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SECONDARY HYPERTENSION ,[object Object],[object Object]
SECONDARY HYPERTENSION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
RISK FACTORS FOR SECONDARY HYPERTENSION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ENDOCRINE HYPERTENSION ,[object Object],[object Object]
ENDOCRINE HYPERTENSION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
PHEOCROMOCYTOMA ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object]
CATHACHOLAMINE PRODUCTING TUMOURS CHROMAFFIN CELLS  ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
TYROSINE DIHYDROXY PHENYLALANINE (DOPA) CATHACHOLAMINES 3 – METHOXY  4 HYDROXY MANDELIC  ACID  VMA  HYDROXYLATED  DECARBOXYLATED DEGRADED
TREATMENT  ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERALDOSTERISM PRIMARY  SECONDARY  ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],EXCESS OF ALDOSTERONE ECF EXPANSION HTN MARKED SUPPRESSON OF RENIN SECRETION  2/3 ADENOMA < 2CM BILATERAL HYPERPLASIA
CLINICAL  ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
GLUCORTICOID EXCESS HYPERCORTISOLISM CUSHING SYNDROME ACTH DEPENDENT PITUITARY DEPENDENT CUSHING DISEASE) 65% ECTOPIC ACTH PRODUCING 10% ACTH ADMINISTRATION  NON -ACTH DEPENDENT ADRENAL  25%  ADENOMA HYPERPLASIA  CARCINOMA  CORTICOSTEROID ADMINISTRATION
CUSHING’S SYNDROME ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIAGNOSIS  ,[object Object],[object Object],[object Object],[object Object],[object Object]
IMAGING
SURGICAL
ENDOCRINE HYPERTENSION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
CASE 2 ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],MAK-AKU 2008
MAK-AKU 2008
INVESTIGATION DONE AT AKUH ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
 
THIS PATIENT HAS A BP 155/110
TAKE HOME MESSAGE ,[object Object],[object Object],[object Object]
THANKYOU  FOR YOUR ATTENTION  MAK-AKU 2008

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Causes and Risk Factors of Secondary Hypertension