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Critical care Management in
Medical Nursing
Dr.Puvaneswari. K
Professor
Narayana Hrudayalaya College of
Nursing
MEDICAL CONDITIONS CONSIDERED
AS CRITICAL
ANY PERSON WITH LIFE THREATENING CONDITIONS :
 Acute Myocardial Infarction
 Acute renal failure
 Cardiac Tamponade
 Severe shock
 Heart block
 Acute Respiratory Distress Syndrome
 Multiple organ failure and organ dysfunction
 COPD
 Poly trauma
 Severe burns
 Poisoning
IDENTIFICATION OF PATIENT NEEDS IN
MEDICAL CONDITIONS
• Early Warning Scoring (EWS)
• Acute Physiology and Chronic Health
Evaluation (APACHE) Scoring
• Simplified Acute Physiological Score (SAPS)
• Sequential Organ Failure Assessment (SOFA)
• Trauma Revised Injury Severity Score (TRISS)
• Nutritional assessment
• Pressure area risk assessment
• Sedation scoring
• Glasgow coma score (GCS)
• CVP monitoring
PROBLEMS FACED IN ICU MEDICAL
PATIENTS
• Hypoxia
• Hypotension and shock
• Acid base disturbance
• Sepsis / Fever
• Electrolyte imbalances
• Pain
HYPOXIA
HYPOXIA
NORMALINCREASED
PaCO2
INCREASED
• TYPE 11
RESPIRATORY
FAILURE
• COPD
LOW
CHEST X-
RAY
• CARDIOGENIC
PULMONARY OEDEMA
• SLEEP APNOEA
• SEDATIVE
OVERDOSE
• CENTRAL
HYPOTENSION
NORMAL ABNORMAL
CONSIDER PE
PAOP <16
CI
NORMAL
PAOP >16
CI LOW
ARDS
RESPIRATORY
ACIDOSIS
PAaO2 =PAO2-PaO2
HYPOXIA MANAGEMENT
COPD
• Methyl Prednisolone 0.5- 2 mg/Kg Q6H
• Antibiotics
• Aminophylline 5mg/Kg loading .5mg/Kg /hour
• Beta-2 Agonists by MDI
ARDS
• Diuretics
• Inotropics / vasopressor – Dopamine, Dobutamine ,
Nor epinephrine
• Sedation /Paralysis/analgesia
Cardiogenic pulmonary oedema
• Morphine 2-5 mg IV
• Frusemide 40mg IV
• IV nitroglycerine for Ischemia
• IV Nitroprusside 0.5-2 mcg/kg/min
• IV Dobutamine 2.5-15 mcg/kg/min
• IABP
• Mechanical ventilation
HYPOTENSION AND SHOCK
HYPOTENSION FLUID
RESUSCITATION
HT NOT
RESOLVED
HT RESOLVED
• CONTINUE FLUID RESUSCITATION
• ASSESS THE CAUSE
• ADD DOPAMINE AND NOR EPINEPHRINE
VOL.PROBLEM VENT.PROBLEM PUMPING.PROBLEM
• GI BLEED
• DIARROHEA
• SEPSIS
PNUMOTHORAX
PEEP
ARRYTHMIA
• BRADY
• TACHY
• ECG
• ECHO
• CARDIAC ENZYMES
• MI
• CARDIAC TAMPONADE
• PUL.EMBOLISM
Hypotension and shock management
• Fluid therapy- IV 1-1.5 liters or 20-40 mL/kg .
• Lower volumes of crystalloid to prevent over
dilution of blood and coagulation factors
• Pressors – Dopamine, Dobutamine , Epinephrine,
Norephinephrine, Vasopressin
• IABP
• Antibiotics
• Treatment based on cause
ACID-BASE DISTURBANCE
LOW PH
HCO3
PaCO2
HCO3
METABOLIC
ACIDOSIS
RESPIRATORY
ACIDOSIS
LOW HIGH
↓PaCO2
APPROPRIATE
FOR ↓ IN HCO3
↑ 𝐇𝐂𝐎𝟑
APPROPRIATE
FOR ↑ IN PaCO2
LOW
HIGH
• DKA
• DIARRHOEA
• RENAL FAILURE
• SHOCK
• COPD
• GBS
• DRUG OVERDOSE
• AIRWAY
OBSTRUCTION
HIGH PH
HCO3
PaCO2
HCO3
METABOLIC
ALKALOSIS
RESPIRATORY
ALKALOSIS
HIGH LOW
↑PaCO2
APPROPRIATE
FOR ↓ IN HCO3
↓ 𝐇𝐂𝐎𝟑
APPROPRIATE
FOR ↓ IN PaCO2
HIGH
LOW
VOMITTING, DIURETIC THERAPY,
EXCESS GASTRIC SUCTIONING
HYPERVENTILATION,
SEPTICAEMIA, RESP.ALKALOSIS
DISORDER PRIMARY
ALTERATION
SECONDARY
RESPONSE
Metabolic
Acidosis
in plasma
HCO3
in plasma pCO2
Metabolic
Alkalosis
in plasma
HCO3
increase in
pCO2
Respiratory
Acidosis
in plasma pCO2 in plasma HCO3
Respiratory
Alkalosis
in plasma pCO2 in plasma HCO3
ACID BASE DISTURBANCE MANAGEMENT
Respiratory alkalosis
• Correct the underlying cause
• Reassurance or sedation, rebreathing into a closed
system (e.g., a paper bag)
• Correction of hypocapnia ( Reset the device)
Respiratory acidosis
• Treat the cause . eg Bronchodilators, Respiratory
stimulants
• Supplemental O2
• Mechanical ventilation and NIPPV
Metabolic alkalosis
• Treat the cause
• Use k- sparing diuretics and Acetazolamide
• Correct the deficiency (i e give chloride, water , K+)
• Expand ECF Volume with N/saline and KCl .
Metabolic acidosis
• Accurate diagnosis and treat the cause
• Oxygen administration or Intubation and
Mechanical ventilation.
• Replace losses (e.g. of fluids and electrolytes)
ACID BASE DISTURBANCE MANAGEMENT
SEPSIS / FEVER
HISTORY AND PHYSICAL
EXAMINATION
SEPSIS WORKUP- CBC,LFT, CULTURE OF ALL SPECIMENS,
CULTURE FROM INFLAMMED SITES , CHEST
RADIOGRAPHY
CULTURE +VE
NARROW
ANTIBIOTICS,
WORK UP
CULTURE – VE
CONTINUE EMPIRIC
ANTIBIOTICS
CULTURE –VE
CONTINUE ANTIBIOTICS
BASED ON BEST GUESS
CULTURE +VE
NARROW
ANTIBIOTICS
REPEAT HISTORY AND
PE SEPTIC WORK UP,
CHANGE ALL LINES
FEVER
RESOLVES
OCCULT INFECTIOUS CAUSE
• CHOLECYSTITIS
• INTRA ABDOMINAL ABSECESS
NON INFECTIOUS CAUSE
• DVT,
• PANCREATITIS,
• DRUG FEVER
SOURCE
ISOLATED
NO
SOURCE
FEBRILE
SEPSIS / FEVER MANAGEMENT
• Symptomatic management- Antibiotic therapy ,
Antifungal therapy.
• Fluid resuscitation ( 20 mL/kg of isotonic
crystalloid, followed by boluses of up to 1000 mL
of crystalloid or 500 mL of colloid given over 30
minutes).
• Vasopressors and Inotropes
• Steroids
• Antipyretic therapy eg acetaminophen
ELECTROLYTE DISTURBANCES
DIAGNOSTIC APPROACH FOR
HYPERNATREMIA
URINE
OUTPUT
URINE
OSMOLALITY
URINE
OSMOLALITY
HYPERTONIC FLUID LOSS
• INSENSIBLE FLUID LOSS
• GI LOSS
• PRIOR RENAL LOSS
DIABETIC
INSIPIDUS
OSMOTIC
DIURESIS
HIGH HIGH LOW
LOW HIGH
HYPERNATREMIA MANAGEMENT
• Calculate BWD= DBW- TBW
• Replace BWD in addition to maintenance fluid
• For oliguria/ hypotensive patient – Normal saline
• For stable patients- D5W
• Replace 50% BWD over 24
hours. Rest over 24-48 hours.
• Follow serum sodium q6h
HYPOKALEMIA
INCREASED LOSS
DISTRIBUTION DEFECT
AML, HYPERGLYCAEMIA,
ALKALOSISURINARY SPOT K<10
URINARY SPOT K< 10
NONRENAL LOSS
DIARROHOEA, BILIARY LOSS,
SMALL INTESTINE FISTULAS
URINARY SPOT K>20
RENAL LOSS
CHECK BP
HTN NORMAL BP
CHECK PLASMA
RENIN
CHECK PLASMA HCO3
INCREASED
SEC.HYPER
ALDOSTER
DECREASED
PRIMARY
HYPER
ALDOSTER
• VOMITTING
WITH
METABOLIC
ALKALOSIS
• DIURETICS,
• NORMOTENSIVE,
• HYPERALDOSTER
INCREASED
CHECK URINARY CI
HYPOKALEMIA MANAGEMENT
• Find and Correct cause
• Angiotensin-converting enzyme (ACE) inhibitors,
which inhibit renal potassium excretion.
• Potassium-sparing diuretics.
• Syp .KCL po 40 meq. And
IV 40 meq @ 10 meq/hour
• Check ECG
PAIN
PAIN ASSESSMENT
Sedatives /Anxiolytics
Patient controlled analgesia
Epidural analgesia
Narcotics
Non steroidal anti inflammatory drugs
NURSES ROLE IN MANGEMENT OF
PATIENTS WITH MEDICAL CONDITIONS
• Assessment
• Diagnosis
• Planning
• Implementation
• Evaluation
RESPIRATORY MANAGEMENT
• Oxygen therapy
• Mobilisation of secretions
• Ventilator support
FLUID AND ELECTROLYTE MAINTENANCE
• Administer IV fluids
• Administer blood products
• Maintenance of intake and output
NUTRITIONAL MANAGEMENT
• Enteral feeding with need based nutrients
• Total parenteral nutrition
HYGIENIC CARE
• Meeting /Assisting ADL
• Back care
• Special care for ventilated clients
POSITIONING
• Prevention of bed sore
• Therpeutic positions
• Safety
PREVENTION OF INFECTION
• Aseptic technique
• Invasive site care.
• Hand washing.
PREVENTION OF COMPLICATIONS
• Monitoring
• Early identification of warning signs
• Patient safety
EXERCISES
• Respiratory , ROM exercises
• Early mobilisation
• Leg exercises
FAMILY MANAGEMENT
• Involve in decision making
• Family coping
• Transcultural and al manage
Critical care Management in Medical Nursing

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Critical care Management in Medical Nursing

  • 1. Critical care Management in Medical Nursing Dr.Puvaneswari. K Professor Narayana Hrudayalaya College of Nursing
  • 2. MEDICAL CONDITIONS CONSIDERED AS CRITICAL ANY PERSON WITH LIFE THREATENING CONDITIONS :  Acute Myocardial Infarction  Acute renal failure  Cardiac Tamponade  Severe shock  Heart block
  • 3.  Acute Respiratory Distress Syndrome  Multiple organ failure and organ dysfunction  COPD  Poly trauma  Severe burns  Poisoning
  • 4. IDENTIFICATION OF PATIENT NEEDS IN MEDICAL CONDITIONS • Early Warning Scoring (EWS) • Acute Physiology and Chronic Health Evaluation (APACHE) Scoring • Simplified Acute Physiological Score (SAPS) • Sequential Organ Failure Assessment (SOFA)
  • 5. • Trauma Revised Injury Severity Score (TRISS) • Nutritional assessment • Pressure area risk assessment • Sedation scoring • Glasgow coma score (GCS) • CVP monitoring
  • 6. PROBLEMS FACED IN ICU MEDICAL PATIENTS • Hypoxia • Hypotension and shock • Acid base disturbance • Sepsis / Fever • Electrolyte imbalances • Pain
  • 8. HYPOXIA NORMALINCREASED PaCO2 INCREASED • TYPE 11 RESPIRATORY FAILURE • COPD LOW CHEST X- RAY • CARDIOGENIC PULMONARY OEDEMA • SLEEP APNOEA • SEDATIVE OVERDOSE • CENTRAL HYPOTENSION NORMAL ABNORMAL CONSIDER PE PAOP <16 CI NORMAL PAOP >16 CI LOW ARDS RESPIRATORY ACIDOSIS PAaO2 =PAO2-PaO2
  • 9. HYPOXIA MANAGEMENT COPD • Methyl Prednisolone 0.5- 2 mg/Kg Q6H • Antibiotics • Aminophylline 5mg/Kg loading .5mg/Kg /hour • Beta-2 Agonists by MDI ARDS • Diuretics • Inotropics / vasopressor – Dopamine, Dobutamine , Nor epinephrine • Sedation /Paralysis/analgesia
  • 10. Cardiogenic pulmonary oedema • Morphine 2-5 mg IV • Frusemide 40mg IV • IV nitroglycerine for Ischemia • IV Nitroprusside 0.5-2 mcg/kg/min • IV Dobutamine 2.5-15 mcg/kg/min • IABP • Mechanical ventilation
  • 12. HYPOTENSION FLUID RESUSCITATION HT NOT RESOLVED HT RESOLVED • CONTINUE FLUID RESUSCITATION • ASSESS THE CAUSE • ADD DOPAMINE AND NOR EPINEPHRINE VOL.PROBLEM VENT.PROBLEM PUMPING.PROBLEM • GI BLEED • DIARROHEA • SEPSIS PNUMOTHORAX PEEP ARRYTHMIA • BRADY • TACHY • ECG • ECHO • CARDIAC ENZYMES • MI • CARDIAC TAMPONADE • PUL.EMBOLISM
  • 13. Hypotension and shock management • Fluid therapy- IV 1-1.5 liters or 20-40 mL/kg . • Lower volumes of crystalloid to prevent over dilution of blood and coagulation factors • Pressors – Dopamine, Dobutamine , Epinephrine, Norephinephrine, Vasopressin • IABP • Antibiotics • Treatment based on cause
  • 15. LOW PH HCO3 PaCO2 HCO3 METABOLIC ACIDOSIS RESPIRATORY ACIDOSIS LOW HIGH ↓PaCO2 APPROPRIATE FOR ↓ IN HCO3 ↑ 𝐇𝐂𝐎𝟑 APPROPRIATE FOR ↑ IN PaCO2 LOW HIGH • DKA • DIARRHOEA • RENAL FAILURE • SHOCK • COPD • GBS • DRUG OVERDOSE • AIRWAY OBSTRUCTION
  • 16. HIGH PH HCO3 PaCO2 HCO3 METABOLIC ALKALOSIS RESPIRATORY ALKALOSIS HIGH LOW ↑PaCO2 APPROPRIATE FOR ↓ IN HCO3 ↓ 𝐇𝐂𝐎𝟑 APPROPRIATE FOR ↓ IN PaCO2 HIGH LOW VOMITTING, DIURETIC THERAPY, EXCESS GASTRIC SUCTIONING HYPERVENTILATION, SEPTICAEMIA, RESP.ALKALOSIS
  • 17. DISORDER PRIMARY ALTERATION SECONDARY RESPONSE Metabolic Acidosis in plasma HCO3 in plasma pCO2 Metabolic Alkalosis in plasma HCO3 increase in pCO2 Respiratory Acidosis in plasma pCO2 in plasma HCO3 Respiratory Alkalosis in plasma pCO2 in plasma HCO3
  • 18. ACID BASE DISTURBANCE MANAGEMENT Respiratory alkalosis • Correct the underlying cause • Reassurance or sedation, rebreathing into a closed system (e.g., a paper bag) • Correction of hypocapnia ( Reset the device) Respiratory acidosis • Treat the cause . eg Bronchodilators, Respiratory stimulants • Supplemental O2 • Mechanical ventilation and NIPPV
  • 19. Metabolic alkalosis • Treat the cause • Use k- sparing diuretics and Acetazolamide • Correct the deficiency (i e give chloride, water , K+) • Expand ECF Volume with N/saline and KCl . Metabolic acidosis • Accurate diagnosis and treat the cause • Oxygen administration or Intubation and Mechanical ventilation. • Replace losses (e.g. of fluids and electrolytes) ACID BASE DISTURBANCE MANAGEMENT
  • 21. HISTORY AND PHYSICAL EXAMINATION SEPSIS WORKUP- CBC,LFT, CULTURE OF ALL SPECIMENS, CULTURE FROM INFLAMMED SITES , CHEST RADIOGRAPHY CULTURE +VE NARROW ANTIBIOTICS, WORK UP CULTURE – VE CONTINUE EMPIRIC ANTIBIOTICS CULTURE –VE CONTINUE ANTIBIOTICS BASED ON BEST GUESS CULTURE +VE NARROW ANTIBIOTICS REPEAT HISTORY AND PE SEPTIC WORK UP, CHANGE ALL LINES FEVER RESOLVES OCCULT INFECTIOUS CAUSE • CHOLECYSTITIS • INTRA ABDOMINAL ABSECESS NON INFECTIOUS CAUSE • DVT, • PANCREATITIS, • DRUG FEVER SOURCE ISOLATED NO SOURCE FEBRILE
  • 22. SEPSIS / FEVER MANAGEMENT • Symptomatic management- Antibiotic therapy , Antifungal therapy. • Fluid resuscitation ( 20 mL/kg of isotonic crystalloid, followed by boluses of up to 1000 mL of crystalloid or 500 mL of colloid given over 30 minutes). • Vasopressors and Inotropes • Steroids • Antipyretic therapy eg acetaminophen
  • 24. DIAGNOSTIC APPROACH FOR HYPERNATREMIA URINE OUTPUT URINE OSMOLALITY URINE OSMOLALITY HYPERTONIC FLUID LOSS • INSENSIBLE FLUID LOSS • GI LOSS • PRIOR RENAL LOSS DIABETIC INSIPIDUS OSMOTIC DIURESIS HIGH HIGH LOW LOW HIGH
  • 25. HYPERNATREMIA MANAGEMENT • Calculate BWD= DBW- TBW • Replace BWD in addition to maintenance fluid • For oliguria/ hypotensive patient – Normal saline • For stable patients- D5W • Replace 50% BWD over 24 hours. Rest over 24-48 hours. • Follow serum sodium q6h
  • 26. HYPOKALEMIA INCREASED LOSS DISTRIBUTION DEFECT AML, HYPERGLYCAEMIA, ALKALOSISURINARY SPOT K<10 URINARY SPOT K< 10 NONRENAL LOSS DIARROHOEA, BILIARY LOSS, SMALL INTESTINE FISTULAS URINARY SPOT K>20 RENAL LOSS CHECK BP HTN NORMAL BP CHECK PLASMA RENIN CHECK PLASMA HCO3 INCREASED SEC.HYPER ALDOSTER DECREASED PRIMARY HYPER ALDOSTER • VOMITTING WITH METABOLIC ALKALOSIS • DIURETICS, • NORMOTENSIVE, • HYPERALDOSTER INCREASED CHECK URINARY CI
  • 27. HYPOKALEMIA MANAGEMENT • Find and Correct cause • Angiotensin-converting enzyme (ACE) inhibitors, which inhibit renal potassium excretion. • Potassium-sparing diuretics. • Syp .KCL po 40 meq. And IV 40 meq @ 10 meq/hour • Check ECG
  • 28. PAIN
  • 30. Sedatives /Anxiolytics Patient controlled analgesia Epidural analgesia Narcotics Non steroidal anti inflammatory drugs
  • 31. NURSES ROLE IN MANGEMENT OF PATIENTS WITH MEDICAL CONDITIONS • Assessment • Diagnosis • Planning • Implementation • Evaluation
  • 32. RESPIRATORY MANAGEMENT • Oxygen therapy • Mobilisation of secretions • Ventilator support FLUID AND ELECTROLYTE MAINTENANCE • Administer IV fluids • Administer blood products • Maintenance of intake and output NUTRITIONAL MANAGEMENT • Enteral feeding with need based nutrients • Total parenteral nutrition
  • 33. HYGIENIC CARE • Meeting /Assisting ADL • Back care • Special care for ventilated clients POSITIONING • Prevention of bed sore • Therpeutic positions • Safety PREVENTION OF INFECTION • Aseptic technique • Invasive site care. • Hand washing.
  • 34. PREVENTION OF COMPLICATIONS • Monitoring • Early identification of warning signs • Patient safety EXERCISES • Respiratory , ROM exercises • Early mobilisation • Leg exercises FAMILY MANAGEMENT • Involve in decision making • Family coping • Transcultural and al manage