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ECG: Atrial Flutter
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16 arrhythmias2009
16 arrhythmias2009
internalmed
Basics of electrophysiology study for heart.
Electrophysiology study basics
Electrophysiology study basics
Satyam Rajvanshi
In this ppt, I am going to discuss various pacemaker malfunction and how to diagnose them.
Evaluation and management of Pacemaker malfunction
Evaluation and management of Pacemaker malfunction
PRAVEEN GUPTA
Electrophysiology study of AVNRT arrhythmia in heart
Electrophysiology AVNRT
Electrophysiology AVNRT
Satyam Rajvanshi
Biatrial enlargement is diagnosed when criteria for both right and left atrial enlargement are present on the same ECG. The diagnosis of biatrial enlargement requires criteria for LAE and RAE to be met in either lead II, lead V1 or a combination of leads.
Atrial enlargement
Atrial enlargement
Ramachandra Barik
Supraventricular tachycardias
Supraventricular tachycardias
Praveen Nagula
Approach to svt
Approach to svt
Approach to svt
Amir Mahmoud
Cardiology, internal medicine
Atrial fibrillation (af) and flutter
Atrial fibrillation (af) and flutter
Domina Petric
Recommandé
16 arrhythmias2009
16 arrhythmias2009
internalmed
Basics of electrophysiology study for heart.
Electrophysiology study basics
Electrophysiology study basics
Satyam Rajvanshi
In this ppt, I am going to discuss various pacemaker malfunction and how to diagnose them.
Evaluation and management of Pacemaker malfunction
Evaluation and management of Pacemaker malfunction
PRAVEEN GUPTA
Electrophysiology study of AVNRT arrhythmia in heart
Electrophysiology AVNRT
Electrophysiology AVNRT
Satyam Rajvanshi
Biatrial enlargement is diagnosed when criteria for both right and left atrial enlargement are present on the same ECG. The diagnosis of biatrial enlargement requires criteria for LAE and RAE to be met in either lead II, lead V1 or a combination of leads.
Atrial enlargement
Atrial enlargement
Ramachandra Barik
Supraventricular tachycardias
Supraventricular tachycardias
Praveen Nagula
Approach to svt
Approach to svt
Approach to svt
Amir Mahmoud
Cardiology, internal medicine
Atrial fibrillation (af) and flutter
Atrial fibrillation (af) and flutter
Domina Petric
Pulmonary stenosis presentation
Pulmonary stenosis presentation
Pulmonary stenosis presentation
Nizam Uddin
Atrial flutter- an uncommon arrhythmia and understanding of its pathophysiology, types and treatment options...
Atrial flutter (AFl) – management principals
Atrial flutter (AFl) – management principals
Prithvi Puwar
Electrophysiology evaluation of SA node
Sinus Node Dysfunction
Sinus Node Dysfunction
Raghu Kishore Galla
Bundle branch blocks
Bundle branch blocks
Adarsh
AV nodal reentrant tachycardia (AVNRT), or atrioventricular nodal reentrant tachycardia, is a type of tachycardia (fast rhythm) of the heart. It is a type of supraventricular tachycardia (SVT), meaning that it originates from a location within the heart above the bundle of His. AV nodal reentrant tachycardia is the most common regular supraventricular tachycardia. It is more common in women than men (approximately 75% of cases occur in females). The main symptom is palpitations. Treatment may be with specific physical maneuvers, medication, or, rarely, synchronized cardioversion. Frequent attacks may require radiofrequency ablation, in which the abnormally conducting tissue in the heart is destroyed. AVNRT occurs when a reentry circuit forms within or just next to the atrioventricular node. The circuit usually involves two anatomical pathways: the fast pathway and the slow pathway, which are both in the right atrium. The slow pathway (which is usually targeted for ablation) is located inferior and slightly posterior to the AV node, often following the anterior margin of the coronary sinus. The fast pathway is usually located just superior and posterior to the AV node. These pathways are formed from tissue that behaves very much like the AV node, and some authors regard them as part of the AV node. The fast and slow pathways should not be confused with the accessory pathways that give rise to Wolff-Parkinson-White syndrome (WPW syndrome) or atrioventricular reciprocating tachycardia (AVRT). In AVNRT, the fast and slow pathways are located within the right atrium close to or within the AV node and exhibit electrophysiologic properties similar to AV nodal tissue. Accessory pathways that give rise to WPW syndrome and AVRT are located in the atrioventricular valvular rings. They provide a direct connection between the atria and ventricles, and have electrophysiologic properties similar to ventricular myocardium.
AVNRT
AVNRT
Ramachandra Barik
Ecg criteria of chamber enlargement
Ecg criteria of chamber enlargement
Adarsh
Premature Atrial complexes
Premature Atrial complexes
Mohamed Salih Aziz
Echo assessment of RV function
Echo assessment of RV function
Echo assessment of RV function
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Echo assessment of aortic valve disease
Echo assessment of aortic valve disease
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IDIOPATHIC FASCICULAR VT
FASCICULAR VENTRICULAR TACHYCARDIA( VT)
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Stanley Medical College, Department of Medicine
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Vasoreactive testing in pulmonary hypertension
Vasoreactive testing in pulmonary hypertension
Himanshu Rana
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Electrophysiology AVRT
Satyam Rajvanshi
Aortic regurgitation
Aortic regurgitation
Dhinil Dares
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Peripheral Vascular Examination
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Shkar Ahmed
Contenu connexe
Tendances
Pulmonary stenosis presentation
Pulmonary stenosis presentation
Pulmonary stenosis presentation
Nizam Uddin
Atrial flutter- an uncommon arrhythmia and understanding of its pathophysiology, types and treatment options...
Atrial flutter (AFl) – management principals
Atrial flutter (AFl) – management principals
Prithvi Puwar
Electrophysiology evaluation of SA node
Sinus Node Dysfunction
Sinus Node Dysfunction
Raghu Kishore Galla
Bundle branch blocks
Bundle branch blocks
Adarsh
AV nodal reentrant tachycardia (AVNRT), or atrioventricular nodal reentrant tachycardia, is a type of tachycardia (fast rhythm) of the heart. It is a type of supraventricular tachycardia (SVT), meaning that it originates from a location within the heart above the bundle of His. AV nodal reentrant tachycardia is the most common regular supraventricular tachycardia. It is more common in women than men (approximately 75% of cases occur in females). The main symptom is palpitations. Treatment may be with specific physical maneuvers, medication, or, rarely, synchronized cardioversion. Frequent attacks may require radiofrequency ablation, in which the abnormally conducting tissue in the heart is destroyed. AVNRT occurs when a reentry circuit forms within or just next to the atrioventricular node. The circuit usually involves two anatomical pathways: the fast pathway and the slow pathway, which are both in the right atrium. The slow pathway (which is usually targeted for ablation) is located inferior and slightly posterior to the AV node, often following the anterior margin of the coronary sinus. The fast pathway is usually located just superior and posterior to the AV node. These pathways are formed from tissue that behaves very much like the AV node, and some authors regard them as part of the AV node. The fast and slow pathways should not be confused with the accessory pathways that give rise to Wolff-Parkinson-White syndrome (WPW syndrome) or atrioventricular reciprocating tachycardia (AVRT). In AVNRT, the fast and slow pathways are located within the right atrium close to or within the AV node and exhibit electrophysiologic properties similar to AV nodal tissue. Accessory pathways that give rise to WPW syndrome and AVRT are located in the atrioventricular valvular rings. They provide a direct connection between the atria and ventricles, and have electrophysiologic properties similar to ventricular myocardium.
AVNRT
AVNRT
Ramachandra Barik
Ecg criteria of chamber enlargement
Ecg criteria of chamber enlargement
Adarsh
Premature Atrial complexes
Premature Atrial complexes
Mohamed Salih Aziz
Echo assessment of RV function
Echo assessment of RV function
Echo assessment of RV function
Dr. Md. Ahasanul Kabir Shahin
Echo assessment of aortic valve disease
Echo assessment of aortic valve disease
Echo assessment of aortic valve disease
Nizam Uddin
IDIOPATHIC FASCICULAR VT
FASCICULAR VENTRICULAR TACHYCARDIA( VT)
FASCICULAR VENTRICULAR TACHYCARDIA( VT)
Malleswara rao Dangeti
Restrictive cardiomyopathy
Restrictive cardiomyopathy
Restrictive cardiomyopathy
Nizam Uddin
Mitral Stenosis identification, quantification, etiology and associated findings in echocardiography
Echo Mitral Stenosis
Echo Mitral Stenosis
Mashiul Alam
Stress echocardiography
Stress echocardiography
Muhammad Mobarock Hossain
MI ECG LOCALISATION ST ELEVATION DIFFERENTIAL DIAGNOSIS
Myocardial infarction (MI) ecg localisation
Myocardial infarction (MI) ecg localisation
Malleswara rao Dangeti
ECG: Pericarditis
ECG: Pericarditis
Stanley Medical College, Department of Medicine
Rawalpindi Medical college
Supraventricular tachyarrythmias
Supraventricular tachyarrythmias
Rawalpindi Medical College
Echo assessment of lv systolic function and swma
Echo assessment of lv systolic function and swma
Fuad Farooq
Left ventricular diastolic dysfunction in echocardiography
Left ventricular diastolic dysfunction in echocardiography
Left ventricular diastolic dysfunction in echocardiography
Yukta Wankhede
Vasoreactivity testing in Pulmonary Hypertension as a guide to treatment and responsiveness to calcium channel blockers
Vasoreactive testing in pulmonary hypertension
Vasoreactive testing in pulmonary hypertension
Himanshu Rana
Electrophysiology of AV reentrant tachycardia in heart
Electrophysiology AVRT
Electrophysiology AVRT
Satyam Rajvanshi
Tendances
(20)
Pulmonary stenosis presentation
Pulmonary stenosis presentation
Atrial flutter (AFl) – management principals
Atrial flutter (AFl) – management principals
Sinus Node Dysfunction
Sinus Node Dysfunction
Bundle branch blocks
Bundle branch blocks
AVNRT
AVNRT
Ecg criteria of chamber enlargement
Ecg criteria of chamber enlargement
Premature Atrial complexes
Premature Atrial complexes
Echo assessment of RV function
Echo assessment of RV function
Echo assessment of aortic valve disease
Echo assessment of aortic valve disease
FASCICULAR VENTRICULAR TACHYCARDIA( VT)
FASCICULAR VENTRICULAR TACHYCARDIA( VT)
Restrictive cardiomyopathy
Restrictive cardiomyopathy
Echo Mitral Stenosis
Echo Mitral Stenosis
Stress echocardiography
Stress echocardiography
Myocardial infarction (MI) ecg localisation
Myocardial infarction (MI) ecg localisation
ECG: Pericarditis
ECG: Pericarditis
Supraventricular tachyarrythmias
Supraventricular tachyarrythmias
Echo assessment of lv systolic function and swma
Echo assessment of lv systolic function and swma
Left ventricular diastolic dysfunction in echocardiography
Left ventricular diastolic dysfunction in echocardiography
Vasoreactive testing in pulmonary hypertension
Vasoreactive testing in pulmonary hypertension
Electrophysiology AVRT
Electrophysiology AVRT
En vedette
Aortic regurgitation
Aortic regurgitation
Dhinil Dares
Peripheral Vascular Examination by Dr. Aram Baram
Peripheral Vascular Examination
Peripheral Vascular Examination
Shkar Ahmed
Patent ductus arteriosus (PDA) is a congenital disorder in the heart wherein a neonate's ductus arteriosus fails to close after birth. Early symptoms are uncommon, but in the first year of life include increased work of breathing and poor weight gain. With age, the PDA may lead to congestive heart failure if left uncorrected. The ductus arteriosus is a normal fetal blood vessel that closes soon after birth. In a patent ductus arteriosus (PDA) the vessel does not close and remains "patent" (open) resulting in irregular transmission of blood between two of the most important arteries close to the heart, the aorta and the pulmonary artery. PDA is common in neonates with persistent respiratory problems such as hypoxia, and has a high occurrence in premature children. In hypoxic newborns, too little oxygen reaches the lungs to produce sufficient levels of bradykinin and subsequent closing of the DA. Premature children are more likely to be hypoxic and thus have PDA because of their underdeveloped heart and lungs. A patent ductus arteriosus allows a portion of the oxygenated blood from the left heart to flow back to the lungs by flowing from the aorta (which has higher pressure) to the pulmonary artery. If this shunt is substantial, the neonate becomes short of breath: the additional fluid returning to the lungs increases lung pressure to the point that the neonate has greater difficulty inflating the lungs. This uses more calories than normal and often interferes with feeding in infancy. This condition, as a constellation of findings, is called congestive heart failure. In some cases, such as in transposition of the great vessels (the pulmonary artery and the aorta), a PDA may need to remain open. In this cardiovascular condition, the PDA is the only way that oxygenated blood can mix with deoxygenated blood. In these cases, prostaglandins are used to keep the patent ductus arteriosus open
Patent ductus arteriosus
Patent ductus arteriosus
Ramachandra Barik
Cardiology Department
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Unstable Angina Pectoris
Faradhillah Adi Suryadi
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Pratap Tiwari
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Jyotindra Singh
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حسين عزالدين
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Hope it could help ^^
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Mitral stenosis
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Glycated haemoglobin ppt by Basalingappa BG
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TRANS ESOPHAGEAL ECHOCARDIOGRAPHY
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Raynaud's phenomenon
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Glucosuria
Glucosuria
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