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Abruptio
Placenta
1
Definition
• It is one form of Antepartum Hemorrhage
where the bleeding occurs due to
premature separation of a normally
situated placenta
2
Incidence
• 0.5% - 1% of all deliveries
• Decreasing trend
3
Risk factors
• Increased age and parity
• Preeclampsia
• Chronic hypertension
• Preterm ruptured membranes
• Multi fetal gestation
• Polyhydramnios
4
Risk factors
• Cigarette smoking
• Thrombophilias
• Cocaine use
• Prior abruption
• Uterine leiomyoma
• External Trauma
5
Retroplacental decidual hematoma
• Separation, compression and destruction
of adjacent placenta
• Varieties: Concealed, Revealed & mixed
• Couvelaire uterus
6
PATHOLOGY
Clinical Features
Sign / symptom Frequency%
Vaginal bleeding 78
Uterine tenderness
back pain
66
Fetal distress 60
Preterm labor 22
Hypertonic contractions 17
Hyper tonus 17
Dead fetus 15
7
Diagnosis…
Essentially a clinical diagnosis
•Painful bleeding
•Abdominal findings
•Cardiotocographic Monitoring
• AFP
8
9
Cardiotocograph
Role of ultrasound – limited
•Negative findings do not exclude
the diagnosis
•U/S is mainly used to confirm fetal
viability, Presentation & position.
•To exclude Placenta praevia
10
Classification
Grade 0: Retrospective diagnosis.
Grade 1: External bleeding present, no fetal
or maternal complication
Grade 2: External bleeding +/-,
tense & tender uterus, Fetal
distress/death
Grade 3: External bleeding +/-
tense & tender uterus, fetal death with
maternal shock .
DIC, Renal failure
11
Consumptive Coagulopathy
• Delee (1901) Temporary hemophilia
Parameters: Fibrinogen < 150 mg/dl,
• elevated FDP, D-dimers, decrease in other
coagulation factors
Mechanism: DIC & retro placental clot
formation
• Seen in 30% cases of abruption severe
enough to kill the fetus
12
Acute Renal failure
• Pathology: Acute tubular necrosis (75%)
& acute cortical necrosis (25%)
• Mechanism: Severe hypovolemia , DIC
along with Underlying preeclampsia
• Prevention: Prompt & vigorous
replacement of blood and circulating blood
volume
13
Differential Diagnosis
1. Without pain: Placenta Previa
2. Without Bleeding: Acute degeneration
or torsion of a fibroid, hematoma of
rectus sheath, rupture of an
appendicular abscess.
3. With mild pain & bleeding: Labour with
heavy show
4. Rupture Uterus
14
Management
Initial assessment
• Monitor vital signs: BP – poor guide to
the extent of bleeding.
• Mark the fundal height & measure
abdominal girth
• cardiotocographic monitoring of fetus
Investigations: HB, PCV, blood grouping
& typing, BT CT, Clot retraction & lyses,
DIC profile
• Foley catheterization & hourly output
chart
• watch for bleeding 15
Management
“Swift & decisive”
Resuscitate the mother
• Start an IV line, transfuse Ringer lactate, N
Saline
• Two lines if bleeding is severe
• Replace blood loss and maintain circulation
• Maintain PCV at 30% & urine output >30
ml/hr.
• CVP in difficult cases
• Delivery
16
Management
Caesarian section
• Live & mature fetus
• Delivery not imminent
• Fetal distress
• No response to induction of labour
• Bleeding
17
Management
Vaginal delivery
• Preferred if fetus is dead.
• In Selected cases with a live fetus
• ARM with oxytocin
• Watch for PPH
• RH prophylaxis
18
Management
Expectant line of management
• Doubtful diagnosis
• Minor abruption
• Preterm gestation
• Intensive surveillance & Induction at
or before term
19
Perinatal mortality
• Main danger is to the fetus. If the
abruption is severe enough to threaten the
mother, the fetus will usually be dead
25 fold increase in PMR
• Still birth
• Prematurity
• Hypoxia
• Cerebral palsy
20
Maternal deaths due to hemorrhage
Causes of hemorrhage Percentage
Abruption 19
Uterine rupture 16
Atonic PPH 15
Coagulopathies 14
Placenta praevia 7
Uterine bleeding 6
placenta accreta/percreta 6
Retained placenta 4
21
Maternal mortality
Case report: woman, who seemed well
enough to wait in an emergency dept for 2
hrs. When the doctor saw her at the end
of this time she was dead!!!
“a fit woman may be able to compensate
for severe hemorrhage until collapse
occurs as a terminal event”
• A reminder – need to maintain high
standards in obstetric care
22
23

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Abruptio placenta- Define, cause, sign and symptoms, Risk Factors, Incidence,pathology, Classification, Prevention and Treatment, management in PPT

  • 2. Definition • It is one form of Antepartum Hemorrhage where the bleeding occurs due to premature separation of a normally situated placenta 2
  • 3. Incidence • 0.5% - 1% of all deliveries • Decreasing trend 3
  • 4. Risk factors • Increased age and parity • Preeclampsia • Chronic hypertension • Preterm ruptured membranes • Multi fetal gestation • Polyhydramnios 4
  • 5. Risk factors • Cigarette smoking • Thrombophilias • Cocaine use • Prior abruption • Uterine leiomyoma • External Trauma 5
  • 6. Retroplacental decidual hematoma • Separation, compression and destruction of adjacent placenta • Varieties: Concealed, Revealed & mixed • Couvelaire uterus 6 PATHOLOGY
  • 7. Clinical Features Sign / symptom Frequency% Vaginal bleeding 78 Uterine tenderness back pain 66 Fetal distress 60 Preterm labor 22 Hypertonic contractions 17 Hyper tonus 17 Dead fetus 15 7
  • 8. Diagnosis… Essentially a clinical diagnosis •Painful bleeding •Abdominal findings •Cardiotocographic Monitoring • AFP 8
  • 10. Role of ultrasound – limited •Negative findings do not exclude the diagnosis •U/S is mainly used to confirm fetal viability, Presentation & position. •To exclude Placenta praevia 10
  • 11. Classification Grade 0: Retrospective diagnosis. Grade 1: External bleeding present, no fetal or maternal complication Grade 2: External bleeding +/-, tense & tender uterus, Fetal distress/death Grade 3: External bleeding +/- tense & tender uterus, fetal death with maternal shock . DIC, Renal failure 11
  • 12. Consumptive Coagulopathy • Delee (1901) Temporary hemophilia Parameters: Fibrinogen < 150 mg/dl, • elevated FDP, D-dimers, decrease in other coagulation factors Mechanism: DIC & retro placental clot formation • Seen in 30% cases of abruption severe enough to kill the fetus 12
  • 13. Acute Renal failure • Pathology: Acute tubular necrosis (75%) & acute cortical necrosis (25%) • Mechanism: Severe hypovolemia , DIC along with Underlying preeclampsia • Prevention: Prompt & vigorous replacement of blood and circulating blood volume 13
  • 14. Differential Diagnosis 1. Without pain: Placenta Previa 2. Without Bleeding: Acute degeneration or torsion of a fibroid, hematoma of rectus sheath, rupture of an appendicular abscess. 3. With mild pain & bleeding: Labour with heavy show 4. Rupture Uterus 14
  • 15. Management Initial assessment • Monitor vital signs: BP – poor guide to the extent of bleeding. • Mark the fundal height & measure abdominal girth • cardiotocographic monitoring of fetus Investigations: HB, PCV, blood grouping & typing, BT CT, Clot retraction & lyses, DIC profile • Foley catheterization & hourly output chart • watch for bleeding 15
  • 16. Management “Swift & decisive” Resuscitate the mother • Start an IV line, transfuse Ringer lactate, N Saline • Two lines if bleeding is severe • Replace blood loss and maintain circulation • Maintain PCV at 30% & urine output >30 ml/hr. • CVP in difficult cases • Delivery 16
  • 17. Management Caesarian section • Live & mature fetus • Delivery not imminent • Fetal distress • No response to induction of labour • Bleeding 17
  • 18. Management Vaginal delivery • Preferred if fetus is dead. • In Selected cases with a live fetus • ARM with oxytocin • Watch for PPH • RH prophylaxis 18
  • 19. Management Expectant line of management • Doubtful diagnosis • Minor abruption • Preterm gestation • Intensive surveillance & Induction at or before term 19
  • 20. Perinatal mortality • Main danger is to the fetus. If the abruption is severe enough to threaten the mother, the fetus will usually be dead 25 fold increase in PMR • Still birth • Prematurity • Hypoxia • Cerebral palsy 20
  • 21. Maternal deaths due to hemorrhage Causes of hemorrhage Percentage Abruption 19 Uterine rupture 16 Atonic PPH 15 Coagulopathies 14 Placenta praevia 7 Uterine bleeding 6 placenta accreta/percreta 6 Retained placenta 4 21
  • 22. Maternal mortality Case report: woman, who seemed well enough to wait in an emergency dept for 2 hrs. When the doctor saw her at the end of this time she was dead!!! “a fit woman may be able to compensate for severe hemorrhage until collapse occurs as a terminal event” • A reminder – need to maintain high standards in obstetric care 22
  • 23. 23