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HISTORY TAKING & PHYSICAL
EXAMINATION
Dr.Bilal Jamal Kamal
MB ChB
FIBMS
MRCP(UK)
Part2
HITORY TAKING
Tips for effective conversations:
1. Choose a quiet & private space
2. Speak clearly & audibly
3. Ask open questions to start with
4. Don’t interrupt your patient
5. Try & appear unharried
6. Use silence to encourage explanations
7. Do not use jargon or emotive words
8.Clarify & summarize what you understand
9. Acknowledge emotions
10. Seek ideas, concerns & expectations
History taking usually takes 15 min, but for
students, it may take upto 30 min.
Arrange seating in a non-confrontational way.
Look for non-verbal cues e.g. distress & mood.
Active listening:
Hear your patients story, ask open questions to
start with, e.g. What has brought you along to
see me today?
Active listening means encouraging the patient
to talk by looking interested, making
encouraging comments.
Closed questions, e.g. Have you had cough
today? Seek specific information as part of a
systemic enquiry. They invite yes or no
answers. Both types of questions have their
places.
1. Demographic data: name, age, gender,
address, marital status, date of addmission
2. Chief complain
3. History of present illness;
Characteristic of pain (SOCRATES)
Site, somatic is well localized, visceral is diffuse
Onset, speed of onset & any associated
circumstances
Character, sharp, dull, burning, stabbing
Radiation
Associated symptoms
Timing (duration, course, pattern)
Exacerbating & relieving factors
Severity
Past history:
Have you had any serious illness that brought
you to see doctor?
Have you had any operations?
Have you attended any hospital clinics?
Drug history
Ask about prescribed drugs, OTC, herbal,
traditional.
Ask about
compliance,adherance,dosage,regimen,durati
on.
DRUG ALLERGIES
Family history:
Diseases run in the family??
Social history:
Helps you to understand the context of the patients
life & possible relevant factors.
Lifestyle: exercise, diet, travel history, sexual history,
smooking, alcohol, non-prescribed drugs
Occupation
House
Leisure activities
Systemic enquiry
Cardiovascular system: chest pain,breathlessness
Palpitation,leg swelling
Respiratory: cough,dyspnea,sputum,blood in
sputum, chest pain
GIT: mouth, difficulty in swelling,nausea,vomitin
Blood in vomiting,heartburn,bowel motion
Genitourinary: Dysurea,blood in urine,
Frequency, libido
CNS: headache,fits,weakness,visual diturbance
Memory change
Endocrine: heat & cold intolerance, sweating
Musculoskeletal: joint pain, stiffness, mobility
Women: menstrual history
PHYSICAL EXAMINATION
• Privacy is essential when you examine a patient
• Talk quietly but ensure good communication
• The room should be warm and well lit
• Subtle abnormalities of complexion such as mild
jaundice are easier to detect in natural light
• cannot lie flat.
• Seek permission and sensitively, but adequately,
expose the areas of the body to be examined;
cover the rest of the patient with a blanket or
sheet to ensure that he or she does not become
cold. Avoid unnecessary exposure and
embarrassment
• The sequence of examination is:
• Inspection
• Palpation
• Percussion
• Auscultation
Introduce yourself to the patient (Handshake &
first impressions)
History taking from the patient
Differential diagnosis
Examine the patient(General & systemic)
Confirm or refute your diagnosis
First Impressions:
The physical examination starts as soon as you see
the patient. Assess patients’ general demeanour
and external appearance, and watch how they
rise from their chair and walk into the room.
The handshake:
Introduce yourself and shake hands. This may
provide diagnostic clues . Greet your patient in a
friendly but professional manner. Note if his right
hand works; in patients with a right hemiparesis
you may need to shake his left hand. Avoid too
firm a grip, particularly in patients with arthritis.
Facial expression and general
demeanour:
Actively recognise the features of anxiety, fear,
anger.
Clothing:
Clothing gives clues about personality, state of
mind and social circumstances. Young people
wearing dirty clothes may have problems with
alcohol or drug addiction.
Complexion:
Facial colour depends on oxyhaemoglobin, reduced
haemoglobin, melanin and carotene. Unusual
skin colours are due to abnormal pigments.
Haemoglobin: Untanned European skin is pink due
to the red pigment oxyhaemoglobin in the
superficial capillary–venous plexuses. A pale
complexion may be misleading(because it can be
caused by vasoconstriction during faint or fear)
but can suggest anaemia. The pallor of anaemia is
best seen in the mucous membranes of the
conjunctivae, lips and tongue and in the nail
beds. Facial plethora is caused by raised
haemoglobin concentration with elevated
haematocrit (polycythaemia).
Cyanosis:
Cyanosis is a blue discoloration of the skin and
mucous membranes that occurs when the
absolute concentration of deoxygenated
haemoglobin is increased.
• Central cyanosis
• Central cyanosis of the lips.This is seen at the
lips and tongue . It corresponds to an arterial
oxygen saturation (SpO2) of <90% and usually
indicates underlying cardiac or pulmonary ot
due to increment of deoxygenated Hb > 5 g/dl
• Peripheral cyanosis
This occurs in the hands, feet or ears, usually
when they are cold. In healthy people it occurs
in cold conditions when prolonged peripheral
capillary flow allows greater oxygen extraction
and hence increased levels of
deoxyhaemoglobin. In combination with
central cyanosis, it is most often seen with
poor peripheral circulation due to shock, heart
failure.
• Melanin
Causes of abnormal melanin productionSkin
colour is greatly influenced by the deposition
of melanin.
Vitiligo:
This chronic condition produces bilateral
symmetrical depigmentation, commonly of
the face, neck and extensor aspects of the
limbs, resulting in irregular pale patches of
skin.
Albinism:
This is an inherited disorder in which patients
have little or no melanin in their skin or hair
• Overproduction of melanin
This can be due to excess of the pituitary
hormone, adrenocorticotrophic hormone, as
in adrenal insufficiency
• Pregnancy and oral contraceptives
These may produce chloasma (blotchy
pigmentation of the face)
Carotene: Hypercarotenaemia occurs in people
who eat large amounts of raw carrots and
tomatoes, and in hypothyroidism. A yellowish
discoloration is seen on the face, palms and
soles, but not the sclerae, and this
distinguishes it from jaundice
Bilirubin: Jaundice is detectable when serum
bilirubin concentration is elevated and the
sclerae, mucous membranes and skin become
yellow.
Iron: Haemochromatosis increases skin
pigmentation due to iron deposition and
increased melanin production
m
The Hands:
Examination sequence:
■ Inspect the dorsal and then palmar aspects of
both hands.
■ Note changes in the:
■ skin
■ nails
■ soft tissues (evidence of muscle wasting)
■ tendons
■joints.
■ Feel the temperature.
Abnormal findings :
Deformity
Colour
Temperature
Skin
Finger clubbing Clubbing: is painless soft-tissue
swelling of the terminal phalanges. The
enlargement increases convexity of the nail
with loss of nail fold angle.
Cause of finger clubbing:
Lung diseses: abscess, bronchiectasis, fibrosis
Heart diseases: cyanotic congenital heart
disease, Infective endocarditis
GIT: Cirrhosis , IBD, Celiac disease
THE TONGUE:
Examination sequence :
Ask the patient to put out his tongue.
Look at the size, shape, movements, colour and
surface
THE LYMPH NODES :
Lymph nodes may be palpable in normal people,
especially in the submandibular, axilla and groin
regions . Distinguish between normal and
pathological nodes. Pathological
lymphadenopathy may be local or generalised,
and is of diagnostic and prognostic significance in
the staging of lymphoproliferative and other
malignancies.
HISTORY TAKING & PHYSICAL EXAMINATION.pptx
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HISTORY TAKING & PHYSICAL EXAMINATION.pptx

  • 1. HISTORY TAKING & PHYSICAL EXAMINATION Dr.Bilal Jamal Kamal MB ChB FIBMS MRCP(UK) Part2
  • 2. HITORY TAKING Tips for effective conversations: 1. Choose a quiet & private space 2. Speak clearly & audibly 3. Ask open questions to start with 4. Don’t interrupt your patient 5. Try & appear unharried 6. Use silence to encourage explanations 7. Do not use jargon or emotive words 8.Clarify & summarize what you understand 9. Acknowledge emotions 10. Seek ideas, concerns & expectations
  • 3. History taking usually takes 15 min, but for students, it may take upto 30 min. Arrange seating in a non-confrontational way. Look for non-verbal cues e.g. distress & mood. Active listening: Hear your patients story, ask open questions to start with, e.g. What has brought you along to see me today? Active listening means encouraging the patient to talk by looking interested, making encouraging comments.
  • 4. Closed questions, e.g. Have you had cough today? Seek specific information as part of a systemic enquiry. They invite yes or no answers. Both types of questions have their places.
  • 5. 1. Demographic data: name, age, gender, address, marital status, date of addmission 2. Chief complain 3. History of present illness; Characteristic of pain (SOCRATES) Site, somatic is well localized, visceral is diffuse Onset, speed of onset & any associated circumstances Character, sharp, dull, burning, stabbing Radiation
  • 6. Associated symptoms Timing (duration, course, pattern) Exacerbating & relieving factors Severity
  • 7. Past history: Have you had any serious illness that brought you to see doctor? Have you had any operations? Have you attended any hospital clinics?
  • 8. Drug history Ask about prescribed drugs, OTC, herbal, traditional. Ask about compliance,adherance,dosage,regimen,durati on. DRUG ALLERGIES
  • 9. Family history: Diseases run in the family?? Social history: Helps you to understand the context of the patients life & possible relevant factors. Lifestyle: exercise, diet, travel history, sexual history, smooking, alcohol, non-prescribed drugs Occupation House Leisure activities
  • 10. Systemic enquiry Cardiovascular system: chest pain,breathlessness Palpitation,leg swelling Respiratory: cough,dyspnea,sputum,blood in sputum, chest pain GIT: mouth, difficulty in swelling,nausea,vomitin Blood in vomiting,heartburn,bowel motion Genitourinary: Dysurea,blood in urine, Frequency, libido CNS: headache,fits,weakness,visual diturbance Memory change Endocrine: heat & cold intolerance, sweating Musculoskeletal: joint pain, stiffness, mobility Women: menstrual history
  • 11. PHYSICAL EXAMINATION • Privacy is essential when you examine a patient • Talk quietly but ensure good communication • The room should be warm and well lit • Subtle abnormalities of complexion such as mild jaundice are easier to detect in natural light • cannot lie flat. • Seek permission and sensitively, but adequately, expose the areas of the body to be examined; cover the rest of the patient with a blanket or sheet to ensure that he or she does not become cold. Avoid unnecessary exposure and embarrassment
  • 12. • The sequence of examination is: • Inspection • Palpation • Percussion • Auscultation
  • 13. Introduce yourself to the patient (Handshake & first impressions) History taking from the patient Differential diagnosis Examine the patient(General & systemic) Confirm or refute your diagnosis
  • 14. First Impressions: The physical examination starts as soon as you see the patient. Assess patients’ general demeanour and external appearance, and watch how they rise from their chair and walk into the room. The handshake: Introduce yourself and shake hands. This may provide diagnostic clues . Greet your patient in a friendly but professional manner. Note if his right hand works; in patients with a right hemiparesis you may need to shake his left hand. Avoid too firm a grip, particularly in patients with arthritis.
  • 15. Facial expression and general demeanour: Actively recognise the features of anxiety, fear, anger. Clothing: Clothing gives clues about personality, state of mind and social circumstances. Young people wearing dirty clothes may have problems with alcohol or drug addiction.
  • 16. Complexion: Facial colour depends on oxyhaemoglobin, reduced haemoglobin, melanin and carotene. Unusual skin colours are due to abnormal pigments. Haemoglobin: Untanned European skin is pink due to the red pigment oxyhaemoglobin in the superficial capillary–venous plexuses. A pale complexion may be misleading(because it can be caused by vasoconstriction during faint or fear) but can suggest anaemia. The pallor of anaemia is best seen in the mucous membranes of the conjunctivae, lips and tongue and in the nail beds. Facial plethora is caused by raised haemoglobin concentration with elevated haematocrit (polycythaemia).
  • 17. Cyanosis: Cyanosis is a blue discoloration of the skin and mucous membranes that occurs when the absolute concentration of deoxygenated haemoglobin is increased. • Central cyanosis • Central cyanosis of the lips.This is seen at the lips and tongue . It corresponds to an arterial oxygen saturation (SpO2) of <90% and usually indicates underlying cardiac or pulmonary ot due to increment of deoxygenated Hb > 5 g/dl
  • 18. • Peripheral cyanosis This occurs in the hands, feet or ears, usually when they are cold. In healthy people it occurs in cold conditions when prolonged peripheral capillary flow allows greater oxygen extraction and hence increased levels of deoxyhaemoglobin. In combination with central cyanosis, it is most often seen with poor peripheral circulation due to shock, heart failure.
  • 19. • Melanin Causes of abnormal melanin productionSkin colour is greatly influenced by the deposition of melanin. Vitiligo: This chronic condition produces bilateral symmetrical depigmentation, commonly of the face, neck and extensor aspects of the limbs, resulting in irregular pale patches of skin. Albinism: This is an inherited disorder in which patients have little or no melanin in their skin or hair
  • 20. • Overproduction of melanin This can be due to excess of the pituitary hormone, adrenocorticotrophic hormone, as in adrenal insufficiency • Pregnancy and oral contraceptives These may produce chloasma (blotchy pigmentation of the face)
  • 21. Carotene: Hypercarotenaemia occurs in people who eat large amounts of raw carrots and tomatoes, and in hypothyroidism. A yellowish discoloration is seen on the face, palms and soles, but not the sclerae, and this distinguishes it from jaundice Bilirubin: Jaundice is detectable when serum bilirubin concentration is elevated and the sclerae, mucous membranes and skin become yellow. Iron: Haemochromatosis increases skin pigmentation due to iron deposition and increased melanin production
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  • 25. The Hands: Examination sequence: ■ Inspect the dorsal and then palmar aspects of both hands. ■ Note changes in the: ■ skin ■ nails ■ soft tissues (evidence of muscle wasting) ■ tendons ■joints. ■ Feel the temperature.
  • 26. Abnormal findings : Deformity Colour Temperature Skin Finger clubbing Clubbing: is painless soft-tissue swelling of the terminal phalanges. The enlargement increases convexity of the nail with loss of nail fold angle.
  • 27. Cause of finger clubbing: Lung diseses: abscess, bronchiectasis, fibrosis Heart diseases: cyanotic congenital heart disease, Infective endocarditis GIT: Cirrhosis , IBD, Celiac disease
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  • 30. THE TONGUE: Examination sequence : Ask the patient to put out his tongue. Look at the size, shape, movements, colour and surface THE LYMPH NODES : Lymph nodes may be palpable in normal people, especially in the submandibular, axilla and groin regions . Distinguish between normal and pathological nodes. Pathological lymphadenopathy may be local or generalised, and is of diagnostic and prognostic significance in the staging of lymphoproliferative and other malignancies.