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Legg calve perthes disease
By: Ala’a AlGhanem | 211508057 | B1
Learning Objectives:
 Normal anatomy of hip joint.
 Introduction and epidemiology
 Aetiology and classifications
 Clinical presentation
 Investigations
 Treatment and prognosis
 Complications
Anatomy of hip joint:
Legg calve perthes disease
Idiopathic avascular necrosis of the proximal femoral epiphysis in
children
Legg Calve Perthes
Epidemiology:
2-Sex:
1-incidence:
affects 1 in 1200 children
>
5 : 1
3-Age
4-8 years is most common age of presentation
4-Population:
more commonly seen in urban populations versus rural
5-Location:
bilateral in 12% ( never at the same stage of disease)
the exact cause of disruption of blood supply remains
unknown.
Etiology:
Associated conditions:
-ADHD (33%)
-delayed bone age (98%)
-Thrombophilia (50%)
how Avascular necrosis (AVN) develops:
Risk Factors:
1-positive family history
2-low birth weight
3-abnormal birth presentation
4-children exposed to second hand
smoke
5-Asian, Inuit, and Central European
decent
Classifications:
 Waldenström
 Lateral Pillar (Herring ) Classification
 Catterall Classification
 Salter-Thompson classification
 Stulberg classification
Stages of Legg-Calves-Perthes
(Waldenström)
Initial stage
(infarction)
Fragmentation
Reossification
Healing or
remodeling
Lateral Pillar (Herring ) Classification
Determined at the beginning of fragmentation stage
lateral pillar
maintains full
height
Maintains
>50% height
Maintains
<50% height
Catterall Classification
Based on degree of head involvement
Salter-Thompson classification
Based on radiographic cresent sign
crescent sign involves < 1/2 of
femoral head
crescent sign involves > 1/2 of
femoral head
Class A
Class B
Stulberg classification
normal
Spherical head
with enlargement,
short neck, or
steep acetabulum
Nonspherical
head
Flat head Flat head with
incongruent hip joint
Gold standard for rating residual femoral head deformity and joint
congruence
Clinical presentation:
-insidious onset
-may cause
painless limp
-intermittent
knee, hip, groin
or thigh pain
Physical Exam:
Symptoms:
-Trendelenburg gait
-antalgic limp
limb length
discrepancy is a late
finding
-hip stiffness with loss
of internal rotation
and abduction
-gait disturbance:
Investigations:
 Plain radiographs:
AP of pelvis and frog leg laterals
early findings include:
 medial joint space widening (earliest)
 irregularity of femoral head ossification
 cresent sign (represents a subchondral fracture)
MRI
can provide early diagnosis revealing alterations in the
capital femoral epiphysis and physis.
 Bone scan:
can confirm suspected case of LCP
decreased uptake (cold lesion) can predate changes on radiographs
Arthrogram
a dynamic arthrogram can demonstrate coverage and containment
of the femoral head
Differential Diagnosis
 multiple epiphyseal dysplasia
 spondyloepiphyseal dysplasia
 sickle cell disease
 Gaucher disease
 hypothyroidism
 Meyers dysplasia
Treatment:
 The main Goals of treatment:
 1-keep the femoral head contained and
maintain good motion
 2-containment limits deformity and minimizes loss
of sphericity and lessen subsequent
degenerative changes.
Non-operative:
observation alone, activity restriction, and physical therapy
Indications:
1-children < 8 years of age
2-children with lateral pillar A
3-consider activity restriction and protected weight-bearing
during earlier stages until reossification is complete
techniques:
Cast Brace
Operative:
 Femoral or pelvic osteotomy
Indications:
1-children > 8 years of age, especially lateral pillar B and B/C
improved outcomes with surgery for lateral pillar B and B/C in
children > 8 years
poor outcome for lateral pillar C regardless of treatment.
Pelvic osteotomy:
 Dega Osteotomy
 Salter (Innominate) Osteotomy
Femoral osteotomy:
 Varus femoral osteotomy
Prognosis:
 prognosis worse with:
1-age (bone age) > 6 years at presentation
2-female sex
3-decreased hip range of motion (abduction)
 prognosis improved with:
1-age (bone age) < 6 years at presentation
Complications:
 The head of the femur may lose its normal, spherical
shape and/or collapse.
 Also, degenerative joint disease can occur (i.e. as
occurs in osteoarthritis).
 The affected leg may lose some of its motion and may
become shorter than the normal leg.
References:
Johns Hopkins Pediatric Orthopaedics
Patient
Thank You

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legg calve Perthes disease

  • 1. Legg calve perthes disease By: Ala’a AlGhanem | 211508057 | B1
  • 2. Learning Objectives:  Normal anatomy of hip joint.  Introduction and epidemiology  Aetiology and classifications  Clinical presentation  Investigations  Treatment and prognosis  Complications
  • 3. Anatomy of hip joint:
  • 4.
  • 5. Legg calve perthes disease Idiopathic avascular necrosis of the proximal femoral epiphysis in children Legg Calve Perthes
  • 7. 3-Age 4-8 years is most common age of presentation 4-Population: more commonly seen in urban populations versus rural 5-Location: bilateral in 12% ( never at the same stage of disease)
  • 8. the exact cause of disruption of blood supply remains unknown. Etiology: Associated conditions: -ADHD (33%) -delayed bone age (98%) -Thrombophilia (50%)
  • 9. how Avascular necrosis (AVN) develops:
  • 10. Risk Factors: 1-positive family history 2-low birth weight 3-abnormal birth presentation 4-children exposed to second hand smoke 5-Asian, Inuit, and Central European decent
  • 11. Classifications:  Waldenström  Lateral Pillar (Herring ) Classification  Catterall Classification  Salter-Thompson classification  Stulberg classification
  • 12. Stages of Legg-Calves-Perthes (Waldenström) Initial stage (infarction) Fragmentation Reossification Healing or remodeling
  • 13. Lateral Pillar (Herring ) Classification Determined at the beginning of fragmentation stage lateral pillar maintains full height Maintains >50% height Maintains <50% height
  • 14. Catterall Classification Based on degree of head involvement
  • 15. Salter-Thompson classification Based on radiographic cresent sign crescent sign involves < 1/2 of femoral head crescent sign involves > 1/2 of femoral head Class A Class B
  • 16. Stulberg classification normal Spherical head with enlargement, short neck, or steep acetabulum Nonspherical head Flat head Flat head with incongruent hip joint Gold standard for rating residual femoral head deformity and joint congruence
  • 17. Clinical presentation: -insidious onset -may cause painless limp -intermittent knee, hip, groin or thigh pain Physical Exam: Symptoms: -Trendelenburg gait -antalgic limp limb length discrepancy is a late finding -hip stiffness with loss of internal rotation and abduction -gait disturbance:
  • 18. Investigations:  Plain radiographs: AP of pelvis and frog leg laterals early findings include:  medial joint space widening (earliest)  irregularity of femoral head ossification  cresent sign (represents a subchondral fracture)
  • 19. MRI can provide early diagnosis revealing alterations in the capital femoral epiphysis and physis.  Bone scan: can confirm suspected case of LCP decreased uptake (cold lesion) can predate changes on radiographs Arthrogram a dynamic arthrogram can demonstrate coverage and containment of the femoral head
  • 20. Differential Diagnosis  multiple epiphyseal dysplasia  spondyloepiphyseal dysplasia  sickle cell disease  Gaucher disease  hypothyroidism  Meyers dysplasia
  • 21. Treatment:  The main Goals of treatment:  1-keep the femoral head contained and maintain good motion  2-containment limits deformity and minimizes loss of sphericity and lessen subsequent degenerative changes.
  • 22. Non-operative: observation alone, activity restriction, and physical therapy Indications: 1-children < 8 years of age 2-children with lateral pillar A 3-consider activity restriction and protected weight-bearing during earlier stages until reossification is complete
  • 24. Operative:  Femoral or pelvic osteotomy Indications: 1-children > 8 years of age, especially lateral pillar B and B/C improved outcomes with surgery for lateral pillar B and B/C in children > 8 years poor outcome for lateral pillar C regardless of treatment.
  • 25. Pelvic osteotomy:  Dega Osteotomy  Salter (Innominate) Osteotomy
  • 26. Femoral osteotomy:  Varus femoral osteotomy
  • 27. Prognosis:  prognosis worse with: 1-age (bone age) > 6 years at presentation 2-female sex 3-decreased hip range of motion (abduction)  prognosis improved with: 1-age (bone age) < 6 years at presentation
  • 28. Complications:  The head of the femur may lose its normal, spherical shape and/or collapse.  Also, degenerative joint disease can occur (i.e. as occurs in osteoarthritis).  The affected leg may lose some of its motion and may become shorter than the normal leg.
  • 29. References: Johns Hopkins Pediatric Orthopaedics Patient