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RK ECG.pptx

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RK ECG.pptx

  1. 1. Electrocardiography Rigil Kent A. Franco, RN, MD 1st Year Resident Emergency Medicine Fatima University Medical Center
  2. 2. Electrocardiography ▲ Graphic recording of electric potentials produced by the heart. ▲ Signals detected by metal electrodes attached to extremities and chest wall and recorded by the ECG machine. ▲ Noninvasive, inexpensive and readily available.
  3. 3. Unipolar Precordial Leads V1 - 4th ICS right sternal margin V2 - 4th ICS left sternal margin V3 - midway between V2 and V4 V4 - 5th ICS MCL V5 - AAL same level as V4 V6 - MAL same level as V4
  4. 4. Leads LI LII LIII AVR AVL AVF V1 V2 V3 V4 V5 V6 View of Heart Lateral wall Inferior wall Inferior wall No specific view Lateral wall Interior wall Anteroseptal wall Anteroseptal wall Anterior wall Anterior wall Anterolateral wall Anterolateral wall
  5. 5. P wave ● represents atrial depolarization ● atrial conduction time ● normal amplitude is 0.5 to 2.5 mm ● normal duration is up to 0.10s in adults ● usually biphasic in V1
  6. 6. P-R Interval ● represents time interval for impulse to reach ventricles from SA node ● measured in limb lead with longest PR interval ● normal is 0.12-.20s in adults (HR = 70-90/min)
  7. 7. QRS Complex Q wave - initial downward deflection R wave - first upward deflection whether preceded by a Q wave or not S wave - downward deflection following the R wave QS wave - single negative deflection representing entire QRS R’ wave - second upward deflection S’ wave – downward deflection following the R’ wave
  8. 8. ST Segment ● represents period from end of ventricular depolarization to start of ventricular repolarization ● between end of QRS and start of T wave ● clinically important if elevated or depressed as it may represent infarction or ischemia ● usually isoelectric but may be depressed –0.5 mm or elevated by 1mm.
  9. 9. S-T depression
  10. 10. T wave ● represents ventricular repolarization ● usually upright in LI, LII and diphasic or inverted in LIII, V1 ● maybe inverted up to V3 in young adults ● T wave in V6 usually > V1 ● physiologic T wave changes maybe seen in body position, fever, skeletal abnormalities, hyperventilation , fever, etc.
  11. 11. Q-T Interval ● represents electrical systole ● time required for ventricular depolarization and repolarization ● varies with age sex and heart rate ● normal QT is 0.35-0.44s in adults ● Corrected QT or QTc Q-Tc = Q-Ts ● Prolonged QTc > 0.425s R-R interval (s)
  12. 12. U wave ● small deflection after the T wave ● represents repolarization of the Purkinje fibers ● tallest in V2 & V3 ● usually does not exceed >1mm in amplitude ● same as T wave polarity ● increased amplitude in LVH, hypokalemia, drugs etc. ● negative U wave specific for heart disease
  13. 13. STEPS IN BASIC ECG READING 1. Determine Rate 2. Determine Rhythm 3. Measure Intervals 4. Determine QRS electrical axis 5. Check for chamber enlargements 6. Inspect QRS complexes for bundle branch block or fascicular block 7. Check for ST and T wave changes 8. Check for miscellaneous ECG findings
  14. 14. • In determining the heart rate in regular rhythm, • count the number of small squares between two successive QRS complexes. • The numerator (1500) is a constant, and when divided by 20 (number of small squares between successive QRS complexes), yields a heart rate of 75 beats per minute. DETERMINING THE RATE
  15. 15. • In determining the heart rate in an irregular rhythm, • count the number of QRS complexes within the 6 second strip. • This value should be multiplied by 10 in order to yield the heart rate in 1 minute. DETERMINING THE RATE
  16. 16. DETERMINING THE RHYTHM
  17. 17. DETERMINING THE RHYTHM
  18. 18. DETERMINING THE RHYTHM
  19. 19. DETERMINING THE RHYTHM
  20. 20. Electrical Axis
  21. 21. Electrical Axis
  22. 22. CHAMBER ENLARGEMENTS
  23. 23. Left Atrial Hypertrophy ● P wave duration > 0.12s ● Biphasic P wave in V1 & V2 with negative terminal portion having depth of >0.1 mV
  24. 24. V1 LII
  25. 25. Right Atrial Enlargement ● P waves tall (> 0.25mV) & peaked in inferior leads ● Biphasic P wave in V1 with first component larger than the second
  26. 26. Right Atrial Enlargement
  27. 27. Right Atrial Enlargement
  28. 28. Right Ventricular Hypertrophy ● R/S ratio in V1 > 1 ● S in V1 < 2mm ● RAD > 110 degrees
  29. 29. Right Ventricular Hypertrophy
  30. 30. Left Ventricular Hypertrophy Sum of R wave in V5 or V6 + S wave in V1 or V2 > 35mm in > 30 year olds >40 mm in 20-30 year olds >60 mm in 6-20 year olds
  31. 31. STEPS IN BASIC ECG READING 1. Determine Rate 2. Determine Rhythm 3. Measure Intervals 4. Determine QRS electrical axis 5. Check for chamber enlargements 6. Inspect QRS complexes for bundle branch block or fascicular block 7. Check for ST and T wave changes 8. Other ECG tracings
  32. 32. PREMATURE ATRIAL CONTRACTIONS • A longer PR interval • Premature atrial contractions may occur in a pattern, such as every other beat (atrial bigeminy), every third beat (atrial trigeminy), and so on. • Common at all ages and usually do not indicate underlying heart disease. • Increased rates of premature atrial contractions are seen in patients with chronic heart or lung disease
  33. 33. PREMATURE VENTRICULAR  Many PVCs occurring in a bigeminal or trigeminal pattern have a fixed coupling interval (within 40 milliseconds) from the preceding sinus beat  The degree is categorized into:  Occasional / Isolated  Multiple of similar morphology / Unifocal  Multiple with different morphology / Unifocal
  34. 34. BRADYDYSRHYTHMIAS
  35. 35. JUNCTIONAL RHYTHM  Usually do not require specific treatment.  Atropine can be used to accelerate the SA node discharge rate and enhance AV nodal conduction.  Accelerated junctional rhythm and junctional tachycardia are managed by treating the underlying cause.
  36. 36. IDIOVENTRICULAR RHYTHM  Regular widened QRS complexes without evidence of atrial activity  Atropine can be tried, although the likelihood of successful treatment is low.  Cardiac pacing is often needed, starting via the transcutaneous route.
  37. 37. SUPRAVENTRICULAR TACHYDYSRHYTHMIAS
  38. 38. ATRIAL FIBRILLATION AND ATRIAL FLUTTER  Treated with Ventricular Rate Control and/or Synchronized Cardioversion  Atrial Fibrillation – 150-200J  Atrial Flutter - May require as little as 25–50 J
  39. 39. MULTIFOCAL ATRIAL TACHYCARDIA  An irregular rhythm resulting from at least three different atrial foci competing to pace the heart, resulting in distinct P-wave morphologies on the ECG.  Found most often in elderly patients with decompensated chronic lung disease, but may also complicate heart failure or sepsis.  Vagal maneuvers are often effective
  40. 40. Paroxysmal supraventricular Tachycardia  Seen more frequently in females, with a peak in the late teenage and young adult years.  Palpitations, lightheadedness, and dyspnea are common symptoms.  Attention to technique is important to maximize success rate. If there is no response to vagal maneuvers, IV adenosine is recommended to convert to sinus rhythm.
  41. 41. ATRIOVENTRICULAR BLOCKS
  42. 42. FIRST-DEGREE ATRIOVENTRICULAR BLOCK  First-degree AV block occasionally is found in normal hearts.  Patients with first-degree AV block without evidence of organic heart disease appear to have no difference in mortality compared with matched controls.
  43. 43. SECOND-DEGREE MOBITZ TYPE I (WENCKEBACH’S) ATRIOVENTRICULAR BLOCK  There is progressive prolongation of AV conduction (and the PR interval) until an atrial impulse is completely blocked.
  44. 44. SECOND-DEGREE MOBITZ TYPE II ATRIOVENTRICULAR BLOCK  The PR interval remains constant across the rhythm strip, both before and after the nonconducted atrial beats.
  45. 45. THIRD-DEGREE ATRIOVENTRICULAR (COMPLETE HEART BLOCK)  In third-degree AV block, there is no AV conduction.
  46. 46. WIDE-COMPLEX TACHYCARDIAS
  47. 47. VENTRICULAR TACHYCARDIA  Ventricular tachycardia is the occurrence of three or more consecutive depolarizations from a ventricular ectopic pacemaker at a rate faster than 100 beats/min.  Monomorphic / Polymorphic
  48. 48. VENTRICULAR FIBRILLATION  Ventricular fibrillation is seen most commonly in patients with severe ischemic heart disease, with or without an acute myocardial infarction.  Treatment of pulseless ventricular tachycardia or fibrillation is with electrical defibrillation along with chest compressions and other advanced life support measures.
  49. 49. CONDUCTION AND ABNORMALITIES
  50. 50. WOLFF-PARKINSON-WHITE
  51. 51. BRUGADA SYNDROME  Brugada syndrome is an inherited disorder of myocardial depolarization that predisposes young individuals to malignant ventricular dysrhythmias and sudden death.  The majority of patients with Brugada syndrome are asymptomatic and are only found via an incidental ECG.
  52. 52. LONG QT SYNDROME  Prolongation of the QT interval.  Do not use medications that possess channel blockade effects, impair cardiac repolarization, prolong the QT interval, and provoke tachydysrhythmias.  Correct underlying electrolyte abnormalities especially K and Mg.
  53. 53. SHORT QT SYNDROME  A rare but highly lethal entity associated with an increased risk of ventricular tachydysrhythmias  Primarily recognized by a shortened QT interval without any need to adjust for heart rate.  Afflicted patients have otherwise structurally and functionally normal hearts.
  54. 54. THANK YOU!

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