Why study etiology?
Elimination of the
etiology is the first
step in treatment
Persistence of the
etiology is one of the
major causes of post-
treatment relapse
What causes malocclusion?
Interaction of genetic and environmental
factors.
Although it is difficult to know the
precise cause of most malocclusions, we do
know in general what the possibilities are,
and these must be considered when
orthodontic problems are being evaluated.
Etiology of malocclusion
Can be classified into:
General factors
Local factors
Or
Skeletal factors
Soft tissue factors
Local or dental factors
Habits
Etiology of malocclusion
General factors:
1. Hereditary.
2. Congenital.
3. Environmental.
4. Predisposing metabolic, climate or disease.
5. Dietary problems.
6. Habits.
7. Posture
8. Trauma and accident.
Etiology of malocclusion/General
factors
1.Heriditary
The child is a product of parents who have dissimilar genetic
make up.
Thus the child may inherit conflicting trait from both parents
resulting in dentofacial deformity.
Some features are thought to be inherited such as:
Toot size.
Arch dimension.
Overjet.
Abnormality in teeth number.
Etiology of malocclusion
Local factors:
1. Abnormalities of tooth
number.
2. Abnormalities of tooth
size.
3. Abnormalities of tooth
shape.
4. Abnormal frenal
attachment.
5. Premature loss of
deciduous tooth.
6. Prolonged retention of
deciduous tooth.
7. Delayed eruption of
permanent teeth.
8. Abnormal eruptive path.
9. Ankyloses.
10. Dental caries.
11. Improper restoration.
Etiology of malocclusion/
Local factors
a. Abnormality in tooth number
1. Hypodontia (missing Teeth):
Prevalence:
0.1-0.9% in primary dentition.
4-6% in permanent dentition (excluding third molars).
25-35% of all third molars are missing.
The permanent teeth most likely to be congenitally missing are the mandibular
second premolars and the maxillary lateral incisors.
Female > Male = 3:2.
Maxillary central and lateral incisors are the teeth most likely to be lost to trauma.
Etiology of malocclusion/Local
factors: abnormalities in number
Types of hypodontia:
a. Hypodontia: congenital absence of 1 - 6 teeth.
b. Oleigodontia: congenital absence of more than 6 teeth.
c. Anodontia: congenital absence of all teeth.
Syndrome associated with hypodontia:
Ectodermal dysplasia.
Down syndrome.
Cleft lip and palate.
Etiology of malocclusion/local
factors: abnormalities in number
2. Supernumerary Teeth:
Presence of extra tooth or teeth.
Also called hyperdontia.
Prevalence:
0.8% in primary dentition
2% in permanent dentition.
Males>Females = 2:1
90% of all supernumerary teeth are found in the anterior
part of the maxilla.
Etiology of malocclusion
Types of supernumerary:
1. According to the position:
A. Mesiodense:
Most common type of
supernumerary teeth.
Conical in shape and Found
in the midline.
Etiology of malocclusion
1. According to the position:
B. Paramolar:
Buccal and palatal to premolar and molar.
Etiology of
malocclusion
2. According to the shape
B. Tuberculate:
These are barrels in shape.
They usually have no roots
thus most of them do not
erupt.
The most common cause of
maxillary incisors impaction is
tuberculate supernumerary.
Etiology of malocclusion
1. according to the shape
D. Odontomas:
They are the most common odontogenic tumors and they usually interfere
with eruption of permanent teeth.
They are usually asymptomatic and are discovered during routine radiographic
examination when there is delayed eruption of permanent tooth.
Usually are two types:
Compound odontoma.
Complex odontoma.
Etiology of malocclusion
D. Odontomas:
a. Compound odontoma:
It is a collection of small
radiopaque masses, some or
all may be tooth-like
structures “denticles”.
62% in the anterior region
of the maxilla and usually
associated with the crown of
an unerupted canine.
Etiology of malocclusion
D. Odontomas:
b. Complex odontoma:
It is composed of
haphazardly arranged dental
hard and soft tissue.
It has no resemblance to a
normal tooth.
It tends to occur in 70% in
the posterior region of the
mandible.
Etiology of malocclusion
Effects of supernumeraries:
Midline diastema.
Crowding.
Labial or palatal deflection of teeth.
Impaction of permanent teeth.
No effect.
Etiology of malocclusion/ local
factors
b. Abnormalities of tooth size
a. Macrodontia:
Teeth larger than normal
Types:
True generalized macrodontia: all teeth are
larger than normal e.g. in pituitary gigantism.
Relative generalized macrodontia: normal or
slightly normal teeth present in jaws that are
smaller than normal.
Localized macrodontia: usually one tooth is
involved, mostly affects maxillary central
incisors.
Etiology of malocclusion/
local factors
b. Abnormalities of tooth size
a. Microdontia:
Teeth smaller than normal
Types:
True generalized microdontia: all teeth are
smaller than normal e.g. in pituitary dwarfism.
Relative generalized microdontia: normal or
slightly normal teeth present in jaws that are
larger than normal.
Localized microdontia: usually one tooth is
involved, mostly affects maxillary lateral
incisors (peg lateral) and third molars.
Etiology of malocclusion/ local
factors
c. Abnormalities of tooth shape
Double teeth
Gemination
Fusion
Etiology of malocclusion/ local
factors
c. Abnormalities of tooth shape
Concrescence
Fusion by cementum
Etiology of malocclusion/ local
factors
c. Abnormalities of tooth
shape
Accessory cusps
Example:
Talon cusp: palatal to lateral
incisors
Etiology of malocclusion/ local
factors
c. Abnormalities of tooth shape
Dense in dente (tooth in tooth)
Etiology of malocclusion/ local
factors
c. Abnormalities of tooth
shape
Dilaceration:
Bending in the root
Can be idiopathic or due to
trauma
Etiology of malocclusion/local
factors
d. Premature loss of deciduous tooth:
Can be due to caries, trauma or less commonly periodontal disease
Effects of early loss of primary teeth:
1. Primary Incisors:
Minimal effect after eruption of deciduous canines.
No space maintainer is usually needed except for aesthetic or speech.
2. Primary Canines:
Can cause midline shift.
Space maintainer or balanced extraction of the contralateral canine is
indicated.
Etiology of malocclusion/ local
factors
3. Primary first molar:
Can cause midline shift specially if it was lost in young age.
Space maintainer is indicated.
4. Primary second molar:
Can space loss by mesial drift of the first molar can occur.
Space maintainer is indicated.
Etiology of malocclusion/ local
factors
e. Prolonged retention of primary tooth
Reasons:
Absence of permanent teeth
Endocrine disturbance such as hypothyroidism
Ankylosed primary tooth that failed to resorption
Etiology of malocclusion/ Local
factors
f. Delayed eruption of permanent teeth:
Reasons
Missing permanent teeth.
Lack of space.
Retained deciduous or premature loss of deciduous tooth.
Presence of physical barrier ( heavy mucosa, supernumerary, tumor, cyst).
Primary failure of eruption
Endocrine disturbance (hypothyroidism)
Etiology of malocclusion/ Local
factors
g. Ankyloses
A condition where part or the whole root surface is directly
fused to the bone with absence of intervening periodontal
ligament.
Causes
Trauma
Infections
Endocrine disturbance
Declaration
The author wish to declare that; these presentations are his original work, all
materials and pictures collection, typing and slide design has been done by the
author.
Most of these materials has been done for undergraduate students, although
postgraduate students may find some useful basic and advanced information.
The universities title at the front page indicate where the lecture was first
presented. The author was working as a lecturer of orthodontics at Ibn Sina
University, Sudan International University, and as a Master student in Orthodontics at
University of Khartoum.
The author declare that all materials and photos in these presentations has been
collected from different textbooks, papers and online websites. These pictures are
presented here for education and demonstration purposes only. The author are not
attempting to plagiarize or reproduced unauthorized material, and the intellectual
properties of these photos belong to their original authors.
Declaration
As the authors reviews several textbooks, papers and other references during
preparation of these materials, it was impossible to cite every textbook and journal
article, the main textbooks that has been reviewed during preparation of these
presentations were:
Contemporary Orthodontics 5th edition; Proffit, William R, Henry W. Fields, and
David M. Sarver.
Handbook of Orthodontics. 1st edition; Cobourne, Martyn T, and Andrew T. DiBiase.
Essentials of orthodontics: Diagnosis and Treatment; Robert N. Staley, Neil T. Reske
Orthodontics: Current Principles & Techniques 5th edition; Graber, Lee W, Robert L.
Vanarsdall, and Katherine W. L. Vig
Orthodontics: The Art and Science. 3rd Edition. Bhalajhi, S.I.
Declaration
For the purposes of dissemination and sharing of knowledge, these
lectures were given to several colleagues and students. It were also
uploaded to SlideShare website by the author. Colleagues and students
may download, use, and modify these materials as they see fit for non-
profit purposes. The author retain the copyright of the original work.
The author wish to thank his family, teachers, colleagues and students
for their love and support throughout his career. I also wish to express
my sincere gratitude to all orthodontic pillars for their tremendous
contribution to our specialty.
Finally, the author welcome any advices and enquires through his
email address: Mohanad-07@hotmail.com