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1)    Define Rheumatic Heart disease
2)    Explain the pathophysiology and Aetiology of Rheumatic
      Heart Disease
3)    Explain the clinical manifestation of Rheumatic Heart
      Disease
4)    State the diagnostic evaluation/studies of RHD
5)    Explain the Nursing assessment
6)    State the nursing diagnoses
7)    Explain the nursing management and interventions
8)    Medical management
9)    Health education
10)   State complications of RHD
0 According to Lewis et al, RHD is the chronic
  condition/disease resulting from rheumatic fever
  that is characterised by swelling and deformity of
  valves.
0 RHD is an acute, recurrent inflammatory disease that
  causes damage to the heart as a sequela to group A
  beta-hemolytic streptococcal infection, particularly
  the valves, resulting in valve leakage (insufficiency)
  and/or obstruction (narrowing or stenosis).
contd/:
0 RHD Is the a chronic condition characterised by
  scarring and deformity of the heart valves following
  rheumatic fever infection.
0 Rheumatic fever is an inflammatory disease of the
  heart potentially involving all the layers of the heart ie
  endocardium ,myocardium and pericardium
 Streptococcal infections
    o Gram-positive non motile spherical bacteria
      occurring in chains.
    o Most species are saprophytes and some are pathogenic
    o Many pathogenic species are haemolytic
             o They have the ability to destroy red blood cells
ARTICLE PUBLISHED ON N.º 240 OF JOURNAL OF GENERAL HOSPITAL ROCHESTER
INTERNET:WWW. www.interscience.wiley.com)---21/01/2013
0 Rheumatic fever is a sequela to group A streptococcal
  infection that occurs in about 3% of untreated
  infections. (Nettina S.M et al,2006)
0 It is a preventable disease through the detection and
  adequate treatment of streptococcal pharyngitis.
0 Connective tissue of the heart, blood vessels, joints,
  and subcutaneous tissues are affected.
0 Lesions in connective tissue are known as Aschoff
  bodies, which are localized areas of tissue necrosis
  surrounded by immune cells.
contd/:
0 Heart valves are affected, resulting in valve leakage
  and narrowing.
0 Compensatory changes in the chamber sizes and
  thickness of chamber walls occur.
0 Heart involvement (carditis) also includes
  pericarditis, myocarditis, and endocarditis
 Streptococcal infections        inflammation
of the heart’s tissues & fever      affects the
 heart’s valves        resulting in valve leakage
and narrowing         as a Compensatory
 mechanism changes in the chamber sizes and
thickness of chamber walls occur.
0 Symptoms of streptococcal pharyngitis may precede
 rheumatic symptoms e.g.
   Sudden onset of sore throat; throat reddened with
    exudate
   Swollen, tender lymph nodes at angle of jaw
   Headache and fever >38 degrees celsius
   Abdominal pain (children)
   Some cases of streptococcal throat infection are
    relatively asymptomatic
0 Warm and swollen joints (polyarthritis)
contd/:
0 Chorea -(irregular, jerky, involuntary, unpredictable
  muscular movements especially affecting the head, face or
  limbs)
0 Erythema marginatum -(transient meshlike macular
  rash on trunk and extremities
0 Subcutaneous nodules (hard, painless nodules over
  extensor surfaces of extremities; rare)
0 Fever >38 degrees celsius.
0 Prolonged Pulse Rate (heart beat) interval demonstrated
  by ECG.
0 Heart murmurs; pleural and pericardial rubs.
0 Throat culture-to determine presence of
  streptococcal organisms
0 Sedimentation rate, WBC count and differential,
  and CRP increased during acute phase of infection
0 Elevated antistreptolysin-O (ASO) titer
0 ECG-prolonged Pulse Rate interval or heart block
SUBJECTIVE DATA
Past health history:
  Recent streptococcal infection
  Previous history of RHD/RF
Health perception-health management:
  Family history of rheumatic fever
Nutritional-metabolic:
  Anorexia and weight loss
Activity-exercise:
  Palpitations, generalized weakness,fatigue,ataxia etc
contd
Cognitive –perceptual:
  Chest pain
  Migratory joint pain
  Tenderness (especially large joints)


      OBJECTIVE DATA
General
   fever
Integumentary:
   Subcutaneous nodules
   Erythema marginatum
Cardiovascular
   Tachycardia,
   pericardial friction rub
   Muffed heart sounds
   Gallop rhythm
   Murmurs &
   Peripheral edema
 Neurologic:
   Chorea-involuntary, purposeless,rapid motions,facial
    grimaces.
o Musculoskeletal:
   Signs of mono or polyarthritis including swelling
    ,heat,redness,limitation of motion
    (especially,knees,ankles,elbows,shoulders,wrists etc)
1) Ineffective breathing pattern related to
   musculoskeletal fatigue ,low level of consciousness
   as manifested by irregular breathing patterns and
   use of accessory muscles.
2) Altered thermoregulation (Hyperthermia) related
   to micro organisms invasion as manifested by high
   temperature of 38 degrees Celsius.
3) Decreased cardiac output related to valve
   dysfunction of heart failure as evidenced by low
   blood pressure
4)Altered comfort pain(joint pain) related to swollen
joint as evidenced by patient’s verbalisation and
inability to stretch joints well.
5)Activity intolerance related to arthralgia secondary to
joint pain, pain from pericarditis and heart failure as
manifested by facial expression
6)Ineffective therapeutic regimen management related
to lack of knowledge concerning the need for long-term
prophylactic antibiotic therapy and possible disease
sequelae as manifested by patient asking a lot of
questions.
0 Heart failure
0 Atrial fibrillation
0 Infective endocarditis
0 Atrial and ventricular arrhythmias
Ineffective breathing pattern
0 Observe for cyanosis, dyspnoea, hypoxia, and
  confusion, indicating worsening condition.
0 Place patient in an upright position to obtain
  greater lung expansion and improve aeration.
  Frequent turning and increased activity (up in
  chair, ambulate as tolerated) should be
  employed.
0 Administer oxygen at concentration to
  maintain Pao2 at acceptable level i.e. 4 to 6
0 Avoid high concentrations of oxygen in patients with
 COPD, particularly with evidence of CO2 retention; use
 of high oxygen concentrations may worsen alveolar
 ventilation by depressing the patient's only remaining
 ventilatory drive. If high concentrations of oxygen are
 given, monitor alertness and Pao2 and Paco2 levels for
 signs of CO2 retention.
0 Follow ABG levels/Sao2 to determine oxygen need and
 response to oxygen therapy
REDUCING FEVER
0 Do tepid sponging to reduce fever through conduction and
  evaporation and administer antipyretic e.g. ASA.
0 Administer penicillin therapy as prescribed to eradicate
  hemolytic streptococcus; an alternative drug may be prescribed if
  patient is allergic to penicillin, or sensitivity testing and
  desensitization may be done.
0 Give salicylates or NSAIDs as prescribed to suppress rheumatic
  activity by controlling toxic manifestations, to reduce fever, and to
  relieve joint pain.
0 Assess for effectiveness of drug therapy.
  0 Take and record temperature every 3 hours.
0 Evaluate patient's comfort level every 3 hours
contd
   Maintaining Adequate Cardiac Output
0 Assess for signs and symptoms of acute rheumatic
  carditis.
  0 Be alert to patient's complaints of chest pain,
    palpitations, and/or precordial tightness.
  0 Monitor for tachycardia (usually persistent when
    patient sleeps) or bradycardia.
  0 Be alert to development of second-degree heart block or
    Wenckebach's disease (acute rheumatic carditis causes
    Pulse Rate interval prolongation).
contd
0 Auscultate heart sounds every 4 hours.
   0 Document presence of murmur or pericardial friction
     rub.
   0 Document extra heart sounds (S3 gallop, S4 gallop).
0 Monitor for development of chronic rheumatic
 endocarditis, which may include valvular disease and
 heart failure
PAIN MANAGEMENT
Total bed rest /quiet environment for the
 comfortability of the patient.
Patient sleep to the side which is less painful
Administer prescribed analgesic drugs eg PCM 1g
 tds/24hrs
Diversion therapy- avoid the patient’s mind
 concentrating on his/her pain
NURSING INTERVENTIONS
         contd
        MAINTAINING ACTIVITY
0 Maintain bed rest for duration of fever or if signs of active
  carditis are present.
0 Allow the patient to do the physical exercises which he/she
  can manage to do due to his/her easily fatigue.
0 Provide diversional activities that prevent exertion.
0 Discuss need for tutorial services with parents to help
  child keep up with school work.
patient education and health
          maintenance
0 Counsel patient to maintain good nutrition.
0 Counsel patient on hygienic practices.
   0 Discuss proper handwashing, disposal of tissues,
     laundering of handkerchiefs (decrease risk of exposure
     to microbes).
   0 Discuss importance of using patient's own toothbrush,
     soap, and washcloths when living in group situations.
0 Counsel patient on importance of receiving adequate
  rest.
0 Instruct patient to seek treatment immediately should
  sore throat occur.
0 Support patients in long-term antibiotic therapy to
  prevent relapse (5 years for most adults).
0 Instruct patient with valvular disease to use
  prophylactic penicillin therapy before certain
  procedures and surgery
0 Explore with patient his ability to pay for medical
  treatment. If appropriate, contact social services for
  patient. (Financial difficulties may inhibit patient from
  seeking early treatment of symptoms.)
Evaluation: Expected Outcomes

0 Afebrile
0 Denies chest pain; normal sinus rhythm
0 Maintains bed rest while febrile
COMMENT/CONCLUSION
0 Tell as many other people as possible about this
  disease (rheumatic heart disease).
0 It could save their lives !!!
0 DON'T ever think that you are not prone to rheumatic
  heart disease as your age is less than 25 or 30.
  Nowadays due to the change in the life style,
  rheumatic heart disease is found among people of all
  age groups.
AS NURSES,TELL AS MANY OTHER PEOPLE AS POSSIBLE ABOUT
THIS DISEASE OF RHEUMATIC HEART DISEASE (RHD)
IT COULD SAVE THEIR LIVES !!! REMEMBER HEART IS
THE ENGINE OF THE BODY. ANY CONDITION WHICH
CAN AFFECT THE HEART CAN LEAD TO DEATH!!!
REFERENCES
1) LEWIS ,HEITKEMPER,DIRKSEN,O’BRIEN &
   BUCHER,(2007) MEDICAL -SURGICAL
   NURSING,ASSESSMENT AND MANAGEMENT OF
   CLITICAL PROBLEMS.7TH EDITION.MOSBY ELSEVIER.
2) JOYCE M.BLACK AND JANE HOKANSON HAWKS,(2009)
   MEDICAL-SURGICAL NURSING CLINICAL
   MANAGEMENT FOR POSITIVE OUTCOMES.8TH
   EDITION.MOSBY ELSEVIER.
3) NETTINA,SANDRA M,MILLS ELIZABETH
   JACQUUELINE,(2006) LIPPINCOTT MANUAL OF
   NURSING PRACTICE.8TH EDITION.WILLIAMS & WILKINS.
4) INTERNET: Wiley
   (http://www.interscience.wiley.com)---
   21/01/2013

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Rheumatic heart disease

  • 1.
  • 2.
  • 3. 1) Define Rheumatic Heart disease 2) Explain the pathophysiology and Aetiology of Rheumatic Heart Disease 3) Explain the clinical manifestation of Rheumatic Heart Disease 4) State the diagnostic evaluation/studies of RHD 5) Explain the Nursing assessment 6) State the nursing diagnoses 7) Explain the nursing management and interventions 8) Medical management 9) Health education 10) State complications of RHD
  • 4. 0 According to Lewis et al, RHD is the chronic condition/disease resulting from rheumatic fever that is characterised by swelling and deformity of valves. 0 RHD is an acute, recurrent inflammatory disease that causes damage to the heart as a sequela to group A beta-hemolytic streptococcal infection, particularly the valves, resulting in valve leakage (insufficiency) and/or obstruction (narrowing or stenosis).
  • 5. contd/: 0 RHD Is the a chronic condition characterised by scarring and deformity of the heart valves following rheumatic fever infection. 0 Rheumatic fever is an inflammatory disease of the heart potentially involving all the layers of the heart ie endocardium ,myocardium and pericardium
  • 6.  Streptococcal infections o Gram-positive non motile spherical bacteria occurring in chains. o Most species are saprophytes and some are pathogenic o Many pathogenic species are haemolytic o They have the ability to destroy red blood cells
  • 7. ARTICLE PUBLISHED ON N.º 240 OF JOURNAL OF GENERAL HOSPITAL ROCHESTER INTERNET:WWW. www.interscience.wiley.com)---21/01/2013
  • 8. 0 Rheumatic fever is a sequela to group A streptococcal infection that occurs in about 3% of untreated infections. (Nettina S.M et al,2006) 0 It is a preventable disease through the detection and adequate treatment of streptococcal pharyngitis. 0 Connective tissue of the heart, blood vessels, joints, and subcutaneous tissues are affected. 0 Lesions in connective tissue are known as Aschoff bodies, which are localized areas of tissue necrosis surrounded by immune cells.
  • 9. contd/: 0 Heart valves are affected, resulting in valve leakage and narrowing. 0 Compensatory changes in the chamber sizes and thickness of chamber walls occur. 0 Heart involvement (carditis) also includes pericarditis, myocarditis, and endocarditis
  • 10.  Streptococcal infections inflammation of the heart’s tissues & fever affects the heart’s valves resulting in valve leakage and narrowing as a Compensatory mechanism changes in the chamber sizes and thickness of chamber walls occur.
  • 11. 0 Symptoms of streptococcal pharyngitis may precede rheumatic symptoms e.g.  Sudden onset of sore throat; throat reddened with exudate  Swollen, tender lymph nodes at angle of jaw  Headache and fever >38 degrees celsius  Abdominal pain (children)  Some cases of streptococcal throat infection are relatively asymptomatic 0 Warm and swollen joints (polyarthritis)
  • 12. contd/: 0 Chorea -(irregular, jerky, involuntary, unpredictable muscular movements especially affecting the head, face or limbs) 0 Erythema marginatum -(transient meshlike macular rash on trunk and extremities 0 Subcutaneous nodules (hard, painless nodules over extensor surfaces of extremities; rare) 0 Fever >38 degrees celsius. 0 Prolonged Pulse Rate (heart beat) interval demonstrated by ECG. 0 Heart murmurs; pleural and pericardial rubs.
  • 13. 0 Throat culture-to determine presence of streptococcal organisms 0 Sedimentation rate, WBC count and differential, and CRP increased during acute phase of infection 0 Elevated antistreptolysin-O (ASO) titer 0 ECG-prolonged Pulse Rate interval or heart block
  • 14. SUBJECTIVE DATA Past health history:  Recent streptococcal infection  Previous history of RHD/RF Health perception-health management:  Family history of rheumatic fever Nutritional-metabolic:  Anorexia and weight loss Activity-exercise:  Palpitations, generalized weakness,fatigue,ataxia etc
  • 15. contd Cognitive –perceptual:  Chest pain  Migratory joint pain  Tenderness (especially large joints) OBJECTIVE DATA General  fever
  • 16. Integumentary:  Subcutaneous nodules  Erythema marginatum Cardiovascular  Tachycardia,  pericardial friction rub  Muffed heart sounds  Gallop rhythm  Murmurs &  Peripheral edema
  • 17.  Neurologic:  Chorea-involuntary, purposeless,rapid motions,facial grimaces. o Musculoskeletal:  Signs of mono or polyarthritis including swelling ,heat,redness,limitation of motion (especially,knees,ankles,elbows,shoulders,wrists etc)
  • 18. 1) Ineffective breathing pattern related to musculoskeletal fatigue ,low level of consciousness as manifested by irregular breathing patterns and use of accessory muscles. 2) Altered thermoregulation (Hyperthermia) related to micro organisms invasion as manifested by high temperature of 38 degrees Celsius. 3) Decreased cardiac output related to valve dysfunction of heart failure as evidenced by low blood pressure
  • 19. 4)Altered comfort pain(joint pain) related to swollen joint as evidenced by patient’s verbalisation and inability to stretch joints well. 5)Activity intolerance related to arthralgia secondary to joint pain, pain from pericarditis and heart failure as manifested by facial expression 6)Ineffective therapeutic regimen management related to lack of knowledge concerning the need for long-term prophylactic antibiotic therapy and possible disease sequelae as manifested by patient asking a lot of questions.
  • 20. 0 Heart failure 0 Atrial fibrillation 0 Infective endocarditis 0 Atrial and ventricular arrhythmias
  • 21. Ineffective breathing pattern 0 Observe for cyanosis, dyspnoea, hypoxia, and confusion, indicating worsening condition. 0 Place patient in an upright position to obtain greater lung expansion and improve aeration. Frequent turning and increased activity (up in chair, ambulate as tolerated) should be employed. 0 Administer oxygen at concentration to maintain Pao2 at acceptable level i.e. 4 to 6
  • 22. 0 Avoid high concentrations of oxygen in patients with COPD, particularly with evidence of CO2 retention; use of high oxygen concentrations may worsen alveolar ventilation by depressing the patient's only remaining ventilatory drive. If high concentrations of oxygen are given, monitor alertness and Pao2 and Paco2 levels for signs of CO2 retention.
  • 23. 0 Follow ABG levels/Sao2 to determine oxygen need and response to oxygen therapy
  • 24. REDUCING FEVER 0 Do tepid sponging to reduce fever through conduction and evaporation and administer antipyretic e.g. ASA. 0 Administer penicillin therapy as prescribed to eradicate hemolytic streptococcus; an alternative drug may be prescribed if patient is allergic to penicillin, or sensitivity testing and desensitization may be done. 0 Give salicylates or NSAIDs as prescribed to suppress rheumatic activity by controlling toxic manifestations, to reduce fever, and to relieve joint pain. 0 Assess for effectiveness of drug therapy. 0 Take and record temperature every 3 hours. 0 Evaluate patient's comfort level every 3 hours
  • 25. contd Maintaining Adequate Cardiac Output 0 Assess for signs and symptoms of acute rheumatic carditis. 0 Be alert to patient's complaints of chest pain, palpitations, and/or precordial tightness. 0 Monitor for tachycardia (usually persistent when patient sleeps) or bradycardia. 0 Be alert to development of second-degree heart block or Wenckebach's disease (acute rheumatic carditis causes Pulse Rate interval prolongation).
  • 26. contd 0 Auscultate heart sounds every 4 hours. 0 Document presence of murmur or pericardial friction rub. 0 Document extra heart sounds (S3 gallop, S4 gallop). 0 Monitor for development of chronic rheumatic endocarditis, which may include valvular disease and heart failure
  • 27. PAIN MANAGEMENT Total bed rest /quiet environment for the comfortability of the patient. Patient sleep to the side which is less painful Administer prescribed analgesic drugs eg PCM 1g tds/24hrs Diversion therapy- avoid the patient’s mind concentrating on his/her pain
  • 28. NURSING INTERVENTIONS contd MAINTAINING ACTIVITY 0 Maintain bed rest for duration of fever or if signs of active carditis are present. 0 Allow the patient to do the physical exercises which he/she can manage to do due to his/her easily fatigue. 0 Provide diversional activities that prevent exertion. 0 Discuss need for tutorial services with parents to help child keep up with school work.
  • 29. patient education and health maintenance 0 Counsel patient to maintain good nutrition. 0 Counsel patient on hygienic practices. 0 Discuss proper handwashing, disposal of tissues, laundering of handkerchiefs (decrease risk of exposure to microbes). 0 Discuss importance of using patient's own toothbrush, soap, and washcloths when living in group situations.
  • 30. 0 Counsel patient on importance of receiving adequate rest. 0 Instruct patient to seek treatment immediately should sore throat occur. 0 Support patients in long-term antibiotic therapy to prevent relapse (5 years for most adults).
  • 31. 0 Instruct patient with valvular disease to use prophylactic penicillin therapy before certain procedures and surgery 0 Explore with patient his ability to pay for medical treatment. If appropriate, contact social services for patient. (Financial difficulties may inhibit patient from seeking early treatment of symptoms.)
  • 32. Evaluation: Expected Outcomes 0 Afebrile 0 Denies chest pain; normal sinus rhythm 0 Maintains bed rest while febrile
  • 33. COMMENT/CONCLUSION 0 Tell as many other people as possible about this disease (rheumatic heart disease). 0 It could save their lives !!! 0 DON'T ever think that you are not prone to rheumatic heart disease as your age is less than 25 or 30. Nowadays due to the change in the life style, rheumatic heart disease is found among people of all age groups.
  • 34. AS NURSES,TELL AS MANY OTHER PEOPLE AS POSSIBLE ABOUT THIS DISEASE OF RHEUMATIC HEART DISEASE (RHD) IT COULD SAVE THEIR LIVES !!! REMEMBER HEART IS THE ENGINE OF THE BODY. ANY CONDITION WHICH CAN AFFECT THE HEART CAN LEAD TO DEATH!!!
  • 35. REFERENCES 1) LEWIS ,HEITKEMPER,DIRKSEN,O’BRIEN & BUCHER,(2007) MEDICAL -SURGICAL NURSING,ASSESSMENT AND MANAGEMENT OF CLITICAL PROBLEMS.7TH EDITION.MOSBY ELSEVIER. 2) JOYCE M.BLACK AND JANE HOKANSON HAWKS,(2009) MEDICAL-SURGICAL NURSING CLINICAL MANAGEMENT FOR POSITIVE OUTCOMES.8TH EDITION.MOSBY ELSEVIER. 3) NETTINA,SANDRA M,MILLS ELIZABETH JACQUUELINE,(2006) LIPPINCOTT MANUAL OF NURSING PRACTICE.8TH EDITION.WILLIAMS & WILKINS. 4) INTERNET: Wiley (http://www.interscience.wiley.com)--- 21/01/2013