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Applied aspects of
Tridoshasidhanta
Dr. Vinodkumar M V
Professor, Dept. of Samhita, Sanskrit & Sidhanta
VPSV Ayurveda College Kottakkal, Kerala
ayurvin@gmail.com
Tridosha
Explains relatedness of a living organism to all
possible variables in and out its matrix
Characters under continuous variation
Biological dynamics
Not necessarily follow physical dynamics
Shift from Philosophy to Practice
Phiolosophy, the
raw material
Panchabhoota, refined
material extracted from
raw material
Tridosha, fine product from
refined material
Treatment, final
application, based on
need
From 2 to 3
 Usually Universe is explained through dichotomic expressions like
 Ushna X Seeta (Agneeshomeeya)
 Snigdha X Ruksha (Snehasara)
 Tridosha explains Trichotomy
 Shift from Dichotomy to Trichotomy is important
Ushna
Seeta
Rooksha
Snigdha
Pitta
?
Kapha
Stability
Vaata
Mobility
Seeta+Ruksha – Vata
Seeta+Snigdha - Kapha
Ushna+Sngdha - Pitta
Where is Ushna+Ruksha
A rare,
abnormal,
destructive
phenomenon
Seeta-
snigdha
Seeta-
rooksha
Ushna-
snigdha
TRIGUNA
PANCHABHOOTA
TRIDOSHA
Evolution
Universal
perspective
Material
perspective
Biological
perspective
Relative status in health and
diseases
Dosha – They are easily disturbed and put disturbance to the system
Dhatu – Normally they support the biological system
Mala – They sometimes contaminate the body
Sthoona – Behave like three pillars, to support the biological system
TRIDOSHA – is it ‘DRAVYA’ or not?
THE RECIPROCAL
DEFINITION OF DRAVYA
WHEREVER THERE IS QUALITY &
ACTIVITY
THERE SHOULD BE AN UNDERLYING
DRAVYA
IF ‘DRAVYA’ IS NOT
PERCEIVABLE
GO FOR ITS QUALITIES AND
ACTIVITIES
Is sugar
directly
perceivable
it the tea?
Tridosha - Definitions
NOT
In terms of structure
BUT
Qualities and activities
Gunas (Qualities)
 Gunas not for description but for APPLICATION
 Main basis to apply saamaanya-visesha sidhaanta
 Exclusively Gurvaadiguna;
 Exceptions - Pitta & Kapha (visra & mritsna)
 There is a cause-effect relation between Guna & Karma
Dormant & Dominant Gunas
 Almost all the functions of Doshas are expressions of
underlying Gunas
 All the Gunas of Doshas are not equally active in all the
functions
 Eg: Normal functions of Vata (Utsaha-achwasa-niswasa etc.)
predominate with Calaguna only
 Then where are the other Gunas?
 So, Gunas should be understood distinctively as dominant
Gunas & dormant Gunas
Rule - 1
 Guna dormant in normal state may become
dominant in abnormal state
 Eg: Vata – Normal function – Utsaha etc – dominant
Guna – Cala
 Vata – increased presence (Vriddhilakshana)
 Kaarsya – Laghu
 Karshnya – Ruksha
 Ushnakamitwa – Seeta
 Kampa – Cala
 Sakritgraha – Ruksha, Seeta etc.
Different Gunas
exhibit their
functions
KOSHT’HA
NORMAL
INCREASE
CALA
ROOKSHA
NORMAL
PERISTALTIC
MOVEMENT
CONSTIPATION
For Vata
Rule - 2
 Quality dormant in one activity may become dominant in
another activity in the same state of Dosha
 See the following examples for Vata
SANGAM
STAMBHAM
S’EETA
KAMPAMCHALA
REDUCED
MOVEMENT
HYPER
MOBILITY
Rule - 3
 Quality dominant in one site/situation may be
dormant in another site/situation
Site VAATA PITTA KAPHA
AGNI Vishamaagni due to
CHALA
(ANAVASTHITATWA)
Teekshna due to
USHNA TEEKSHNA
Manda due to GURU
MANDA
KOSHT’HA Kroora due to
ROOKSHA S’EETA
Mridu due to DRAVA
SARA
Madhyama due to
SEETA SNIGDHA
PITTA
TEEKSHNA
PRAKAASA
USHNA
SNEHA
Pakti
Ooshma
Darsana
Prabha
Medha
Dhee
Saurya
Tanumaardava
Paachaka
Ranjaka
Bhraajaka
Aalochaka
Saadhaka
25042011 21RoTP TPRA
MIND Sharpness
Mobility
Stabilty
Easiness
Receptivity
Paatava
Utsaaha
Medha
Dhee
Smriti
Kshama
Descrimination
Judgement
Retention
Memory
Vaata
Kapha
Pitta
Variations of Dosha
(Doshabheda)
 Sites (Sthaana)
 Functions (Karma)
 Stage (Avasthaa)
 Cause (Hetu)
 Manifestation (Aakriti)
 Management (Saadhana)
 Combination (Samyoga)
Ref: Doshabhedeeya A H Soo 12 (Sarvangasundara)
Chief locations
 Related to feeding, processing & evacuation
 Related to sites of production in the gut
Importance of gut in biology
INLET
OUTLET
VACCUM AT THE
BOTTOM
PROCESSING
Heaviness
Lightness
MAN
Sites – clinical importance
 Includes dhaatu, kosht’ha, indriya etc.
 Clinical - Compare with rogamaarga
 Vaata – mostly madhyama
 Kapha-pitta – mostly – saakha,kosht’ha
 Related to prognosis – tulya/atulyadooshyata
 Grouping of syndrome
 eg: aamavaata – “sleshmasthaanam pradhaavati”
 Sleshmaas’ayaanaam soonyatvam
Functional varieties (karmabheda)
 Five types of doshas
 Classification - on the basis of function
Functional sites
 Main repository (moolasthaana) & functional territories
(gocharasthaana)
 Almost totally different from structural sites
 Function more important than site
 Physiological importance stressed initially
 Pathologically - Becomes more important in the context of
aavarana
 Explains the harmonious co-existence of doshas
Stage (avastha)
 Accumulation (chaya)
 Circulation (prakopa)
 Normalization (pras’ama)
 Variants – s’eeta and ushna
 Explains seasonal phenomena
 Practically explainable
Varieties of cause (hetubheda)
 Three types of causative agents
 Includes food, physical activities, mental activities,
facultative activities, seasonal variations etc.
 Not dosha specific
 Not disease specific
 Only headings
Varieties of management
(saadhanabheda)
 Snigdhopas’aya – Vaata, vaata-pitta
 Rookshopas’aya – kapha, kapha-pitta
Varieties of association (samyogabheda)
 Samsarga
 Sannipaata
 Then what is anubandha…?
 Because any two of the doshas have some common features
 In the process of disease one dosha may take up another
dosha with it
Dosha vikriti
 Decrease (Kshaya)
 Increase (vriddhi)
 Signature features (Aatmaroopa)
 Morbid activities (Kupitakarma)
 Independent diseases (naanaatmajavyaadhi)
 Dependent diseases (saamaanyajavyaadhi)
Increase and decrease of doshas
are understood through increase
of some or other qualities
Symptoms of Increase (vriddhilakshana)
 General indicators of particular dosha
 Students’ guideline for dosha
 Not a clinician’s guideline
 Not disease-specific
 But, sometimes help for differential diagnosis
 Important during an ‘identity crisis’
Clinical importance
 Not suggesting the process of disease but, indicating
doshaja variety of a disease or to identify
anubandhadosha
 Eg: kshut, trit (pitta)
 Some times very important in designing treatment
 Trishna in jvara
 Alpanidrata in depression
Signature features (aatmaroopa)*
 Mentioned in Carakasamhita
 Sure indicators of particular dosha
 सर्वेष्र्वपि खल्र्वेतेषु र्वातपर्वकारेषूक्तेष्र्वन्येषु चानुक्तेषु र्वायोररदमात्मरूिमिररणामम
कममणश्च स्र्वलक्षणं, यदुिलभ्य तदर्वयर्वं र्वा पर्वमुक्तसन्देहा र्वातपर्वकारमेर्वाध्यर्वस्यन्न्त
कु शलााः (C.S.Su 20/12)
 Responsible for different activities when localized in tissues
 Guna-karma relationship between aatmaroopa-prakupitakarma
Signature features (aatmaroopa)
Vaata
Raukshya
Saitya
Laaghava
Vaisadya
Gati
Amoortatva
Anavasthitatva
Pitta
Aushnya
Taikshnya
Dravatva
Anatisneha
Suklaarunavarjya-
varna
Visragandha
Katukaamlarasa
Saratva
Kapha
Sneha
Saitya
Sauklya
Gaurava
Maadhurya
Sthairya
Paichilya
Maartsnya
Ref: Carakasamhita Sutrasthana 20/12,14,16
Morbid activities (prakupita karma)
 Indicators of pathological processes at different
sites of action
 Basic understanding of mechanism of dosha
action
 Should be understood in comparison with site of
action
 Related to sthaanasamsraya of dosha in
different sites
 Dhaatugatha avastha of doshas can be taken as
a simple model
Pathogenesis
Aatmaroopa
Dosha Dooshya
Prakupitakarma
sthaanasamsraya
Disease
Saamaanyaja
naanaatmaja
Diseases – two types
 Aatmaroopa of a single dosha expressed at a site
leads to Naanaatmajavyaadhi
 nakhabheda - raukshya at nakha –
 Anavasthitachittatva – anavasthitatva of manas
 Davathu – aushnya of indriyas
 Aatmaroopa of more than one dosha expressed at
a site leads to Saamaanyayaadhi
Vaata – prakupitakarma
Ref: A H Sutra
Sramsa/Bhrams’a
 Caused by laghutva & chalatva
 Loosening /dislocation/displacement
 Loosening – muscle tone/ flaccidity
 Dislocation – joint
 Muscle dislocation – prolapses, intussusception, ptosis,
hernia etc.
 Dislocation – in aasayaapaksrshagati, atisaara
(visramsayati abdhaatum)
Vyaasa - dilation
 Due to predominance of aakaas’a & lightness
 Production of avakaas’a in dhaatukshaya
 Mechanism of hypertrophy
 Dilation of lumens – aneurysm, cardiomegaly,
brochiectasis etc.
 In the sampraapti of raajayakshma – “ativivritya vaa”
Svaapa
 “kriyaasu achaitanyam”
 Sensory loss
Saada
 Functional insufficiency (kriyaasu asamartha)
 Deficiencies of different factors Eg: insulin deficiency
(type II)
Sankocha & Sanga –
Partial and complete obstruction
 Due to seeta, rooksha qualities
 Mostly of a lumen
 Mostly mechanical
 Otherwise called rodham
 GI tact - udara, gunma, udaavarta, ruddhapadakaamala
 Respiratory tract – swaasa, kaasa
 Mootravahasrotas – mootraaghaata, asmari
 Circulatory system – atherosclerosis etc.
Vartana
 Pindeekarana – mass formation
 Gunma, Space occupying lesions etc.
 Kampa
 due to chalatva
 tremor
 Paarushya
 due to kharatva
 harshness (of voice)
 Soushirya
 due to saukshmya
 porosity (mainly in bone tissue)
 S’osha
 due to rooksha & laghu
 wasting, dystrophy, degeneration
 Spandana
 Due to chalatva
 Fasciculations, involuntar y movements
Stambha
 Due to seeta+rooksha
 Solid medium - Rigidity, stiffness
 Liquid medium – arrest of flow - (like in coagulation)
 Malastambha
sosha
Bhrams’a
Paarushya
vyaasa
sanga
Diseases
Naanaatmaja
Absence of other doshas
in history, clinical
examination or
anupas’aya-anupas’aya
Saamaanyaja
Presence of other doshas
in history, clinical
examination or upas’aya-
anupas’aya
Independent diseases
(naanaatmajavyaadhi)
 Independent of other morbid factors
 Mostly localized diseases
 Minimum systemic involvement/pathogenesis
 Linear correlation with aatmaroopa
General diseases (saamaanyajavyaadhi)
 Involvement of more morbid factors
 Process of pathogenesis well identified
 Involvement of co-morbid factors like srotas, agni, aama,
ojus etc.
Conversion
 Some diseases may have naanaatmaja and saamaanyaja stages
Eg: Gridhrasi, pakshaaghaata etc.
 Sometimes, at a particular stage of treatment a saamaanyaja
disease can turn naanaatmaja (keveladoshaavastha)
 Most of the saamaanyajavyaadhi can not become naanaatmaja
Eg: swaasa, udara, raajayakshma etc.
Paaka
Sruti, kleda
Sanga,
sankocha
Vyaasa
Saushirya
Scar, loss of elasticity
(kaathinya)
Guna Aatmaroopa
Prakupita-
karma
DooshyaVyaadhi
Naanaatmajam
Saamaanyam
Mutual interaction
 Aama
 Aavarana
Exceptions
 Vikritivishamasamavaaya
Beyond tridosha
 Rishta
 Bhootagraha
TRIDOSHA – POSSIBLE VARIATIONS
Normal
Prakriti
Normal variation
Chaya
Prakopa
TRIDOSHA – POSSIBLE VARIATIONS
Normal
Prakriti
Normal variation
Chaya
Prakopa
COMBINATIONS
SAMSARGA SANNIPAATA
SIX COMBINATIONS OF SAMSARGA
TEN COMBINATIONS OF SANNIPAATA
TEN COMBINATIONS OF SANNIPAATASIX COMBINATIONS OF SAMSARGA
THANK YOU
Applied aspects of Tridosha Siddhanta
Applied aspects of Tridosha Siddhanta

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Applied aspects of Tridosha Siddhanta

Notes de l'éditeur

  1. Susrutas description