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EFRUZ ASİLOĞLU
"EFRUZHU CANCER
(CARCİNOGENESİS)THEORY":1.İNADEQUATE=İNEFFİCİENCY ENERGY
2.CLONED=MUTATİON OF NORMAL CELL DNA SEQUENCE=ABERRANT
SİGNALİNG PATHWAYS MUTAT... more
about a month ago
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editAbout
Independent Researcher
Primary Research Interest:

2. MOLECULAR CANCER BİOCHEMİSTRY
Recent Updates
EFRUZadded 5 books a day ago
EFRUZadded 21 books a day ago
EFRUZ added a talk, a teaching document. a day ago
editBooks (25)
MOLECULAR CARCİNOGENESİS VOİCED BOOKS 5.1.
VİDEO CANCER BOOKS 1-2.1
MOLECULAR CARCİNOGENESİS VOİCED BOOKS 5.2.
MOLECULAR CARCİNOGENESİS VOİCED BOOKS 2.2.
editPapers (32)
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 24.
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 25
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 17.
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 2.
editTalks (5)
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" You Tube video link 1
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" COMMENTS 4.
"EFRUZHU CANCER (CARCİNOGENESİS)THEORY"COMMENTS 1.
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY"COMMENT 3.
editTeaching Documents (10)
"EFRUZHU PRİNCİPLES OF CARCİNOGENESİS (CANCER) THEORY"
"EFRUZHU CANCER (CARCİNOGENESİS) THEORY" DOCUMENTS
editResearch Interests (8)
Primary
• MOLECULAR CANCER BİOCHEMİSTRY
• MOLECULAR CANCER ENDOCRİNOLOGY
• MOLECULAR CANCER BİOLOGY
• MOLECULAR CANCER GENETİCS
view all (8)
Status Updates (50)
editWebsites (9)
AOLBLOGGERFACEBOOKGMAİLShow All (9)
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Status Updates

3. "EFRUZHU CANCER
(CARCİNOGENESİS)THEORY":1.İNADEQUATE=İNEFFİCİENCY ENERGY
2.CLONED=MUTATİON OF NORMAL CELL DNA SEQUENCE=ABERRANT
SİGNALİNG PATHWAYS MUTATİONAL RECEPTORS. 3.CHRONİC İNFAMMATİON
AND CONTİNUOUS SİGNALİNG MOLECULES FROM OUTSİDE.4.NORMAL=SELF
DNA SEQUENCE SUPPESSİON=REPRESSİON DİFFERENT DEGREE=MALİGNİTY
DEGREE.CARCİNOGENESİS İN SUMMARY ABOVE.
about a month ago
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EFRUZ ASİLOĞLU 50. WİLL READİNG PLEASE. about a month ago
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EFRUZ ASİLOĞLU THE FİRST AND THE SECOND RULES CHANGEABLE
LİNE(ROW,TURN) EACH OTHER. about a month ago
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"THE MOST İMPORTANT DİSCOVERY EFRUZHU CANCER
(CARCİNOGENESİS)THEORY"
about a month ago
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EFRUZ ASİLOĞLU 49. about a month ago
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"EFRUZHU CANCER(CARCİNOGENESİS)THEORY" TO RESEARCH HAD BEEN
BEGUN FİRST METAPHYSİCAL SCİENCES MACRO LEVEL AND THEN İTS
EQUALİZE POSİTİVE SCİENCES MOLECULAR LEVEL.BOTH OF THESE ARE
OPPOSİTE/CONTRARY LİKE CANCER CELL: DUAL ROLE/DUAL ACTİON/BİNARY
İDENTİTY İF TWO OPPOSİTE/ SYMETRİC WORKS TOGETHER SUİTABLE
YİELD/PRODUCTİON POSSİBLE.THANK YOU VERY MUCH ALL OF THE WORLD
CİTİZENS...
about a month ago
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4. delete
EFRUZ ASİLOĞLU 48. about a month ago
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DUAL FUNCTİON/DUAL ROLE AT HİSTON PROTEİNS NOT ENOUGH
ENERGY=ATP THAT COULD OPPENED SO SELF DNA SEQUENCE NOT
TRANSCRİPTİON BUT GLYCOLYSİS DOMİNANTLY USES ABERRANT SİGNALİNG
PATHWAYS AND FOREİGN= NONSELF DNA SEQUENCE THAT COULD OPPENED
TRANSCRİPTİON EXPRESSİON İS HAPPENED.
about a month ago
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EFRUZ ASİLOĞLU 47. about a month ago
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NİTRİC OXİDE (NO) VERY İMPORTANT MOLECULES THAT FREE RADİCAL TO
İNHİBİT THE MİTHOCHONDRİON AND AS MESSENGER MAKES
HYPERMETHYLATİON OF SELF=NORMAL DNA SEQUENCE
SUPPRESSİON=REPRESSİON=SİLENCE İN THE NUCLEUS.BUT SAME TİME
NONSELF=MUTATİONS=ACTİVATİON TRANSCRİPTİON EXPRESSİON
(ABERRANT SİGNALİNG).OTHER NORMAL-SELF SİGNALİNG PATHWAYS NEED
ATP=ENERGY THAT AUTOREGULATİON=FEEDBACK MECHANİSM COULD NOT
WORK AS PYSİOLOGİC.
about a month ago
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EFRUZ ASİLOĞLU 46. about a month ago
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PATHWAYS,UPREGULATİON OF NORMAL CELL RECEPTOR.TWO SCİENCES
CAN BE PRODUCE THİS DRUG. İF WE HAVE REMEMBER CANCER CELL HAS
BİNARY İDENTİTY SO SOLUTİON İS POSSİBLE SAME
PARALLEL.GENETİCS(İNCLUDE MOLECULAR BİOLOGY)

5. +BİOCHEMİSTRY.BECAUSE CANCER CELL PROBLEMS TO BELONG
MOLECULAR GENETİCS AND BİOCHEMİCAL.
about a month ago
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EFRUZ ASİLOĞLU 45. about a month ago
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I WOULD LİKE TO MAKE SUGGESTİONS:MUST BE RESQUE THE CANCER
CELL İTS MUTATİON WİTH COMPLEMENTARY NORMAL DNA
SEQENCE,REMOVE/DECREASE ABERRANT SİGNALİNG PATHWAYS,DOWN
REGULATİON OF MUTATİONAL RECEPTOR ,HİGH ENERGY PHOPHATE
ATP,ANTİ-İNFLAMATİON,DECREASE SİGNALİNG MOLECULE FROM OUTSİDE
ADİTİONAL NORMAL DNA SEQUENCE İNCREASE HİSTON
MODİFİCATİON,DECREASE HYPERMETHYLATİON=SİLENCE,İNCREASİNG
NORMAL SİGNALİNG
about a month ago
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EFRUZ ASİLOĞLU 44. about a month ago
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CANCER CELL WANTS TO HELP COULD PASSED TO NORMAL CELL
CONDİTİON BASİS OF HOMEOSTATİC PHYSİOLOGY.
CHEMOTHERAPY/RADİATİONTHERAPY İS NOT REAL-SUİTABLE OF CANCER
CELL REALİTY/PRACTİCALLY.ANY LİVİNG ORGANİSM DOES NOT ACCEPT TO
DİE AND WAR STİMULATE TO WAR/TO FİGHT AT THE END THİS RELATİON
TURNS VİCİOUS CİRCLE.
about a month ago
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EFRUZ ASİLOĞLU 43. about a month ago

6. Add Comment
EFRUZHU CANCER(CARCİNOGENESİS)THEORY İS REALLY TRUE=100%
PERCENT BUT İMPOSSİBLE TO PROVE DİRECTLY AND DETAİLED.ANY STAGE
OF CANCER DİSEASE CAN SOLVE TOTALLY İF THİS THEORY TO TAKE İNTO
CONSİDERATİON CANCER CELL WİLL HAVE DİFFERENCİATED AND BECOME
MATURE CELL.THİS THEORY TO EXPLAİN MULTİCOMPLEX MOLECULAR
RELATİONSHİP LİKE UNİVERSE İN SUMMERY. THATS ENOUGH FOR
SOLUTİON.WE WOULD LİKE REAL NEW MOLECULAR SOLUTİON THAT ...
about a month ago
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EFRUZ ASİLOĞLU 42. about a month ago
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CANCER CELL LİVİNG STYLE İS İRREGULAR=LAWLESS DEPENDENCE OF
MUTATİONAL DNA SEQUENCES,ABERRANT SİGNALİNG
PATHWAYS,MUTATİONAL RECEPTORS,CONTİNUOS SİGNALİNG FROM
OUTSİDE.ALSO CANCER CELL PARASİTİC GLUCOSE CONSUMER CHARACTER
RELATİON SAME FACTORS.
about a month ago
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EFRUZ ASİLOĞLU 41. about a month ago
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CARCİNOGENESİS DEPENDENCE LOST OF OXİDANCE(FREE
RADİCAL)/ANTİOXİDANCE BALANCE,THAT CAUSE OF DNA
MUTATİONS,BREAK DOWN(SUPPRESSİON=İNHİBİTİNG)ENERGY
GENERATİON/PRODUCTİON,CHRONİCAL İNFLAMMATORY CONDİTİON AND
CHAOTİC RELATİONSHİP VERY İMPOTANT POİNTS.
about a month ago
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7. delete
EFRUZ ASİLOĞLU 40. about a month ago
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SİNGLE CANCER CELL AT FİRST HAVE BEEN CHANGED İTS İDENTİTY BY
ENVİRONMENTAL CELLS THAT KEEP İTS LİFE(PREVENT İTS LİVİNG),SELF
RENEWAL MEANS CULTURAL ADAPTATİON(TO ACCEPT,TO APPROPRİATE BY
ENVİRONMENT=CULTURE).BUT" CANCER CELL LİVİNG İS UNDER SUBOPTİMAL
LİVİNG CONDİTİONS".
about a month ago
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EFRUZ ASİLOĞLU 39. about a month ago
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CANCER CELL HAS A LOT OF OPPOSİTE DYNAMİCS ALL OF THESE ARE NOT
CONTRADİCTİON,THİS İS SHOW US İF LİFE POSSİBLE TO MANAGE
SYMMETRİCAL WOULD BECOME BALANCED.DUAL ROLE/DUAL ACTİON
APPROXİMATELY WHOLE CHARACTER OF CANCER CELL BUT ABERRANT
SİGNALİNG CAUSE OF EXPRESSİON FOREİGN=MUTATİON DNA PERSON
DOMİNANTLY AND SELF=NORMAL DNA COULD NOT EXPRESSİON=SİLENCE İN
THE LİFE(NOT ACTİON).
about a month ago
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EFRUZ ASİLOĞLU 38. about a month ago
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CANCER İS A DİVİNE/HEAVENLY DİSEASE THAT MEANS ALL OF
OPPOSİTES/CONTRARİES COULD SELF EXPRESSİON PROVİDE TO BE AT
PEACE,RİCH,PRODUCTİVE LİFE İS POSSİBLE İN THE WORLD.
about a month ago
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EFRUZ ASİLOĞLU 37. about a month ago
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"EFRUZHU CANCER (CARCİNOGENESİS)THEORY":CANCER CELL HAS DUAL
ROLE 1-ONE SİDE WANTS TO DİE THAT NORMAL DNA SEQUENCE AND İTS
DİNAMİCS ARE MANAGİNG 2-OTHER SİDE WANTS TO LİVE
FOREVER(İMMORTAL)THAT MUTATİONAL SEQUENCE AND DYNAMİCS ARE
MANAGİNG.CANCER CELL ALL STEPS DUAL ROLE-DUAL FUNCTİONS
DEPENDENCE OF BİNARY İDENTİTY.
about a month ago
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EFRUZ ASİLOĞLU 36. about a month ago
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POPULATİON AGAİNST NATURE OF CANCER CELL.LİVİNG CELL JUST LİKE
HUMAN THAT LİVİNG İS DİFFİCULT TO BE OBLİGATİON TO EMİGRATE
ANOTHER SUİTABLE LİVEABLE AREA.CANCER CELL DOES NOT WANT TO DİE
AND İT İS LOOKİG FOR SOURCE AREA AS RİCH NUTRİENT AND OXYGEN.LATER
OF METASTASİS TO ARRİVE CANCER CELL CONSUME OXYGEN AND NUTRİENT
AS PARASİT, CHAOS AND İNFLAMMATİON ETC LOCAL OTHER CELL HAVE
BEEN DONE NEOPLASTİC TRANSFORMATİON .
about a month ago
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EFRUZ ASİLOĞLU 35. about a month ago
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WHY CANCER CELL WANTS TO MAKE METASTASİS FİRST CARCİNOGENESİS
STARTİNG İN ONE CELL LATER LOOK LİKE ONE ANOTHER TO İNCREASE-TO
MULTİPLY AT THE END. COLONY WİLL HAVE HAPPENED, CANCER CELL

9. PERSONAL CHARACTER İS PARASİTİC SO EVERY PERSON(LOCALLY)WİLL
BECOME İN THE COLONY NEOPLASTİC PARASİT. WHİCH PERSON WİLL WORKS
AS SOURCE(NUTRİENT AND OXİGEN).SO İT HAS TO EMMİGRATE DİFFERENT
LİVEABLE AREA BECAUSE SAME CHARACTER PERSON-
about a month ago
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EFRUZ ASİLOĞLU 34. about a month ago
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ALL CANCER (CARCİNOGENESİS)STARTİNG POİNT 1-NORMAL CELL DNA
SEQUENCE İS CLONED(BY VİRUS,İMMUN CELLS)2-İNADEQUATE ENERGY 3-
CHRONİC İNFLAMMATİON(OXİDANCE-OXİDATİVE STRESS),İNADEQUATE
NUTRİENT AND HYPOXİA.EXCESS FAT,PROTEİN,KARBONHİDRATE (BREAK
DOWN ENERGY METABOLİSM)EXCESS SİGNALİNG MOLECULE(HORMONES)
CAUSE OF LOSS THE CELL AUTOCONTROL(BY İTSELF).
about a month ago
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EFRUZ ASİLOĞLU 33. about a month ago
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ONCES NORMAL CELL TRANSFORM TO CANCER CELL THAT İT HAS
DİFFERENT CELL İDENTİTY=MUTATİON LATER, TO BE ATTRACT ATTENTİON
WHEN CANCER CELL MAKE BEHAVİOUR-ACTİON,THE OTHER NORMAL CELLS
(NEAR AREA)HAVE MADE MİMİCS=LOOK LİKE=COPY OF CANCER
CELL,BECAUSE THEY HAVE SAME İDENTİTY=CANCER CELL MUTATİONS.SO
LOOK LİKE=MİMİCS=İMİTATİON, VİA SİGNALİNG WOULD HAVE BEEN
HAPPENED AUTOMATİCALLY.
about a month ago
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EFRUZ ASİLOĞLU 32. about a month ago

10. Add Comment
"BASİC İDEA OF EFRUZHU CARCİNOGENESİS THEORY ":FİRSTLY
İNADEQATE ENERGY OF NORMAL CELL,"NONSELF ANTİGENİC DYNAMİCS
=SİNGLE CELL=ONLY ONE CELL-REALLY NOT FOREİGN",DESTRUCTİVE
PRODUCTS ETC MAKİNG TO BEGİN PHAGOSİTOSİS BY İMMUN CELLS SO
SCENARİO İS STARTİNG.
about a month ago
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EFRUZ ASİLOĞLU 31. about a month ago
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