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PRESENTATION ON
EMERGENCY, TRAUMA
AND
MULTI-SYSTEM ORGAN
FAILURE
Presented to:
Dr. Pallavi Pathania
Associate Professor
Medical Surgical Nursing
Shimla Nursing College
Presented by:
Reena Sharma
M.sc.(Nursing) 1st year
Shimla Nursing College
Annandale, Shimla
INDEX
SR. NO. CONTENT
1. Introduction
2. DIC
3. Trauma
4. Burn
5. Poising
6. Conclusion
7. Summarization
8 Recapitalization
9 Assignment
10 Bibliography
INTRODUCTION:
ā€¢ Emergencies can happen anywhere at any time.
Small-scale incidents occur frequently and are
dealt with effectively by the emergency
services. A Trauma service provides care for
people who have sustained physical injuries.
These injuries are often the result of an accident
but can be sustained in other circumstances.
CONTā€¦
MODS(MULTI-ORGAN DYSFUNCTIONING
SYNDROME)
It is progressive dysfunction of two or more major
organ systems in a critically ill patient that makes it
impossible to maintain homeostasis without medical
intervention and that is typically a complication of
sepsis and is a major factor in predicting mortality
.
Or
It is also known as multiple organ failure
(MOF), total organ failure (TOF) or
multisystem organ failure (MSOF), is
altered organ function in an acutely ill
patient requiring medical intervention to
achieve homeostasis
Contā€¦
DEFINITIONS:
EMERGENCY:
something dangerous or serious, such as an
accident, that happens suddenly or unexpectedly and
needs fast action in order to avoid harmful results.
OR
ā€¢ An emergency is a situation that poses an immediate
risk to health, life, property, or environment.
MOST COMMON MEDICAL EMERGENCIES
ā€¢ Bleeding
ā€¢ Trauma
ā€¢ Burn
ā€¢ Poising
ā€¢ Breathing difficulties
Contā€¦
ā€¢ Someone collapses
ā€¢ Fit and/or epileptic seizure
ā€¢ Severe pain
ā€¢ Heart attack
ā€¢ A stroke
(DIC)
DISSEMINATED
INTRAVASCULAR
COGULOPATHY
INTRODUCTION
ā€¢ In disseminated intravascular coagulation,
abnormal clumps of thickened blood (clots)
form inside blood vessels. These abnormal
clots use up the blood's clotting factors,
which can lead to massive bleeding in other
places. Causes include inflammation,
infection and cancer.
DEFINITION:
ā€¢ Disseminated intravascular coagulation
(DIC) is a serious disorder in which the
proteins that control blood clotting
become overactive.
OR
ā€¢ Disseminated intravascular
coagulation (DIC) is a condition in
which blood clots form throughout
the body, blocking small blood
vessels.
Contā€¦
RISK FACTORS:
ā€¢ Blood transfusion reaction
ā€¢ Cancer, especially certain types of leukemia
ā€¢ Inflammation of the pancreas (pancreatitis)
ā€¢ Infection in the blood, especially by bacteria
or fungus
ā€¢ Liver disease
ā€¢ Pregnancy complications (such as
placenta that is left behind after
delivery)
Contā€¦
CLASSIFICATION
ACUTE
ā€¢ It happened rapidly, the coagulopathy is dominant and
major symptoms are bleeding and shock, mainly seen
in severe infection, amniotic fluid embolism
CHRONIC
ā€¢ it happened slowly and last several weeks, thrombosis
and clotting may predominate
ā€¢ mainly seen in cancer.
PATHOPHYSIOLOGY:
Clinical findings
ļ¶Bleeding
ļ¶Bruising
ļ¶Deep vein thrombosis
ļ¶Purpura
ļ¶Petechiae
ļ¶Oliguria
ļ¶Acute renal failure
ļ¶Dyspnea
ļ¶cyanosis
ļ¶Respiratory failure
ļ¶Convulsion
ļ¶Coma
ļ¶Progressive cardiac failure
ļ¶shock
Contā€¦
DIAGNOSTIC EVALUATION:
ā€¢ History taking
ā€¢ Physical examination
LABORATORY TEST FINDINGS
Platelet count Markedly decreased
Prothrombin time Increased
Activated partial thromboplastin time Increased
Fibrin degradation products Markedly Increased
Fibrinogen Normal or decreased
D-dimer Markedly increased
Contā€¦
TREATMENT
Replacement therapy
Other Treatment
Heparin therapy
Treatment of the
underlying disorder
Treatment of the underlying disorder:
ā€¢ Avoid of either acute or chronic DIC.
ā€¢ Avoid delay treat vigorously (e.g. shock,
delay treat vigorously (e.g., shock, sepsis,
obstetrical problems
Contā€¦
Contā€¦
Replacement therapy:
ā€¢ Coagulation factor deficiency require
replacement with FFP (fresh frozen plasma).
ā€¢ Platelet transfusion should be used to
maintain a platelet count greater than
30000/Ī¼l, and 50000/Ī¼l.
Contā€¦.
Heparin therapy:
ā€¢ In some cases heparin therapy is contraindicated,
but when DIC is producing serious clinical
consequences Dose:500~750u/h is necessary.
ā€¢ Heparin therapy must be used in combination with
replacement therapy, it can lead to severe bleeding
Contā€¦.
Other treatment:
ā€¢ Aminocaproic acid, 1g/h iv Aminocaproic acid, 1g/h
iv
ā€¢ Tranexamic acid, 10mg/kg, iv,q8h,
Aminocaproic acid can never be used without heparin in
DIC because of the risk of thrombosis
ACUTE DIC:
Without bleeding or evidence of ischemia: No
treatment
With bleeding :
ā€¢ Blood components as needed
ā€¢ Fresh frozen plasma
ā€¢ Cryoprecipitate
ā€¢ Platelet transfusions
With ischemia:
Anticoagulants after bleeding risk is corrected
with blood products
Chronic DIC:
Without thromboembolism :No specific therapy
needed
But prophylactic drugs: eg, (low dose heparin, low
molecular weight heparin)
May be used for patients at high risk of thrombosis.
CONT..
With thromboembolism :Heparin or low-molecular-
weight heparin, trial of warfarin sodium (Coumadin).
(If warfarin is unsuccessful, long-term use of low ā€“
molecular weight heparin may be helpful.)
COMPLICATION OF DIC:
ļ¶Severe bleeding
ļ¶Stroke
ļ¶Ischemia of extremities or organs
ļ¶Waterhouse-Friderichse syndrome
ļ¶Sheehanā€™s syndrome
ļ¶Kasabach-merritt syndrome
TRAUMA
TRAUMA
DEFINITION:
Trauma is the response to a deeply distressing
or disturbing event that overwhelms an
individualā€™s ability to cope, causes feelings of
helplessness, diminishes their sense of self and
their ability to feel the full range of emotions
and experiences.
OR
Severe emotional shock and pain caused by
an extremely upsetting experience.
Contā€¦
TYPES OF TRAUMA :
ā€¢ BLUNT TRAUMA
ā€¢ PENETRATING TRAUMA
ā€¢ DECELERATION INJURIES
ā€¢ EXTERNAL FORCE INJURIES
ETIOLOGY OF TRAUMA :
UN-HELMETED MOTORCYCLES OR BICYCLE CRASH
MOTOR VEHICLE CRASH
FALL
ASSULT
PATHOPHYSIOLOGY:
All trauma leads to
Decreased organ perfusion,
Cellular ischemia,
Edema and inflammation. Once begun,
Inflammation becomes a disease process independent of its origin,
Multiple organ failure and
Death even after a patient has been completely resuscitated.
CLINICAL MANIFESTATION:
ā€¢ Fractures
ā€¢ Contusions
ā€¢ Open wound
ā€¢ Vomiting or nausea
ā€¢ Dizziness
ā€¢ Edema
Cont..
ā€¢ Increased heart rate
ā€¢ Low blood pressure
ā€¢ Fever
ā€¢ Disorientation or confusion
ā€¢ Loss of consciousness
ā€¢ Feeling of coldness as temperature
drops
ā€¢ Increased metabolism
Diagnostic evaluation:
ā€¢ History taking
ā€¢ Physical examination
ā€¢ X-ray
ā€¢ CT Scan
ā€¢ Ultrasonography
ā€¢ Echocardiography
ā€¢ MRI Scan
Contā€¦
MANAGEMENET:
The primary survey consists of the following
steps:
ā—Airway assessment and protection (maintain
cervical spine stabilization when appropriate)
ā—Breathing and ventilation assessment (maintain
adequate oxygenation)
ā—Circulation assessment (control hemorrhage and
maintain adequate end-organ perfusion)
ā—Disability assessment (perform
basic neurologic evaluation)
ā—Exposure, with environmental
control (undress patient and search
everywhere for possible injury,
while preventing hypothermia)
Contā€¦
1) AIRWAY:
ā€¢ Severely injured patients can develop airway
obstruction or inadequate ventilation leading to
hypoxia and death within minutes.
ā€¢ Assessment ā€” In a conscious patient, initial airway
assessment can be performed as fellow:
ā€¢ Inspect and palpate the anterior neck for lacerations,
hemorrhage, crepitus, swelling, or other signs of
injury.
ASSESSMENT IS DONE BY LEMON
mnemonic
ļƒ˜L: LOOK: Facial and neck injuries can distort
external and internal structures making it
difficult to visualize the glottis or insert an
endotracheal tube.
ļƒ˜E: EVALUATE : This refers to the intraoral,
mandibular, and hyoid-to-thyroid notch
distances.
Contā€¦
Cont..
ā€¢ M: MALLAMPATI: A standard
calculation of the mallampati score
cannot be performed in many
trauma patients; injured patients
requiring emergency intubation
often cannot open their mouths
spontaneously
CONT..
ļƒ˜O: OBSTRUCTION/OBESITY: Any number of
injuries can obstruct the airway including internal
or external hematomas or soft tissue edema from
smoke inhalation.
ļƒ˜N: NECK MOBILITY: Neck stabilization is
necessary in most trauma patients. It is important
to note that the risk of neurologic injury from
hypoxemia is much greater than the risk of spinal
injury due to neck extension during intubation.
Intubation :
ā€¢ Tracheal intubation of the injured patient is often
complicated by the need to maintain cervical
immobilization, the presence of obstructions
such as blood, vomitus, and debris, and possibly
by direct trauma to the airway.
ā€¢ Intubation improves oxygenation, thereby
helping to meet increased physiologic demands
ā€¢ Cricothyrotomy ā€” Clinicians who
manage trauma must be prepared to
perform a cricothyrotomy when
orotracheal intubation cannot be
accomplished
Contā€¦
ā€¢ Breathing and ventilation ā€” Once airway
patency is ensured, assess the adequacy of
oxygenation and ventilation.
ā€¢ Chest trauma accounts for 20 to 25 percent of
trauma-related deaths, in large part due to its
harmful effects on oxygenation and ventilation
2) Breathing and ventilation
3) CIRCULATION:
ā€¢ Recognition and management of hemorrhage ā€”
Once the airway and breathing are stabilized, perform
an initial evaluation of the patient's circulatory status by
palpating central pulses
ā€¢ While circulation is assessed, two large-bore (16 gauge
or larger) intravenous (IV) catheters are placed, most
often in the antecubital fossa of each arm
4) Disability assessment:
Once problems related to the airway,
breathing, and circulation are addressed,
perform a focused neurologic examination.
This should include a description of the
patientā€™s
ļ¶Level of consciousness using the
glass-cow coma scale (GCS) score,
ļ¶assessments of pupillary size and
reactivity,
ļ¶ Gross motor function,
ļ¶ sensation
Contā€¦
5) Exposure and environmental control:
ā€¢ Emergency department (ED) and
operating room (OR) temperatures of
at least 29.4Ā°C (85Ā°F) during the
treatment of these patients .
ā€¢ Rapidly remove wet clothing, make
liberal use of warm blankets and active
external warming devices, and warm IV
fluids and blood.
ā€¢ Hypothermia should be prevented if
possible and treated immediately once
identified.
Contā€¦
LEVEL OF TRAUMA CARE:
ā€¢ Level I: Provides every aspect of trauma care from
prevention through rehabilitation
ā€¢ Level II: Provides initial definitive care
ā€¢ Level III: Provides assessment, resuscitation,
emergency care, stabilization
ā€¢ Level IV: Provides advanced trauma life support prior to
transfer
TRIAGE:
ā€¢ Four common triage categories:
IMMEDIATE (RED)
DELAYED (YELLOW)
MINIMAL (GREEN)
EXPECTANT (BLACK)
INTRODUCTION:
ā€¢ A burn is a type of injury to skin, or other
tissues, caused by heat, cold, electricity,
chemicals, friction, or radiation.
Most burns are due to heat from hot liquids,
solids, or fire. Burns are generally
preventable. Treatment depends on the
severity of the burn
DEFINITION:
ā€¢ Damage to the skin or deeper tissues
caused by sun, hot liquids, fire, electricity
or chemicals.
OR
ā€¢ A burn is a type of injury to skin, or other
tissues, caused by heat, cold, electricity,
chemicals, friction, or radiation
ETIOLOGY OF BURN:
DRY HEAT CHEMICAL
FLAME
FROSTIBITE
ELECTRICAL
CONTACT
IONIZING
RADIATION
CLASSIFICATION OF BURN:
DEPENDING UPON THE
PERCENTAGE OF BURN:
1.MILD
2.MODERATE
3.SEVERE
DEPENDING UPON THE THIKNESS OF
SKIN INVOLVED :
1. FIRST DEGREE
2. SECOND DEGREE
3. THIRD DEGREE
4. FOURTH DEGREE
1.Depending on the percentage of burns:
ā€¢ MILD:
ā€¢ Partial thickness burns:
ļ±<15% in adults and
ļ±<10% In children
ā€¢ Full thickness burns:
ļ±<2% it Can be treated on outpatient
department
Contā€¦
ā€¢ MODERATE:
ā€¢ Second degree burn of 15-25% burns
ā€¢ Third degree burn between 2-10% burns
ā€¢ Burns which are not involving eyes, ears
Face, hand, feet and perineum
Contā€¦
ā€¢ SEVERE:
ā€¢ Second degree burns more then 25% in adults
and More then 20% in children
ā€¢ All third degree burns more then10%
ā€¢ All electrical burns and inhalation burns
ā€¢ Burns with fracture
ā€¢ Burns involving eyes, ears, feet, hands and
perineum
2.Depending on thickness of skin involved:
1ST First Degree(superficial) burn:
ā€¢ It affect only the outer layer of skin. The
epidermis
ā€¢ Epidermis looks red and painful
ā€¢ No blisters formation
ā€¢ Heals rapidly In 5-7 days by
epithelialization Without scarring
Contā€¦
2nd degree burns( Partial thickness)
ā€¢ It involve the epidermis and part of the lower
layer of skin , the dermis.
ā€¢ Affected area is red, mottled, painful
ā€¢ Blister formation
ā€¢ Heals in 14-21 days by epithelialization With
scaring
Contā€¦
3rd degree(full thickness) burns:
ā€¢ It destroy the epidermis and dermis.
ā€¢ They may go to the innermost layer of
skin, the subcutaneous tissue.
ā€¢ Affected area is painless and insensitive
with Thrombosis of superficial vessels
ā€¢ It requires grafting
Cont..
4th degree burns:
ā€¢ It involves underlying Tissues,
Muscles, bones
ā€¢ There is no feeling in the area since
the nerve ending are destroyed..
Lack son's thermal wound theory:
Zone of coagulationā€”
ļ‚§ This occurs at the point of maximum damage.
ļ‚§ In this zone there is irreversible tissue loss due to
coagulation of the constituent proteins.
Zone of stasisā€”
ļ‚§ The surrounding zone of stasis is characterised by decreased
tissue perfusion.
ļ‚§ The tissue in this zone is potentially salvageable.
Contā€¦
Zone of hyperemiaā€”
ļ‚§In this outermost zone tissue
perfusion is increased.
ļ‚§The tissue here will invariably
recover unless there is severe
sepsis or prolonged hypoperfusion.
Sign and symptoms:
ā€¢ Cardiovascular changesā€”
ā€¢ Capillary permeability is increased,
ā€¢ leading to loss of intravascular proteins and fluids into
the interstitial compartment.
ā€¢ Peripheral and splanchnic vasoconstriction occurs.
ā€¢ Myocardial contractility is decreased,
ā€¢ organ hypoperfusion.
ā€¢ Respiratory changesā€”
ā€¢ Inflammatory
mediators cause
bronchoconstriction,
ā€¢ in severe burns adult
respiratory distress
syndrome can occur.
Contā€¦
ā€¢ Metabolic changesā€”
ā€¢ The basal metabolic rate increases up
to three times its original rate.
ā€¢ This, coupled with splanchnic
hypoperfusion, necessitates early and
aggressive enteral feeding to decrease
catabolism and maintain gut integrity.
Cont..
ā€¢ Immunological changesā€”
ā€¢ Non-specific down regulation of
the immune response occurs,
ā€¢ affecting both cell mediated and
humoral pathways
Contā€¦
ā€¢ GASTROINTESTINAL SYSTEM:
ā€¢ mucosal atrophy
ā€¢ decreased absorption & increased intestinal
permeability
ā€¢ Increased bacterial translocation
ā€¢ Septicemia
ā€¢ Curling;s ulcer.
ā€¢ Abdominal compartment syndrome
Contā€¦
Renal system:
ā€¢ Diminished blood flow and cardiac output leads to
decreased renal blood flow and GFR
ā€¢ Toxins released from the wound along with sepsis causes
acute tubular necrosis.
ā€¢ Myoglobin released from muscles (in case of electric
injury or often from eschar) is most injurious to kidneys.
ā€¢ Earlier resuscitation decreases renal failure and
improves associated mortality
Pathophysiology
Heat
causes
coagulation necrosis of skin and subcutaneous tissue
Release of vasoactive peptides
Altered capillary permeability
Loss of fluid Severe hypovolaemia
Contā€¦
Decreased cardiac Decreased myocardial output
function
Decreased renal blood Oliguria flow (Renal failure)
Altered pulmonary resistance causing pulmonary edema
Infection
Systemic inflammatory response syndrome (SIRS)
Multiorgan dysfunction syndrome (MODS).
Assessment of burn:
ā€¢ 1) Wallace`s rules of nine
ā€¢ It is used for early assessment
2) The Lund and Browder chart:
ā€¢ Better method for assessing the burns wound.
ā€¢ Here each part of the body is individually assessed
3) Rule of palm:
ā€¢ Patientā€™s entire hand area is 1%.
ā€¢ Clean piece of paper is cut to
the size of hand and through that
percentage of burns is assessed.
Management of burn at home:
Management of burn:
ā€¢ Management of burn is depend upon the severity of burn:
ļ¶ Airway:
ļ¶Breathing: beware of inhalation and rapid airway compromise
ļ¶Circulation:
ļ¶Fluid replacement:
ļ¶Disability: compartment syndrome
ļ¶ Exposure: percentage area of burn.
1) Establishment of adequate airway:
ā€¢ Administer oxygen therapy.
ā€¢ Arterial blood gas analysis.
ā€¢ Endotracheal intubation is done.
ā€¢ Use of pressure ventilators.
ā€¢ Secretion from respiratory passage.
2) Fluids management:
ā€¢ Initiate fluids for ongoing resuscitation and fluid losses
using the Parkland formula with half of this total given
in the first 8 hours after injury
4ml of crystalloid solution (kg of body weight)
Ɨ(burn)=ml in first 24 hours
ā€¢ Example: In the case of a patient weighing 70ā€‰kg with a
50% TBSA burn, (4 Ɨ 70 Ɨ 50) = 14 000ā€‰mL needed in
the first 24 hours. Half is needed in the first 8 hours after
injury and remaining is giving after 16 hours.
ā€¢ Consensus formula:
ā€¢ RL (2-4) mlƗ kg body weight Ɨ% total
body surface area (TBSA burn)
ā€¢ First 8 hours- half solution
ā€¢ Next 16 hours ā€“ remaining half to be given.
Contā€¦
ā€¢ Even formula:
ā€¢ Colloids: 1ml Ɨ kg body weight Ɨ% TBSA burn
ā€¢ Electrolytes: 1ml Ɨ kg body weight Ɨ% TBSA burn
ā€¢ Glucose (5% in water):2000 ml for insensible loss
Contā€¦
ā€¢ Brooke army formula:
ā€¢ Collides: 0.5ml Ɨ kg body weight Ɨ% TBSA burn
ā€¢ Electrolytes (RL) 1.5ml Ɨ kg body weight Ɨ% TBSA
burn
ā€¢ Glucose (5% in water):2000 ml for insensible loss
Contā€¦
3) Pain and anxiety medications:
ā€¢ Penicillin prophylaxis
ā€¢ Healing burns can be incredibly painful
ā€¢ E.g. ā€“ Morphine sulphate
4) Burn creams and Ointments:
E.g. ā€“
ļƒ˜Bacitracin
ļƒ˜Silver sulfadiazine
ļƒ˜ Providine-iodine ointment
ļƒ˜ 0.5% silver nitrate solution
5) Dressing:
6) Tetanus shot:
ā€¢ If you have not had a tetanus shot in the past
five years and your burn is superficial partial-
thickness or deeper, you need a tetanus
booster vaccine
Surgical treatment:
ā€¢ Skin grafts ā€“ a skin grafts is a
surgical procedure in which a
sections of your own healthy skin
are used to replace the scar tissue
caused by deep burns .
ā€¢ Donor skin from deceased donors or
pigs can be used as a temporary
solution .
Plastic surgery:
ā€¢ Plastic surgery(reconstruction)
can improve the appearance of
burn scars and increase the
flexibility of joints affected by
scarring.
Causes of death:
ā€¢ Hypovolaemia (refractory and uncontrolled) and shock
ā€¢ Renal failure
ā€¢ Pulmonary oedema and ARDS
ā€¢ Septicaemia
ā€¢ Multiorgan failure
ā€¢ Acute airway block in head and neck burns
POISONING
INTRODUCTION:
ā€¢ Poisoning is when a person is exposed to a substance that
can damage their health or endanger their life. In 2013-14,
almost 150,000 people were admitted to hospital with
poisoning in England. Most cases of poisoning happen at
home and children under five have the highest risk of
accidental poisoning.
ā€¢ In around one in four reported cases, the person
intentionally poisoned themselves as a deliberate act
of Self-harm .
Definition of Poisoning:
ā€¢ Poisoning is a condition or a process in which
an organism becomes chemically harmed severely (poisoned) by
a toxic substance or venom of an animal.
OR
ā€¢ A Poison is any chemical that harms the body. It can be:
ļ‚§ Accidental
ļ‚§ Occupational
ļ‚§ Recreational
ļ‚§ Intentional(killing)
ļ‚§ Natural or manufactured toxins
Mode of absorption:
ā€¢ Ingestion
ā€¢ Inhalation
ā€¢ Injection
ā€¢ Splashing in to the eye
ā€¢ Absorbed through the skin (inuction)
ā€¢ Insufflation ( the act of blowing gas or a
powder in to a body cavity)
Route of poisoning:
ā€¢ Inhalation
ā€¢ Absorption
ā€¢ Injections
ā€¢ ingestion
TYPES OF POISIOING:
1) ACIDS
2) ALKALIES
3) MEDICATION
4) METAL POISIONING
5) ORGANOPHOSPHOROUS POISONING
6) PETROLEUM PRODUCTS
7) OILS
1)ACIDS:
ļ¶NITRIC ACIDS
ļ¶H2SO4
ļ¶HCL
ļ¶CARBOLIC ACID
ļ¶ACETIC ACID
2)ALKALIES:
ļ¶DRAIN CLEARNERS
ļ¶DISHWASHING:
-DETERGENTS
-AMMONIA
ļ¶BLEACHERS
3)MEDICATION:
ļ¶ASPIRIN & ASPIRIN CONTAINING
MEDICATIONS
ļ¶NSAIDS
ļ¶HALLUCINOGEN
ļ¶BARBITURATES
ļ¶ALCOHOL
4)METAL POISONING:
ļ¶IRON
ļ¶COPPER
ļ¶CYANIDE
ļ¶LEAD
ļ¶ETHYLENE GLYCOL
LEAD POISONING:
5)OP POISONING:
ļ¶INSECTICIDES NAPHALENE
ļ¶PESTICIDES
ļ¶OPIUM
ļ¶CASTOR OIL
ļ¶MUSHROOM
ļ¶TOBACCO
ļ¶CANNABIS
ORGANOPHOSPHATE & CARBAMATE POISIONG:
ā€¢ It is state of Acetylcholine excess
ā€¢ It is a combination of:
ļ¶Muscarinic receptor
ļ¶Nicotinic receptor
ļ¶Nicotinic receptor
ļ¶CNS(unspecified)
CLINICAL MANIFESTATION:
ļ¶Muscarinic effects
ļ¶Nicotinic effects
ļ¶General symptoms
Muscarinic effects ( antidote: atropine)
ā€¢ Miosis
ā€¢ Salivation
ā€¢ Bradycardia
ā€¢ Diarrhea
ā€¢ Sweating
ā€¢ pulmonary edema
ā€¢ hypersecretion
Nicotinic effects:
ā€¢ Fasciculation ( brief spontaneous
contraction of a few muscle fibers)
ā€¢ Muscle paralysis
ā€¢ CNS symptoms
ā€¢ Coma
ā€¢ Bradycardia
ā€¢ Hypotension
ā€¢ Respiratory depression
General sign and symptoms of poisoning:
ā€¢ The symptoms of poisoning will depends on the type
of poison and the amount taken in, but general things
to look out for include:
ļ¶vomiting
ļ¶stomach pains
ļ¶confusion
ļ¶drowsiness and fainting fits
ā€¢ Convulsions
ā€¢ Crepitations in the chest for evidence of
aspiration ( soft-fine crackling sound heard in
the lungs through the stethoscope)
ā€¢ Hypotension
ā€¢ Bradycardia
ā€¢ bradypnoea
Contā€¦
Diagnosis:
ā€¢ History taking
ā€¢ Physical examination
ā€¢ Respiration and cyanosis
ā€¢ Condition of skin
ā€¢ Size of the pupil- small in OP Poisoning and
large in cocaine
ā€¢ Small near the patient and gastric content
Management:
ā€¢ Catherization, RT, establish patent airway
ā€¢ IV line vascular access, gastric lavage
ā€¢ Administration of activated charcoal orally
or via gastric tube within 60 minutes of
poison ingestion.
ā€¢ Many toxins adhere to charcoal and
excreted through GI tract rather than
absorption in to the circulation
Skin and outer decontamination:
ā€¢ It involves the removal of toxins from eyes and skin
using copious amount of water or saline
ā€¢ Dry substances should be brushed from the skin.
ā€¢ Remove the clothes before water is used.
ā€¢ ((Personal protective devices should be used like
goggles gowns respirators etc.)
Bowel management:
ā€¢ Cathartics (Sorbitol) are given
together with the first dose of charcoal
to stimulate intestinal motility.
ā€¢ Whole bowel irrigation involves the
administration of bowel evacuant
solution(eg: golytely).
ā€¢ This solution is administered every 4-5
hrs. until stool are clear.
ā€¢ It is effective for swallowed objects foe
cocaine filled balloon or condoms, and
heavy metals such as lead and mercury
ANTIDOTES
List of antidots:
ļƒ¼ Poison ļƒ¼ Antidotes
ļ¶ Organophosphate ļ¶ Atropine
ļ¶ Cyanide ļ¶ Amyl nitrite
ļ¶ Digoxin ļ¶ Anti digoxin
ļ¶ Paracetamol ļ¶ Acetylcysteine
ļ¶ Irritant gases ļ¶ Budesonide
ļ¶ Lead ļ¶ Calcium disodium edetate
ļ¶ Benzodiazepine ļ¶ flumazenil
ļ¶ Methanol ļ¶ Ethanole
Contā€¦
ļƒ¼ Poison ļƒ¼ Antidotes
ļ¶ insulin ļ¶ Glucose
ļ¶ Opioids analgesics ļ¶ naloxone
ļ¶ Carbon monoxide ļ¶ Oxygen
ļ¶ copper ļ¶ Penicillamine
ļ¶ Warfarin ļ¶ Clotting factors
ļ¶ Isoniazid ļ¶ Vita 6
ļ¶ Caffeine ļ¶ Esmolol
ļ¶ penicillin ļ¶ Epinephrine
ļƒ¼ Poison ļƒ¼ Antidotes
ļ¶ Curare ļ¶ Tensilon
ļ¶ Vincristine ļ¶ Hyaluronidase
ļ¶ Mistinon ļ¶ Atropine sulphate
ļ¶ Meyaproterenol ļ¶ Esmolol
ļ¶ Narcotics ļ¶ Naloxone
ļ¶ Chlorates ļ¶ Methylene blue
ļ¶ Thallium ļ¶ Prussian blue
ļ¶ Formaldehyde ļ¶ Folic acid
Contā€¦
Promotion of poison excretion:
ā€¢ Increase fluid intake
ā€¢ Hemodialysis
ā€¢ Hemoperfusion
ā€¢ Peritoneal diaylsis
Supportive therapy:
Nurses responsibility:
ā€¢ Inform police
ā€¢ Report MLC
ā€¢ Collect information from the surrounding
persons
ā€¢ Preserve the suspending material like bottle
containing pills or liquid
ā€¢ Preserve the vomited material.
REHABILITATION
INTRODUCTION:
ā€¢ Rehabilitation is care that can help you get back,
keep, or improve abilities that you need for daily
life. These abilities may be physical, mental, and/or
cognitive (thinking and learning). You may have
lost them because of a disease or injury, or as a side
effect from a medical treatment. Rehabilitation can
improve your daily life and functioning.
DEFINITION
ā€¢ Rehabilitation is ā€œa set of
measures that assist individuals
who experience, or are likely to
experience, disability to achieve
and maintain optimal functioning
in interaction with their
environmentsā€
TYPES OF REHABILITAION:
There are many type of rehabilitation. In poison depend upon which organ is affected
according to that we provide rehabilitation to the patient:
Neurological rehabilitation
Stroke rehabilitation
Cardiac Rehabilitation
Drug rehabilitation
Alcohol rehabilitation
CONTā€¦
Medical rehabilitation
Physical rehabilitation
Vocational rehabilitation
Vestibular rehabilitation
NEED REHABILATION:
ā€¢ Injuries and trauma: burns, fractures (broken bones), traumatic
brain injury, and spinal cord injuries , poison .
ā€¢ Stroke
ā€¢ Severe infections
ā€¢ Major surgery
ā€¢ Side effects from medical treatments, such as
from cancer treatments
ā€¢ Certain birth defects and genetic disorders
ā€¢ Developmental disabilities
ā€¢ Chronic pain, including back and neck pain
CONTā€¦
FELLOW-UP:
The follow-up clinic or cardiac rehab appointment is
successful if:
ā€¢ Patient arrives at appointment within 7 days of
discharge from hospital.
ā€¢ Discharge summary (including summary of
hospitalization, updated medication list) available to
follow-up provider.
ā€¢ Patient brings his/her medications or a medication list to
clinic visit.
ļ¶Reason for referral available to cardiac rehab
center and patient brings referral letter or
provider prescription
ļ¶Rates of physician follow-up within 1 week of
discharge were low and varied substantially
across hospitals.
CONTā€¦
ļ¶Patients discharged from hospitals with more
consistent early follow-up with 7 days have
lower risk of 30-day readmission.
ļ¶Enhanced transition planning and ensuring
that patients are evaluated within a week of
discharge represents an achievable target for
hospital quality improvement.
CONTā€¦
Conclusion:
ā€¢ I conclude that emergency is a situation
which can be occur anywhere at any time
which can cause severe trauma to the
patient and their family member.
immediate treatment should be required
otherwise it will lead to a death.
Summarization:
Today we discussed about:
ļ¶Emergency
ļ¶MODS
ļ¶DIC
ļ¶Trauma
ļ¶Burn
ļ¶Poising
ļ¶Conclusion
ļ¶Recapitalization
ļ¶Bibliography
Recapitalization:
1) Define the common emergency ?
2) Define the classification of burn?
3) Define the antidot of paracetamol?
BIBLIOGRAPHY:
ā€¢ Suddharthā€™s & brunner Textbook of medical surgical nursing published by Wolters
Kluwer edition south Asian page no. 1250-1255
ā€¢ Black M. joyce Textbook of medical surgical nursing published by elsvier edition 1st page
no.. 1780-1800
ā€¢ www.slideshare.net ā€ŗ shisha_sk ā€ŗ burn-ppt-Shashi Viewed on 02/04.2020
ā€¢ www.slideshare.net ā€ŗ AseemBadarudeen ā€ŗ poisoning-49486135 viewed on 3/04/2020
ā€¢ www.slideshare.net ā€ŗ haifa12 ā€ŗ rehabilitation-54792687 viewed on 5/04/2020
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Disseminated intravascular coagulation

  • 1. PRESENTATION ON EMERGENCY, TRAUMA AND MULTI-SYSTEM ORGAN FAILURE Presented to: Dr. Pallavi Pathania Associate Professor Medical Surgical Nursing Shimla Nursing College Presented by: Reena Sharma M.sc.(Nursing) 1st year Shimla Nursing College Annandale, Shimla
  • 2. INDEX SR. NO. CONTENT 1. Introduction 2. DIC 3. Trauma 4. Burn 5. Poising 6. Conclusion 7. Summarization 8 Recapitalization 9 Assignment 10 Bibliography
  • 3. INTRODUCTION: ā€¢ Emergencies can happen anywhere at any time. Small-scale incidents occur frequently and are dealt with effectively by the emergency services. A Trauma service provides care for people who have sustained physical injuries. These injuries are often the result of an accident but can be sustained in other circumstances.
  • 4. CONTā€¦ MODS(MULTI-ORGAN DYSFUNCTIONING SYNDROME) It is progressive dysfunction of two or more major organ systems in a critically ill patient that makes it impossible to maintain homeostasis without medical intervention and that is typically a complication of sepsis and is a major factor in predicting mortality .
  • 5. Or It is also known as multiple organ failure (MOF), total organ failure (TOF) or multisystem organ failure (MSOF), is altered organ function in an acutely ill patient requiring medical intervention to achieve homeostasis Contā€¦
  • 6. DEFINITIONS: EMERGENCY: something dangerous or serious, such as an accident, that happens suddenly or unexpectedly and needs fast action in order to avoid harmful results. OR ā€¢ An emergency is a situation that poses an immediate risk to health, life, property, or environment.
  • 7. MOST COMMON MEDICAL EMERGENCIES ā€¢ Bleeding ā€¢ Trauma ā€¢ Burn ā€¢ Poising ā€¢ Breathing difficulties
  • 8. Contā€¦ ā€¢ Someone collapses ā€¢ Fit and/or epileptic seizure ā€¢ Severe pain ā€¢ Heart attack ā€¢ A stroke
  • 10. INTRODUCTION ā€¢ In disseminated intravascular coagulation, abnormal clumps of thickened blood (clots) form inside blood vessels. These abnormal clots use up the blood's clotting factors, which can lead to massive bleeding in other places. Causes include inflammation, infection and cancer.
  • 11. DEFINITION: ā€¢ Disseminated intravascular coagulation (DIC) is a serious disorder in which the proteins that control blood clotting become overactive.
  • 12. OR ā€¢ Disseminated intravascular coagulation (DIC) is a condition in which blood clots form throughout the body, blocking small blood vessels. Contā€¦
  • 13. RISK FACTORS: ā€¢ Blood transfusion reaction ā€¢ Cancer, especially certain types of leukemia ā€¢ Inflammation of the pancreas (pancreatitis) ā€¢ Infection in the blood, especially by bacteria or fungus
  • 14. ā€¢ Liver disease ā€¢ Pregnancy complications (such as placenta that is left behind after delivery) Contā€¦
  • 15. CLASSIFICATION ACUTE ā€¢ It happened rapidly, the coagulopathy is dominant and major symptoms are bleeding and shock, mainly seen in severe infection, amniotic fluid embolism CHRONIC ā€¢ it happened slowly and last several weeks, thrombosis and clotting may predominate ā€¢ mainly seen in cancer.
  • 17. Clinical findings ļ¶Bleeding ļ¶Bruising ļ¶Deep vein thrombosis ļ¶Purpura ļ¶Petechiae ļ¶Oliguria ļ¶Acute renal failure
  • 19. DIAGNOSTIC EVALUATION: ā€¢ History taking ā€¢ Physical examination
  • 20. LABORATORY TEST FINDINGS Platelet count Markedly decreased Prothrombin time Increased Activated partial thromboplastin time Increased Fibrin degradation products Markedly Increased Fibrinogen Normal or decreased D-dimer Markedly increased Contā€¦
  • 21. TREATMENT Replacement therapy Other Treatment Heparin therapy Treatment of the underlying disorder
  • 22. Treatment of the underlying disorder: ā€¢ Avoid of either acute or chronic DIC. ā€¢ Avoid delay treat vigorously (e.g. shock, delay treat vigorously (e.g., shock, sepsis, obstetrical problems Contā€¦
  • 23. Contā€¦ Replacement therapy: ā€¢ Coagulation factor deficiency require replacement with FFP (fresh frozen plasma). ā€¢ Platelet transfusion should be used to maintain a platelet count greater than 30000/Ī¼l, and 50000/Ī¼l.
  • 24. Contā€¦. Heparin therapy: ā€¢ In some cases heparin therapy is contraindicated, but when DIC is producing serious clinical consequences Dose:500~750u/h is necessary. ā€¢ Heparin therapy must be used in combination with replacement therapy, it can lead to severe bleeding
  • 25. Contā€¦. Other treatment: ā€¢ Aminocaproic acid, 1g/h iv Aminocaproic acid, 1g/h iv ā€¢ Tranexamic acid, 10mg/kg, iv,q8h, Aminocaproic acid can never be used without heparin in DIC because of the risk of thrombosis
  • 26. ACUTE DIC: Without bleeding or evidence of ischemia: No treatment With bleeding : ā€¢ Blood components as needed ā€¢ Fresh frozen plasma ā€¢ Cryoprecipitate ā€¢ Platelet transfusions With ischemia: Anticoagulants after bleeding risk is corrected with blood products
  • 27. Chronic DIC: Without thromboembolism :No specific therapy needed But prophylactic drugs: eg, (low dose heparin, low molecular weight heparin) May be used for patients at high risk of thrombosis.
  • 28. CONT.. With thromboembolism :Heparin or low-molecular- weight heparin, trial of warfarin sodium (Coumadin). (If warfarin is unsuccessful, long-term use of low ā€“ molecular weight heparin may be helpful.)
  • 29. COMPLICATION OF DIC: ļ¶Severe bleeding ļ¶Stroke ļ¶Ischemia of extremities or organs ļ¶Waterhouse-Friderichse syndrome ļ¶Sheehanā€™s syndrome ļ¶Kasabach-merritt syndrome
  • 31. TRAUMA DEFINITION: Trauma is the response to a deeply distressing or disturbing event that overwhelms an individualā€™s ability to cope, causes feelings of helplessness, diminishes their sense of self and their ability to feel the full range of emotions and experiences.
  • 32. OR Severe emotional shock and pain caused by an extremely upsetting experience. Contā€¦
  • 33. TYPES OF TRAUMA : ā€¢ BLUNT TRAUMA ā€¢ PENETRATING TRAUMA ā€¢ DECELERATION INJURIES ā€¢ EXTERNAL FORCE INJURIES
  • 34. ETIOLOGY OF TRAUMA : UN-HELMETED MOTORCYCLES OR BICYCLE CRASH MOTOR VEHICLE CRASH FALL ASSULT
  • 35. PATHOPHYSIOLOGY: All trauma leads to Decreased organ perfusion, Cellular ischemia, Edema and inflammation. Once begun, Inflammation becomes a disease process independent of its origin, Multiple organ failure and Death even after a patient has been completely resuscitated.
  • 36. CLINICAL MANIFESTATION: ā€¢ Fractures ā€¢ Contusions ā€¢ Open wound ā€¢ Vomiting or nausea ā€¢ Dizziness ā€¢ Edema
  • 37. Cont.. ā€¢ Increased heart rate ā€¢ Low blood pressure ā€¢ Fever ā€¢ Disorientation or confusion ā€¢ Loss of consciousness ā€¢ Feeling of coldness as temperature drops ā€¢ Increased metabolism
  • 38. Diagnostic evaluation: ā€¢ History taking ā€¢ Physical examination ā€¢ X-ray
  • 39. ā€¢ CT Scan ā€¢ Ultrasonography ā€¢ Echocardiography ā€¢ MRI Scan Contā€¦
  • 40. MANAGEMENET: The primary survey consists of the following steps: ā—Airway assessment and protection (maintain cervical spine stabilization when appropriate) ā—Breathing and ventilation assessment (maintain adequate oxygenation) ā—Circulation assessment (control hemorrhage and maintain adequate end-organ perfusion)
  • 41. ā—Disability assessment (perform basic neurologic evaluation) ā—Exposure, with environmental control (undress patient and search everywhere for possible injury, while preventing hypothermia) Contā€¦
  • 42. 1) AIRWAY: ā€¢ Severely injured patients can develop airway obstruction or inadequate ventilation leading to hypoxia and death within minutes. ā€¢ Assessment ā€” In a conscious patient, initial airway assessment can be performed as fellow: ā€¢ Inspect and palpate the anterior neck for lacerations, hemorrhage, crepitus, swelling, or other signs of injury.
  • 43. ASSESSMENT IS DONE BY LEMON mnemonic ļƒ˜L: LOOK: Facial and neck injuries can distort external and internal structures making it difficult to visualize the glottis or insert an endotracheal tube. ļƒ˜E: EVALUATE : This refers to the intraoral, mandibular, and hyoid-to-thyroid notch distances. Contā€¦
  • 44. Cont.. ā€¢ M: MALLAMPATI: A standard calculation of the mallampati score cannot be performed in many trauma patients; injured patients requiring emergency intubation often cannot open their mouths spontaneously
  • 45. CONT.. ļƒ˜O: OBSTRUCTION/OBESITY: Any number of injuries can obstruct the airway including internal or external hematomas or soft tissue edema from smoke inhalation. ļƒ˜N: NECK MOBILITY: Neck stabilization is necessary in most trauma patients. It is important to note that the risk of neurologic injury from hypoxemia is much greater than the risk of spinal injury due to neck extension during intubation.
  • 46. Intubation : ā€¢ Tracheal intubation of the injured patient is often complicated by the need to maintain cervical immobilization, the presence of obstructions such as blood, vomitus, and debris, and possibly by direct trauma to the airway. ā€¢ Intubation improves oxygenation, thereby helping to meet increased physiologic demands
  • 47. ā€¢ Cricothyrotomy ā€” Clinicians who manage trauma must be prepared to perform a cricothyrotomy when orotracheal intubation cannot be accomplished Contā€¦
  • 48. ā€¢ Breathing and ventilation ā€” Once airway patency is ensured, assess the adequacy of oxygenation and ventilation. ā€¢ Chest trauma accounts for 20 to 25 percent of trauma-related deaths, in large part due to its harmful effects on oxygenation and ventilation 2) Breathing and ventilation
  • 49. 3) CIRCULATION: ā€¢ Recognition and management of hemorrhage ā€” Once the airway and breathing are stabilized, perform an initial evaluation of the patient's circulatory status by palpating central pulses ā€¢ While circulation is assessed, two large-bore (16 gauge or larger) intravenous (IV) catheters are placed, most often in the antecubital fossa of each arm
  • 50. 4) Disability assessment: Once problems related to the airway, breathing, and circulation are addressed, perform a focused neurologic examination. This should include a description of the patientā€™s ļ¶Level of consciousness using the glass-cow coma scale (GCS) score,
  • 51. ļ¶assessments of pupillary size and reactivity, ļ¶ Gross motor function, ļ¶ sensation Contā€¦
  • 52. 5) Exposure and environmental control: ā€¢ Emergency department (ED) and operating room (OR) temperatures of at least 29.4Ā°C (85Ā°F) during the treatment of these patients .
  • 53. ā€¢ Rapidly remove wet clothing, make liberal use of warm blankets and active external warming devices, and warm IV fluids and blood. ā€¢ Hypothermia should be prevented if possible and treated immediately once identified. Contā€¦
  • 54. LEVEL OF TRAUMA CARE: ā€¢ Level I: Provides every aspect of trauma care from prevention through rehabilitation ā€¢ Level II: Provides initial definitive care ā€¢ Level III: Provides assessment, resuscitation, emergency care, stabilization ā€¢ Level IV: Provides advanced trauma life support prior to transfer
  • 55. TRIAGE: ā€¢ Four common triage categories: IMMEDIATE (RED) DELAYED (YELLOW) MINIMAL (GREEN) EXPECTANT (BLACK)
  • 56.
  • 57. INTRODUCTION: ā€¢ A burn is a type of injury to skin, or other tissues, caused by heat, cold, electricity, chemicals, friction, or radiation. Most burns are due to heat from hot liquids, solids, or fire. Burns are generally preventable. Treatment depends on the severity of the burn
  • 58. DEFINITION: ā€¢ Damage to the skin or deeper tissues caused by sun, hot liquids, fire, electricity or chemicals. OR ā€¢ A burn is a type of injury to skin, or other tissues, caused by heat, cold, electricity, chemicals, friction, or radiation
  • 59. ETIOLOGY OF BURN: DRY HEAT CHEMICAL FLAME FROSTIBITE ELECTRICAL CONTACT IONIZING RADIATION
  • 60. CLASSIFICATION OF BURN: DEPENDING UPON THE PERCENTAGE OF BURN: 1.MILD 2.MODERATE 3.SEVERE DEPENDING UPON THE THIKNESS OF SKIN INVOLVED : 1. FIRST DEGREE 2. SECOND DEGREE 3. THIRD DEGREE 4. FOURTH DEGREE
  • 61. 1.Depending on the percentage of burns: ā€¢ MILD: ā€¢ Partial thickness burns: ļ±<15% in adults and ļ±<10% In children ā€¢ Full thickness burns: ļ±<2% it Can be treated on outpatient department
  • 62. Contā€¦ ā€¢ MODERATE: ā€¢ Second degree burn of 15-25% burns ā€¢ Third degree burn between 2-10% burns ā€¢ Burns which are not involving eyes, ears Face, hand, feet and perineum
  • 63. Contā€¦ ā€¢ SEVERE: ā€¢ Second degree burns more then 25% in adults and More then 20% in children ā€¢ All third degree burns more then10% ā€¢ All electrical burns and inhalation burns ā€¢ Burns with fracture ā€¢ Burns involving eyes, ears, feet, hands and perineum
  • 64. 2.Depending on thickness of skin involved: 1ST First Degree(superficial) burn: ā€¢ It affect only the outer layer of skin. The epidermis ā€¢ Epidermis looks red and painful ā€¢ No blisters formation ā€¢ Heals rapidly In 5-7 days by epithelialization Without scarring
  • 65. Contā€¦ 2nd degree burns( Partial thickness) ā€¢ It involve the epidermis and part of the lower layer of skin , the dermis. ā€¢ Affected area is red, mottled, painful ā€¢ Blister formation ā€¢ Heals in 14-21 days by epithelialization With scaring
  • 66. Contā€¦ 3rd degree(full thickness) burns: ā€¢ It destroy the epidermis and dermis. ā€¢ They may go to the innermost layer of skin, the subcutaneous tissue. ā€¢ Affected area is painless and insensitive with Thrombosis of superficial vessels ā€¢ It requires grafting
  • 67. Cont.. 4th degree burns: ā€¢ It involves underlying Tissues, Muscles, bones ā€¢ There is no feeling in the area since the nerve ending are destroyed..
  • 68.
  • 69. Lack son's thermal wound theory: Zone of coagulationā€” ļ‚§ This occurs at the point of maximum damage. ļ‚§ In this zone there is irreversible tissue loss due to coagulation of the constituent proteins. Zone of stasisā€” ļ‚§ The surrounding zone of stasis is characterised by decreased tissue perfusion. ļ‚§ The tissue in this zone is potentially salvageable.
  • 70. Contā€¦ Zone of hyperemiaā€” ļ‚§In this outermost zone tissue perfusion is increased. ļ‚§The tissue here will invariably recover unless there is severe sepsis or prolonged hypoperfusion.
  • 71. Sign and symptoms: ā€¢ Cardiovascular changesā€” ā€¢ Capillary permeability is increased, ā€¢ leading to loss of intravascular proteins and fluids into the interstitial compartment. ā€¢ Peripheral and splanchnic vasoconstriction occurs. ā€¢ Myocardial contractility is decreased, ā€¢ organ hypoperfusion.
  • 72. ā€¢ Respiratory changesā€” ā€¢ Inflammatory mediators cause bronchoconstriction, ā€¢ in severe burns adult respiratory distress syndrome can occur.
  • 73. Contā€¦ ā€¢ Metabolic changesā€” ā€¢ The basal metabolic rate increases up to three times its original rate. ā€¢ This, coupled with splanchnic hypoperfusion, necessitates early and aggressive enteral feeding to decrease catabolism and maintain gut integrity.
  • 74. Cont.. ā€¢ Immunological changesā€” ā€¢ Non-specific down regulation of the immune response occurs, ā€¢ affecting both cell mediated and humoral pathways
  • 75. Contā€¦ ā€¢ GASTROINTESTINAL SYSTEM: ā€¢ mucosal atrophy ā€¢ decreased absorption & increased intestinal permeability ā€¢ Increased bacterial translocation ā€¢ Septicemia ā€¢ Curling;s ulcer. ā€¢ Abdominal compartment syndrome
  • 76. Contā€¦ Renal system: ā€¢ Diminished blood flow and cardiac output leads to decreased renal blood flow and GFR ā€¢ Toxins released from the wound along with sepsis causes acute tubular necrosis. ā€¢ Myoglobin released from muscles (in case of electric injury or often from eschar) is most injurious to kidneys. ā€¢ Earlier resuscitation decreases renal failure and improves associated mortality
  • 77. Pathophysiology Heat causes coagulation necrosis of skin and subcutaneous tissue Release of vasoactive peptides Altered capillary permeability Loss of fluid Severe hypovolaemia
  • 78. Contā€¦ Decreased cardiac Decreased myocardial output function Decreased renal blood Oliguria flow (Renal failure) Altered pulmonary resistance causing pulmonary edema Infection Systemic inflammatory response syndrome (SIRS) Multiorgan dysfunction syndrome (MODS).
  • 79. Assessment of burn: ā€¢ 1) Wallace`s rules of nine ā€¢ It is used for early assessment
  • 80. 2) The Lund and Browder chart: ā€¢ Better method for assessing the burns wound. ā€¢ Here each part of the body is individually assessed
  • 81. 3) Rule of palm: ā€¢ Patientā€™s entire hand area is 1%. ā€¢ Clean piece of paper is cut to the size of hand and through that percentage of burns is assessed.
  • 82. Management of burn at home:
  • 83. Management of burn: ā€¢ Management of burn is depend upon the severity of burn: ļ¶ Airway: ļ¶Breathing: beware of inhalation and rapid airway compromise ļ¶Circulation: ļ¶Fluid replacement: ļ¶Disability: compartment syndrome ļ¶ Exposure: percentage area of burn.
  • 84. 1) Establishment of adequate airway: ā€¢ Administer oxygen therapy. ā€¢ Arterial blood gas analysis. ā€¢ Endotracheal intubation is done. ā€¢ Use of pressure ventilators. ā€¢ Secretion from respiratory passage.
  • 85. 2) Fluids management: ā€¢ Initiate fluids for ongoing resuscitation and fluid losses using the Parkland formula with half of this total given in the first 8 hours after injury 4ml of crystalloid solution (kg of body weight) Ɨ(burn)=ml in first 24 hours ā€¢ Example: In the case of a patient weighing 70ā€‰kg with a 50% TBSA burn, (4 Ɨ 70 Ɨ 50) = 14 000ā€‰mL needed in the first 24 hours. Half is needed in the first 8 hours after injury and remaining is giving after 16 hours.
  • 86. ā€¢ Consensus formula: ā€¢ RL (2-4) mlƗ kg body weight Ɨ% total body surface area (TBSA burn) ā€¢ First 8 hours- half solution ā€¢ Next 16 hours ā€“ remaining half to be given. Contā€¦
  • 87. ā€¢ Even formula: ā€¢ Colloids: 1ml Ɨ kg body weight Ɨ% TBSA burn ā€¢ Electrolytes: 1ml Ɨ kg body weight Ɨ% TBSA burn ā€¢ Glucose (5% in water):2000 ml for insensible loss Contā€¦
  • 88. ā€¢ Brooke army formula: ā€¢ Collides: 0.5ml Ɨ kg body weight Ɨ% TBSA burn ā€¢ Electrolytes (RL) 1.5ml Ɨ kg body weight Ɨ% TBSA burn ā€¢ Glucose (5% in water):2000 ml for insensible loss Contā€¦
  • 89. 3) Pain and anxiety medications: ā€¢ Penicillin prophylaxis ā€¢ Healing burns can be incredibly painful ā€¢ E.g. ā€“ Morphine sulphate
  • 90. 4) Burn creams and Ointments: E.g. ā€“ ļƒ˜Bacitracin ļƒ˜Silver sulfadiazine ļƒ˜ Providine-iodine ointment ļƒ˜ 0.5% silver nitrate solution
  • 92. 6) Tetanus shot: ā€¢ If you have not had a tetanus shot in the past five years and your burn is superficial partial- thickness or deeper, you need a tetanus booster vaccine
  • 93. Surgical treatment: ā€¢ Skin grafts ā€“ a skin grafts is a surgical procedure in which a sections of your own healthy skin are used to replace the scar tissue caused by deep burns . ā€¢ Donor skin from deceased donors or pigs can be used as a temporary solution .
  • 94. Plastic surgery: ā€¢ Plastic surgery(reconstruction) can improve the appearance of burn scars and increase the flexibility of joints affected by scarring.
  • 95. Causes of death: ā€¢ Hypovolaemia (refractory and uncontrolled) and shock ā€¢ Renal failure ā€¢ Pulmonary oedema and ARDS ā€¢ Septicaemia ā€¢ Multiorgan failure ā€¢ Acute airway block in head and neck burns
  • 97. INTRODUCTION: ā€¢ Poisoning is when a person is exposed to a substance that can damage their health or endanger their life. In 2013-14, almost 150,000 people were admitted to hospital with poisoning in England. Most cases of poisoning happen at home and children under five have the highest risk of accidental poisoning. ā€¢ In around one in four reported cases, the person intentionally poisoned themselves as a deliberate act of Self-harm .
  • 98. Definition of Poisoning: ā€¢ Poisoning is a condition or a process in which an organism becomes chemically harmed severely (poisoned) by a toxic substance or venom of an animal. OR ā€¢ A Poison is any chemical that harms the body. It can be: ļ‚§ Accidental ļ‚§ Occupational ļ‚§ Recreational ļ‚§ Intentional(killing) ļ‚§ Natural or manufactured toxins
  • 99. Mode of absorption: ā€¢ Ingestion ā€¢ Inhalation ā€¢ Injection ā€¢ Splashing in to the eye ā€¢ Absorbed through the skin (inuction) ā€¢ Insufflation ( the act of blowing gas or a powder in to a body cavity)
  • 100. Route of poisoning: ā€¢ Inhalation ā€¢ Absorption ā€¢ Injections ā€¢ ingestion
  • 101. TYPES OF POISIOING: 1) ACIDS 2) ALKALIES 3) MEDICATION 4) METAL POISIONING 5) ORGANOPHOSPHOROUS POISONING 6) PETROLEUM PRODUCTS 7) OILS
  • 104. 3)MEDICATION: ļ¶ASPIRIN & ASPIRIN CONTAINING MEDICATIONS ļ¶NSAIDS ļ¶HALLUCINOGEN ļ¶BARBITURATES ļ¶ALCOHOL
  • 108. ORGANOPHOSPHATE & CARBAMATE POISIONG: ā€¢ It is state of Acetylcholine excess ā€¢ It is a combination of: ļ¶Muscarinic receptor ļ¶Nicotinic receptor ļ¶Nicotinic receptor ļ¶CNS(unspecified)
  • 110. Muscarinic effects ( antidote: atropine) ā€¢ Miosis ā€¢ Salivation ā€¢ Bradycardia ā€¢ Diarrhea ā€¢ Sweating ā€¢ pulmonary edema ā€¢ hypersecretion
  • 111. Nicotinic effects: ā€¢ Fasciculation ( brief spontaneous contraction of a few muscle fibers) ā€¢ Muscle paralysis ā€¢ CNS symptoms ā€¢ Coma ā€¢ Bradycardia ā€¢ Hypotension ā€¢ Respiratory depression
  • 112. General sign and symptoms of poisoning: ā€¢ The symptoms of poisoning will depends on the type of poison and the amount taken in, but general things to look out for include: ļ¶vomiting ļ¶stomach pains ļ¶confusion ļ¶drowsiness and fainting fits
  • 113. ā€¢ Convulsions ā€¢ Crepitations in the chest for evidence of aspiration ( soft-fine crackling sound heard in the lungs through the stethoscope) ā€¢ Hypotension ā€¢ Bradycardia ā€¢ bradypnoea Contā€¦
  • 114. Diagnosis: ā€¢ History taking ā€¢ Physical examination ā€¢ Respiration and cyanosis ā€¢ Condition of skin ā€¢ Size of the pupil- small in OP Poisoning and large in cocaine ā€¢ Small near the patient and gastric content
  • 115. Management: ā€¢ Catherization, RT, establish patent airway ā€¢ IV line vascular access, gastric lavage ā€¢ Administration of activated charcoal orally or via gastric tube within 60 minutes of poison ingestion. ā€¢ Many toxins adhere to charcoal and excreted through GI tract rather than absorption in to the circulation
  • 116. Skin and outer decontamination: ā€¢ It involves the removal of toxins from eyes and skin using copious amount of water or saline ā€¢ Dry substances should be brushed from the skin. ā€¢ Remove the clothes before water is used. ā€¢ ((Personal protective devices should be used like goggles gowns respirators etc.)
  • 117. Bowel management: ā€¢ Cathartics (Sorbitol) are given together with the first dose of charcoal to stimulate intestinal motility. ā€¢ Whole bowel irrigation involves the administration of bowel evacuant solution(eg: golytely).
  • 118. ā€¢ This solution is administered every 4-5 hrs. until stool are clear. ā€¢ It is effective for swallowed objects foe cocaine filled balloon or condoms, and heavy metals such as lead and mercury
  • 120. List of antidots: ļƒ¼ Poison ļƒ¼ Antidotes ļ¶ Organophosphate ļ¶ Atropine ļ¶ Cyanide ļ¶ Amyl nitrite ļ¶ Digoxin ļ¶ Anti digoxin ļ¶ Paracetamol ļ¶ Acetylcysteine ļ¶ Irritant gases ļ¶ Budesonide ļ¶ Lead ļ¶ Calcium disodium edetate ļ¶ Benzodiazepine ļ¶ flumazenil ļ¶ Methanol ļ¶ Ethanole
  • 121. Contā€¦ ļƒ¼ Poison ļƒ¼ Antidotes ļ¶ insulin ļ¶ Glucose ļ¶ Opioids analgesics ļ¶ naloxone ļ¶ Carbon monoxide ļ¶ Oxygen ļ¶ copper ļ¶ Penicillamine ļ¶ Warfarin ļ¶ Clotting factors ļ¶ Isoniazid ļ¶ Vita 6 ļ¶ Caffeine ļ¶ Esmolol ļ¶ penicillin ļ¶ Epinephrine
  • 122. ļƒ¼ Poison ļƒ¼ Antidotes ļ¶ Curare ļ¶ Tensilon ļ¶ Vincristine ļ¶ Hyaluronidase ļ¶ Mistinon ļ¶ Atropine sulphate ļ¶ Meyaproterenol ļ¶ Esmolol ļ¶ Narcotics ļ¶ Naloxone ļ¶ Chlorates ļ¶ Methylene blue ļ¶ Thallium ļ¶ Prussian blue ļ¶ Formaldehyde ļ¶ Folic acid Contā€¦
  • 123. Promotion of poison excretion: ā€¢ Increase fluid intake ā€¢ Hemodialysis ā€¢ Hemoperfusion ā€¢ Peritoneal diaylsis
  • 125. Nurses responsibility: ā€¢ Inform police ā€¢ Report MLC ā€¢ Collect information from the surrounding persons ā€¢ Preserve the suspending material like bottle containing pills or liquid ā€¢ Preserve the vomited material.
  • 127. INTRODUCTION: ā€¢ Rehabilitation is care that can help you get back, keep, or improve abilities that you need for daily life. These abilities may be physical, mental, and/or cognitive (thinking and learning). You may have lost them because of a disease or injury, or as a side effect from a medical treatment. Rehabilitation can improve your daily life and functioning.
  • 128. DEFINITION ā€¢ Rehabilitation is ā€œa set of measures that assist individuals who experience, or are likely to experience, disability to achieve and maintain optimal functioning in interaction with their environmentsā€
  • 129. TYPES OF REHABILITAION: There are many type of rehabilitation. In poison depend upon which organ is affected according to that we provide rehabilitation to the patient: Neurological rehabilitation Stroke rehabilitation Cardiac Rehabilitation Drug rehabilitation Alcohol rehabilitation
  • 130. CONTā€¦ Medical rehabilitation Physical rehabilitation Vocational rehabilitation Vestibular rehabilitation
  • 131. NEED REHABILATION: ā€¢ Injuries and trauma: burns, fractures (broken bones), traumatic brain injury, and spinal cord injuries , poison . ā€¢ Stroke ā€¢ Severe infections ā€¢ Major surgery
  • 132. ā€¢ Side effects from medical treatments, such as from cancer treatments ā€¢ Certain birth defects and genetic disorders ā€¢ Developmental disabilities ā€¢ Chronic pain, including back and neck pain CONTā€¦
  • 133. FELLOW-UP: The follow-up clinic or cardiac rehab appointment is successful if: ā€¢ Patient arrives at appointment within 7 days of discharge from hospital. ā€¢ Discharge summary (including summary of hospitalization, updated medication list) available to follow-up provider. ā€¢ Patient brings his/her medications or a medication list to clinic visit.
  • 134. ļ¶Reason for referral available to cardiac rehab center and patient brings referral letter or provider prescription ļ¶Rates of physician follow-up within 1 week of discharge were low and varied substantially across hospitals. CONTā€¦
  • 135. ļ¶Patients discharged from hospitals with more consistent early follow-up with 7 days have lower risk of 30-day readmission. ļ¶Enhanced transition planning and ensuring that patients are evaluated within a week of discharge represents an achievable target for hospital quality improvement. CONTā€¦
  • 136. Conclusion: ā€¢ I conclude that emergency is a situation which can be occur anywhere at any time which can cause severe trauma to the patient and their family member. immediate treatment should be required otherwise it will lead to a death.
  • 137. Summarization: Today we discussed about: ļ¶Emergency ļ¶MODS ļ¶DIC ļ¶Trauma ļ¶Burn ļ¶Poising ļ¶Conclusion ļ¶Recapitalization ļ¶Bibliography
  • 138. Recapitalization: 1) Define the common emergency ? 2) Define the classification of burn? 3) Define the antidot of paracetamol?
  • 139. BIBLIOGRAPHY: ā€¢ Suddharthā€™s & brunner Textbook of medical surgical nursing published by Wolters Kluwer edition south Asian page no. 1250-1255 ā€¢ Black M. joyce Textbook of medical surgical nursing published by elsvier edition 1st page no.. 1780-1800 ā€¢ www.slideshare.net ā€ŗ shisha_sk ā€ŗ burn-ppt-Shashi Viewed on 02/04.2020 ā€¢ www.slideshare.net ā€ŗ AseemBadarudeen ā€ŗ poisoning-49486135 viewed on 3/04/2020 ā€¢ www.slideshare.net ā€ŗ haifa12 ā€ŗ rehabilitation-54792687 viewed on 5/04/2020
  • 140. Write down the nursing management of burn