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Developmental
Anomalies
of teeth
M.Ekram
Chapter 18
White & Pharoh
Page 330
Reference:
M.Ekram
Causes of Developmental Anomalies
1. Genetic: inherited or gene mutation.
2. Environmental causes:
Infection, trauma, radiation.
hormonal disturbances.
nutritional deficiencies.
hypoxia
miscellaneous
M.Ekram
Classification
Size Shape Number Structure
Disturbance
in eruption
Microdontia
macrodontia
gemination,
fusion.
Concrescence.
Dilaceration.
Talon cusp.
dense-in-dent.
Dens
invaginatus
Taurodontism.
Missing teeth
supernumerary
Amelogenesis
dentinogenisis
imperfecta.
Dentin
dysplasia
Trasposition
premature
delayed
eruption
M.Ekram
Anomalies in tooth size
Microdontia
True:
Generalized → all teeth.
e.g. in dwarfism
Single, group of teeth → #2 (peg shape )
→ # 8 (fused roots)
Relative Microdontia: as normal sized teeth in
large mandible
Clinically: identified easily
M.Ekram
Radiographically:
small sized crown and root
Microdontia (cont.)
M.Ekram
Macrodontia
True
generalized Localized
all teeth in facial hemi-
in Gigantism hypertrophy
clinically: single macrodont is
difficult to be identified from
gemination or fusion
Relative
normal or slightly
large teeth in small
jaw
M.Ekram
Developmental Anomalies in
Teeth Number
Increased number
supernumerary teeth.
Predeciduous dent.
Post permanent dent.
Decreased number
Hypodontia
Oligodontia
Anodontia.
partial-
anodontia
M.Ekram
Supernumerary Teeth
Arise from extra-teeth buds.
Rare in deciduous teeth.
May resemble the tooth anatomically or
not.
M.Ekram
Supernumerary Teeth
Sites
any where, But the most
common are:
Mesiodense: between
#1,#1. Small, conical
inverted, impacted,
short root.
Paramolars: located
around the premolar-
molars.
M.Ekram
Supernumerary Teeth
Distomolar: Distal to # 8,
small, rudimentary.
M.Ekram
Clinical Picture
May prevent eruption or displacement of normal teeth
or cause tooth resorption.
Multiple Supernumerary Teeth Are manifested in :
1. Gardner’s Syndrome: multiple supernumerary and
impacted teeth, multiple polyps of large
intestine,multiple osteomas , sebaceous cysts.
M.Ekram
Abnormalities
Bifid ribs and other skeletal abnormalities.
Multiple jaw cysts.
Multiple nevoid basal cell carcinomas.
Characteristic frontal
bossing and broad
nasal root
Bifid ribs Multiple cyst - like radiolucencies
1. Gorlin-Goltz Syndrome: (basal cell nevous-
bifid rib syndrome).
M.Ekram
3. Cleido-cranial dysostosis: large skull with
mental retardation, open fontanells, small
maxilla, high arched palate, absent or
hypoplastic clavicles.
M.Ekram
Predeciduous Dentition
Hornified structure at birth
or just after.
Due to extra-teeth buds.
Most common in lower
anterior area.
Present just above the
alveolar ridge, so easily
removed.
NB: we should differentiate
between it and deciduous
teeth before attempting
removal.
M.Ekram
Post-permanent Dentition
Third Dentition
After extraction of all teeth and
construction of denture, new teeth
begin to erupt.
They may be either impacted.
Or supernumerary unerupted teeth.
M.Ekram
M.Ekram
M.Ekram
Decrease Number of Teeth
Missing teeth - Anodontia
Total Anodontia:
failure of odontogenesis
rare but may accompany:
H. Ectodermal Dysplasia:
with dry skin, absence of
sweet glands, scanty hair,
patient can not tolerate
heat.
Partial Anodontia:
few teeth: Hypodontia
Many teeth: Oligodontia.
More common than total A.
Frequency:
#8 > #2 >#5 in permanent.
Upper # B > lower # B
M.Ekram
Hereditary
Radiation to the head in a very early stage of
development with either a total destruction of
teeth buds, or the teeth are partially developed
and hypo-calcified with stunted growth.
Diagnosis:
proper history and exclusion of extraction help
in diagnosis. Then radiographic examination
confirms the clinical ex.
Etiology (Anodontia)
M.Ekram
Anodontia
M.Ekram
Ectodermal dysplasia
Fine scanty hairs Anodontia
M.Ekram
Anodontia
M.Ekram
Ectodermal dysplasia
M.Ekram
Anomalies in eruption of teeth
Transposition
1. Permanent canine and 1st premolar.
2. Second premolar (between 6 & 7).
3. Central & lateral incisors.
N.B. Transposition was not reported in Pry teeth but may be
with hypodontia or supernumerary teeth.
Teeth commonly involved
M.Ekram
Anomalies in tooth Shape
Gemination (twinning)
Partial division
tooth with a single root and
a crown that is divided
totally or partially.
Complete division
complete separation with
formation of 2 teeth with
crowns and roots.
N.B. the result is a normal teeth
No + supernumerary tooth.
A single tooth bud divides by an
invagination
M.Ekram
Complete
separationPartial division
M.Ekram
Clinical Picture of Gemination
Site:
anterior (deciduous or
permanent).
Structure:
enamel and dentin may
be hypoplastic or
hypocalcified.
M.Ekram
Radiographic Picture of Gemination
Large tooth with well
defined enamel
separating the two
parts.
One large pulp
chamber or partially
divided one.
M.Ekram
Gemination
Divided pulp chamber One pulp chamber
M.Ekram
Clinical implications of Gemination
Poor esthetics due to a partially divided tooth.
Hypoplastic enamel and increased caries
susceptibility
Malocclusion and periodontal problems.
M.Ekram
Fusion
Union of two normally separated teeth.
Theories and types
1- fusion occurs early before
calcification:
the tooth formed is one single large
tooth.
2- fusion occurs late after
calcification:
there may be union in the roots only
with two separate or single root canal.
M.Ekram
Clinical Picture of Fusion
Decreased number of
teeth by one.
Occurs more in
deciduous teeth.
More in anterior.
The crown may be
bifid
M.Ekram
What happens when fusion occurs between
normal tooth and supernumerary tooth ?
it will be very difficult to be differentiated
from gemination !!!!!
M.Ekram
Radiographic picture of fusion
Single root with cleft crown
(fusion occurs late after calcification had started)
M.Ekram
fusion
Fusion In deciduous teeth
2 separate root canals
M.Ekram
Differences between
fusion and gemination
Fusion
Number of teeth is less by
one except if fusion
occurs with extra-tooth.
Tooth with 2 separate
Root Canals with one or
two roots.
Gemination
Number of teeth is
correct or increased by
one.
Tooth with large pulp
chamber with either
clefted or partially
divided crown.
N.B. Also from macrodontia
M.Ekram
M.Ekram
Concrescence
It is a form of fusion but by cementum
only.
Etiology
trauma and crowding of teeth
with resorption of interdental bone
so that teeth are united with
deposition of cementum.
It may occur before or after
development of teeth (true &
acquired).
Significance: during teeth extraction.
M.Ekram
Taurodontism
Describes the increase in
length of the crown on the
expense of root with
elongated pulp chamber
so, it shows increase
distance between the
CEJ and root furcation.
The name derived from similarity of teeth to that of
Cud-chewing animals, so termed Bull-like teeth.
M.Ekram
Etiology: mutation resulting from odotoblastic
deficiency during formation of roots.
Clinically : indistinguishable
M.Ekram
Radiographic features
1.tooth rectangular in shape.
2. Large elongated pulp chamber.
3. Lack of usual constriction at cervical area.
4. Furcation is few mms away from the apex (short root).
M.Ekram
Dilaceration
This term refers to an angle or sharp
bend either in the crown or root.
Etiology: Developmental or could be due to
trauma
Trauma of deciduous teeth may cause
dilaceration of the permanents.
Clinically
Site: any where in the root and may be in
the crown.
More in maxillary anterior teeth.
M.Ekram
Clinical and Radiographic Picture
(Dilaceration)
Teeth may not erupt.
If in the crown: bad
esthetics.
If in the root: has no
significance except in
endodontics and
extraction.
Radiographically
appears if dilaceration is to the mesial or distal.
Appears as well defined RO area with LD in the center
(apical foramen) giving the appearance of “bulls’ eye”
M.Ekram
Dilaceration
Buccal or lingual dilaceration
Distal dilacerationDistal dilaceration
Clear angulation
M.Ekram
Differential Diagnosis of Dilaceration
some times it is difficult to be differentiated from
condensing osteitis,
idiopathic osteosclerosis.
fused roots.
N.B. we take radiographs from different angles
M.Ekram
Dense-in-Dent
It is due to enfolding of the
enamel organ to the
interior during
development and before
calcification.
Etiology:
1- focal growth stimulation
or focal growth
retardation in certain
areas.
2- External trauma.
M.Ekram
Clinical Picture of Dens-in-Dent
Incidence: tooth #2> #1
Site: most common in crowns, but may be in the roots
due to folding of epithelial root sheath of Hertwing.
It Appears as a palatal pit.
N.B. the most extreme form of this anomaly is referred to
as Dilated odontome.
M.Ekram
Clinical Picture of Dens-in-Dent
Clinical significance:
 The palatal pit is difficult to
clean.
very thin enamel separate
the defect from the pulp
chamber.
There is a risk of caries
and pulp necrosis.
M.Ekram
Coronally
pear-shaped area of enamel and
dentin.
Narrow constriction at the
opening .
Closely approximated to the pulp
in depth.
Radiographic Picture of Dens-in-Dent
M.Ekram
Radicular
lined with cementum,
may reach to the apical
foramen (not as defined
as the coronally).
Radiographic Picture of Dens-in-Dent
M.Ekram
Dense-in-Dent
Reported Case
M.Ekram
Dense evaginatus
Leong’s premolar
Clinically:
Occurs in premolars, as
a tubercle of enamel in
the middle of the
occlusal surface.
covered with enamel
and composed of
dentin and a core of
pulp.
It is due to out folding
of the enamel organ.
M.Ekram
Dense evaginatus
Radiographically: extension of pulp covered by
E & D.
Significance:
occlusal interference.
prevention of complete eruption of the
opposing tooth.
wearing with subsequently pulp exposure.
M.Ekram
Talon Cusp
It is projecting from the cingulum of
upper or lower anterior teeth.
Clinical Picture
Surrounded by 2 grooves.
Blends to the surface of the
tooth.
Significance
* May Interfere with occlusion.
* Caries susceptibility
M.Ekram
Radiographic picture of Talon Cusp
Differential diagnosis : Supernumerary teeth (buccal
object rule).
M.Ekram
Enamel Pearl
Is a small globule of enamel1-3 mm
in diameter that occurs on the roots
of molars (furcation area).
DD
1. Isolated piece of calculus
2. Pulp stone (buccal object rule –
vertically).
Anomalies in Tooth
Structure
M.Ekram
Developmental anomaly that affects enamel
formation.
Marked changes in E in either part or all the
teeth in both dentitions.
Dentin and roots are usually normal.
Causes delayed eruption of the affected teeth.
Increases tendency for impaction.
Amelogenesis Imperfecta (AI)
M.Ekram
AI: Types
E. Hypoplastic type. in matrix formation.
E. Hypocalcification in mineralization.
E. Hypomaturation.
Hypomaturation. Hypocalcifid type. (mottled E).
M.Ekram
Clinical Picture (AI)
Hypoplastic type
Thin enamel & dentin is
shown, so the teeth are
dark or brown in color.
Loss of normal tooth
contour and teeth become
undersized.
Lack of proximal contact.
Occlusal surfaces are
usually flat (attrition).
M.Ekram
E Hypomaturation type
Clinical features
Enamel is of normal thickness but mottled.
Enamel can be pierced by probe.
Patient may be affected by different degrees.
Teeth may show vertical grooves.
Teeth may be chalky or dark in color.
radiographically.
Enamel is of normal thickness but with density
similar to D.
M.Ekram
Clinical picture (AI)
E Hypocalcification type
Clinical features
Staining of the tooth
occurs due to increased
permeability.
Associated with deposition of secondary
dentine in the pulp. Therefore ,
resembles DI
Staining of the tooth
occurs due to increased permeability.
M.Ekram
E Hypocalcification
Normal enamel thickness with less density than D.
Radiographically.
M.Ekram
Radiographically
The diagnosis depends mainly
on the clinical examination.
The teeth are Normal or Square-
shaped with thin opaque mottled E .
(according to the thickness of enamel).
Enamel may be lost and very difficult
to differentiate it from DI.
M.Ekram
DD of AI
From DI why ?
If formation of secondary dentin with
obliteration of the pulp is associated with
Enamel abrasion, this will resemble DI.
However bulbous tooth + obliterated short
roots rule out AI.
M.Ekram
Dentinogenesis imperfecta (DI)
It is a developmental anomaly affecting dentin in both
sexes and both dentitions.
Hereditary opalescent dentin
M.Ekram
Type I Type II
usually + O I Never with O I
Unless by chance
Types of Dentinogenesis imperfecta
(DI)
M.Ekram
General clinical picture (D I)
Type I: more in deciduous teeth > permanent
Type II: equal in both
Tooth Color:
Teeth vary in color from
brownish, yellowish brown
or to even violet with
unusual translucency.
Enamel:
May be lost due to
abnormal DEJ, so dentin
undergo discoloration and
rapid attrition.
opalescent dentin
M.Ekram
Radiographic Picture (DI)
Bulbous teeth with variable degrees
of attrition.
In the early stage of development, the
pulp appears more wide then quickly
shows calcification.
Partial or total obliteration of pulp due
to deposition of dentin in both
deciduous and permanent teeth.
Short blunted roots.
Occasionally associated with
multiple periapical R L. without
actual pulp exposure but not as
frequent as in D dysplasia.
M.Ekram
Radiographic Picture (DI)
M.Ekram
Osteogenesis imperfecta
Hereditary disorder
Clinical features
Skeletal deformities
Blue sclera
Progressive osteopenia
Class III malocclusion
Impacted 1st & 2nd molars.
Dentinogenesis imperfecta
(25% of cases)
M.Ekram
Dentin Dysplasia
(Rootless teeth)
Very rare, characterized by normal E,
defective D, abnormal pulp
morphology.
Very short conical roots with
obliteration of pulp.
Associated with periapical RL.
M.Ekram
Rootless Teeth
M.Ekram
Dentin Dysplasia: Types
Type I (Radicular)
Roots are either short or of
abnormal shape (specules).
Obliteration of pulp before
eruption.
Associated with periapical
radiolucencies.
Teeth of normal color
Teeth malalignment
Tooth exfoliation
Type II (Coronal)
Crowns as in DI
Obliteration of pulp after
eruption
Pulp chambers may
become flame-like shaped.
Anterior teeth and
premolars may develop
thistle-tube shaped pulp
chamber.
M.Ekram
Rootless Teeth Type I
M.Ekram
Rootless Teeth Type II
Flame-like pulp chamber Thistle-like pulp
M.Ekram
DD of Dentin Dysplasia
D Imperfecta
Crown: bulbous bell-
shaped
Root: short
Obliterated pulp chamber
D Dysplasia
Crown: Normal size and
shape.
Root: short or normal
Type II shows obliteration
after eruption
Pulp chamber is thistle-
tube- shaped or flame-like.
Rarifying osteitis is more
frequent.
Both causes discoloration of teeth and obliteration
of pulp chamber.
M.Ekram
Regional Odontodysplasia
(Ghost teeth)
It is a relatively rare disorder of unknown
etiology affecting both E & D of both
dentitions.
E and D are both hypoplastic and
hypocalcified.
Results in arresting development of the
involved teeth.
One or several teeth in a localized area are
affected.
M.Ekram
Ghost tooth
Clinically:
It affects maxillary anterior
teeth more than mandibular.
Central > lateral > canine.
Teeth have irregular shape
with defective mineralization.
Teeth show delayed eruption.
Increase incidence of caries,
pulp infection and tooth
fracture.
M.Ekram
Ghost tooth
Radiographically
thin E & D with large
pulp.
Sometimes E is very
hypo-dens so that it may
not be evident on
radiographs.
M.Ekram
DD of Odontodysplasia
D Imperfecta
Family history
E is normal
Involves all teeth
Odontodysplasia
No family history
E is hypoplastic
Few or group of teeth
in the arch are only
affected.
It may resemble DI But:
M.Ekram
Turner’s Hypoplasia
Usually involve single tooth.
Permanent > deciduous.
Ranges from mild discoloration or pitting of E,
to severe defects and abnormal anatomy.
Due to infection or trauma to deciduous.
M.Ekram
Congenital Syphilis
Affects permanent
dentition more than
deciduous.
Deciduous teeth are
usually normal as
abortion would occur if
treponeamal spirocheats
get access through the
placenta to the fetus.
M.Ekram
Clinical features
Anterior teeth : Hutchinson’s
teeth.
#1 >#2 >#6
Screw driver with rounding of
the mesial and distal incisal
angles.
Notching of the incisal edge.
Molars: Mulberry molars or
Moon's molars
narrow crown with globular
shaped cusps.
E. hypoplasia due to syphilis has a very
pathognomonic features.
M.Ekram
Thank you
Questions ?

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Radiographic assessment of teeth developmental anomalies

  • 2. M.Ekram Chapter 18 White & Pharoh Page 330 Reference:
  • 3. M.Ekram Causes of Developmental Anomalies 1. Genetic: inherited or gene mutation. 2. Environmental causes: Infection, trauma, radiation. hormonal disturbances. nutritional deficiencies. hypoxia miscellaneous
  • 4. M.Ekram Classification Size Shape Number Structure Disturbance in eruption Microdontia macrodontia gemination, fusion. Concrescence. Dilaceration. Talon cusp. dense-in-dent. Dens invaginatus Taurodontism. Missing teeth supernumerary Amelogenesis dentinogenisis imperfecta. Dentin dysplasia Trasposition premature delayed eruption
  • 5. M.Ekram Anomalies in tooth size Microdontia True: Generalized → all teeth. e.g. in dwarfism Single, group of teeth → #2 (peg shape ) → # 8 (fused roots) Relative Microdontia: as normal sized teeth in large mandible Clinically: identified easily
  • 6. M.Ekram Radiographically: small sized crown and root Microdontia (cont.)
  • 7. M.Ekram Macrodontia True generalized Localized all teeth in facial hemi- in Gigantism hypertrophy clinically: single macrodont is difficult to be identified from gemination or fusion Relative normal or slightly large teeth in small jaw
  • 8. M.Ekram Developmental Anomalies in Teeth Number Increased number supernumerary teeth. Predeciduous dent. Post permanent dent. Decreased number Hypodontia Oligodontia Anodontia. partial- anodontia
  • 9. M.Ekram Supernumerary Teeth Arise from extra-teeth buds. Rare in deciduous teeth. May resemble the tooth anatomically or not.
  • 10. M.Ekram Supernumerary Teeth Sites any where, But the most common are: Mesiodense: between #1,#1. Small, conical inverted, impacted, short root. Paramolars: located around the premolar- molars.
  • 11. M.Ekram Supernumerary Teeth Distomolar: Distal to # 8, small, rudimentary.
  • 12. M.Ekram Clinical Picture May prevent eruption or displacement of normal teeth or cause tooth resorption. Multiple Supernumerary Teeth Are manifested in : 1. Gardner’s Syndrome: multiple supernumerary and impacted teeth, multiple polyps of large intestine,multiple osteomas , sebaceous cysts.
  • 13. M.Ekram Abnormalities Bifid ribs and other skeletal abnormalities. Multiple jaw cysts. Multiple nevoid basal cell carcinomas. Characteristic frontal bossing and broad nasal root Bifid ribs Multiple cyst - like radiolucencies 1. Gorlin-Goltz Syndrome: (basal cell nevous- bifid rib syndrome).
  • 14. M.Ekram 3. Cleido-cranial dysostosis: large skull with mental retardation, open fontanells, small maxilla, high arched palate, absent or hypoplastic clavicles.
  • 15. M.Ekram Predeciduous Dentition Hornified structure at birth or just after. Due to extra-teeth buds. Most common in lower anterior area. Present just above the alveolar ridge, so easily removed. NB: we should differentiate between it and deciduous teeth before attempting removal.
  • 16. M.Ekram Post-permanent Dentition Third Dentition After extraction of all teeth and construction of denture, new teeth begin to erupt. They may be either impacted. Or supernumerary unerupted teeth.
  • 19. M.Ekram Decrease Number of Teeth Missing teeth - Anodontia Total Anodontia: failure of odontogenesis rare but may accompany: H. Ectodermal Dysplasia: with dry skin, absence of sweet glands, scanty hair, patient can not tolerate heat. Partial Anodontia: few teeth: Hypodontia Many teeth: Oligodontia. More common than total A. Frequency: #8 > #2 >#5 in permanent. Upper # B > lower # B
  • 20. M.Ekram Hereditary Radiation to the head in a very early stage of development with either a total destruction of teeth buds, or the teeth are partially developed and hypo-calcified with stunted growth. Diagnosis: proper history and exclusion of extraction help in diagnosis. Then radiographic examination confirms the clinical ex. Etiology (Anodontia)
  • 25. M.Ekram Anomalies in eruption of teeth Transposition 1. Permanent canine and 1st premolar. 2. Second premolar (between 6 & 7). 3. Central & lateral incisors. N.B. Transposition was not reported in Pry teeth but may be with hypodontia or supernumerary teeth. Teeth commonly involved
  • 26. M.Ekram Anomalies in tooth Shape Gemination (twinning) Partial division tooth with a single root and a crown that is divided totally or partially. Complete division complete separation with formation of 2 teeth with crowns and roots. N.B. the result is a normal teeth No + supernumerary tooth. A single tooth bud divides by an invagination
  • 28. M.Ekram Clinical Picture of Gemination Site: anterior (deciduous or permanent). Structure: enamel and dentin may be hypoplastic or hypocalcified.
  • 29. M.Ekram Radiographic Picture of Gemination Large tooth with well defined enamel separating the two parts. One large pulp chamber or partially divided one.
  • 31. M.Ekram Clinical implications of Gemination Poor esthetics due to a partially divided tooth. Hypoplastic enamel and increased caries susceptibility Malocclusion and periodontal problems.
  • 32. M.Ekram Fusion Union of two normally separated teeth. Theories and types 1- fusion occurs early before calcification: the tooth formed is one single large tooth. 2- fusion occurs late after calcification: there may be union in the roots only with two separate or single root canal.
  • 33. M.Ekram Clinical Picture of Fusion Decreased number of teeth by one. Occurs more in deciduous teeth. More in anterior. The crown may be bifid
  • 34. M.Ekram What happens when fusion occurs between normal tooth and supernumerary tooth ? it will be very difficult to be differentiated from gemination !!!!!
  • 35. M.Ekram Radiographic picture of fusion Single root with cleft crown (fusion occurs late after calcification had started)
  • 36. M.Ekram fusion Fusion In deciduous teeth 2 separate root canals
  • 37. M.Ekram Differences between fusion and gemination Fusion Number of teeth is less by one except if fusion occurs with extra-tooth. Tooth with 2 separate Root Canals with one or two roots. Gemination Number of teeth is correct or increased by one. Tooth with large pulp chamber with either clefted or partially divided crown. N.B. Also from macrodontia
  • 39. M.Ekram Concrescence It is a form of fusion but by cementum only. Etiology trauma and crowding of teeth with resorption of interdental bone so that teeth are united with deposition of cementum. It may occur before or after development of teeth (true & acquired). Significance: during teeth extraction.
  • 40. M.Ekram Taurodontism Describes the increase in length of the crown on the expense of root with elongated pulp chamber so, it shows increase distance between the CEJ and root furcation. The name derived from similarity of teeth to that of Cud-chewing animals, so termed Bull-like teeth.
  • 41. M.Ekram Etiology: mutation resulting from odotoblastic deficiency during formation of roots. Clinically : indistinguishable
  • 42. M.Ekram Radiographic features 1.tooth rectangular in shape. 2. Large elongated pulp chamber. 3. Lack of usual constriction at cervical area. 4. Furcation is few mms away from the apex (short root).
  • 43. M.Ekram Dilaceration This term refers to an angle or sharp bend either in the crown or root. Etiology: Developmental or could be due to trauma Trauma of deciduous teeth may cause dilaceration of the permanents. Clinically Site: any where in the root and may be in the crown. More in maxillary anterior teeth.
  • 44. M.Ekram Clinical and Radiographic Picture (Dilaceration) Teeth may not erupt. If in the crown: bad esthetics. If in the root: has no significance except in endodontics and extraction. Radiographically appears if dilaceration is to the mesial or distal. Appears as well defined RO area with LD in the center (apical foramen) giving the appearance of “bulls’ eye”
  • 45. M.Ekram Dilaceration Buccal or lingual dilaceration Distal dilacerationDistal dilaceration Clear angulation
  • 46. M.Ekram Differential Diagnosis of Dilaceration some times it is difficult to be differentiated from condensing osteitis, idiopathic osteosclerosis. fused roots. N.B. we take radiographs from different angles
  • 47. M.Ekram Dense-in-Dent It is due to enfolding of the enamel organ to the interior during development and before calcification. Etiology: 1- focal growth stimulation or focal growth retardation in certain areas. 2- External trauma.
  • 48. M.Ekram Clinical Picture of Dens-in-Dent Incidence: tooth #2> #1 Site: most common in crowns, but may be in the roots due to folding of epithelial root sheath of Hertwing. It Appears as a palatal pit. N.B. the most extreme form of this anomaly is referred to as Dilated odontome.
  • 49. M.Ekram Clinical Picture of Dens-in-Dent Clinical significance:  The palatal pit is difficult to clean. very thin enamel separate the defect from the pulp chamber. There is a risk of caries and pulp necrosis.
  • 50. M.Ekram Coronally pear-shaped area of enamel and dentin. Narrow constriction at the opening . Closely approximated to the pulp in depth. Radiographic Picture of Dens-in-Dent
  • 51. M.Ekram Radicular lined with cementum, may reach to the apical foramen (not as defined as the coronally). Radiographic Picture of Dens-in-Dent
  • 53. M.Ekram Dense evaginatus Leong’s premolar Clinically: Occurs in premolars, as a tubercle of enamel in the middle of the occlusal surface. covered with enamel and composed of dentin and a core of pulp. It is due to out folding of the enamel organ.
  • 54. M.Ekram Dense evaginatus Radiographically: extension of pulp covered by E & D. Significance: occlusal interference. prevention of complete eruption of the opposing tooth. wearing with subsequently pulp exposure.
  • 55. M.Ekram Talon Cusp It is projecting from the cingulum of upper or lower anterior teeth. Clinical Picture Surrounded by 2 grooves. Blends to the surface of the tooth. Significance * May Interfere with occlusion. * Caries susceptibility
  • 56. M.Ekram Radiographic picture of Talon Cusp Differential diagnosis : Supernumerary teeth (buccal object rule).
  • 57. M.Ekram Enamel Pearl Is a small globule of enamel1-3 mm in diameter that occurs on the roots of molars (furcation area). DD 1. Isolated piece of calculus 2. Pulp stone (buccal object rule – vertically).
  • 59. M.Ekram Developmental anomaly that affects enamel formation. Marked changes in E in either part or all the teeth in both dentitions. Dentin and roots are usually normal. Causes delayed eruption of the affected teeth. Increases tendency for impaction. Amelogenesis Imperfecta (AI)
  • 60. M.Ekram AI: Types E. Hypoplastic type. in matrix formation. E. Hypocalcification in mineralization. E. Hypomaturation. Hypomaturation. Hypocalcifid type. (mottled E).
  • 61. M.Ekram Clinical Picture (AI) Hypoplastic type Thin enamel & dentin is shown, so the teeth are dark or brown in color. Loss of normal tooth contour and teeth become undersized. Lack of proximal contact. Occlusal surfaces are usually flat (attrition).
  • 62. M.Ekram E Hypomaturation type Clinical features Enamel is of normal thickness but mottled. Enamel can be pierced by probe. Patient may be affected by different degrees. Teeth may show vertical grooves. Teeth may be chalky or dark in color. radiographically. Enamel is of normal thickness but with density similar to D.
  • 63. M.Ekram Clinical picture (AI) E Hypocalcification type Clinical features Staining of the tooth occurs due to increased permeability. Associated with deposition of secondary dentine in the pulp. Therefore , resembles DI Staining of the tooth occurs due to increased permeability.
  • 64. M.Ekram E Hypocalcification Normal enamel thickness with less density than D. Radiographically.
  • 65. M.Ekram Radiographically The diagnosis depends mainly on the clinical examination. The teeth are Normal or Square- shaped with thin opaque mottled E . (according to the thickness of enamel). Enamel may be lost and very difficult to differentiate it from DI.
  • 66. M.Ekram DD of AI From DI why ? If formation of secondary dentin with obliteration of the pulp is associated with Enamel abrasion, this will resemble DI. However bulbous tooth + obliterated short roots rule out AI.
  • 67. M.Ekram Dentinogenesis imperfecta (DI) It is a developmental anomaly affecting dentin in both sexes and both dentitions. Hereditary opalescent dentin
  • 68. M.Ekram Type I Type II usually + O I Never with O I Unless by chance Types of Dentinogenesis imperfecta (DI)
  • 69. M.Ekram General clinical picture (D I) Type I: more in deciduous teeth > permanent Type II: equal in both Tooth Color: Teeth vary in color from brownish, yellowish brown or to even violet with unusual translucency. Enamel: May be lost due to abnormal DEJ, so dentin undergo discoloration and rapid attrition. opalescent dentin
  • 70. M.Ekram Radiographic Picture (DI) Bulbous teeth with variable degrees of attrition. In the early stage of development, the pulp appears more wide then quickly shows calcification. Partial or total obliteration of pulp due to deposition of dentin in both deciduous and permanent teeth. Short blunted roots. Occasionally associated with multiple periapical R L. without actual pulp exposure but not as frequent as in D dysplasia.
  • 72. M.Ekram Osteogenesis imperfecta Hereditary disorder Clinical features Skeletal deformities Blue sclera Progressive osteopenia Class III malocclusion Impacted 1st & 2nd molars. Dentinogenesis imperfecta (25% of cases)
  • 73. M.Ekram Dentin Dysplasia (Rootless teeth) Very rare, characterized by normal E, defective D, abnormal pulp morphology. Very short conical roots with obliteration of pulp. Associated with periapical RL.
  • 75. M.Ekram Dentin Dysplasia: Types Type I (Radicular) Roots are either short or of abnormal shape (specules). Obliteration of pulp before eruption. Associated with periapical radiolucencies. Teeth of normal color Teeth malalignment Tooth exfoliation Type II (Coronal) Crowns as in DI Obliteration of pulp after eruption Pulp chambers may become flame-like shaped. Anterior teeth and premolars may develop thistle-tube shaped pulp chamber.
  • 77. M.Ekram Rootless Teeth Type II Flame-like pulp chamber Thistle-like pulp
  • 78. M.Ekram DD of Dentin Dysplasia D Imperfecta Crown: bulbous bell- shaped Root: short Obliterated pulp chamber D Dysplasia Crown: Normal size and shape. Root: short or normal Type II shows obliteration after eruption Pulp chamber is thistle- tube- shaped or flame-like. Rarifying osteitis is more frequent. Both causes discoloration of teeth and obliteration of pulp chamber.
  • 79. M.Ekram Regional Odontodysplasia (Ghost teeth) It is a relatively rare disorder of unknown etiology affecting both E & D of both dentitions. E and D are both hypoplastic and hypocalcified. Results in arresting development of the involved teeth. One or several teeth in a localized area are affected.
  • 80. M.Ekram Ghost tooth Clinically: It affects maxillary anterior teeth more than mandibular. Central > lateral > canine. Teeth have irregular shape with defective mineralization. Teeth show delayed eruption. Increase incidence of caries, pulp infection and tooth fracture.
  • 81. M.Ekram Ghost tooth Radiographically thin E & D with large pulp. Sometimes E is very hypo-dens so that it may not be evident on radiographs.
  • 82. M.Ekram DD of Odontodysplasia D Imperfecta Family history E is normal Involves all teeth Odontodysplasia No family history E is hypoplastic Few or group of teeth in the arch are only affected. It may resemble DI But:
  • 83. M.Ekram Turner’s Hypoplasia Usually involve single tooth. Permanent > deciduous. Ranges from mild discoloration or pitting of E, to severe defects and abnormal anatomy. Due to infection or trauma to deciduous.
  • 84. M.Ekram Congenital Syphilis Affects permanent dentition more than deciduous. Deciduous teeth are usually normal as abortion would occur if treponeamal spirocheats get access through the placenta to the fetus.
  • 85. M.Ekram Clinical features Anterior teeth : Hutchinson’s teeth. #1 >#2 >#6 Screw driver with rounding of the mesial and distal incisal angles. Notching of the incisal edge. Molars: Mulberry molars or Moon's molars narrow crown with globular shaped cusps. E. hypoplasia due to syphilis has a very pathognomonic features.