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IntestinalIntestinal
Obstruction inObstruction in
ChildrenChildren
ObjectivesObjectives
• Presentation of obstruction
• To know different causes
• A brief about each cause
Duodenal HematomaDuodenal Hematoma
• Causes:
• Blunt trauma
• RTA
• Associated injuries include
• ----laceration to the left lobe of liver and to the
pancreases
• Bleeding Disorders(Henoch-schonlein purpura)
• It can cause complete or partial obstruction
IMAGING
•Ba Meal
•( Thickened mucosal folds, localized filling defects
due to intramural hematoma)
•CT Abdomen
•(for assessment of acute trauma and hematoma
directly, or for abnormal duodenal enhancement)
Enhanced CTEnhanced CT
Intramural duodenal hematoma almost completelyIntramural duodenal hematoma almost completely
obscuring the lumenobscuring the lumen
DUPLICATION CYSTDUPLICATION CYST
• An abnormal portion of intestine which is attached
to or intrinsic with normal bowel
• Incomplete recanalization at around 8wks
• Any where in the GIT
• 1/3 involve distal small bowel
Types
• Tubular
• Spherical
• communication
• Presentation
depends on the size and site
• Esp. those assoc. with stomach or duodenum
present with
• Abd. Pain
• Vomiting
• May act as a lead point for Intussusception
• GI Bleeding ( From ectopic mucosa)
ImagingImaging
• Radiography
(mass effect with displacement of adjacent bowel
loops)
• Ultrasound
(simple hypoechoic cyst, Gut wall signature)
Hypoechoic cyst with double ‘gut wallHypoechoic cyst with double ‘gut wall
signature’(inner echogenic mucosa & outersignature’(inner echogenic mucosa & outer
hypoechoic smooth muscle layerhypoechoic smooth muscle layer
Differentials
•-mesenteric
•-omental
•-choledochal
•-renal
•-ovarian
Mesenteric/OmentalMesenteric/Omental
cyst(lymphangiomas)cyst(lymphangiomas)
• Developmental anomalies of lymphatic system
(mesentry/ omentum)
Presentation
• -similar to duplication cyst
• On U/S multiloculated cyst with thin septation
• Tx– surgical resection
Mesenteric Cyst
Meckel ‘s DiverticulumMeckel ‘s Diverticulum
Persistence of prox. Vitelline duct
• True diverticulum
• From anti mesenteric border
• Rule of two’s
Complication
• -acute inflammation (mimicking appendicitis)
• -GI bleed
• -lead point for intussusception
Supine & prone radiographs of the upper GI barium seriesSupine & prone radiographs of the upper GI barium series
AppendicitisAppendicitis
• Peak incidence 12-15 years
Presention
• -ill defined abd. Pain in RIF
• -fever and vomiting
IMAGINGIMAGING
Radiography
•May be normal or localized dilated bowel loops
•5-10% radiodense appendicolith identified
Ultrasound
•Non compressible blind ending tubular structure
approx 6mm or more
•Increased echogenicity of mesenteric fat
•Hyperemia on color Doppler
•Free fluid / mesenteric lymph nodes
Right iliac fossa mixed echogenicity inflammatoryRight iliac fossa mixed echogenicity inflammatory
mass and echogenic focus with acousticmass and echogenic focus with acoustic
shadowingshadowing
Hypoechoic tubular structure 7mm in diameterHypoechoic tubular structure 7mm in diameter
adjacent to iliac vesselsadjacent to iliac vessels
CT findings are
•--localized or multi-focal abscess
ComplicationComplication
• Appendicolith
• Pelvic abcess
• Generalized peritonitis
• Portal vein thrombosis
• Multiple hepatic abcess ( rare)
Differential
•-- mesenteric adenitis
•--Crohn’s disease
•--Infection
•--ovarian torsion/cyst
HENOCH SCHONLEINHENOCH SCHONLEIN
PURPERAPURPERA
Small bowel vasculitis
•Jejunum most frequently involved
•Unknown etiology/postinfectious/post drug
therapy
Presentations with
•Purpuric rash over the buttocks & legs
•Abdominal pain
•glomerulonephritis
Henoch schonlein pupuraHenoch schonlein pupura
Ultrasound & barium follow throughUltrasound & barium follow through
JejunalJejunal bowel wall thickeningbowel wall thickening
Complications
•Transient small bowel intussusception(rare)
•Echogenic kidney suggest renal involvement
OTHEROTHER
INFECTIONS
•(giardia,compylobacter,yersinia,salmonella etc)
GRAFT VERSUS HOST REACTION
•(mostly effecting small bowel)
CROHN’S DISEASE
•(mostly effecting terminal ilium & cecum)
Polyps and polyposisPolyps and polyposis
syndromessyndromes
Isolated juvenile polyps
•Single or multiple
•Under 10 years of age
•Found in sigmoid colon and rectum
•Unlike adults they are hamartomas
•Present with painless rectal bleeding
leading to iron deficiency anemia
•Not premalignant
• Double contrast barium enema
• Endoscopy
• A pedunculated polyp with a long stalk is seen
Barium enema showing a pedunculated polyp in theBarium enema showing a pedunculated polyp in the
descending colondescending colon
Juvenile polyposisJuvenile polyposis
• Positive family hx (most cases)
• Five or more polyps
• Associated with higher long term risk of colonic
carcinoma
Peutz jeghers syndromePeutz jeghers syndrome
• Autosomal dominant
• Occur anywhere from stomach to rectum (mostly
small intestine)
• Associated with mucocutaneous pigmentation and
GI hamartomas
Small bowl follow through
• -multiple filling defects
Complications
•Intussusception around polyps(usually transient)
•Small bowel obstruction
•Gastrointestinal adenocarcinoma & non GI neoplasm
involving pancreas, breast or reproductive organs
• Familial polyposis coli
• Gardner syndrome
• Both are dominanly inherited
• Multiple adenomatous polyps are found (numerous
in colon)
• High malignant potential
• Prophylactic proctocolectomy usually
recommended
TURCOT’S SYNDROMETURCOT’S SYNDROME
• Autosomal recessive condition
• Colonic adenomas associated with CNS glioma
Small bowel malignanciesSmall bowel malignancies
Burkit type non Hodgkin lymphoma
•Mostly involve Ileocecal region
•Male predominance
•Peak incidence 5-8yrs
Presenting symptoms are
•Abdominal pain
•Palpable mass
•Failure to thrive
ULTRASOUND
•Thickened hypoechoic bowel loops are seen often
forming adherent masses with infiltration of adjacent
omentum & mesentery
•Hepatospenomegaly
•Retroperitoneal lymphadenopathy
CAUSES OF COLITIS INCAUSES OF COLITIS IN
CHILDHOODCHILDHOOD
• INFECTIOUS
• (compylobacter,E.coli,salmonella,shigella etc)
• INFLAMMATORY BOWEL DISEASE
• TYPHILITIS
• HEAMOLYTIC URAEMIC SYNDROME
• PSEUDOMEMBRANOUS COLITIS
• GRAFT VERSUS HOST REACTIONS
• ISCHAEMIC COLITS
• IRRADIATION COLITIS
CROHN’S DISEASECROHN’S DISEASE
• Involve any part of GIT from mouth to anus (usually
sparing the rectum)
• Prepubertal child or adolescent are effected
Extraintestinal features more prominent
• weight loss
• anorexia
• short stature
• Delayed puberty
GI SYMPTOMS
•Diarrhoea
•Abdominal pain
IMAGINGIMAGING
• ENDOSCOPY
• BARIUM ENEMA(largely replaced by endoscopy)
• aphthoid ulceration
• mucosal ulceration is deep, discontinuous &
asymmetrical
• generally have thicker colon than ulcerative colitis
• LEUCOCYTE SCINTOGRAPHY(extent of disease)
CT SCAN
•transmural bowel wall thickening
•creeping fat within the mesentery
•strictures
•fistulas
•localised collection
MRI
•assessment of disease extent
Innumerable aphthoid ulcer in crohn’s diseaseInnumerable aphthoid ulcer in crohn’s disease
Enema in crohn’s disease showing extensiveEnema in crohn’s disease showing extensive
cobblestoning due to linear ulceration &mucosalcobblestoning due to linear ulceration &mucosal
edema. Rectum is sparededema. Rectum is spared
ULCERATIVE COLITISULCERATIVE COLITIS
• Relapsing and remitting proctits
• Rectum is always effected
• Effects young adults(15-25yrs) with second smaller
peak at approx 60yrs
CLINICAL FINDING
•bloody diarrhoea
•abdominal pain
•failure to thrive
IMAGINGIMAGING
Double contrast barium enema
Proctosigmoidoscopy
•loss of normal mucosal vascular pattern (earliest
detectable change)
•ulceration is continuous & superficial
•(deep ulceration does occur)
•haustral blunting
• Luminal narrowing
• Colonic shortening(due to muscular abnormality
rather than fibrosis)
CT SCAN
not for primary diagnosis once toxic megacolon is
established
Double contrast barium enema shows granularDouble contrast barium enema shows granular
mucosa (changes of early disease)mucosa (changes of early disease)
Complication
•Risk of colonic ca is high approx 20% per decade
•toxic megacolon
TYPHILITISTYPHILITIS
• Inflammatory condition
• Predominantly effects right colon in neutropenic
patients
ON ULTRASOUND
• Thickened hypoechoic cecum and ascending
colon
• Echogenic mucosa and hyperaemia
CT SCAN
• -shows bowel wall thickening
Bowel wall thickening & fat strandingBowel wall thickening & fat stranding
HAEMOLYTIC URAEMICHAEMOLYTIC URAEMIC
SYNDROMESYNDROME
• Commonest cause of acute renal failure in children
• Diarrheal illness caused by E.coli leading to
• Microangiopathic anemia
• Thrombocytopenia and acute renal failure
IMAGINGIMAGING
Ultrasound
•Association of colonic thickening &echogenic
kidneys is highly suggestive of diagnosis
•Doppler flow within the bowel wall is reduced (atleast
in prodromal phase)
INTUSSUSCEPTIONINTUSSUSCEPTION
• Invagination of a segment of bowel(the
intussusceptum) into the contiguous segment(the
intussuscipiens)
Site
• Ileocolic(approx 90% cases)
• Ileoileocolic,colocolic,ileoileal
Peak age incidence
• 6 months to 2yrs
Classic presentation
•Episodic abdominal pain
•Screaming episodes associated with passage of
blood & mucus(current jelly)
•Haemodynamic instability due to considerable fluid
shift
IMAGINGIMAGING
Abdominal radiograph
•Absence of bowel gas in the right iliac fossa with
rounded soft tissue mass
•A crescent of air at the apex of intussusception
•Or small bowl obstruction
Ultrasound(highly sensitive)
•a mass with multiple hyperechoic concentric rings
Paucity of bowel gas in the right iliac fossa andPaucity of bowel gas in the right iliac fossa and
soft tissue masssoft tissue mass
Transverse ultrasound showing multipleTransverse ultrasound showing multiple
hypoechoic concentric rings, central echogenichypoechoic concentric rings, central echogenic
mesentery and few small echogenic lymph nodesmesentery and few small echogenic lymph nodes
• Small crescents of peritoneal fluid may be trapped
b/w the layers of intussusception
• Colour flow with in the mass suggests bowel viability
• Small lymph nodes are frequently found within the
intussusception
TREATMENTTREATMENT
RADILOLOGICAL REDUCTION
•Absolute contraindications are peritonitis and
perforation
PNEUMATIC REDUCTION(air enema)
•Replaced the barium in most paediatric centres(70-
90% success rate)
THANKSTHANKS

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