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Austin Journal of Clinical Neurology

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Austin Journal of Clinical Neurology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of neurology, neurophysiology and stroke.
The aim of the journal is to provide a forum for neurologists, researchers, physicians, and other health professionals to find most recent advances in the areas of clinical and experimental neurology. Austin Journal of Clinical Neurology accepts original research articles, review articles, case reports, clinical images and rapid communication on all the aspects of clinical neurology and nervous system diseases.
Austin Journal of Clinical Neurology strongly supports the scientific upgradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing, mutual promotion of multidisciplinary science.
Austin Journal of Clinical Neurology is an open access, peer reviewed, scholarly journal dedicated to publish articles in all areas of neurology, neurophysiology and stroke.

Publié dans : Santé
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Austin Journal of Clinical Neurology

  1. 1. HISTORY “CEREBRAL PALSY” was first identified by a English Surgeon “WILLIAM LITTLE” in 1860. It is also called as Little’s Disease
  2. 2. DEFINITION Cerebral Palsy is considered as a neurological disorder caused by a non- progressive brain injury or malformation that occurs by abnormal development or damage to motor control centers of the brain.
  3. 3. ETIOLOGY Prenatal Perinatal Postnatal/childhood
  4. 4. 1. PRENATAL Maternal diseases Obstetrical complication Abuse of drugs Cerebral malformation Maternal diseases Prolonged rupture of membranes Gestational diseases Premature delivery Preeclampsia Infections Severe protienuria Fetal heart rate depression Long labor Asphyxia
  5. 5. 2. PERINATAL Prematurity and associated problems Low birth weight Meconium aspiration Cerebral trauma Severe hyperglycemia Blood incompatibilities Hyperbilirubinemia Meconium aspiration Complicated delivery
  6. 6. 3. POSTNATAL Brain injury Stroke Meningitis or encephalitis Traumatic brain injury Seizures Vascular malformations Cerebral anoxia Sickle cell anemia Near drowning Toxins
  7. 7. CLASSIFICATION OF CP CEREBRAL PALSY is classified into two types as below: Depending upon Topographical Distribution Depending on tone or movement patterns (physiologic)
  8. 8. DEPENDING UPON TOPOGRAPHICAL DISTRIBUTION Monoplegia Diplegia Paraplegia Quadriplegia Triplegic Hemiplegic Tetraplegic/Double hemiplegic
  9. 9. DEPENDING ON TONE OR MOVEMENT PATTERNS (PHYSIOLOGIC) Spastic CP Athetoid/Dyskinetic CP Ataxic CP Flaccid/Hypotonic CP Choreiform CP Rigid CP Mixed CP
  10. 10. DIAGNOSIS Physical Evaluation Magnetic Resonance Imaging(MRI) Computerized Tomography(CT) scan EEG X-ray skull-intracranial calcification Neurological assessment Ultrasound
  11. 11. TREATMENT Physiotherapy Occupational therapy Speech therapy Surgery Adjunctive therapy Recreation therapy
  12. 12.  Reduce spasticity  Increase mobility and flexibility  Prevent deformity  Improve physical activity  Improve functional skills  Intake of proper diet and medications MEASURES TAKEN BY A CP PATIENT
  13. 13. MAINTAIN PROPER DIET

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