9. OTALGIA (EARACHE)
Pain in the ear called otalgia .
LOCAL CAUSES
External ear.
• Furuncle, impacted wax, otitis externa, otomycosis, myringitis bullosa,
herpes zoster and malignant neoplasms.
Middle ear.
• Acute otitis media, eustachian tube obstruction, mastoiditis, extradural
abscess, aero-otitis media and carcinoma middle ear.
CAUSES
10. As ear receives nerve supply
from
• Vth (auriculotemporal
branch)
• IXth (tympanic branch)
• Xth (auricular branch)
• cranial nerves - from C2
(lesser occipital) and C2
and C3 (greater auricular),
REFERRED CAUSES
Vth
Vth
Vth
Xth
IXth
IXth
11. PSYCHOGENIC CAUSES
Via Vth cranial nerve
(a) Dental. Caries tooth, apical abscess, impacted molar, malocclusion.1
(b) Oral cavity. Benign or malignant ulcerative lesions of oral cavity or tongue.
(c) Temporomandibular joint disorders - osteoarthritis, recurrent dislocation and ill-
fitting denture
Via IXth cranial nerve
(a) Oropharynx. Acute tonsillitis, peritonsillar abscess, tonsillectomy. Benign or
malignant ulcers of soft palate, tonsil and its pillars.
(b) Base of tongue. Tuberculosis or malignancy.
(c) Elongated styloid process
Via Xth cranial nerve.
Malignancy or ulcerative lesion of vallecula, epiglottis, larynx or laryngopharynx and
oesophagus.
Via C2 and C3 spinal nerves.
Cervical spondylosis, injuries of cervical spine and caries spine.
13. श्रमात क्षयाद रुतक्षकषायभोजनात समीरर्ः शब्दपथे प्रधतधष्ठतः
धवररक्तशीषणस्य च शीतसेधवनः करोधत धह क्ष्वेडमतीव कर्णयोः
कर्णक्ष्वेड
धनदान
दोष - वात
- सधनपत (मा.धन.)
लक्षर् –
वायुः धपतादीधभ युक्तों वेर्ु नोषोंपमं स्वनम
करोधत कर्णयोः क्ष्वेड कर्णक्ष्वेड स उच्यते (मा.धन.)
क्ष्वेड का वर्णन वागभट ने नहीं धकया
धचधकत्सा – कर्णनाद समान
14. धचधकत्सा
सामान्य धचधकत्सा – कर्ण रोग
सामान्य धचधकत्सा – 4 रोग
कर्ण पूरर् – कर्ण शूल समान
अरंड धशग्रू वरुतर् मूल स्वरस
योग रत्नाकर – अपामागण तेल पूरन
वातज शूल समान
15. Tinnitus
Tinnitus is ringing sound or noise in the ear.
The characteristic feature is that the origin of this sound is within the patient.
Usually, it is unilateral but may also affect both ears.
It may vary in pitch and loudness and has been variously described by the patient
as roaring, hissing, swishing, rustling or clicking type of noise.
Tinnitus is more annoying in quiet surroundings, particularly at night,
when the masking effect of ambient noise from the environment is lost
TYPES OF TINNITUS
Two types of tinnitus are described:
Subjective - which can only be heard by the patient.
Objective - which can even be heard by the examiner with the use of a
stethoscope
17. Objective tinnitus
seen less frequently
Vascular lesions
glomus tumour
carotid artery aneurysm cause swishing tinnitus synchronous with
pulse.
Temporarily abolished by pressure on the common carotid artery
Patulous eustachian tube
Tinnitus synchronous with respiration
Idiopathic stapedial or tensor tympani
myoclonus
Dental
Clicking of TM joint
tinnitus is psychogenic and no cause can be found in the ear or central
nervous system.
18. TREATMENT OF TINNITUS
Tinnitus is a symptom and not a disease.
Where possible, its cause should be discovered and treated.
Sometimes, even clicking clock or a similar device may mask the tinnitus
and help the patient to go to sleep
TINNITUS MASKERS
• can be used in patients who have no hearing
loss. They are worn like a hearing aid.
TINNITUS INSTRUMENT
• It is a combination of a hearing aid and a masker in
one device.
19. TINNITUS RETRAINING THERAPY
(TRT)
• basis for habituation therapy.
• It occurs at two levels.
Habituation of reaction.
• It is uncoupling of brain and body from negative reactions to tinnitus
Habituation of tinnitus.
• It is blocking the tinnitus-related neuronal activity to reach level of
consciousness
Therapy consists of two major components: (i) counselling (ii) sound
therapy.
TRT needs a long period of 18–24 months but gives a significant
improvement in more than 80% of patients
22. वागभट ने कर्णनाद की उपेक्षा से कर्णबाधधयण की उत्पाती मानी है
बालक वृद्ध तथा एक वषण पुराना बाधधयण - असाध्य
धचधकत्सा
सामान्य धचधकत्सा – कर्ण रोग
कर्णशूले प्रर्ादे च बाधधयण क्ष्वेड योरधप । चतुर्ाणमधप रोगार्ां सामान्यं भेषजं धवदुः
वातज शूल समान धचधकत्सा – कफ का अनुबंध होने पर वमन कर क
े धूमपान
प्रधतस्याय की समस्त धचधकत्सा
वागभट अनुसार धचधकत्सा क्रम
स्नेहन – स्वेदन – नस्य – धसरोबस्ती – बस्ती कमण
कर्ण पूरर्
धबल्वादी तेल
धहंगवादी तेल
धनगुणण्डी तेल
23. Hearing Loss
Peripheral(VIIIth nerve) Central(Central auditorypathways
CLASSIFICATION
Hearing Loss
Organic Nonorganic
Conductive Sensorineural
Sensory(cochlear) Neural
24. CONDUCTIVE HEARING LOSS
Any disease process which interferes with the conduction of sound to reach
cochlea causes conductive hearing loss.
The lesion may lie in the external ear and tympanic membrane, middle ear or
ossicles up to stapediovestibular joint
25. AETIOLOGY
The cause may be congenital or
acquired
• Meatal atresia
• Fixation of stapes footplate
• Fixation of malleus head
• Ossicular discontinuity
• Congenital cholesteatoma
congenital
External ear
• Any obstruction in the ear canal, e.g. wax,
foreignbody, furuncle, acute inflammatory
swelling, benign or malignant tumour.
acquired
Middle ear
• Perforation of tympanic membrane
• Fluid in the middle ear, e.g. otitis media,
• Mass in middle ear, e.g. benign or malignant tumour
• Disruption of ossicles, e.g. trauma to ossicular chain,
cholesteatoma
• Fixation of ossicles, e.g. otosclerosis, tympanosclerosis,
• Eustachian tube blockage, e.g. retracted tympanic
membrane, serous otitis media
26. MANAGEMENT
characteristics of conductive hearing loss
Negative Rinne test, i.e. BC > AC.
Weber lateralized to poorer ear.
Normal absolute bone conduction.
Low frequencies affected more.
Audiometry shows bone conduction better than
air conduction with air-bone gap. Greater the air-
bone gap, more is the conductive loss .
Loss is not more than 60 dB.
Speech discrimination is good
Frequency in Hertz
125 250 500 1k 2k 4k 8k
Hearing
loss
in
dB
0
10
20
30
40
50
60
70
80
90
Most cases of conductive hearing loss can be managed by medical or
surgically
27. Removal of canal obstructions
• impacted wax, foreign body, osteoma or
exostosis, keratotic mass, benign or malignant
tumours.
Removal of fluid.
• Myringotomy with or without grommet
insertion.
Removal of mass from middle ear.
• Tympanotomy and removal of small middle
ear tumours or cholesteatoma behind intact
tympanic membrane.
Stapedectomy
• otosclerotic fixation of stapes footplate.
Tympanoplasty
• Repair of perforation, ossicular chain or both.
Hearing aid.
• In cases, where surgery is not possible,
28. SENSORINEURAL HEARING LOSS
Sensorineural hearing loss (SNHL) results from lesions of the cochlea,
VIIIth nerve or central auditory pathways.
It may be present at birth (congenital) or start later in life (acquired).
29. AETIOLOGY
CONGENITAL
• It is present at birth and is the result of anomalies of the inner
ear or damage to the hearing apparatus
ACQUIRED
• It appears later in life
Infections of labyrinth—viral, bacterial or spirochaetal
Trauma to labyrinth or VIIIth nerve
surgery
Noise-induced hearing loss
Ototoxic drugs
Presbycusis
Ménière’s disease
Acoustic neuroma
Sudden hearing loss
Familial progressive SNHL
Systemic disorders, e.g. diabetes, hypothyroidism, kidney disease, autoimmune
disorders, multiple sclerosis
30. characteristics of sensorineural hearing loss
A positive Rinne test, i.e. AC > BC.
Weber lateralized to better ear.
Bone conduction reduced on Schwabach and absolute
bone conduction tests.
More often involving high frequencies.
No gap between air and bone conduction curve on
audiometry Loss may exceed 60 dB.
Speech discrimination is poor.
There is difficulty in hearing in the presence of noise
31. MANAGEMENT
Early detection of SNHL is important as measures can be taken to stop its
progress
early rehabilitation programme,
Syphilis of the inner ear is treatable with high doses of penicillin and
steroids
Ototoxic drugs should be used with care and discontinued
Noiseinduced hearing loss can be prevented by removed from the noisy
surroundings
Rehabilitation of hearing impaired with
hearing aids
Air conduction hearing aid.
33. NOISE TRAUMA
• excessive noise can be divided into two
groups:
- Acoustic
trauma
- Noise-induced hearing loss
(NIHL)
Acoustic trauma
• Permanent damage to hearing can be caused by a
single brief exposure to very intense sound without
this being preceded by a temporary threshold shift
Noise-induced hearing loss (NIHL)
• Hearing loss, in this case, follows
chronic exposure to less intense
sounds
34. SUDDEN HEARING LOSS
Sudden SNHL is defined as 30 dB or more of SNHL over at least three
contiguous frequencies occurring within a period of 3 days or less.
Mostly it is unilateral
• Remember the mnemonic
“In The Very Ear Too No Major Pathology”
Infections
Trauma
Vascular
Ear (otologic)
Toxic
Miscellaneous
Psychogenic
PRESBYCUSIS
• Sensorineural hearing loss associated with physiological aging process in
the ear is called presbycusis
35. NONORGANIC HEARING LOSS
In this type of hearing loss, there is no organic lesion
It is either due to malingering or is psychogenic
Patient may present with any of the three clinical situations:
(i) Total hearing loss in both ears
(ii) total loss in only one ear
(iii)exaggerated loss in one or both ears
Stenger test
take two tuning forks of equal frequency, strike and keep them say 25 cm from each ear.
Patient will claim to hear it in the normal ear.
Now bring the tuning fork on the side of feigned deafness to within 8 cm, keeping the tuning
fork on the normal side at the same distance
The patient will deny hearing anything even though tuning fork on normal side is where it
could be heard earlier
Acoustic reflex
threshold
37. कर्णस्राव
धनदान
दोष -
लक्षर् –
धशरो अधभनातद अथवा धनमज्जतों जले प्रपाकाद अथवा अधप धवदधेः
स्त्रवेतू पूयं श्रवर्ो अधनलवृतः स कर्ण सस्त्राव इधत प्रकीतततेः
वागभट ने वर्णन नहीं धकया है
38. धचधकत्सा
धशरोधवरेचन
धूपन
पूरर्
प्रमाजणन
धावन
प्रमाजणन
राजवृक्षादी गर्
सुरसादी गर्
पंचक्षीरीगर्
पूरर्
शेवालादी तेल
धप्रयन्गवादी तेल
स्त्री दू ध + रसंजन + मधु
समुद्रफ
े न चूर्ण
शंबूक तेल – कर्णनाड़ी नाशक
गंध तेल
नाड़ीव्रर्वत / दुष्ट व्रर्वत
धूपन – गुगगलू अगरू
कर्णस्त्राव,पूधतकर्ण,क
ृ धमकर्ण – समान धचधकत्सा
39. ACUTE SUPPURATIVE OTITIS MEDIA
acute inflammation of middle ear by pyogenic organisms.
middle ear implies middle ear cleft, i.e. Eustachian tube, middle ear, attic, aditus,
antrum and mastoid air cells
AETIOLOGY
more common - infants and children
lower socioeconomic group
Typically disease follows viral infection of upper respiratory tract but soon
the pyogenic organisms invade the middle ear
ROUTES OF INFECTION
Via eustachian tube - It is the most common
route
Via external ear
Blood-borne- uncommon
40. PREDISPOSING FACTORS
• Recurrent attacks of common cold, upper respiratory tract infections and
exanthematus fevers like measles,
• diphtheria or whooping cough.
• Infections of tonsils and adenoids.
• Chronic rhinitis and sinusitis.
• Nasal allergy.
• Tumours of nasopharynx, packing of nose or nasopharynx
• for epistaxis.
• Cleft palate
BACTERIOLOGY
Streptococcus pneumoniae
Haemophilus influenzae
Moraxella catarrhalis
41. PATHOLOGY AND CLINICAL FEATURES
5 stages of ASOM :
Stage of tubal occlusion
• Oedema and hyperaemia of nasopharyngeal end of
eustachian tube blocks
• the tube leading to absorption of air and negative
intratympanic pressure.
• There is retraction of tympanic membrane with some
degree of effusion in the middle ear but fluid may not
be clinically appreciable
Symptoms.
Deafness and earache are the two symptoms but they are not
marked.
no fever
Signs
Tympanic membrane is retracted
Tuning fork tests show conductive
42. Stage of pre-suppuration.
• tubal occlusion – prolonged - pyogenic organisms
invade tympanic cavity causing hyperaemia of its
lining.
• Inflammatory exudate appears in the middle ear.
• Tympanic membrane becomes
congested
Symptoms.
• marked earache which may disturb sleep and is of
throbbing nature.
• Deafness and tinnitus are also present, but
complained only by adults.
• Usually, child runs high degree of fever and is
restless.
Signs
• congestion of pars tensa.
• Leash of blood vessels appear along the handle of
malleus and at the periphery of tympanic membrane cart-
wheel appearance.
43. Stage of suppuration.
• marked by formation of pus in the middle ear and to some extent in
mastoid air cells.
• Tympanic membrane starts bulging to the point of rupture
Symptoms
Earache becomes excruciating
Deafness increases
child may run fever of 102–103°F.
Signs
Tympanic membrane appears red and bulging with loss of
landmarks.
Handle of malleus may be engulfed by the swollen and protruding
tympanic membrane
44. Stage of resolution.
The tympanic membrane ruptures with release of pus and subsidence of
symptoms.
Inflammatory process begins to resolve.
If proper treatment is started early or if the infection was mild, resolution
may start even without rupture of tympanic membrane.
Symptoms.
With evacuation of pus
earache is relieved,
fever comes down and child feels better.
Signs
External auditory canal may contain blood-tinged discharge which later
becomes mucopurulent.
small perforation is seen in anteroinferior quadrant of pars tensa.
45. Stage of complication.
• If virulence of organism is high or resistance of patient poor
acute mastoiditis
Subperiosteal abscess,
facial paralysis,
labyrinthitis,
petrositis,
Extradural abscess,
meningitis,
brain abscess
lateral sinus thrombophlebitis
46. TREATMENT
Antibacterial therapy
• indicated in all cases with fever and severe
earache
Amoxicillin Novamox, Biomox 40 mg/kg 3
Ampicillin Biocillin 50–100 mg/kg 4
Co-amoxiclav Augmentin, Enhancin 40 mg/kg 2–3
Erythromycin Emycin, Althrocin 30–50 mg/kg 4
Cefaclor (II generation) Keflor, Distaclor 20 mg/kg 2–3
Cefixime (III generation) Taxim-0, Biotax-0 8 mg/kg 1 or 2
Ceftibuten (III generation) Procadax 9 mg/kg 1
Drug Trade names Total daily dose Divided dose
• Antibacterial therapy must be continued for a minimum of 10 days, till tympanic
membrane regains normal appearance and hearing returns to normal
• Early discontinuance of therapy with relief of earache and fever, or therapy given in inadequate
doses may lead to secretory otitis media and residual hearing loss.
47. Decongestant nasal
drops
Analgesics and antipyretics.
- xylometazoline (Otrivin)
- Paracetamol
Ear toilet. - dry-mopped with sterile cotton buds
Myringotomy.
All cases of acute suppurative otitis media should be carefully
followed till drum membrane returns to its normal appearance and
conductive deafness disappears
48. Review after 48–72 h
Acute otitis media
Antibacterial therapy
Earache and fever
persist or increase
Good response
Another antibacterial
therapy for 10 days or
myringotomy and
culture and specific
antimicrobial for
10 days
Continue same
for 10 days
Persistent fluid but
earache and fever abate
Complete
resolution
Periodic checks
for 12 weeks
Complete resolution Persistent
effusion
Treat as otitis media
with effusion
49. पूधतकर्ण
धनदान
दोष - कफ धपत
लक्षर् –
च्छस्थतेः कफ
े स्रोतधस धपत तेजसां धवलायमाने भ्रंश सम्प्रतापवान
अवेदनों वाअप्य अथवा सवेदनों ननं स्त्रवेत पूधत च पूधत कर्णकः
कर्ण धवद्रधध पाक
े न कर्े वा वाररपूरर्ात
पूयं स्त्रवधत यः पूधत स पूधतकर्णक ःः (भा. प्र )
धचधकत्सा
सामान्य
कर्णस्त्राव समान
कर्ण पूरर् – स्त्री दुग्ध + रसंजान
50. CHRONIC SUPPURATIVE OTITIS
MEDIA
Chronic suppurative otitis media (CSOM) is a long-standing infection of a
part or whole of the middle ear cleft characterized by ear discharge and a
permanent perforation.
A perforation becomes permanent when its edges are covered by
squamous epithelium
TYPES OF CSOM
two types
Tubotympanic
- safe or benign type
- involves anteroinferior part of middle ear cleft,
- There is no risk of serious complications
Atticoantral.
- unsafe or dangerous type
- involves posterosuperior part of the cleft (i.e. attic, antrum and mastoid)
- Risk of complications is high in this variety
51. TUBOTYMPANIC TYPE
AETIOLOGY
- sequela of acute otitis media
- Ascending infections via the eustachian tube - tonsils, adenoids and
infected sinuses
- result of allergy to ingestants such as milk, eggs, fish,
BACTERIOLOGY
• Pseudomonas aeruginosa,
• Proteus,
• Escherichia coli
• Staphylococcus aureus,
52. CLINICAL FEATURES
• Ear discharge.
- mucoid or mucopurulent,
- constant or intermittent
• Hearing loss.
- conductive type; severity varies but rarely exceeds 50
dB
- the patient reports of a paradoxical effect, i.e. hears better in the presence
of discharge than when the ear is dry. This is due to “round window shielding
effect” produced by discharge which helps to maintain phase differential
• Perforation
• Middle ear
mucosa
- seen when the perforation is large
53. INVESTIGATIONS
- Examination under microscope
- Audiogram.
- Culture and sensitivity of ear
discharge.
- Mastoid X-rays/CT scan temporal bone
54. TREATMENT
- Aural toilet
Remove all discharge and debris from the ear.
It can be done by dry mopping with absorbent cotton buds, suction
clearance under microscope or irrigation
- Ear drops
• Antibiotic ear drops containing neomycin, chloromycetin or
gentamicin are used.
• combined with steroids + local anti-inflammatory effect.
- Systemic antibiotics
- Precautions - keep water out of the ear during bathing, swimming and hair
wash
- Avoid Hard nose blowing
- Surgical treatment.
55. ATTICOANTRAL TYPE
AETIOLOGY
- Aetiology of atticoantral disease is same as of cholesteatoma
CLINICAL FEATURES
Ear
discharge
- Usually scanty,
- but always foul-smelling due to bone destruction
- Discharge may be so scanty that the patient may not even be
aware of it
Hearing loss
- Hearing is normal when ossicular chain is intact
- when cholesteatoma caused by destroyed ossicles
Bleeding
56. SIGNS
Perforatio
n - attic or posterosuperior marginal type
Retraction
pocket
Cholesteatoma
• Pearly-white flakes of cholesteatoma
INVESTIGATIONS
- Examination under microscope
- Audiogram.
- Culture and sensitivity of ear discharge.
- Mastoid X-rays/CT scan temporal bone
57. FEATURES INDICATING COMPLICATIONS IN CSOM
• Pain
• Vertigo
• Persistent headache
• Fever, nausea and vomiting
• Irritability and neck rigidity
• Diplopia
Surgical
TREATMENT
• Canal wall down procedures
• Canal wall up procedures.
Reconstructive surgery
Conservative treatment
58. Tubotympanic or safe type Atticoantral or unsafe type
• Discharge Profuse, mucoid,
odourless
Scanty, purulent, foul
smelling
• Perforation Central Attic or marginal
• Polyp Pale Red and fleshy
• Cholesteatoma Absent Present
• Complications Rare Common
59. कर्णधवद्रधध
धनदान
प्रकार - 2 – आगंतुज+ दोषज
लक्षर् –
क्षत अधभनात प्रभवस्तु धवद्रधध भवेत तथा दोष क
ृ तों अपरः पुनः
स रक्त धपत अरुतर्ं अस्त्रं स्त्रवेत प्रतोद धूमयान दाहचोषवान
धचधकत्सा
सामान्य धवद्रधधवत
आम धवद्रधध धचधकत्सा – व्रर्शोथ उपक्रम – धवरेचन से अपतपणर् तक
पक्व धवद्रधध धचधकत्सा – भेदन पीड़न – व्रर्वत
सधन्नपात धवद्रधध – असाध्य
क्षतज धवद्रधध – धपतज कर्णशूलवत
60. localized acute otitis externa (Furuncle)
A furuncle is a staphylococcal infection of the hair follicle.
As the hair are confined only to the cartilaginous part of the meatus,
furuncle is seen only in this part of meatus.
Usually single but may be multiple.
Symptoms
• Severe ear pain and tenderness
• Movements of the pinna are painful. Jaw
movements, as in chewing, also cause pain in the
ear.
Sign
• posterior meatal wall causes oedema over the
mastoid with obliteration of the retroauricular
groove.
• Periauricular lymph node enlarged
61. Treatment
• systemic antibiotics,
• analgesics
• local heat.
• An ear pack of 10% glycerine provides splintage and reduces
pain.
• If abscess has formed, incision and drainage should be done.
• recurrent furunculosis, diabetes should be excluded,
62. कर्णपाक
धनदान
दोष - धपत
लक्षर् – भवेत प्रपाकः खलु धपत कोपतो धवकोथ क्लेदकरश्च कर्णयो
कर्ण पाकस्तू धपतेन कोथ धवक्लेदक
ृ त भवेत
कर्ण धवद्रधध पाकाद वा जायते च अंबु पुरर्ात (मा.धन.)
- वागभट ने वर्णन नहीं धकया है
धचधकत्सा
धपतज धवसपण समान
गोयाणधद नृत
शीतल लेप
उत्पल चंदन मंधजष्टा लेप
63. It is diffuse inflammation of meatal skin which may spread to pinna and
epidermal layer of tympanic membrane
Trauma - scratching the ear canal with hair pins or matchsticks,
unskilled instrumentation to remove foreign bodies
Common organisms
• Staphylococcus aureus, Pseudomonas pyocyaneus, Bacillus
proteus and Escherichia coli but more often the infection is
mixed.
Diffuse otitis externa
Aetiolog
y • commonly seen in hot and humid climate and in swimmers.
• Two factors commonly responsible for this condition are:
(i) trauma to the meatal skin and
(ii) invasion by pathogenic organisms.
64. Acute phase
• hot burning sensation in ear
• Pain aggravated by movements of jaw.
• Oozing thin serous discharge
• Meatal lining becomes inflamed and swollen.
• Conductive hearing loss.
• In severe cases, regional lymph nodes become
enlarged and tender
Chronic phase
• irritation and strong desire to itch.
• Discharge is scanty and may dry up to form crusts.
• Meatal skin which is thick and swollen show scaling
and fissuring.
Clinical features.
Diffuse otitis externa may be acute or chronic with varying degrees of severity.
65. Ear toilet
• most important single factor in the treatment
of diffuse otitis externa.
• All exudate and debris should gently
removed.
• Ear toilet can be done by dry mopping,
suction clearance or irrigating the canal with
warm,
• sterile normal saline.
Medicated wicks.
• wick soaked in antibiotic steroid and inserted
in ear canal and patient
• Wick is changed daily for 2–3 days
Antibiotics.
• Broad-spectrum systemic antibiotics.
Analgesics- For relief of pain.
Treatment
66. कर्णगूथ
धनदान
दोष - k p
लक्षर् –
धवशोधषत श्लेष्मर्ी धपततेजसां नृर्ा भवेत स्त्रोतधस कर्णगुथकः
धचधकत्सा
• प्रक्लेधन – तेल – अपामागण क्षार तेल
• धवलायन - स्वेदन से
• शोधन – शलाका से – आहरर् कमण
67. Impacted wax
Wax is composed of secretion of sebaceous glands, ceruminous glands, hair,
epithelial debris and dirt
Sebaceous glands provide fluid rich in fatty acids while secretion of
ceruminous gland is rich in lipids.
Secretion of both these glands mixes with the epithelial cells and keratin to
form wax.
Wax has a protective function
It has acidic pH and is bacteriostatic and
Normally, only a small amount of wax is secreted, which dries up and is
later expelled from the meatus by movements of the jaw.
68. Etiology
• people sweat more
• narrow and tortuous ear canal
• stiff hair
• Obstructive lesion of the canal
Symotoms
impairment of hearing
sense of blocked ear.
Tinnitus and giddiness may result from impaction of wax against the
tympanic membrane.
Reflex cough due to stimulation of auricular branch of vagus
onset of symptoms may be sudden when water enters the ear canal
during bathing
69. Treatment
syringing
instrumental manipulation.
Hard impacted - wax solvents.
Cerumen hook, scoop or Jobson- Horne probe are often used
First, a space is created between the wax and meatal
wall, the instrument is passed beyond the wax, and whole
plug then dragged out in a single piece
If it breaks, syringing may be used to remove the fragments.
5% sodium bicarbonate + glycerine + water – 2/3
times a day
Hydrogen peroxide, liquid paraffin
Commercial drops - ceruminolytic agents - paradichlorobenzene
70. कर्णक
ं डु
धनदान
दोष - k
लक्षर् –
कफ
े न क
ं डु प्रधचतेन कर्णयो भ्रशं भवेत स्त्रोतधस कर्ण संधनत
धचधकत्सा
ना – नाड़ी स्वेद
व – वमन
न – नस्य
धु – धूमपान
मु – मूधण धवरेचन
71. Otomycosis
Otomycosis is a fungal infection of the ear canal
Common organism
Aspergillus niger,
A. Fumigatus
Candida albicans.
Mostly seen in hot and humid climate
Secondary fungal growth is also seen in patients using topical antibiotics for
treatment of otitis externa or middle ear suppuration
72. Clinical features
itching,
discomfort or pain in the ear,
watery discharge with a musty odour
ear blockage.
Sign
• fungal mass may appear white, brown or black
o A niger - black headed
o A fumigatus - pale blue or green
o Candida - white or creamy
Treatment
ear toilet - By syringing, suction or mopping
antifungal agents Nystatin - Candida.
broad-spectrum antifungal - clotrimazole
74. शागणधर ने क
ृ धम कर्ण को कर्ण हच्छल्लका नाम से धलखा है
पतंगः शतपदश्च कर्णश्रोत प्रधवश्य धह
अररत व्याक
ु लत्वं च भ्रशं क
ु वणच्छन्त वेदनां
कर्ो धनस्तूधते तस्य तथा फरफरायते
कीटचरधतरुतक तीव्रा धनष्पन्दे मंद वेदना
धचधकत्सा
क
ृ धमनाशक धचधकत्सा
• अपकषणर्
• प्रक
ृ धत धवधात
• धनदान पररवजणन
• कर्णपूरर् – सषणप तेल , गोमूत्र+हरताल, सुयणमुखीस्वरस, धनगुंडी स्वरस
75. Foreign bodies of ear
(a)Nonliving.
Children may insert a variety of foreign bodies in the
ear;
• a piece of paper or sponge,
• grain seeds (rice, wheat, maize),
• slate pencil,
• piece of chalk or metallic ball bearings.
An adult
• broken end of matchstick
(b) Living.
• Flying or crawling insects like mosquitoes, beetles, cockroach or ant
76. Methods of removing a foreign body
include:
(i) Forceps removal
(ii) Syringing
(iii) Suction
(iv) Microscopic removal with special
instruments
(v) Postaural approach
Soft and irregular foreign bodies removed by fine crocodile forceps.
seed grains and smooth objects can be removed with syringing
Living objects –
• No attempt should be made to catch them alive.
• First, the insect should be killed by instilling oil (a household remedy),
spirit or chloroform water.
• Once killed, the insect can be removed by any of the method dsescribed
78. Catarrhal inflammation of the Eustachian tube is one of the most common, unpleasant and long but
not very grave illnesses of the fall, winter and spring months.
Catarrhal inflammation of the Eustachian tube
At change of altitude, for instance, during an airplane flight, especially at take-off and landing, we
often feel unpleasant pressure and blocking in our ears.
Symptoms
• Rhinitis – nasal discharge
• Ear ache
• feeling of the blocking of the ear
• Hearing loss
• Tinnitus
Sign
• Tympanic membrane – retracted
• Conductive deafness sign
Treatment
• According to cause
• Antibiotics
• Analgesic
• Chewing
81. Perichondritis
Etiology
• infection secondary to lacerations, haematoma or surgical incisions.
• diffuse otitis externa
• furuncle
• Pseudomonas and mixed flora are the common pathogens.
Inflamation of peri chondrium of pinna called
perichondritis
symptoms
• red, hot and painful pinna which feels
stiff.
• Later abscess may form between the cartilage and
perichondrium
Treatment
• early stages - systemic antibiotics
• When abscess has formed – incision and
drained
82. कर्ण अशण
धनदान
प्रकार – 4
लक्षर् –
-धवणरुतद्धाध्यशनस्त्रीप्रसङ्गोत्कटुकासनपृष्ठयानवेगधवधारर्ाधदधभधवणशेषैः
तत्र कर्णजेषु बाधधयं शूलं पुधतकर्णता च
धचधकत्सा
- चतुधवणधोऽशणसां साधनोपायः
- भेषजं क्षारोऽधनः शस्त्र
भेषजसाध्य – अधचरकाल जात अन्यल्पदोष धलङ्गोपद्रवाधर्
क्षार – मृदु प्रसृत अवगाढ अन्य उच्छच्छ
र ताधन
अधन – कक
ण श च्छस्थर पृथु कधिन
शस्त्रेर् – तनुमूला अन्य उच्छच्छ
र ताधन क्लेदवच्छन्त च
83. A polyp is a smooth mass of oedematous and inflamed mucosa which has
protruded through a perforation and presents in the external canal
Ear Polyp
It is usually pale in contrast to pink, fleshy polyp seen in atticoantral
disease
Etiology
• sequela of acute otitis media
•Cholesteatoma
•Foreign object
•Inflammation
•Tumor
Symptoms
• Patients usually present with otorrhea,
• conductive hearing loss,
• otalgia,
• bleeding and a sensation of a mass