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Hemorrhage & Shock
Its Management In
Oral Surgery
Instructor – Dr.Jesus George 1
Hemorrhage
 Bleeding
 Escape of blood from a blood vessel due to
damage or injury in vasculature
2
Types
 A-depending upon the nature of the
vessel
 Arterial hemorrhage
 Bright red color
 Pulsation of the artery can be seen
 Venous hemorrhage
 Dark red color,
 Non-pulsating with steady flow(constant
flow)
 Difficult to control because vein gets
retracted
3
Cont.
 Capillary hemorrhage
 Red colour
 Slowly oozes out
 It becomes significant if there is
bleeding tendencies
 Bony bleeding
 From nutrient vessels in bone
 Usually follows removal of lower 3rd
molar
4
Cont.
 B. Depending upon timing of
hemorrhage
 Primary hemorrhage -occurs at the time
of surgery
 Reactionary or intermediate
hemorrhage -occurs after 24 hrs of
surgery(causes-hypertension in
postoperative period, sneezing,coughing)
 Secondary hemorrhage -occurs after
48-72 hrs after surgery (infection)
5
Cont.
 C-depending upon the duration of
hemorrhage
 Acute hemorrhage -occurs suddenly
(esophageal variceal bleeding due to
portal hypertension)
 Chronic hemorrhage-(hemorrhoids or
piles or chronic duodenal ulcer
6
Cont.
 D-depending upon the nature of
bleeding
 External hemorrhage - epistaxis
 Internal hemorrhage - splenic rupture
7
Laboratory tests for screening
 Bleeding time (aspirin therapy)
 Platelet count (liver disease, c/c leukemia)
 Prothrombin time (heparin therapy, liver
disease)
8
Hemostasis
 Control or arrest of bleeding
9
Local hemostatic measures
 Mechanical methods
 Thermal methods
 Chemical methods
10
Mechanical methods
 Pressure
 Use of hemostats
 Sutures & ligation
11
Thermal methods
 Cautery
 Electrosurgery
 Cryosurgery
12
Chemical methods
 Astringents (ferric sulfate in ethyl cellulose)
 Styptics (epsilon amino caproic acid local &
systemic)
 Bone wax (bees wax(7)+olive
oil(2)+phenol(1),helps in platelet aggregation)
 Gelfoam (compressed sponge made up of porcine
skin & have physical action)
 Adrenaline
 Surgicel (knitted fabric, interaction with platelets)
 Fibrin glue (thrombin+fibrinogen+factor8)
13
Prevention of hemorrhage during &
after extraction
 Primary hemorrhage – tender handling
of soft tissues & bone.
 Reactionary hemorrhage – semi supine
or sitting posture, diazepam 5 mg at
bedtime or antihypertensive drugs after
consultation with the physician
 Secondary hemorrhage - antibiotics
14
Control of hemorrhage from major
arteries
 Greater palatine artery
 Pressure pack
 A round bolus of gauze is made of
adequate size , so that it does not cause
gagging
 It is kept in place by tie over sutures for
24 to 48 hours
 Pressure pack can be safely removed
after 48 hours
15
Cont.
 Lingual artery
 Local clamping of the artery & application
of electrocautery usually controls bleeding
 Superficial temporal artery
 Electrocautery
16
Hereditary coagulopathies
 Hemophilia -a
 Hemophilia -b
17
Hemophilia -a
 Caused by deficiency of factor 8
 Sex-linked recessive inherited disorder
 Seen in males
 Hemophilia-a is graded as mild ,
moderate ,& severe
 Mild - factor 8 level ranges from 6 to 30
percent
 Moderate - factor 8 level ranges from 1
to 5 percent
18
Cont.
 Severe - factor 8 level ranges from nil to
1 percent
 Clinical features - prolonged bleeding
tendencies even after a mild trauma
 Patients are higher risk for bleeding after
oral surgical procedures
19
Hemophilia b
 It is caused by deficiency of factor 9
 It is congenital disorder& transmitted as
sex--linked recessive trait
 Clinical manifestations of this disease
are similar to hemophilia a
20
Shock
 Shock is a pathophysiologic condition
clinically recognized as a state of
inadequate perfusion due to
inadequate blood flow,there is
inadequate delivery of nutrients to the
tissues & inadequate removal of
cellular waste products from tissue cells,
results in disruption of vital organ
functions.
21
Clinical features
 Mild - <20% blood loss
 Cold, calmy,moist skin
 ↓ in BP
 Rapid pulse
 Collapsed neck veins
 Concentrated urine
22
Cont.
 Moderate - >20-40%
 Feeling of thirsty
 Hypotension
 Rapid pulse
 Oliguria or anuria
 Severe - >40 %
 Agitated & confused patient
 Hypotension
 Rapid pulse
 Rapid respiration
23
Classification
 Hypovolemic shock
 Cardiogenic shock
 Septic shock
 Neurogenic shock
 Anaphylactic shock
24
Hypovolemic shock
 Decrease in the circulating or effective
intravascular volume
 Most common type of shock in
maxillofacial trauma
 Hypovolemic shock is classified into
 1.Haemorrhagic shock
 2.Non- hemorrhagic shock
25
Cont.
 Hemorrhagic shock is due to loss of
blood from the body causing decreased
venous return & thus decreasing the
cardiac output
26
Cont.
 Non-hemorrhagic shock-there is
massive fluid shift from intravascular
compartment to extra vascular
compartment causing decreased blood
volume &thus decreasing tissue perfusion.
 can result from burns, crush injuries,
pancreatitis, peritonitis & pleural effusion,
water loss due to severe diarrhea, vomiting,
hyperglycemia & nephritis
27
Cont.
 Treatment
 Control bleeding
 Legs raised & body supine is preferred
posture as this increases venous return &
cardiac index
 Fluid replacement; normal saline or ringer
lactate
28
Cardiogenic shock
 Inadequate cardiac output , impaired
oxygen delivery & reduced tissue
perfusion, caused by loss of effective
contractile function of myocardium
 Treatment
 Dopamine is the vasopressor of choice. It
is diluted in normal saline or 5%dextrose
drip
29
Septic shock
 Produced by microorganisms or toxins
 May be produced by bacteria ,virus &
fungi
 Inflammatory response causing
vasodilatation & hypovolemia
 Clinically: fever, tachycardia&
respiratory alkalosis
30
Cont.
 Treatment
 Early & effective volume replacement
 Restoration of tissue perfusion
 Control of infection with antibiotic therapy
 I.V betamethasone or dexamethasone
31
Neurogenic shock
 Vasovagal syncope or emotional fainting
 Blockage of sympathetic nervous system
→dialatation of blood vessels →hypovolemia
→shock
 Predisposing factors – fear, anxiety, painPredisposing factors – fear, anxiety, pain
- fatigue- fatigue
- fasting- fasting
- hot & humid weather- hot & humid weather
32
Cont.
 Signs & symptoms(Clinical feature):Signs & symptoms(Clinical feature):
 Pale, cold & moist skinPale, cold & moist skin
 Slow pulse initially followed by rapid pulseSlow pulse initially followed by rapid pulse
 DizzinessDizziness
 WeaknessWeakness
 Nausea & sweatingNausea & sweating
 Loss of consciousnessLoss of consciousness
 Dilated pupilDilated pupil
33
Cont.
 Prevention
 Reassurance
 Achieve confidence of the patient
 Anxiolytic medication prior to surgery
(diazepam 5 mg 1 tablet night before the
surgery & one 1/2hr before surgery)
 patient should not come with empty stomach
34
Cont.
 ManagementManagement
 Monitoring pulseMonitoring pulse
 Lowering head to improve cerebellar circulationLowering head to improve cerebellar circulation
 Loosening of tight cloths at neckLoosening of tight cloths at neck
 Smelling of salt of aromatic ammoniaSmelling of salt of aromatic ammonia
 Flashing the face with cold waterFlashing the face with cold water
 Glucose drinksGlucose drinks
 Inj.dexamethasoneInj.dexamethasone
 Reassurance of patientReassurance of patient
35
Anaphylactic shock
 Caused by anaphylaxis of penicillin&
streptomycin
 Treatment
 Oxygen
 Airway
 Injection adrenaline
 Cardiopulmonary resuscitation
36

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14 hemorrhage & shock

  • 1. Hemorrhage & Shock Its Management In Oral Surgery Instructor – Dr.Jesus George 1
  • 2. Hemorrhage  Bleeding  Escape of blood from a blood vessel due to damage or injury in vasculature 2
  • 3. Types  A-depending upon the nature of the vessel  Arterial hemorrhage  Bright red color  Pulsation of the artery can be seen  Venous hemorrhage  Dark red color,  Non-pulsating with steady flow(constant flow)  Difficult to control because vein gets retracted 3
  • 4. Cont.  Capillary hemorrhage  Red colour  Slowly oozes out  It becomes significant if there is bleeding tendencies  Bony bleeding  From nutrient vessels in bone  Usually follows removal of lower 3rd molar 4
  • 5. Cont.  B. Depending upon timing of hemorrhage  Primary hemorrhage -occurs at the time of surgery  Reactionary or intermediate hemorrhage -occurs after 24 hrs of surgery(causes-hypertension in postoperative period, sneezing,coughing)  Secondary hemorrhage -occurs after 48-72 hrs after surgery (infection) 5
  • 6. Cont.  C-depending upon the duration of hemorrhage  Acute hemorrhage -occurs suddenly (esophageal variceal bleeding due to portal hypertension)  Chronic hemorrhage-(hemorrhoids or piles or chronic duodenal ulcer 6
  • 7. Cont.  D-depending upon the nature of bleeding  External hemorrhage - epistaxis  Internal hemorrhage - splenic rupture 7
  • 8. Laboratory tests for screening  Bleeding time (aspirin therapy)  Platelet count (liver disease, c/c leukemia)  Prothrombin time (heparin therapy, liver disease) 8
  • 9. Hemostasis  Control or arrest of bleeding 9
  • 10. Local hemostatic measures  Mechanical methods  Thermal methods  Chemical methods 10
  • 11. Mechanical methods  Pressure  Use of hemostats  Sutures & ligation 11
  • 12. Thermal methods  Cautery  Electrosurgery  Cryosurgery 12
  • 13. Chemical methods  Astringents (ferric sulfate in ethyl cellulose)  Styptics (epsilon amino caproic acid local & systemic)  Bone wax (bees wax(7)+olive oil(2)+phenol(1),helps in platelet aggregation)  Gelfoam (compressed sponge made up of porcine skin & have physical action)  Adrenaline  Surgicel (knitted fabric, interaction with platelets)  Fibrin glue (thrombin+fibrinogen+factor8) 13
  • 14. Prevention of hemorrhage during & after extraction  Primary hemorrhage – tender handling of soft tissues & bone.  Reactionary hemorrhage – semi supine or sitting posture, diazepam 5 mg at bedtime or antihypertensive drugs after consultation with the physician  Secondary hemorrhage - antibiotics 14
  • 15. Control of hemorrhage from major arteries  Greater palatine artery  Pressure pack  A round bolus of gauze is made of adequate size , so that it does not cause gagging  It is kept in place by tie over sutures for 24 to 48 hours  Pressure pack can be safely removed after 48 hours 15
  • 16. Cont.  Lingual artery  Local clamping of the artery & application of electrocautery usually controls bleeding  Superficial temporal artery  Electrocautery 16
  • 17. Hereditary coagulopathies  Hemophilia -a  Hemophilia -b 17
  • 18. Hemophilia -a  Caused by deficiency of factor 8  Sex-linked recessive inherited disorder  Seen in males  Hemophilia-a is graded as mild , moderate ,& severe  Mild - factor 8 level ranges from 6 to 30 percent  Moderate - factor 8 level ranges from 1 to 5 percent 18
  • 19. Cont.  Severe - factor 8 level ranges from nil to 1 percent  Clinical features - prolonged bleeding tendencies even after a mild trauma  Patients are higher risk for bleeding after oral surgical procedures 19
  • 20. Hemophilia b  It is caused by deficiency of factor 9  It is congenital disorder& transmitted as sex--linked recessive trait  Clinical manifestations of this disease are similar to hemophilia a 20
  • 21. Shock  Shock is a pathophysiologic condition clinically recognized as a state of inadequate perfusion due to inadequate blood flow,there is inadequate delivery of nutrients to the tissues & inadequate removal of cellular waste products from tissue cells, results in disruption of vital organ functions. 21
  • 22. Clinical features  Mild - <20% blood loss  Cold, calmy,moist skin  ↓ in BP  Rapid pulse  Collapsed neck veins  Concentrated urine 22
  • 23. Cont.  Moderate - >20-40%  Feeling of thirsty  Hypotension  Rapid pulse  Oliguria or anuria  Severe - >40 %  Agitated & confused patient  Hypotension  Rapid pulse  Rapid respiration 23
  • 24. Classification  Hypovolemic shock  Cardiogenic shock  Septic shock  Neurogenic shock  Anaphylactic shock 24
  • 25. Hypovolemic shock  Decrease in the circulating or effective intravascular volume  Most common type of shock in maxillofacial trauma  Hypovolemic shock is classified into  1.Haemorrhagic shock  2.Non- hemorrhagic shock 25
  • 26. Cont.  Hemorrhagic shock is due to loss of blood from the body causing decreased venous return & thus decreasing the cardiac output 26
  • 27. Cont.  Non-hemorrhagic shock-there is massive fluid shift from intravascular compartment to extra vascular compartment causing decreased blood volume &thus decreasing tissue perfusion.  can result from burns, crush injuries, pancreatitis, peritonitis & pleural effusion, water loss due to severe diarrhea, vomiting, hyperglycemia & nephritis 27
  • 28. Cont.  Treatment  Control bleeding  Legs raised & body supine is preferred posture as this increases venous return & cardiac index  Fluid replacement; normal saline or ringer lactate 28
  • 29. Cardiogenic shock  Inadequate cardiac output , impaired oxygen delivery & reduced tissue perfusion, caused by loss of effective contractile function of myocardium  Treatment  Dopamine is the vasopressor of choice. It is diluted in normal saline or 5%dextrose drip 29
  • 30. Septic shock  Produced by microorganisms or toxins  May be produced by bacteria ,virus & fungi  Inflammatory response causing vasodilatation & hypovolemia  Clinically: fever, tachycardia& respiratory alkalosis 30
  • 31. Cont.  Treatment  Early & effective volume replacement  Restoration of tissue perfusion  Control of infection with antibiotic therapy  I.V betamethasone or dexamethasone 31
  • 32. Neurogenic shock  Vasovagal syncope or emotional fainting  Blockage of sympathetic nervous system →dialatation of blood vessels →hypovolemia →shock  Predisposing factors – fear, anxiety, painPredisposing factors – fear, anxiety, pain - fatigue- fatigue - fasting- fasting - hot & humid weather- hot & humid weather 32
  • 33. Cont.  Signs & symptoms(Clinical feature):Signs & symptoms(Clinical feature):  Pale, cold & moist skinPale, cold & moist skin  Slow pulse initially followed by rapid pulseSlow pulse initially followed by rapid pulse  DizzinessDizziness  WeaknessWeakness  Nausea & sweatingNausea & sweating  Loss of consciousnessLoss of consciousness  Dilated pupilDilated pupil 33
  • 34. Cont.  Prevention  Reassurance  Achieve confidence of the patient  Anxiolytic medication prior to surgery (diazepam 5 mg 1 tablet night before the surgery & one 1/2hr before surgery)  patient should not come with empty stomach 34
  • 35. Cont.  ManagementManagement  Monitoring pulseMonitoring pulse  Lowering head to improve cerebellar circulationLowering head to improve cerebellar circulation  Loosening of tight cloths at neckLoosening of tight cloths at neck  Smelling of salt of aromatic ammoniaSmelling of salt of aromatic ammonia  Flashing the face with cold waterFlashing the face with cold water  Glucose drinksGlucose drinks  Inj.dexamethasoneInj.dexamethasone  Reassurance of patientReassurance of patient 35
  • 36. Anaphylactic shock  Caused by anaphylaxis of penicillin& streptomycin  Treatment  Oxygen  Airway  Injection adrenaline  Cardiopulmonary resuscitation 36