2. ACUTE SUPPURATIVE OTITIS MEDIA ( ASOM)
It is an acute suppurative inflammation of the
mucoperiosteal layer of middle ear cleft by
suppurative organisms ;
Middle ear cleft includes-Eustachian tube,
middle ear, attic ,aditus,antrum and mastoid air cells;
Duration less than 3 weeks;
Also known as acute otitis media or ASOM .
3. ETIOLOGY AND PREDISPOSING FACTORS
Age group - Infants and children;
Infection through eustachian tube to the middle ear in following
conditions like –recurrent attacks of upper respiratory tract
infections
Nose – acute or chronic rhinitis ;
Sinuses – rhinosinusitis ;
Nasopharynx –adenioditis , nasal packing for epistaxis , tumors;
Oropharynx – tonsilitis , pharyngitis ;
Vomitus or infected milk;
Infected water in the swimmimg pools;
Sniffing or forcible blowing of nose ;
Common in winter;
4.
5. ROUTES OF INFECTION
Infection via eustachian tube is the most common
route of spread of infection which travel via lumen of
the tube or along subepithelial peritubal lymphatics.
Higher incidence in infants and children due to
shorter, wider and more horizontal eustachian tube ;
Abundance of lymphoid tissue in infants and
children producing obstruction to eustachian tube ;
Breast or bottle feeding in young infant in horizontal
position force fluids through the tube into middle ear ;
6. 2) via external ear - traumatic perforation of tympanic
membrane due to any cause open a route to
middle ear infection;
3) Via blood borne - rare ;
8. PATHOLOGY AND CLINICAL FEATURES
Described in stages
Stage of tubal occlusion
Stage of pre suppuration
Stage of suppuration
Stage of resolution
Stage of complications
9. Stage of Tubal occlusion
oedema and hyperaemia of nasopharyngeal end of
eustachian tube ;blocks the tube leading to absorption of
gases at first oxygen is absorbed later other gases like CO2
,nitrogen also diffuse out into the blood ;
resulting in negative intratympanic pressure and
retraction of tympanic membrane ;
10. Clinical features of stage of tubal occlusion
Symptoms –
earache – mild , pulsatile , worse at night
Reduced feeding
Fullness of the ear;
Fever may or may not be present ;
Hard of hearing- adults
Associated symptoms of upper respiratory tract like –
running nose ( cold ),nasal obstruction
cough
11. Signs– Tympanic membrane is retracted
Dull and lusterless tympanic membrane
Cone of light - absent
with handle of malleus assuming a more horizontal
position apparently foreshortened
prominence of lateral process of malleus ;
Anterior and posterior malleolar folds become more
prominent;
Loss of light reflex;
Mobility of tympanic membrane is reduced performed by
seigelization or valsalva
12.
13. Tunning fork test – Rinne’s test BC > AC – Conductive
deafness;
Weber’s test –lateralized to diseased ear
15. STAGE OF PRESUPPURATION
PATHOLOGY - If tubal occlusion is prolonged
pyogenic organisms invade tympanic cavity
causing hyperemia of its lining ;
Inflammatory exudate appears in the middle ear
Tympanic membrane becomes congested;
16. CLINICAL FEATURES OF STAGE OF PRE SUPPURATION
SYMPTOMS
Marked earache which disturbs sleep; usually
throbbing in nature ;
Fullness of ear;
Hard of hearing /decreased hearing
ringing sensation in the ear
High grade of fever ;
Cold /cough
17. Signs of stage of presuppuration
Congestion of pars tensa
Leash of blood vessels appear along the handle of
malleus and at the periphery of tympanic membrane
imparting cart – wheel appearance ;
Later entire tympanic membrane becomes uniformly
red
Tuning fork tests – Rinne ‘s test – BC> AC conductive
deafness
Weber’s test- lateralized to diseased ear;
18.
19. STAGE OF SUPPURATION
PATHOLOGY – The collected exudate in the middle ear
will increase producing tension on the
tympanic membrane
Pressure necrosis in the pars tensa of tympanic
membrane leading to small central perforation
Ear starts draining
( serosanguinous later mucopurulent ) mucoperiosteum
of middle ear cleft will be thickened by neoformation
of capillaries in young fibrous tissue infiltrated with
lymphocytes , plasma cells and polymorphs;
20. Clinical features of stage of suppuration
Symptoms
Earache is excruciating later once ear discharge starts
pain reduces ;
Ear discharge – thin watery discharge
(serosanguinuous )later mucopurulent ;
Hard of hearing
Fever;
21. SIGNS
Tympanic membrane appears red and bulging with loss of
landmarks ;
Handle of malleus may be engulfed by swollen and protruding
tympanic membrane ;
Yellow spot may be seen on tympanic membrane where rupture
is imminent ;
Ear discharge – in the ear canal is thin at first later mucopurulent
after dry mopping -Tympanic membrane shows small central
perforation ;
Pulsatile discharge may be present through the perforation
called – light house sign ;
Mastoid tenderness may be present;
22.
23. STAGE OF RESOLUTION
PATHOLOGY- Inflammatory process begins to resolve
as resistance of host overtakes the virulence of organisms and because
of proper antibiotic therapy , acute infection begins to subside ;if
proper treatment started early resolution may start even without
rupture of tympanic membrane ; pathological process resolves
A large coalescent cavity fills first with vascular
connective tissue later becomes pale –
chicken fat granulation and finally replaced by osteoid tissues or
pneumatic cells may reform ;
Symptoms are relieved –
cessation ear discharge ,
earache relieved ,
fever subsides ; well being improved
24. Signs of resolution
Hyperemia of tympanic membrane subsides return to
normal colour and landmarks ;
Small perforation seen in anteroinferior quadrant of
pars tensa ;
25. Stage of complications
If virulence of organisms is high or resistance of patient is
poor ,resolution may not take place , disease spreads
beyond the confines of middle ear ;
Complications may occur either by erosion of bone or by
hyperemic decalcification or thromboembolic phenomena;
Can be intracranial or extracranial complications
Like – acute mastoditis , subperiosteal abscess ,
facial paralysis , labyrinthitis , petrositis, extradural abscess,
meningitis , brain abscess or lateral sinus thrombophlebitis
26. MANAGEMENT OF ASOM
INVESTIGATIONS
In the early stages no need of any investigations only if
symptoms are not subsiding then following can be done;
PTA – Conductive deafness with hearing loss of ( 20-30 dB)
Impedence audiometry shows B type curve;
CBP – if fever is not subsided –Leukocytosis;
CULTURE AND SENSITIVITY OF EAR DISCHARGE
Xray PNS- To rule out sinusitis in recurrent cases
XRAY MASTOID – cloudy ,haziness of mastoid air cells ;
27. Pure tone audiogram and Impedance
audiogram –
acute suppurative otitis media
28. TREATMENT
Antibiotics of choice – Amoxiclav
40 mg / kg in 2-3 divided doses;
Those allergic to penicillins can be given with cefaclor, cotrimoxazole or erythromycin ;
Decongestant nasal drops – oxymetazoline or xylometazoline nasal drops to reduce
eustachian tube edema and promote ventilation of middle ear;
Oral nasal decongestants – pseudoephedrine 30mg twice daily or combination with anti
histaminics ;
Anti histaminics – levo cetrizine or fexofenadine ;
Analgesics and antipyretics – paracetamol;
Dry mopping in suppurative stage and topical antibiotics added follow up must be done;
Avoid exposure to cold
Keep ear dry by placing cotton while taking bath;
Avoid swimming
Proper positioning of baby while breast feeding must be instructed;
If any pharyngitis or tonsillitis salt water gargling must be instructed
Steam inhalation in case of sinusitis
29. surgical
Myringotomy
Indications – bulging tympanic membrane with acute
pain;
Incomplete resolution despite antibiotics when
tympanic membrane remains full with persistent
conductive deafness;
Persistent effusion beyond 12 weeks ;
30. Incising the tympanic membrane to evacuate pus
Circumferential incision is given in case of ASOM
In the antero-inferior quadrant grommet is placed for
weeks till it is spontaneously extruded;
32. To treat underlying cause –
adenoidectomy
Tonsillectomy
Cleft palate repair
in recurrent cases –
Submucosal resection of nasal septum
Submucosal diathermy ;
33. ACUTE NECROTIZING OTITIS
MEDIA(ANOM)
It’s a form of acute suppurative otitis media
Seen in children;
Suffering from measles , scarlet fever or influenza
Cause: upper respiratory tract infection or
exanthematous fever caused by
beta – hemolytic streptococcus;
34. Pathogenesis:
There is rapid destruction of whole tympsanic
membrane with its annulus , mucosa of promontry ,
ossicular chain and even mastoid air cells;
Healing is by fibrosis or ingrowth of squamous
epithelium leading to secondary acquired
cholesteatoma;
35. SYMPTOMS
Ear discharge – profuse , foul smelling, purulent
ear pain
Decreased hearing;
Symptoms of upper respiratory tract infection like –
fever , cold , skin rashes may be present;
36. Signs
purulent ear discharge in the external auditory meatus
– foul smelling;
After dry mopping- tympanic membrane shows
total perforation;
Mastoid tenderness;
Tunning fork test – rinne’s test BC > AC – Conductive
deafness;
Weber’s test –lateralized to diseased ear
38. MANAGEMENT OF ANOM
INVESTIGATIONS
PTA – Conductive deafness with hearing loss of ( 20-30
dB)
CBP – if fever is not subsided –Leukocytosis;
CULTURE AND SENSITIVITY OF EAR DISCHARGE
Xray PNS- To rule out sinusitis in recurrent cases
XRAY MASTOID – cloudy ,haziness of mastoid air cells ;
39. TREATMENT
Antibiotics of choice – Amoxiclav
40 mg / kg in 2-3 divided doses;
Those allergic to penicillins can be given with cefaclor, cotrimoxazole
or erythromycin ;
Decongestant nasal drops – oxymetazoline or xylometazoline nasal
drops to reduce eustachian tube edema and promote ventilation of
middle ear;
Oral nasal decongestants – pseudoephedrine 30mg twice daily or
combination with anti histaminics ;
Anti histaminics – levo cetrizine or fexofenadine ;
Analgesics and antipyretics – paracetamol;
Dry mopping in suppurative stage and topical antibiotics added follow
up must be done;
Avoid exposure to cold
Keep ear dry by placing cotton while taking bath;
40. If antibacterial therapy fails even after 10 days;
Cortical mastoidectomy is done ;