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THERORIES OF TOOTH
ERUPTION
Resource Faculty
Prabhat Ranjan Pokharel
Head of department
Rajesh Gyawali
Assistant professor
Jamal Giri
Assistant professor
DEPARTMENT OF ORTHODONTICS
PRESENTER
Nabin Chaudhary
Contents
• Root elongation theory(Hammock ligament theory)
• Bone remodeling
• Periodontal ligament contraction theory
• Vascular pressure theory
• Pulp constriction theory
• Dental follicle theory
Physiologic tooth movement
• Tooth eruption
• Migration or drift of tooth
• Changes in tooth position during
mastication
• Active eruption
• Passive eruption
• Anatomical crown
• Clinical crown
Phases of tooth eruption
• Pre-eruptive phase
• Eruptive phase(4 stages:root form-
ation,penetration,movement,occlusal
contact)
• Post-eruptive phase
Patterns of tooth movement
• Pre-eruptive phase
1. Bodily(drifting) movement
2. Eccentric growth
• Eruptive phase
1. Axial(occlusal) movement
2. Drifting(bodily) movement
3. Tilting(tipping) movement
4. Rotating movement
• Post-eruptive phase
1. axial
ROOT ELONGATION THEORY
 proliferating root impinges on a fixed base, thus converting an apically directed force of the
tooth into occlusal movement
 Fallacies
o rootless tooth erupts
o Some teeth erupt more than the total length of the roots
o Teeth erupt even after the complete formation of the root
Therefore root formation is accommodated during tooth eruption and is a consequence,
not a cause, of the eruption process.
• According to Harry Sicher, a band of fibrous tissue(cushion hammock
ligament) exists below the root apex spanning from one side of
alveolar wall to another.
Ligament was never found histologically
BONE REMODELING
• The end result of bone remodeling is a considerable bone deposition
at the bottom of the socket
• selective formation and resorption of bone brings about eruption.
• Fallacies :
• alveolar bone remodeling which occurs around the root,endby bone
deposition, is the outcome and not the cause of axial tooth
movement
PERIODONTAL LIGAMENT
CONTRACTION THEORY
• CONTRACTILE ELEMENTS - COLLAGEN AND FIBROBLASTS
CONSTRICTION
Force initiated by fibroblast
Transmitted to extracellular compartment via fibronexus and
collagen bundles by root formation
ERUPTION
• Fallacies
• Examples of PDL being present but tooth not erupting and rootless
teeth erupting have been reported.
• Fallacies
• Examples of PDL being
present but tooth not
erupting and rootless
teeth erupting have
been reported.
A –fibroblasts
B –adherence type
intercellular junctions
C – fibronexus, attached
to collagen by
fibronectin
D – deposition of
collagen
E – resorption of collagen
PULP CONSTRICTION THEORY
• Growth of root dentin and constriction of pulp --- sufficient pressure
to move tooth occlusally
• Fallacies
• Pulpless teeth erupt at same rate as normal teeth
• Premature extraction of deciduous molar: premolar do erupt
without any growth of dentin or pulp constriction
VASCULAR PRESSURE THEORY
• THE VASCULAR PRESSURES are present in pulpal tissues as well as in the
periodontal ligament
• The pressure exerted by the blood vessels at the apex of the tooth help
in eruption of teeth
• Fallacies
• The pressure exerted is not enough to help in eruption of teeth
• Teeth erupt even when the vascular supply is cut
DENTAL FOLLICLE THEORY
• Specific cellular changes occur in and around the follicle when a
tooth erupts.
• Source for new bone-forming cells and osteoclasts
• Teeth eruption is delayed or absent in animal models and human
diseases that cause a defect in osteoclast differentiation
• Tooth eruption is definitely a multifactorial process involving all
• or few factors of the above or other factors like
 Control of Endocrine Glands
 Pressure from muscular action
 Effect of Nutrition
 Inherent tendency of teeth to erupt
Facts
• Rate of eruption
• Intraosseous=1-10µm/day
• Extraosseous=75 μm/day
• Root formation ( tomes 1872 )
• Bone remodeling ( brash 1928 )
• Dental follicle ( marks and cahill 1984)
• Periodontal ligament ( thomas 1967 )
• Hydrostatic pressure ( sutton and graze 1985 )
• Pulpal pressure ( v . Korff 1935 )

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486 theories of tooth eruption

  • 1. THERORIES OF TOOTH ERUPTION Resource Faculty Prabhat Ranjan Pokharel Head of department Rajesh Gyawali Assistant professor Jamal Giri Assistant professor DEPARTMENT OF ORTHODONTICS PRESENTER Nabin Chaudhary
  • 2. Contents • Root elongation theory(Hammock ligament theory) • Bone remodeling • Periodontal ligament contraction theory • Vascular pressure theory • Pulp constriction theory • Dental follicle theory
  • 3. Physiologic tooth movement • Tooth eruption • Migration or drift of tooth • Changes in tooth position during mastication • Active eruption • Passive eruption • Anatomical crown • Clinical crown
  • 4. Phases of tooth eruption • Pre-eruptive phase • Eruptive phase(4 stages:root form- ation,penetration,movement,occlusal contact) • Post-eruptive phase
  • 5. Patterns of tooth movement • Pre-eruptive phase 1. Bodily(drifting) movement 2. Eccentric growth • Eruptive phase 1. Axial(occlusal) movement 2. Drifting(bodily) movement 3. Tilting(tipping) movement 4. Rotating movement • Post-eruptive phase 1. axial
  • 6. ROOT ELONGATION THEORY  proliferating root impinges on a fixed base, thus converting an apically directed force of the tooth into occlusal movement  Fallacies o rootless tooth erupts o Some teeth erupt more than the total length of the roots o Teeth erupt even after the complete formation of the root Therefore root formation is accommodated during tooth eruption and is a consequence, not a cause, of the eruption process.
  • 7. • According to Harry Sicher, a band of fibrous tissue(cushion hammock ligament) exists below the root apex spanning from one side of alveolar wall to another. Ligament was never found histologically
  • 8. BONE REMODELING • The end result of bone remodeling is a considerable bone deposition at the bottom of the socket • selective formation and resorption of bone brings about eruption. • Fallacies : • alveolar bone remodeling which occurs around the root,endby bone deposition, is the outcome and not the cause of axial tooth movement
  • 9. PERIODONTAL LIGAMENT CONTRACTION THEORY • CONTRACTILE ELEMENTS - COLLAGEN AND FIBROBLASTS CONSTRICTION Force initiated by fibroblast Transmitted to extracellular compartment via fibronexus and collagen bundles by root formation ERUPTION
  • 10. • Fallacies • Examples of PDL being present but tooth not erupting and rootless teeth erupting have been reported.
  • 11. • Fallacies • Examples of PDL being present but tooth not erupting and rootless teeth erupting have been reported. A –fibroblasts B –adherence type intercellular junctions C – fibronexus, attached to collagen by fibronectin D – deposition of collagen E – resorption of collagen
  • 12. PULP CONSTRICTION THEORY • Growth of root dentin and constriction of pulp --- sufficient pressure to move tooth occlusally • Fallacies • Pulpless teeth erupt at same rate as normal teeth • Premature extraction of deciduous molar: premolar do erupt without any growth of dentin or pulp constriction
  • 13. VASCULAR PRESSURE THEORY • THE VASCULAR PRESSURES are present in pulpal tissues as well as in the periodontal ligament • The pressure exerted by the blood vessels at the apex of the tooth help in eruption of teeth • Fallacies • The pressure exerted is not enough to help in eruption of teeth • Teeth erupt even when the vascular supply is cut
  • 14. DENTAL FOLLICLE THEORY • Specific cellular changes occur in and around the follicle when a tooth erupts. • Source for new bone-forming cells and osteoclasts • Teeth eruption is delayed or absent in animal models and human diseases that cause a defect in osteoclast differentiation
  • 15. • Tooth eruption is definitely a multifactorial process involving all • or few factors of the above or other factors like  Control of Endocrine Glands  Pressure from muscular action  Effect of Nutrition  Inherent tendency of teeth to erupt
  • 16. Facts • Rate of eruption • Intraosseous=1-10µm/day • Extraosseous=75 μm/day • Root formation ( tomes 1872 ) • Bone remodeling ( brash 1928 ) • Dental follicle ( marks and cahill 1984) • Periodontal ligament ( thomas 1967 ) • Hydrostatic pressure ( sutton and graze 1985 ) • Pulpal pressure ( v . Korff 1935 )