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PRESENTED BY ERESH
COLLEGE OF
AGRICULTURE RAICHUR
Structural defence
mechanism in plants
1.Pre-existing
2. Post infectional
The first line of defence in plants is present on
its surface
Pre-existing structural defence
mechanism
 First layer in the host to defence
 The structures of epidermal
cells&cellwalls
 The size,location and shape ,no.of
stomata and lenticels,hydathodes
structures
1.waxes:
synhesized by epidermal cells
Complex mixture of long chain acyl lipids.
Extremely hydrophobic
Wax forms outer coating of the cuticle
Often crystallizes in pattern of rods or tubes or
plates
It prevents germination of fungal spores and
bactria
No parasite is known to produce enzymes that
can degrade the cuticular waxes
2.Cuticle and epidermal cells
 The cuticle is composed of insoluble
membranes
 Covered with
1.insoluble waxes
2.cutin embeded
3.cutin+wax blended
 Thick cuticle and tough outer wall of
epidermal cell resistance to direct penetration
of pathogen
 Eg:Barbery-Puccinia graminis tritici-
resistence is attributed to the tough outer
epidermal cells
 Pyricularia grisea-leaf blast of paddy-the
3.Sclerenchyma cells
o Composed of walls thickened with
lignin
o Have brittle cells helps in mechanical
support to plants
o These cells effectively blocks spread
of fungal and bacterial pathogen that
cause angular leaf spots
o Eg: some wheat varieties resistant to
stem rust –stem contain high
sclerenchyma cells
4.Natural openings
1.stomata:
 The minute pores in the epidermis of
leaf through which exchange of gases
takes place
 The structures of stomata provides
resistance to penetration of pathogen
 Eg: citrus variety szinkum –resistant to
citrus canker
 Cultivar Hope-resistant to wheat rust
2.lenticels
 Lenticels are airy cells in the bark of
stems and roots
 The shape and internal structure of
lenticels can increase or decrease the
incidence of fruit diseases
 Lenticels of small size restrict entry of
pathogen
3.Hydathodes:
Early entry points of bacterial pathogen
Eg: xanthomonas campestris
pv.campestris
4.Nectarithodes:
Present tip of infloroscence
Eg: Erwinia amylovora-fire blight of
apple
2.Post –infectional defence
mechnism
 Present within but produced after
infection
 1.histological barriers
 2.cellular barriers
Histological barriers
 A)cork layer: stronger rough surface
of the host
 A layer of meristamatic cells which
form on the surface of the host
 Toxical material spread stops infection
 Stops flow of water & nutrients
towards infection part
 Eg:scab of gauva and citrus canker
 Rhizopus rot of sweet potato
b.Abscission layer
 Means fall off or shred off
 Gap formed b/w infected cell and
healthy cell at the site of infection .
 It surrounds point of locus of infection.
 Gradually infected area shrivels , dyes
carrying with it the pathogen
 Abscission layer-amputative
resistance
 Ex. Xanthomonas pruni and
closterosporium carpophylum on
peach leaves
c.Tyloses
 Over growth of adjacent lining
paravascular cells which protrudes into
xylem vessels through pits
 Tyloses are selective permeable to water
and nutrients
 It blocks further spread of pathogen
 Eg: Fusarium oxysporum f.sp. batatas-
wilt of sweet potato
Cellular defence mechanisms
 Hyphal sheathing :the hyphae
penetrating the cell wall and growing
into the cell lumen are enveloped by a
cellulosic sheath
 Inward stretching of the host cellwall
around the haustorium
 Eg:Botrytis cinera

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Structural Defence Mechanisms in Plants

  • 1. PRESENTED BY ERESH COLLEGE OF AGRICULTURE RAICHUR
  • 2. Structural defence mechanism in plants 1.Pre-existing 2. Post infectional The first line of defence in plants is present on its surface
  • 3. Pre-existing structural defence mechanism  First layer in the host to defence  The structures of epidermal cells&cellwalls  The size,location and shape ,no.of stomata and lenticels,hydathodes
  • 4. structures 1.waxes: synhesized by epidermal cells Complex mixture of long chain acyl lipids. Extremely hydrophobic Wax forms outer coating of the cuticle Often crystallizes in pattern of rods or tubes or plates It prevents germination of fungal spores and bactria No parasite is known to produce enzymes that can degrade the cuticular waxes
  • 5. 2.Cuticle and epidermal cells  The cuticle is composed of insoluble membranes  Covered with 1.insoluble waxes 2.cutin embeded 3.cutin+wax blended  Thick cuticle and tough outer wall of epidermal cell resistance to direct penetration of pathogen  Eg:Barbery-Puccinia graminis tritici- resistence is attributed to the tough outer epidermal cells  Pyricularia grisea-leaf blast of paddy-the
  • 6. 3.Sclerenchyma cells o Composed of walls thickened with lignin o Have brittle cells helps in mechanical support to plants o These cells effectively blocks spread of fungal and bacterial pathogen that cause angular leaf spots o Eg: some wheat varieties resistant to stem rust –stem contain high sclerenchyma cells
  • 7. 4.Natural openings 1.stomata:  The minute pores in the epidermis of leaf through which exchange of gases takes place  The structures of stomata provides resistance to penetration of pathogen  Eg: citrus variety szinkum –resistant to citrus canker  Cultivar Hope-resistant to wheat rust
  • 8. 2.lenticels  Lenticels are airy cells in the bark of stems and roots  The shape and internal structure of lenticels can increase or decrease the incidence of fruit diseases  Lenticels of small size restrict entry of pathogen
  • 9. 3.Hydathodes: Early entry points of bacterial pathogen Eg: xanthomonas campestris pv.campestris 4.Nectarithodes: Present tip of infloroscence Eg: Erwinia amylovora-fire blight of apple
  • 10. 2.Post –infectional defence mechnism  Present within but produced after infection  1.histological barriers  2.cellular barriers
  • 11. Histological barriers  A)cork layer: stronger rough surface of the host  A layer of meristamatic cells which form on the surface of the host  Toxical material spread stops infection  Stops flow of water & nutrients towards infection part  Eg:scab of gauva and citrus canker  Rhizopus rot of sweet potato
  • 12. b.Abscission layer  Means fall off or shred off  Gap formed b/w infected cell and healthy cell at the site of infection .  It surrounds point of locus of infection.  Gradually infected area shrivels , dyes carrying with it the pathogen  Abscission layer-amputative resistance  Ex. Xanthomonas pruni and closterosporium carpophylum on peach leaves
  • 13. c.Tyloses  Over growth of adjacent lining paravascular cells which protrudes into xylem vessels through pits  Tyloses are selective permeable to water and nutrients  It blocks further spread of pathogen  Eg: Fusarium oxysporum f.sp. batatas- wilt of sweet potato
  • 14. Cellular defence mechanisms  Hyphal sheathing :the hyphae penetrating the cell wall and growing into the cell lumen are enveloped by a cellulosic sheath  Inward stretching of the host cellwall around the haustorium  Eg:Botrytis cinera