity infra red light  3-6 pulses of 1.5 sec each  appllied to mucosa
  • 55. Infrared photocoagulation  Coagulate vessels & fix underlying mucosa  Useful for actively bleeding piles  Painless and uncomplicated
  • 56. BICAP Electrocoagulation  Theoretically similar to photocoagulation  Probe must be left in place for ten minutes.  Poor patient tolerance minimizes the effect of this procedure.
  • 57. Lord’s Maximal anal dilatation  Spasmof int sphincter responsible for many symptoms of hemorrhoids  Reserved for large Gr II & Gr III hemorrhoids  NOT eliminate redundant tissue  Risk of incontinence
  • 58. Haemorrhoidolysis  Therapeutic galvanic waves  Produce chemical reaction  Shrink and dissolve hemorrhoidal tissue  Most effective on internal hemorrhoids
  • 59. Indications of surgery  Mainly driven by impact of symptoms on quality of life  3rd and 4th degree piles  2nd degree not cured by conservative means  Fibrosed hemorrhoid  Interno-external hemorrhoid  Bleeding sufficient to cause anemia  Soiling  Ulceration,thrombosis,gangrene
  • 60. Milligan-Morgan (open) Haemorrhoidectomy First described over 2 centuries ago.
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  • 68. Final Operative Aspect in a Haemorrhoidectomy. Wound left open
  • 69. Ferguson’s (Closed) Haemorrhoidectomy  Developed in 1952  Haemorrhoidal tissue excised.  Mucosal wound and skin sutured completely with a continuous absorbable suture.
  • 70.
  • 71.
  • 72. Harmonic Scalpel  sutureless technique  shorter operative time  less post-op pain.  hospital stay not required.  Comparative Increased cost to other techniques.
  • 73. MIPH Longo introduced the technique in 1995.
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  • 93. Advantages  Lesser pain  Quick return to normal activity  Lesser mean hospital stay Risks  Higher chances of recurrence and prolapse  May be unsuccessful in large hemorrhoids  Pelvis sepsis and sphincter dysfunction
  • 94. Laser surgery of hemorrhoid  Pile mass excised or vaporised using laser beam  Allow precision and accuracy  Rapid and unimpaired healing  Lesser bleeding and pain as laser seal off tiny blood vessels and nerves  Can be combined with other modalities
  • 95. HAL-RAR Hemorrhoidectomy  HAL - Doppler guided haemorrhoidal artery ligation  RAR - Recto anal repair proctoplasty (mucopexy)  Combine two methods  Artery ligated 3-4 cm proximal to dentate line  Reducing blood flow to inner hemorrhoidal plexus  Mucopexy combined for grade 3-4 hemorrhoid
  • 97. Complications of surgery  Early complications  Post operative pain lasting 2-3 weeks  Wound infection rarely  Post op bleeding  Swelling of skin bridges  Short term incontinence  Difficult urination
  • 98. Complications of surgery  Late complications  Anal stenosis  Anal fissure  Fecal impaction  Mild incontinence  Submucous abscess  Delayed bleeding  Skin tags  Recurrence
  • 99. Prevention  Eat high fiber diet  Drink Plenty of Liquids  Fiber Supplements  Exercise  Avoid long periods of standing or sitting  Don’t Strain  Go as soon as you feel the urge
  • 100. Thank you for your patience

Notes de l'éditeur

  1. Difficult to evaluate due to the fact many do not seek medical advice because of the embarrassment or because of the fear of the pain of treatemnt.. Prevalence of approximately 5%. Peak of prevalence is between 45 and 65. Development before 20 unusual. Caucasians more affected than Afro-Caribbeans.
  2. Normally easily made on a physical exam. Preferably accompanied by proctoscopy. Rarely a flexible sigmoidocopy is used but this is less sensitive than a proctoscope at detecting haemorrhoids and is used mainly as part of further investigations exclude inflammatory bowel disease/malignancy. Acute pain may require an evaluation under anaesthesia in the operating room. However in the absence of thrombosis acute anal pain is rare in uncomplicated haemorrhoidal disease and may indicate the presence of another disease such as abscess, o fissure. Patients with soiling and incontinence should be investigated with anal manometry to decide on the type of treatment they receive as they are at increased risk of developing incontinence post-surgery. .
  3. Topical Treatment Many over the counter medications available. Include: Pads, ointments, creams, gels, lotions and suppositories. Cocktail of local anaesthetics, corticosteroids, antiseptics, astringents and other ingredients. Treatment of symptoms rather than a cure for the haemorrhoids.
  4. These treatments were first described in the treatment of varicose veins, venous ulcers, and edema. Purified flavonoid fraction is a botanical extract from citrus. It exerts its effects on both diseased and intact vasculature, increasing vascular tone, lymphatic drainage, and capillary resistance; it is also assumed to have antiinflammatory effects and promote wound healing. Lately, several randomized controlled studies evaluated the use of oral micronized, purified flavonoid fraction in the treatment of hemorrhoidal bleeding.In all of the studies, bleeding was relieved rapidly, and no complications were reported.
  5. These treatments were first described in the treatment of varicose veins, venous ulcers, and edema. Purified flavonoid fraction is a botanical extract from citrus. It exerts its effects on both diseased and intact vasculature, increasing vascular tone, lymphatic drainage, and capillary resistance; it is also assumed to have antiinflammatory effects and promote wound healing. Lately, several randomized controlled studies evaluated the use of oral micronized, purified flavonoid fraction in the treatment of hemorrhoidal bleeding.In all of the studies, bleeding was relieved rapidly, and no complications were reported.
  6. Again requires a proctoscope 1 to hold, one to grasp the haemorrhoid. Ligation with a rubber band causes ischaemic necrosis, ulceration and scarring. Fixes connective tissue to the rectal wall. If treating mixed haemorrhoids, analgesia is essential! As the anoderm will be involved which is heavily innervated.
  7. Again requires a proctoscope 1 to hold, one to grasp the haemorrhoid. Ligation with a rubber band causes ischaemic necrosis, ulceration and scarring. Fixes connective tissue to the rectal wall. If treating mixed haemorrhoids, analgesia is essential! As the anoderm will be involved which is heavily innervated.
  8. Again requires a proctoscope 1 to hold, one to grasp the haemorrhoid. Ligation with a rubber band causes ischaemic necrosis, ulceration and scarring. Fixes connective tissue to the rectal wall. If treating mixed haemorrhoids, analgesia is essential! As the anoderm will be involved which is heavily innervated.
  9. During the procedure, a circumferential purse-string suture is placed approximately 2 cm proximal to the dentate line. The stapler is introduced transanally, and the suture is tied around the shaft. On closing and firing the stapler, a circumferential band of excessive rectal mucosa and submucosa proximal to the hemorrhoidal tissue is excised, and the defect in the mucosa is simultaneously closed by the stapler while fixing the mucosa to the underlying rectal wall. This procedure also interrupts the blood supply of the superior hemorrhoidal artery proximal to the hemorrhoidal tissue. So it treats the mucosal prolapse, with concurrent disruption of the blood supply to the hemorrhoidal tissue.
  10. During the procedure, a circumferential purse-string suture is placed approximately 2 cm proximal to the dentate line. The stapler is introduced transanally, and the suture is tied around the shaft. On closing and firing the stapler, a circumferential band of excessive rectal mucosa and submucosa proximal to the hemorrhoidal tissue is excised, and the defect in the mucosa is simultaneously closed by the stapler while fixing the mucosa to the underlying rectal wall. This procedure also interrupts the blood supply of the superior hemorrhoidal artery proximal to the hemorrhoidal tissue. So it treats the mucosal prolapse, with concurrent disruption of the blood supply to the hemorrhoidal tissue.