Adrenal, pancreas[1]

ADRENAL MEDULLA AND CORTEX ,[object Object],[object Object]

Adrenal Steroid Hormones ,[object Object],[object Object],[object Object],[object Object],[object Object]

Structures of adrenal steroids ,[object Object],[object Object],[object Object]

[object Object],[object Object],[object Object],[object Object],[object Object]

Regulation of adrenal steroids ,[object Object],[object Object]

Regulation of adrenal steroids ,[object Object],[object Object],[object Object]

REGULATION OF GLUCOCORTICOIDS ,[object Object],[object Object],[object Object],[object Object],[object Object]

Summary of actions of adrenal steroids ,[object Object],[object Object],[object Object],[object Object]

Actions of Glucocorticoids ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Metabolic effects of cortisol ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Anti-inflammatory and –immune effects of cortisol ,[object Object],[object Object],[object Object],[object Object]

More actions of cortisol ,[object Object],[object Object],[object Object],[object Object]

Relative potencies of drugs and dangers ,[object Object],[object Object],[object Object],[object Object],[object Object]

Actions of aldosterone ,[object Object],[object Object]

Action of aldosterone on kidney

Regulation of aldosterone Secretion ,[object Object],[object Object],[object Object],[object Object]

Zona Glomerulosa: Disorders ,[object Object],[object Object],[object Object]

Disorders ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Hyperadrenalism: Cushing’s Syndrome Hyper secretion of cortisol 1.Independent of ACTH i.e GC excess with low ACTH Causes :Treatment with Gcs Tumor of Ad cortex 2. ACTH dependent ( Cushing Disease ) GC excess with increased ACTH Causes :- Tumor in Pituitary, lungs, kidneys, or pancreas Treatment with ACTH

Cushing’s Disease ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

[object Object],[object Object],[object Object],[object Object]

Adrenal medulla ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Stress ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Life-Span Changes ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Phaeochromocytoma ,[object Object],[object Object]

Pancreas ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Hormones of the pancreas ,[object Object],[object Object],[object Object],[object Object]

Actions of Insulin ,[object Object],[object Object],[object Object]

Insulin decreases blood fatty acid and ketoacid concentrations. ,[object Object],[object Object],[object Object]

Insulin decreases blood amino acid concentration ,[object Object],[object Object]

Other actions of insulin ,[object Object],[object Object],[object Object]

Summary of major actions of insulin: ANABOLIC ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Insulin control ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Insulin Disorders: Diabetes Mellitus ,[object Object],[object Object]

Signs and symptoms ,[object Object],[object Object],[object Object],[object Object]

Continued ….. ,[object Object],[object Object],[object Object]

Type I Diabetes Mellitus ,[object Object],[object Object],[object Object],[object Object],[object Object]

Type II Diabetes Mellitus ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Risk Factors for Type 2 Diabetes Age > 40 Family history of diabetes Ethnicity Obesity; abdominal fat distribution GDM, or infant > 9 lbs Hypertension, hyperlipidemia Previous Impaired Glucose Tolerance

Glucagon ,[object Object],[object Object]

Regulation of Glucagon Secretion ,[object Object],[object Object]

Factors Affecting Glucagon Secretion ,[object Object],[object Object]

Major actions of glucagon ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Pancreatic somatostatin ,[object Object],[object Object],[object Object],[object Object],[object Object]

Pancreatic polypeptide ,[object Object],[object Object],[object Object],[object Object],[object Object]

“ Secondary” Endocrine Organs ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]

Adrenal, pancreas[1]

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Adrenal, pancreas[1]