This document contains an ECG report from the Department of Cardiology at JIPMER in Pondicherry, India. It describes the ECG findings of a patient who presented with narrow complex tachycardia and includes the results of an electrophysiological study and ablation. The EPS found the patient had atrial tachycardia originating from the left atrium posteriorly, and they underwent successful ablation. The document provides details of the case from initial ECG and symptoms to diagnosis and treatment.
8. Narrow complex, regular tachycardia
Rate around 200 beats per minute
P wave were not seen
No significant ST-T wave changes seen
There is no evidence of pre-excitation in the baseline ecg
Diagnosis is Short RP tachycardia
DD AVNRT.
Final diangosis- Patient underwent EPS/ablation at JIPMER, Cardiology department.
EPS was suggestive of Atrial tachycardia with origin from left atrium, posterior in
origin. Patient underwent successful ablation.
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11. Tachycardia ECG-
Regular narrow complex
tachycardia rate 200/minutes,
LBBB morphology
LAD
QRS duration nearly 120 msec
No AV dissociation,
No P wave,
No capture beat
No fusion beat
Sinus ECG
Sinus rhythm at around 75 per
minute,
LAD,
No ST-T wave changes,
No evidence of pre-excitaion
11
12. Patient underwent Electrophysiological study at Department of
Cardiology, JIPMER, Pondicherry, India, by Dr Raja Selvaraj and his
team.
It was suggestive of Antidromic reentrant tachycardia
with mahaim accessory pathway. Patient underwent
successful radiofrequency ablation
Final diagnosis is Mahim fiber tachycardia
12
13. The resting electrocardiogram (ECG) is usually normal
No delta wave with Mahaim fiber conduction
ECG features that suggest Mahaim fibers as the cause of a tachycardia
with a left bundle branch block pattern These include:
QRS axis between 0 and minus 75º
QRS duration of 0.15 seconds or less
R-wave in lead 1
rS complex in lead V1
Precordial transition in lead V4 or later
Cycle length between 220 and 450 milliseconds (heart rates of 130 to 270)
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15. Narrow complex tachycardia
irregularly irregular
No visible P wave seen
Heart rate around 140 beats per minute
ST segment depression with T wave inversion in lead II,III,avF, V4-V6
ECG is suggestive of atrial fibrillation with fast
ventricular effect with digoxin effect
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17. ECG-Sinus rhythm at 100/minute
Inferior axis nearly 90 degree in view of equiphasic QRS in lead I
Diffuse ST segment depression with T wave inversion in lead
I,II,III,avF,avL, V2-V6
ST segment elevation in lead avR, V1
PR interval 120 msec
QT interval 360 msec
QTc interval 464 msec
The ECG suggestive of ACS/USA/ Most likely artery involved is
LMCA
17
20. Description of the ECG
Broad complex regular tachycardia at around 200 beats per minute
Right axis deviation
QRS duration around 280 msec
RBBB morphology
No P wave
No capture wave
No fusion beat
Diagnosis-Broad complex tachycardia, Differential diagnosis- Ventricular tachycardia,
Or It could be Supraventricular tachycardia with aberrancy
Patient underwent EPS study and found to have Bundle branch reentrant tachycarida
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24. Description of the ECG-
Broad complex regular tachycardia at heart rate around 190 beats per minute
QRS of RBBB morphology
QRS duration 240 msec
Normal axis
No capture beats
No fusion beats
No AV dissociation seen
No visible P wave seen
Negative concordance seen from lead V1-V6,
rS seen in lead V6
Final diagnosis- Ventricular tachycardia
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28. Broad complex regular tachycardia with rate around 200 beats per minute,
AV dissociation was present with intermittent visible P wave in lead I,II, V1.
No capture beats
No fusion beat seen
Negative QRS concordance seen in lead V1-V5
QRS complex were of LBBB morphology
QRS axis being inferior (QRS complex are positive in lead II,III,avF)
Diagnosis of this ECG is Ventricular tachycardia with inferior axis
Final diagnosis if RVOT VT
Cardiac MRI(14.02.2017)-Severe biventricular dysfunction, RVEF-25%, LVEF-22%,
RV free wall and sub tricuspid dyskinesia suggestive of Arrythmogenic right ventricular
cardiomyopathy
28
29. Identify the device on the right
side of the chest x-ray
Its use?
Its present status?
29
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Chest X-ray of a patient who is a CRT non-responder and received CCM in
2009 (Optimizer III at that time). He has significant improvement of heart
failure symptoms from NYHA class IV to I. There was left ventricular
reverse remodeling and increase in ejection fraction. He is free of heart
failure rehospitalization in the past 4 years
Liu M, Fang F, Luo XX, Shlomo BH, Burkhoff D, Chan JY, Chan CP,
Cheung L, Rousso B, Gutterman D, Yu CM. Improvement of long-term
survival by cardiac contractility modulation in heart failure patients: A
case–control study. International journal of cardiology. 2016 Mar 1;206:122-
6.