1. Chronic pain
From Wikipedia, the free encyclopedia
Chronic pain
ICD-10
R52.1-R52.2
ICD-9
338.2
Chronic pain is defined as pain that has lasted longer than three to six months,[1][2] though some theorists
and researchers have placed the transition from acute to chronic pain at 12 months. [3] Others apply acute to
pain that lasts less than 30 days, chronic to pain of more than six months duration, and subacute to pain that
lasts from one to six months.[4] A popular alternative definition of chronic pain, involving no arbitrarily
fixed durations is "pain that extends beyond the expected period of healing". [2]
Contents
1 Classification
2 Pathophysiology
3 Management
4 Epidemiology
5 Comorbidities and sequelae
6 Psychology
o 6.1 Psychosocial factors
o 6.2 Personality
o 6.3 Effect on cognition
7 See also
8 References
9 External links
Classification
Main article: Pain#Classification
Chronic pain may be divided into "nociceptive" (caused by activation of nociceptors), and "neuropathic"
(caused by damage to or malfunction of the nervous system). [5]
Nociceptive pain may be divided into "superficial" and "deep", and deep pain into "deep somatic" and
"visceral". Superficial pain is initiated by activation of nociceptors in the skin or superficial tissues. Deep
somatic pain is initiated by stimulation of nociceptors in ligaments, tendons, bones, blood vessels, fasciae
and muscles, and is dull, aching, poorly-localized pain. Visceral pain originates in the viscera (organs).
Visceral pain may be well-localized, but often it is extremely difficult to locate, and several visceral regions
produce "referred" pain when damaged or inflamed, where the sensation is located in an area distant from
the site of pathology or injury. [6]
Neuropathic pain is divided into "peripheral" (originating in the peripheral nervous system) and "central"
(originating in the brain or spinal cord).[7] Peripheral neuropathic pain is often described as "burning",
"tingling", "electrical", "stabbing", or "pins and needles".[8]
Pathophysiology
Under persistent activation nociceptive transmission to the dorsal horn may induce a wind up phenomenon.
This induces pathological changes that lower the threshold for pain signals to be transmitted. In addition it

2. may generate nonnociceptive nerve fibers to respond to pain signals. Nonnociceptive nerve fibers may also
be able to generate and transmit pain signals. In chronic pain this process is difficult to reverse or eradicate
once established.[9]
Chronic pain of different etiologies has been characterized as a disease affecting brain structure and
function. Magnetic resonance imaging studies have shown abnormal anatomical[10] and functional
connectivity, even during rest[11][12] involving areas related to the processing of pain. Also, persistent pain
has been shown to cause grey matter loss, reversible once the pain has resolved.[13][14]
These structural changes can be explained by the phenomenon known as neuroplasticity. In the case of
chronic pain, the somatototic representation of the body is inappropriately reorganized following peripheral
and central sensitization. This maladaptative change results in the experience of allodynia and/or
hyperalgesia. Brain activity in individuals suffering from chronic pain, measured via electroencephalogram
(EEG), has been demonstrated to be altered, suggesting pain-induced neuroplastic changes. More
specifically, the relative beta activity (compared to the rest of the brain) is increased, the relative alpha
activity is decreased, and the theta activity both absolutely and relatively is diminished.[15]
Management
Main article: Pain management
Complete and sustained remission of many neuropathies and most idiopathic chronic pain (pain that
extends beyond the expected period of healing, or chronic pain that has no known underlying pathology) is
rarely achieved, but much can be done to reduce suffering and improve quality of life. [16]
Pain management is the branch of medicine employing an interdisciplinary approach to the relief of pain
and improvement in the quality of life of those living with pain.[17] The typical pain management team
includes medical practitioners, clinical psychologists, physiotherapists, occupational therapists, physician
assistants, and nurse practitioners.[18]Acute pain usually resolves with the efforts of one practitioner;
however, the management of chronic pain frequently requires the coordinated efforts of the treatment
team.[19][20][21]
Psychological treatments including cognitive behavioral therapy[22][23] and acceptance and commitment
therapy[24][25][26] have been shown effective for improving quality of life in those suffering from chronic
pain. Clinical hypnosis, including self-hypnosis, has been shown effective not only for improving quality of
life, but for direct improvement of chronic pain symptoms. [27][28][29]
The emergence of studies relating chronic pain to neuroplasticity also suggest the utilization of
neurofeedback rehabilitation techniques to resolve maladaptive cortical changes and patterns. [15] The
proposed goal of neurofeedback intervention is to abolish maladaptive neuroplastic changes made as a
result of chronic nociception, as measured by abnormal EEG, and thereby relieve the individual's pain.
However, this field of research lacks randomized control trials, and therefore requires further investigation.
Epidemiology
In a recent large-scale telephone survey of 15 European countries and Israel, 19% of respondents over 18
years of age had suffered pain for more than 6 months, including the last month, and more than twice in the
last week, with pain intensity of 5 or more for the last episode, on a scale of 1(no pain) to 10 (worst
imaginable). 4839 of these respondents with chronic pain were interviewed in depth. Sixty six percent
scored their pain intensity at moderate (5–7), and 34% at severe (8–10); 46% had constant pain, 56%
intermittent; 49% had suffered pain for 2–15 years; and 21% had been diagnosed with depression due to the
pain. Sixty one percent were unable or less able to work outside the home, 19% had lost a job, and 13% had
changed jobs due to their pain. Forty percent had inadequate pain management and less than 2% were
seeing a pain management specialist.[30]
In a systematic literature review published by the International Association for the Study of Pain (IASP), 13
chronic pain studies from various countries around the world were analyzed. (Of the 13 studies, there were
three in the United Kingdom, two in Australia, one each in France, the Netherlands, Israel, Canada,
Scotland, Spain, and Sweden, and a multinational.) The authors found that the prevalence of chronic pain
was very high and that chronic pain consumes a large amount of healthcare resources around the globe.
Chronic pain afflicted women at a higher rate than men. They determined that the prevalence of chronic
pain varied from 10.1% to 55.2% of the population. [31]

3. In the United States, the prevalence of chronic pain has been estimated to be approximately 30%.
According to the Institute of Medicine, there are about 116 million Americans living with chronic pain. [1][32]
The Mayday Fund estimate of 70 million Americans with chronic pain is slightly more conservative. [33] In
an internet study, the prevalence of chronic pain in the United States was calculated to be 30.7% of the
population: 34.3% for women and 26.7% for men.[34] These estimates are in reasonable agreement and
indicate a prevalence of chronic pain in the US that is relatively comparable to that of other countries.[citation
needed]
Comorbidities and sequelae
Chronic pain is associated with higher rates of depression and anxiety. [35]Sleep disturbance, and insomnia
due to medication and illness symptoms are often experienced by those with chronic pain.[36] Chronic pain
may contribute to decreased physical activity due to fear of exacerbating pain, often resulting in weight
gain.[35] Such comorbid disorders can be very difficult to treat due to the high potential of medication
interactions, especially when the conditions are treated by different doctors.
Psychology
Psychosocial factors
Those who have a tendency to "catastrophise" in response to pain report greater pain levels induced by an
ice bath (Sullivan et al., 1995).[37] A study by Crombez et al. (1998) showed that those with unhealthy
catastrophising pain beliefs experienced greater impairment in performance in an auditory discrimination
task when a painful stimulus was applied then those with more healthy pain beliefs. [38] In addition there was
a perception of pain at greater intensity during the experiment. This indicates that there is an increase in
attention to pain at the expense of other somatosensation. Indeed Crombez et al. (2005) postulated that in
chronic pain patients are thought to display a “hyper-vigilance” to pain.[38] In addition, pain inhibited visual
attention disengagement in those with a tendency to catastrophise though not in non-catastrophisers (Van
Damme et al., 2004).[39]
Personality
Two of the most frequent personality profiles found in chronic pain patients by the Minnesota Multiphasic
Personality Inventory (MMPI) are the conversion V and the neurotic triad. The conversion V personality,
so called because the higher scores on MMPI scales 1 and 3, relative to scale 2, form a "V" shape on the
graph, expresses exaggerated concern over body feelings, develops bodily symptoms in response to stress,
and often fails to recognize their own emotional state, including depression. The neurotic triad personality,
scoring high on scales 1, 2 and 3, also expresses exaggerated concern over body feelings and develops
bodily symptoms in response to stress, but is demanding and complaining. [40]
Some investigators have argued that it is this neuroticism that causes acute pain to turn chronic, but clinical
evidence points the other way, to chronic pain causing neuroticism. When long term pain is relieved by
therapeutic intervention, scores on the neurotic triad and anxiety fall, often to normal levels.[41][42][43][44]
Self-esteem, often low in chronic pain patients, also shows striking improvement once pain has resolved. [44]
Effect on cognition
Chronic pain's impact on cognition is an under-researched area, but several tentative conclusions have been
published. Most chronic pain patients complain of cognitive impairment, such as forgetfulness, difficulty
with attention, and difficulty completing tasks. Objective testing has found that people in chronic pain tend
to experience impairment in attention, memory, mental flexibility, verbal ability, speed of response in a
cognitive task, and speed in executing structured tasks. In 2007, Shulamith Kreitler and David Niv advised
clinicians to assess cognitive function in chronic pain patients in order to more precisely monitor
therapeutic outcomes, and tailor treatment to address this aspect of the pain experience.[45]
See also
Chronic pain syndrome

4. Suffering
Conditions related to pain
Arthritis
Ehlers Danlos Syndrome
Temporomandibular joint dysfunction
Back pain
Cancer
Chronic Fatigue Syndrome
Clinical depression
Complex Regional Pain Syndrome
Fibromyalgia
Headache
Interstitial Cystitis
Irritable Bowel Syndrome
Myofascial Pain Syndrome
Neuropathic pain
Pelvic pain
Neuropathy
Post-Vasectomy Pain Syndrome
Restless Leg Syndrome
Sciatica
Spasmodic Torticollis
Drugs
Analgesia
Antiepileptics
o Gabapentin
o Pregabalin
o Levetiracetam
o Topiramate
o Lamotrigine
o Zonisamide
Antidepressants
o Duloxetine
o Amitriptyline
o Nortriptyline
o Milnacipran
Local anesthetics
o Ketamine
Acetaminophen (Paracetamol)
NSAIDs
Opioids
Medical cannabis
Other agents:
o Clonidine
o Ziconotide
o Tizanidine
Other approaches in Physical medicine and rehabilitation (Physiatry)
Cryotherapy
Exercise
Hot pack
Occupational therapy
Physical therapy

5. TENS
Alternative therapies
Acupuncture
Chiropractic
Massage therapy
Hypnosis
Behavioral therapy
Prolotherapy
Structural Integration
Surgery
Spinal cord stimulation
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