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MAJOR DISORDERS  OF THE ENDOCRINE SYSTEM Nio C. Noveno, RN, MAN
 
HORMONE REGULATION: NEGATIVE FEEDBACK MECHANISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HORMONE REGULATION: NEGATIVE FEEDBACK MECHANISM
DISORDERS OF THE ENDOCRINE SYSTEM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ANTERIOR PITUITARY DISORDERS
 
 
HYPERPITUITARISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
GROWTH HORMONE HYPERSECRETION ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERPITUITARISM: CLINICAL MANIFESTATIONS ,[object Object],C hest: barrel-shaped R ough facial features O dd sensations: hands and feet M uscle weakness & fatigue E nlargement of organs G rowth of coarse hair A menorrhea; breast milk production L oss of vision; headaches I mpotence; increased perspiration  S noring
HYPERPITUITARISM: CLINICAL MANIFESTATIONS
HYPERPITUITARISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
TRANS-SPHENOIDAL HYPOPHYSECTOMY ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOPITUITARISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOPITUITARISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Sheehan’s syndrome [Post-partum  pituitary necrosis] A complication of delivery Results from severe  blood loss and  hypovolemia    Pituitary ischemia
HYPOPITUITARISM: CLINICAL MANIFESTATIONS ,[object Object],Loss  of vision, strength, libido, & secondary sexual  characteristics
HYPOPITUITARISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
POSTERIOR PITUITARY DISORDERS
 
 
DIABETES INSIPIDUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES INSIPIDUS: DIAGNOSTICS ,[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES INSIPIDUS: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
SYNDROME OF INAPPROPRIATE ADH ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
SIADH: DIAGNOSTICS ,[object Object],[object Object],[object Object],[object Object],[object Object]
SIADH: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID DISORDERS
 
 
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THYROID FUNCTION TESTS ,[object Object],[object Object],[object Object],[object Object]
HYPERTHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERTHYROIDISM: CLINICAL MANIFESTATIONS ,[object Object],R apid weight loss A pprehension V olume deficit; voracious appetite E xophthalmos; erratic menses S ystolic BP elevated; sweating [tremors, tachycardia, palpitations]    in secondary disease    in primary disease TSH
HYPERTHYROIDISM: CLINICAL MANIFESTATIONS
THYROID STORM / THYROTOXIC CRISIS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Occurs in patients with existing but unrecognized  thyrotoxicosis, stressful illness, thyroid surgery, RAI Increased systemic  adrenergic activity:  Severe hypermetabolism
HYPERTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
RAI THERAPY : NURSING IMPLICATIONS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
K OR NA IODIDE, SSKI (LUGOL’S) : NURSING IMPLICATIONS ,[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOTHYROIDISM ,[object Object],[object Object],A utoimmune D evelopmental D ietary I odine deficiency O ncologic D rugs I atrogenic N on-thyroidal E ndocrine
HYPOTHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOTHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOTHYROIDISM: CLINICAL MANIFESTATIONS ,[object Object],E dema (periorbital) R educed BMR  [bradycardia, bradypnea] A pathy; anorexia; anemia I ncreased weight; intolerance to cold L ethargy; loss of libido E nlarged tongue D rooling    in secondary disease    in primary disease TSH
MYXEDEMA COMA ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Precipitating Factors Acute illness  Rapid withdrawal of thyroid medication  Anesthesia / Surgery Hypothermia Opioid use
HYPOTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOTHYROIDISM: NURSING INTERVENTIONS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],Maintain patent airway Administer medications: Synthroid, glucose,  corticosteroids IV fluid replacement Wrap patient in blanket Treat infection or any underlying illness
PARATHYROID DISORDERS
 
 
HYPERPARATHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPERPARATHYROIDISM: CLINICAL MANIFESTATIONS ,[object Object],A pathy L ordosis C ardiac dysrhythmias U pset GIT L ow energylevels I ncreased BP    PO 4  PTH Calcium Alkaline phospatase
HYPERPARATHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOPARATHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
HYPOPARATHYROIDISM: CLINICAL MANIFESTATIONS ,[object Object],E xtremities: tingling F otophobia I ncreased bone density C hvostek sign; cramps I rritability T rousseau sign; tetany    PO 4  PTH Calcium Alkaline phospatase
HYPERTHYROIDISM: MANAGEMENT ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
T rousseau’s & Chvostek’s E levated serum PO 4 ; low Ca 2+ T ingling A lkalosis; Arrhythmias N arrowing of airway I rritability C ramps
HYPOPARATHYROIDISM ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
T  C  AKE ARE ETANY RACHEOSTOMY ALCIUM GLUCONATE ALCIUM 8.6 – 10.6 mg / dL
PHEOCHROMOCYTOMA
ADRENAL GLANDS
ADRENAL MEDULLA ,[object Object],[object Object],[object Object],[object Object]
PHEOCHROMOCYTOMA ,[object Object],[object Object],[object Object]
PHEOCHROMOCYTOMA ,[object Object],[object Object],[object Object],[object Object],[object Object]
PHEOCHROMOCYTOMA BP HR Diaphoresis BMR VMA Glucose
PHEOCHROMOCYTOMA ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
PHEOCHROMOCYTOMA ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ADDISON'S DISEASE
ADRENAL CORTEX HORMONES  ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ADRENAL CORTEX HORMONES ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
 
ADRENAL CORTEX HORMONES ,[object Object],[object Object],[object Object]
 
ADDISON'S DISEASE ,[object Object],[object Object],[object Object]
ADDISON'S DISEASE ,[object Object],[object Object],[object Object],[object Object]
 
ADDISON'S DISEASE ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ADDISON'S DISEASE ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ADDISON’S DISEASE
ADDISON'S DISEASE ,[object Object],[object Object],[object Object],[object Object],[object Object]
CUSHING'S SYNDROME
CUSHING'S DISEASE ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object]
CUSHING’S SYNDROME
CUSHING'S SYNDROME ,[object Object],[object Object],[object Object]
CUSHING'S SYNDROME ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
CUSHING'S SYNDROME ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
P  olyuria olydipsia olyphagia ruritus aresthesia oor healing oor eyesight
Normal Impaired DM FBS <110mg/dl 110-125mg/dl ≥ 126mg/dl 2H OGTT <140mg/dl ≥ 140;  <200mg/dl ≥  200 mg/dl
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object]
Insulin Onset Peak Duration Ultra rapid acting insulin analog (humalog) 10-15 min 1 H 3 H SAI (humulin regular) ½ - 1 H 2-4 H 4-6 (8) H IAI (humulin lente, Humulin NPH) 3-4 H 4-12 H 16-20 H LAI (Protamine zinc, humulin ultralente) 6-8 H 12-16 H 20-30 H Premixed insulin (NPH-regular [80-20, 70-30, 50-50]) ½-1 H 2-12 H 18-24 hrs Insulin glargine  (Lantus ) Slower than NPH No Peak 24 H
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object]
INSULIN ADMINISTRATION ,[object Object],[object Object],Increases blood glucose  levels Glucocorticoids, thiazide diuretics, thyroid agents, oral contraceptives Increase the need for increased insulin dose Illness, infection, and stress
ORAL HYPOGLYCEMIC AGENTS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ORAL HYPOGLYCEMICS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ORAL HYPOGLYCEMICS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
ORAL HYPOGLYCEMICS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object]
GLYCOSYLATED HEMOGLOBIN (HBA 1C ) ,[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
THE CLIENT IS TIRED! ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],achycardia xcessive hunger xcitability remors
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
DIABETES MELLITUS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
MAJOR DISORDERS  OF THE ENDOCRINE SYSTEM THANK YOU! Nio C. Noveno, RN, MAN

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